1  +/- 2.7 mEq/L), consistent with compensated respiratory alkalosis.

2                                     For both respiratory alkalosis (5%--> 3% CO(2)) and metabolic alk

3  learned techniques resulted in intermittent respiratory alkalosis and hypoxia resulting in profoundl

4 Acid-base disturbances, such as metabolic or respiratory alkalosis, are relatively common in critical

5 progesterone and estradiol contribute to the respiratory alkalosis common in cirrhotic patients.

6                       In this model an early respiratory alkalosis developed, followed by a metabolic

7 nder normo- or hypercapnic conditions; under respiratory alkalosis (e.g. hypoxia) RTN neurons are sil