ライフサイエンスコーパス: respiratory burst

1 is required for the TSP1-induced macrophage respiratory burst.

2 ntegrins, also prevented beta-glucan-induced respiratory burst.

3 ivate the proton conductance or for a normal respiratory burst.

4 omote neutrophil adhesion, degranulation and respiratory burst.

5 an and released an efficient plasma membrane respiratory burst.

6 some-lysosome fusion but not by generating a respiratory burst.

7 s with sepsis enhanced bacterial killing and respiratory burst.

8 in-dependent activation of degranulation and respiratory burst.

9 yl-methionyl-leucyl-phenylalanine-stimulated respiratory burst.

10 t N. gonorrhoeae stimulated PMN to produce a respiratory burst.

11 product of lipid peroxidation induced by the respiratory burst.

12 jor role in the activation of the neutrophil respiratory burst.

13 ion of genes that contribute to an effective respiratory burst.

14 al, this bacterium suppresses the neutrophil respiratory burst.

15 utrophils), and the AGE-augmented neutrophil respiratory burst.

16 ducts in conjunction with a minimal residual respiratory burst.

17 (phox-/-)) in which PMN are incapable of the respiratory burst.

18 NADPH oxidase resulting in the absence of a respiratory burst.

19 und site, release reactive oxygen species by respiratory burst.

20 stream intermediates, thereby amplifying the respiratory burst.

21 y neutrophil activation and its accompanying respiratory burst.

22 in eosinophils that is modulated during the respiratory burst.

23 he increase of proton conductance during the respiratory burst.

24 ed in vitro for its effect on the neutrophil respiratory burst.

25 LDL blocked T/HS priming of respiratory burst.

26 al to the activation of H(+) flux during the respiratory burst.

27 ity, stayed the same or decreased during the respiratory burst.

28 cytosis of the particles and generation of a respiratory burst.

29 for the enhanced GSH uptake seen during the respiratory burst.

30 ffect on the enhanced uptake seen during the respiratory burst.

31 by abrogation of the NADPH oxidase-dependent respiratory burst.

32 activation of Ras, and the regulation of the respiratory burst.

33 c-Grb2 interaction, and the FcgammaR-induced respiratory burst.

34 se inhibitors tested against the TNF-induced respiratory burst.

35 sis, Ab-dependent cellular cytotoxicity, and respiratory burst.

36 and eliminated any haplotypic impact on the respiratory burst.

37 in the absence of an NADPH oxidase-mediated respiratory burst.

38 lation at the phagosome, leading to impaired respiratory burst.

39 proinflammatory cytokine production, and the respiratory burst.

40 tes to microbe elimination during macrophage respiratory burst.

41 osomal environment and inhibit the host cell respiratory burst.

42 gmented size, granularity, phagocytosis, and respiratory burst.

43 ucyl-phenylalanine (fMLP)-induced neutrophil respiratory burst.

44 on activation of the NADPH oxidase-dependent respiratory burst.

45 ice that are unable to generate a phagocytic respiratory burst.

46 obust, highly periodic cycles in the form of respiratory bursts.

47 ed the percentage of phagocytes undergoing a respiratory burst (66.0% +/- 6.3% versus 41.0% +/- 8.3%

48 tem's battle against pathogens includes the "respiratory burst," a rapid release of ROS from leukocyt

49 eral blood leukocytes (PBL) and enhanced the respiratory burst, acid phosphatase activity, chemotacti

50 several mechanisms, such as NO synthase, the respiratory burst, acidification, and autophagy, how hum

51 bulin (Ig)G-coated erythrocyte phagocytosis, respiratory burst, actin cup formation, and activation o

52 rradiation was also associated with enhanced respiratory burst activities and an unexpected neutrophi

53 ionyl-leucyl-phenylalanine (fMLP)-stimulated respiratory burst activity and (3)H-DG uptake are tempor

54 nes involved in phagocytic function, such as respiratory burst activity and cytokine secretion.

55 TNFalpha and LPS primed respiratory burst activity and increased membrane expres

56 s in the 6 kindreds, there was no measurable respiratory burst activity and no p67-phox protein detec

57 Respiratory burst activity and phosphorylation of an NAD

58 ocytosis contributes to phagocytosis-induced respiratory burst activity and plays a critical role in

59 ed the ability of Akt to regulate neutrophil respiratory burst activity and to interact with and phos

60 Termination of respiratory burst activity did not reflect dephosphoryla

61 These data suggest that 1) the onset of respiratory burst activity during phagocytosis is linked

62 (phox) phosphorylation, which contributes to respiratory burst activity in human neutrophils.

63 class II beta-chain, and STAT1, and enhanced respiratory burst activity in macrophages.

64 m by which TNFalpha and LPS prime neutrophil respiratory burst activity is by increasing membrane exp

65 TNFalpha failed to prime respiratory burst activity or to increase membrane CD35

66 uptake compared with fMLP without affecting respiratory burst activity, and that fMLP stimulation of

67 ucts (AGEs) enhance NADPH oxidase, and hence respiratory burst activity, of stimulated neutrophils.

68 gp91(phox) are rate-limiting components for respiratory burst activity, our studies may identify rat

69 rmyl methionyl-leucyl-proline (fMLP)-induced respiratory burst activity, suggesting interference by P

70 -induced chemotaxis but has little effect on respiratory burst activity.

71 phage lineages and exhibits phagocytosis and respiratory burst activity.

72 ial for life span extension, chemotaxis, and respiratory burst activity.

73 ssive protection model and induce neutrophil respiratory burst activity.

74 assessment was made of neutrophil function (respiratory burst, adhesion molecule expression, and che

75 Here we optimize the antibody-dependent respiratory burst (ADRB) assay, which assesses the abili

76 ymorphonuclear neutrophil (neutrophil [PMN]) respiratory burst after trauma and hemorrhagic shock (T/

77 ssessed no ability to augment the neutrophil respiratory burst alone.

78 ANCAs activate neutrophils inducing their respiratory burst and a peculiar form of cell death, nam

79 n RAW264.7 macrophages during the phagocytic respiratory burst and A431 cells in response to EGF stim

80 , which regulates activity of the neutrophil respiratory burst and actin assembly.

81 esulted in an impaired ability to activate a respiratory burst and also inhibited chemotaxis.

82 uppression in vitro through the induction of respiratory burst and apoptosis.

83 syk(-/-) neutrophils showed normal respiratory burst and degranulation in response to the b

84 Thus, separate signals control the respiratory burst and degranulation, and a normal rate o

85 The respiratory burst and NO radical (NO.) made distinct con

86 including complement, mononuclear phagocyte respiratory burst and phagocytosis through retargeting o

87 ClC-3 was specifically involved in the respiratory burst and phagocytosis.

88 TF expression in neutrophils contributes to respiratory burst and subsequent trophoblast injury and

89 triggered by the release of H2O2 during the respiratory burst and that induces the uptake of GSH int

90 . phagocytophilum does not suppress a global respiratory burst and that, under identical conditions i

91 al macrophages to E. coli and induced both a respiratory burst and the release of lysozomal enzyme fr

92 0 and PD098059, revealed that priming of the respiratory burst and up-regulation of flavocytochrome b

93 ith impaired neutrophil adhesion, migration, respiratory burst, and degranulation in vitro.

94 an neutrophils, IL-8 induces chemotaxis, the respiratory burst, and granule release, and enhances cel

95 ted responses such as in vitro phagocytosis, respiratory burst, and in vivo thrombocytopenia, we inve

96 CR3 (CD11b/CD18), enhanced the intracellular respiratory burst, and increased levels of Rac2 activati

97 cisella phagosomal escape, inhibition of the respiratory burst, and intracellular survival.

98 ogates the AGE-enhanced activated neutrophil respiratory burst, and it is demonstrably stimulated in

99 included CD11b activation and up-regulation, respiratory burst, and shape changes.

100 such as tight adhesion, spreading, sustained respiratory burst, and specific granule release in vitro

101 Adenosine inhibited the respiratory burst, and, in cocultures, adenosine deamina

102 gamma), and IL-1 alpha; increased neutrophil respiratory burst; and, ultimately, increased clearance

103 eems to mediate the AGE-augmented neutrophil respiratory burst (ascertained by chemiluminescence).

104 lity in the luminol, but not the isoluminol, respiratory burst assays following stimulation with phor

105 r full-length forms was also demonstrated in respiratory burst assays, CD11b Ag expression, and intra

106 The classic respiratory burst at fertilization is the result of prod

107 nt PMN cannot generate an adhesion-dependent respiratory burst, because of markedly diminished integr

108 nyleneiodonium not only blocked a productive respiratory burst but also abrogated the survival advant

109 ation and escape response and the neutrophil respiratory burst but with little increase in the solubl

110 hagocytophilum did not produce a significant respiratory burst, but A. phagocytophilum did not inhibi

111 timulates neutrophil chemotaxis and a robust respiratory burst, but other aspects of this interaction

112 Therefore, HGE bacteria repress the respiratory burst by down-regulating gp91phox, the first

113 and plays a critical role in priming of the respiratory burst by increasing expression of membrane c

114 persists within neutrophils and prevents the respiratory burst by inhibiting gp91(phox).

115 nosa suppresses bacterium-induced neutrophil respiratory burst by interfering with a PKC-dependent, n

116 that PMA activates the H+ efflux during the respiratory burst by modulating the properties of H+ cha

117 opsonized Burkholderia induced a significant respiratory burst by neutrophils compared to unopsonized

118 gp91phox levels and a consequent decline in respiratory burst capability.

119 escence (CL) assays were used to measure the respiratory burst capacity of phagocytes from HIV-infect

120 se in apoptotic frequency and an increase in respiratory burst capacity, consistent with in vivo "pri

121 plained and unexpected defects in neutrophil respiratory burst, chemotaxis and calcium flux, in respo

122 Both have impairments in their neutrophil respiratory burst, chemotaxis response, and calcium flux

123 in modulating neutrophil function, including respiratory burst, chemotaxis, and apoptosis.

124 patients also have defects in the neutrophil respiratory burst, chemotaxis, and calcium flux.

125 ta led to neutropenia; defects in neutrophil respiratory burst, chemotaxis, and calcium flux; and inc

126 mpairment is reflected in reduced neutrophil respiratory burst, chemotaxis, and calcium mobilization.

127 -/-) macrophages having impairments in their respiratory burst, chemotaxis, calcium flux, and phagocy

128 ocytes from infected mice had an ineffective respiratory burst compared with 1%+/-1% (mean+/-SD) of t

129 (p <.05) phorbol myristate acetate elicited respiratory burst compared with buffer or T/SS.

130 d significant elevations in MIP/PAF-elicited respiratory burst compared with T/HS lymph or buffer onl

131 With stimulation of the respiratory burst, cytosolic oxidase components, p47(pho

132 ctivated by IFN-gamma or by macrophages from respiratory burst-deficient mice.

133 sion, adhesion and migratory responsiveness, respiratory burst, degranulation, and calcium mobilizati

134 syk(-/-) neutrophils failed to undergo respiratory burst, degranulation, or spreading in respon

135 To determine the relative contributions of respiratory burst-derived reactive oxygen intermediates

136 CGD mice, lacking a respiratory burst, developed accentuated colitis compare

137 he NBT and Fc-Oxyburst assays could detect a respiratory burst during A. phagocytophila infection.

138 GO:0010200 (response to chitin), GO:0002679 (respiratory burst during defence response) and GO:003555

139 We describe the fungal respiratory burst during host infection, paralleled by s

140 gonococcal susceptibility to the phagocytic respiratory burst during infection and that gonococcal c

141 peritoneal-resident macrophages to maintain respiratory burst during phagocytosis via enhancing mito

142 There was no difference in respiratory burst early after injury.

143 stoichiometric H(+) efflux occurs during the respiratory burst, efforts to stimulate voltage-gated H(

144 tions, in conjunction with activation of the respiratory burst enzyme NADPH oxidase.

145 espite normal or increased activities of the respiratory burst enzymes.

146 The magnitude of respiratory burst found here paralleled the [Ca2+]i resp

147 Also, DNE led to an increase in respiratory burst frequency after AMPA injection into th

148 8-OH-DPAT transiently increased respiratory burst frequency in Lmx1b(f/f/p) preparations

149 RESPIRATORY BURST OXIDASE HOMOLOG F-mediated respiratory burst had a major impact and was a convergin

150 Blocking crystal-activated respiratory burst has, however, no effect on NETs.

151 ive oxygen species (ROS), which requires the respiratory burst homolog RbohB.

152 tis challenge induced a robust intracellular respiratory burst; however, this response did not contri

153 rol appear physiologic because they regulate respiratory burst in a proportional biphasic fashion.

154 ally, vitronectin reduced the silica-induced respiratory burst in AM as determined with chemiluminesc

155 Conversely, phagocytosis did not trigger a respiratory burst in blood monocytes or monocyte-derived

156 cells, cytotoxicity against hepatocytes, and respiratory burst in hepatic leukocytes.

157 ressing cells induced a significantly larger respiratory burst in human neutrophils compared with con

158 nerated during apoptosis inhibited the basal respiratory burst in human neutrophils, and those genera

159 The respiratory burst in interferon-gamma and zymosan-stimul

160 he classic PKC alpha mediates IgG-stimulated respiratory burst in macrophages, whereas the novel PKCs

161 We report here that the respiratory burst in monocytes is accompanied by an incr

162 P. aeruginosa induces a robust respiratory burst in neutrophils that is required for ex

163 f the uracil auxotrophic mutants triggered a respiratory burst in neutrophils, and ingested bacteria

164 those responsible for the pathogen-activated respiratory burst in phagocytes.

165 ivation of the proton conductance during the respiratory burst in phagocytes.

166 ropathogenic Yersinia spp. also inhibits the respiratory burst in PMNs and macrophages, and we show h

167 d association with and induction of a weaker respiratory burst in PMNs from estradiol-treated mice.

168 dose-dependent enhancement on the neutrophil respiratory burst in response to a secondary mechanical

169 OS) from Neisseria meningitidis enhances the respiratory burst in response to formyl-Met-Leu-Phe, an

170 endotoxin, prime neutrophils for an enhanced respiratory burst in response to subsequent stimulation.

171 KC isoforms in IgG-mediated phagocytosis and respiratory burst in the mouse macrophage-like cell line

172 The neutrophil respiratory burst in the presence of A. phagocytophilum

173 Respiratory burst in these cells was Ca2+ dependent and

174 acetate (PMA) is a potent agonist for this "respiratory burst" in human neutrophils.

175 n (Hv1) channels play important roles in the respiratory burst, in pH regulation, in spermatozoa, in

176 uent killing of microbes is initiated by the respiratory burst, in which nicotinamide adenine dinucle

177 nase inhibitor SB-203580 fully inhibited the respiratory burst induced by fMLP or the PlcHR-replete w

178 AcpA is a respiratory burst-inhibiting acid phosphatase from the C

179 AcpA of Francisella spp. is a respiratory-burst-inhibiting acid phosphatase that also

180 These data demonstrate respiratory burst inhibition by A. phagocytophila in viv

181 te acetate, which was fully abrogated by the respiratory burst inhibitor diphenyleneiodonium chloride

182 Both IgG-stimulated phagocytosis and respiratory burst involve activation of protein kinase C

183 The phagocyte respiratory burst is crucial for innate immunity.

184 Defects in the phagocytic cells' respiratory burst lead to life-threatening infections, i

185 e gp91(phox) gene encodes a component of the respiratory burst NADPH oxidase complex and is highly ex

186 7(phox), an essential component of phagocyte respiratory burst NADPH oxidase.

187 NOS2 (NOS2(-/-)), gp91(Phox) subunit of the respiratory burst NADPH-oxidase complex (Phox(-/-)), or

188 Neither activation of the respiratory burst nor phagocytosis of either latex parti

189 PER activation in vitro slightly reduced the respiratory burst of acidophilic granulocytes and drasti

190 regions of Pyk2, specifically inhibited the respiratory burst of cells responding to tumor necrosis

191 HNA-3a antibodies primed the fMLP-activated respiratory burst of HNA-3a+, but not HNA-3a-, PMNs and

192 that SodC protects B. abortus 2308 from the respiratory burst of host macrophages.

193 of their aerobic metabolism and through the respiratory burst of host phagocytes.

194 The respiratory burst of human neutrophils is primed by a nu

195 the addition of 2 mm glutamine increased the respiratory burst of human PMN stimulated with both phor

196 c disruption of IL-27 signaling enhanced the respiratory burst of macrophages.

197 olarization in the presence of Ac-PGP or the respiratory burst of neutrophils in the presence of a me

198 CBD treatment also attenuated the respiratory burst of neutrophils isolated from chronic p

199 ts have revealed a C5a-induced defect in the respiratory burst of neutrophils.

200 Porphyromonas gingivalis (Pg), stimulate the respiratory burst of neutrophils.

201 ly to be due to increased sensitivity to the respiratory burst of phagocytes but is, instead, due to

202 ze interactions of both molecules during the respiratory burst of phagocytes provided an excellent op

203 Granulocytes generate a "respiratory burst" of NADPH oxidase-dependent superoxide

204 cted DC in the presence of inhibitors of the respiratory burst or inhibitors of NO synthase had littl

205 ons, but not for the rapid initiation of the respiratory burst or phagocytosis.

206 It does not correlate with respiratory burst or secretory activity but may reflect

207 ction of superoxide anion, inhibitors of the respiratory burst (or NO production) did not inhibit kil

208 human PMN with inhibitors and scavengers of respiratory burst oxidants only partially reversed antic

209 Mutation of Arabidopsis thaliana respiratory burst oxidase (Atrboh) genes eliminated path

210 The level of BRs was closely related to the respiratory burst oxidase 1 (RBOH1)-encoded NADPH oxides

211 l closure was dependent on the production of RESPIRATORY BURST OXIDASE 1 (RBOH1)-mediated hydrogen pe

212 The CYBB gene, which encodes the respiratory burst oxidase component gp91(phox), is expre

213 s are blocked by inhibitors of ROS-producing respiratory burst oxidase enzymes.

214 be involved in the ROS burst from the plant respiratory burst oxidase homolog (Rboh) of the human ne

215 f Ca(2+) wave propagation in the Arabidopsis respiratory burst oxidase homolog D (AtrbohD) knockout b

216 also induced increased levels of Arabidopsis respiratory burst oxidase homolog D (AtrbohD) mRNA, but

217 g MS, we identified the plant NADPH oxidase, respiratory burst oxidase homolog D (RBOHD), as an in vi

218 as a result of the lower expression level of RESPIRATORY BURST OXIDASE HOMOLOG D (RbohD).

219 re1]) or reactive oxygen species production (respiratory burst oxidase homolog DF [rbohDF]).

220 re associated with increased accumulation of respiratory burst oxidase homolog F (RBOHF)-dependent re

221 Transcripts encoding the Respiratory Burst Oxidase Homolog F, signaling component

222 The RESPIRATORY BURST OXIDASE HOMOLOG F-mediated respiratory

223 nine dinucleotide phosphate, reduced oxidase RESPIRATORY BURST OXIDASE HOMOLOG PROTEIN D (RBOHD) by s

224 n species generation (due to the function of Respiratory Burst Oxidase Homolog proteins D and F) are

225 silencing of a gene encoding NADPH oxidase (Respiratory burst oxidase homolog) in the gox mutants di

226 oxidase homologs of plants termed RBOH (for respiratory burst oxidase homolog).

227 n domains of extracellular ROS donors of the RESPIRATORY BURST OXIDASE HOMOLOGS (RBOH).

228 AtPeps is absent in the double mutant of the respiratory burst oxidase homologs D and F (rbohD rbohF)

229 hrough the transcriptional induction of four Respiratory Burst Oxidase Homologs TUNEL-positive nuclei

230 ADPH oxidase genes, MtRbohA and MtRbohC (for respiratory burst oxidase homologs), is increased in lat

231 d with reduced expression of the Arabidopsis respiratory burst oxidase homologue AtrbohD and the SA b

232 Arabidopsis respiratory burst oxidase homologues (rboh genes) have b

233 The neutrophil respiratory burst oxidase is a multicomponent activatabl

234 We show that lack of A. thaliana respiratory burst oxidase protein F (AtrbohF; an NADPH o

235 The CYBB and NCF2 genes encode the phagocyte respiratory burst oxidase proteins, gp91PHOX and p67PHOX

236 Activation of the phagocyte respiratory burst oxidase requires interaction between t

237 Further studies demonstrated that the respiratory burst oxidase responsible for O-2 production

238 ox) are components of the phagocyte-specific respiratory burst oxidase that are encoded by the NCF2 a

239 gp91phox is the catalytic subunit of the respiratory burst oxidase, an NADPH-dependent, superoxid

240 6 extends into the neighboring gene encoding respiratory burst oxidase, and (2) a commonly used STA6

241 the essential role of Rac in assembly of the respiratory burst oxidase, invasion through this nonopso

242 talytic subunit of the superoxide-generating respiratory burst oxidase, is regulated by subunits p47(

243 talytic subunit of the superoxide-generating respiratory burst oxidase, is stimulated by the regulato

244 actors, the membrane-bound components of the respiratory burst oxidase, membrane-bound adhesion molec

245 codes gp91Phox; a component of the phagocyte respiratory burst oxidase.

246 Activities of enzymes involved in the respiratory burst, oxidase and myeloperoxidase, were exa

247 During the phagocytic respiratory burst, oxygen is converted to potent cytotox

248 in T/HS plasma increased MIP-2/PAF-elicited respiratory burst (p <.05) compared with UC or T/SS plas

249 actericidal phase, which is dependent on the respiratory burst phagocyte oxidase (phox) is succeeded

250 te (NADPH) 2 (NADPH oxidase 2; NOX2), termed respiratory burst (RB).

251 icient neutrophils had an enhanced phagocyte respiratory burst relative to Nbeal2-expressing neutroph

252 crophages, although the phagocytosis-induced respiratory burst remained intact.

253 AGE augmentation of the activated neutrophil respiratory burst requires AA generation, through which

254 cftr gene (Cftr morphants) exhibited reduced respiratory burst response and directed neutrophil migra

255 We show human PMNs generate a respiratory burst response to unopsonized hyphae.

256 reduced in the presence of inhibitors of the respiratory burst response.

257 also potentiated the IAV-induced neutrophil respiratory burst response.

258 HNPs did not increase neutrophil respiratory burst responses to IAV.

259 eater increases in neutrophil uptake of, and respiratory burst responses to, IAV than MBL.

260 arly (CD38 and CD11b) and late (neutrophilic respiratory burst) responses.

261 PKC inhibition during the respiratory burst reversed the activation of both molecu

262 The pir-b-/- neutrophils displayed enhanced respiratory burst, secondary granule release, and a hype

263 Lyn(-/-) neutrophils displayed enhanced respiratory burst, secondary granule release, and a hype

264 cytotoxic effector mechanisms, including the respiratory burst, secretion of inflammatory mediators a

265 obial and cytotoxic functions, including the respiratory burst, secretion, and apoptosis.

266 ological functions, including the phagocytic respiratory burst, sperm motility, apoptosis, and metast

267 el role for RhoG in signaling the neutrophil respiratory burst stimulated by G protein-coupled recept

268 ta indicate that the NADPH oxidase-dependent respiratory burst stimulated by Pseudomonas infection co

269 olesterol availability, Ca(2+) signaling and respiratory burst suggest that Ca(2+) influx and PMN act

270 by genetic abrogation of the host phagocyte respiratory burst, suggesting that the sigmaE regulon pl

271 gested by PMNs and induce a rapid and strong respiratory burst that is comparable to PMA.

272 ation with fresh serum, Hp triggers a modest respiratory burst that is confined to the phagosome, and

273 he macrophage, the bacteria must survive the respiratory burst that produces superoxide.

274 During the "respiratory burst," the NADPH oxidase complex of phagocy

275 ed the relative contribution of PMNs and the respiratory burst to "inflammatory hypoxia" in vivo.

276 beta(1) integrins to include modulating PMN respiratory burst to a pathogen-associated molecular pat

277 not protein kinase C zeta in generating the respiratory burst to beta-glucan.

278 beta(1) integrin ligands did not affect respiratory burst to ingestible beta-glucan-containing p

279 Priming neutrophils for an enhanced respiratory burst together with promoting granule conten

280 TF) expression in neutrophils contributes to respiratory burst, trophoblast injury, and pregnancy los

281 urium did not cause an increase in the early respiratory burst under unprimed or primed conditions, a

282 ole in mediating p38-dependent activation of respiratory burst upon stimulation of Fc gamma RIIIb in

283 tively activates K-ras during induction of a respiratory burst via pathways involving multiple upstre

284 PMN respiratory burst was assessed using the nitro-blue tetr

285 This respiratory burst was associated with increased expressi

286 production also was rescued, and a deficient respiratory burst was corrected.

287 Finally, respiratory burst was dependent on Lyn and membrane raft

288 e role of exocytosis in the human neutrophil respiratory burst was determined using a fusion protein

289 PMN respiratory burst was initiated by using macrophage infl

290 Plasma membrane-associated respiratory burst was measured by reduction of ferricyto

291 The enhanced respiratory burst was phosphoinositide 3-kinase-dependen

292 ffer significantly between the 2 groups, but respiratory burst was significantly less (by 28%) in iro

293 ) antiport regulates pH during the phagocyte respiratory burst, we show here that voltage-gated proto

294 uding cytokine production, phagocytosis, and respiratory burst were globally impaired in macFoxp1tg c

295 unctional, as demonstrated by stimulation of respiratory burst when neutrophils adhered to surfaces c

296 agocytophilum did not inhibit the neutrophil respiratory burst when phorbol myristate acetate was add

297 (-/-) double-knockout mice failed to undergo respiratory burst when plated on mVCAM-1.

298 db/db neutrophils demonstrated a diminished respiratory burst when stimulated with S. aureus.

299 ction cooperatively in phagocytes during the respiratory burst, when reactive oxygen species are prod

300 Endotoxic stimulation induced a respiratory burst with the production of superoxide and