1 f an A1 adenosine receptor antagonist to the respiratory centers.

2 he VMS and they innervate key pontomedullary respiratory centers.

3 tive to neuromodulatory inputs from multiple respiratory centers, and thus they serve as a key nexus

4 lly dyspneic and have an increase in overall respiratory center drive, as a result of increased lung

5 n in forebrain control of compromised bulbar respiratory centers during NREM sleep in PPS.

6 ively deliver recovery-inducing drugs to the respiratory centers exclusively.

7 cally when disconnected from the rest of the respiratory centers in vivo (KYN treatment) and in vitro

8 ed carotid sinus nerve discharge rate to the respiratory centers of the brain, (2) intrinsic hypoxic

9 lutamatergic neurons that innervate the main respiratory centers of the medulla oblongata and pons.

10      They are then conveyed to the brainstem respiratory centers via a brainstem pontine relay locate