ライフサイエンスコーパス: respiratory control

1 nters, and thus they serve as a key nexus of respiratory control.

2 ystematically study sleep-related changes in respiratory control.

3 dulate many vital brain functions, including respiratory control.

4 tion that leads to appropriate modulation of respiratory control.

5 neural function, focusing on NG neurons and respiratory control.

6 are associated with marked changes in cardio-respiratory control.

7 es, and in the carotid body it is crucial to respiratory control.

8 hondrial bioenergetics, Ca(2+) dynamics, and respiratory control.

9 l outer membrane permeability contributes to respiratory control.

10 or (ANT), which provides a possible site for respiratory control.

11 ly behavioural to predominantly chemosensory respiratory control.

12 le electrical circuit model of mitochondrial respiratory control.

13 e phase of the circadian cycle in studies of respiratory control.

14 reflect different physiologic influences on respiratory control.

15 ured (ELISA) in three regions of interest to respiratory control: (1) ventral cervical spinal segment

16 In this study, we showed that ArcA (aerobic respiratory control), a global regulator important for E

17 have a relatively high incidence of central respiratory control abnormalities.

18 is sufficient to disrupt the development of respiratory control and augment the occurrence of apneas

19 rther the role of ATP-mediated signalling in respiratory control and central chemoreception by charac

20 Until recently, the respiratory control and function of intrinsic tongue mus

21 ATP is involved in central respiratory control and may mediate changes in the activ

22 Preterm infants have immature respiratory control and resulting intermittent hypoxia (

23 Additionally, cardiac arrhythmia, respiratory control, and epilepsy genes were screened fo

24 leep apnoea in adults, but the mechanisms of respiratory control are not clearly understood.

25 fects that include CA neurones have abnormal respiratory control at birth.

26 ue CO(2)/H(+) and function as a key locus of respiratory control by integrating information from seve

27 gical studies of the brainstem pre-Botzinger respiratory control center demonstrated an abnormal rhyt

28 that Mecp2 is critical within autonomic and respiratory control centers for survival.

29 of hypoventilation by stimulation of central respiratory control centers.

30 r) were assessed by comparisons with various respiratory control conditions.

31 se during gestation is sufficient to disrupt respiratory control development and promote pathological

32 Respiratory control disorders such as apnea of prematuri

33 ify therapeutic targets for the treatment of respiratory control disorders.

34 ial therapeutic targets for the treatment of respiratory control disorders.

35 ful therapeutic targets for the treatment of respiratory control disorders.

36 We examined the role of respiratory control during O2-induced hypercarbia in pat

37 al characteristics and carotid body-mediated respiratory control during sleep with EMG (EMG+) or with

38 the development of brain regions underlying respiratory control functions.

39 ed in structures important for autonomic and respiratory control, functions that are severely affecte

40 However, its influence upon respiratory control has hardly been studied.

41 re little influenced by central chemosensory respiratory control in awake humans even when at rest un

42 e of circadian variations in respiration and respiratory control in awake humans for the first time u

43 These results provide unique insights into respiratory control in awake humans, and highlight the i

44 tributions to the literature on disorders of respiratory control in infancy and childhood are reviewe

45 ntal nicotine exposure (DNE) impacts central respiratory control in neonates born to smoking mothers.

46 urons from many brainstem nuclei involved in respiratory control increase their firing rate in respon

47 Respiratory control index values on rat liver mitochondr

48 l respiratory function (postischemic percent respiratory control index; NAD(+)-linked: 81.3+/-3.8 ver

49 Postischemic mitochondrial respiratory control indices (RCIs) were significantly be

50 Current theory of respiratory control invokes a role of myoglobin (Mb)-fac

51 he maturational shift away from ADP-mediated respiratory control is regulated by thyroid hormone in v

52 erm exposure to hypoxia generates changes in respiratory control known as ventilatory acclimatization

53 nd lateral mesencephalic reticular nucleus), respiratory control (lateral nucleus of the solitary tra

54 s include small upper airway lumen, unstable respiratory control, low arousal threshold, small lung v

55 Since ageing influences respiratory control mechanisms and serotonergic function

56 d hypercarbia does not indicate a failure of respiratory control mechanisms in the maintenance of PaC

57 critical for proper development of medullary respiratory control mechanisms.

58 but thought to involve depression of central respiratory control mechanisms.

59 eparate brainstem pathways for syringeal and respiratory control of song production, both can affect

60 istent with a functional role for ENK in the respiratory control of the tongue.

61 his period is a critical window during which respiratory control or regulation may be distinctly diff

62 d to be either unique to neurons involved in respiratory control, or at least very unusual for non-re

63 To test the hypothesis that postmetamorphic respiratory control phenotypes arise through permanent d

64 ons (1-4%) and high degree of functionality (respiratory control ratio = 5-6).

65 ol-fed mice showed a significant decrease in respiratory control ratio and an increased sensitivity t

66 inhibition causes a dramatic increase in the respiratory control ratio from 6 to 40 for wild-type oxi

67 The respiratory control ratio of mitochondria from liver of

68 The respiratory control ratio of synaptic and nonsynaptic mi

69 nificant decrease in state 3 respiration and respiratory control ratio that was accompanied by an inc

70 ange of NADH levels, respiratory fluxes, and respiratory control ratio upon transitions elicited by s

71 A significant decrease in the respiratory control ratio was observed in mitochondria i

72 euronal counts, HVR, and brain mitochondrial respiratory control ratio were significantly reduced fol

73 ondrial efficiency as evidenced by increased respiratory control ratio, elevated cytochrome-c oxidase

74 , demonstrated by reduction of state III and respiratory control ratio, increased production of react

75 Despite no change in the respiratory control ratio, substrate control ratios of G

76 the outer membrane and uncorrelated with the respiratory control ratio.

77 3 respirations, and diminished mitochondrial respiratory control ratio.

78 rity of the outer mitochondrial membrane and respiratory control ratio.

79 Respiratory control ratios (RCRs) of GGT(-/-) mice liver

80 deficient rats had lower liver mitochondrial respiratory control ratios and increased levels of oxida

81 Lowered respiratory control ratios were found in daily high-iron

82 sults, the very significant reduction in the respiratory control ratios.

83 l abnormalities and normalized mitochondrial respiratory control, reflecting protection against inner

84 in the subsequent cycles; and (c) models of respiratory control should depict a recurrent inhibitory

85 uscle responsiveness, arousal threshold, and respiratory control stability; loop gain) contributions

86 ith similar state 3 and 4 respiratory rates, respiratory control (state 3/state 4), and ADP/O ratios.

87 eptin in the hindbrain areas involved in the respiratory control such as the nucleus of the solitary

88 lasticity for therapeutic advantage when the respiratory control system is compromised (e.g., sleep a

89 arrest secondary to paralysis of the central respiratory control system or due to paralysis of the re

90 We present the view that the respiratory control system uses these sources of informa

91 nstrated considerable neuroplasticity in the respiratory control system, few studies have explored th

92 em controlled by feedback loops, such as the respiratory control system.

93 have abnormalities in autonomic function and respiratory control that may contribute to premature let

94 which serve an important integrative role in respiratory control; the increased drive provided by enh

95 All three dyes suppressed mitochondrial respiratory control to some extent.

96 ed the "pure" effect of sleep deprivation on respiratory control under strictly controlled behavioral

97 is intrinsically linked to post-inspiratory respiratory control using the unanaesthetized working he

98 this region was not known to be involved in respiratory control, we combined chemical microstimulati

99 7) for one dose; estimates were similar when respiratory controls were used as the control group.

100 e sequelae entrained by disturbance of basic respiratory control whereby a process of which we are no

101 etermine if hypothalamic neurons involved in respiratory control, which were identified in cats by th