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1 of apoE4 domain interaction on mitochondrial respiratory function.
2 d hippocampus were isolated and assessed for respiratory function.
3 abile zinc and interferes with mitochondrial respiratory function.
4 itochondrial RNA stability, translation, and respiratory function.
5 ns in respiratory supercomplex structure and respiratory function.
6 of scapular winging, contractures and normal respiratory function.
7 he effects of their toxic exoproducts impede respiratory function.
8 n, this measurement is limited as a guide to respiratory function.
9 f pulmonary integrity and edema compromising respiratory function.
10 igh-amplitude swelling and exhibited altered respiratory function.
11  obtained by spirometry, were used to assess respiratory function.
12 y barrier cells that hinders regeneration of respiratory function.
13  mutations caused little or no effect on the respiratory function.
14        All presented with a deterioration in respiratory function.
15 ruction would not lead to a deterioration in respiratory function.
16 DNA and protein content, gene expression, or respiratory function.
17 consequent barotrauma, resulting in improved respiratory function.
18 t (LS) is an essential system supporting the respiratory function.
19 SH, but fully restored mitochondrial GSH and respiratory function.
20 ling extubation demonstrate abnormalities of respiratory function.
21 and chest wall are important determinants of respiratory function.
22 ial DNA maintenance, and it is essential for respiratory function.
23  severe respiratory diseases and compromised respiratory function.
24 rsible inhibition of cardiac contractile and respiratory function.
25 ving only a marginal effect on mitochondrial respiratory function.
26 ial protein involved in iron homeostasis and respiratory function.
27 re non-viable, emphasizing the importance of respiratory function.
28 unctional Rating Scale-Revised score and the respiratory function.
29 cal role for Oma1 protease in fine-tuning of respiratory function.
30 coactivator-1alpha (PGC-1alpha) and enhanced respiratory function.
31 ormal structure and displayed a 35-43% lower respiratory function.
32  were clinically satisfactory, with adequate respiratory function.
33 ed to immediate and sustained improvement of respiratory function.
34 sociations of exposure to air pollution with respiratory function.
35 e amount of contractile tissue available for respiratory function.
36 mpiled using simple measures of physical and respiratory function.
37 nal medulla, and for their control of cardio-respiratory function.
38 armacologic activation of TrkB would improve respiratory function.
39 thin the lung interstitium leads to impaired respiratory function.
40 e expertise in caring for patients with poor respiratory function.
41  many nuclear genes specifying mitochondrial respiratory function.
42  it robustly measures the brain, cardiac and respiratory functions.
43 tioning in the case of immune, hormonal, and respiratory functions.
44 ucial roles in modulating cardiovascular and respiratory functions.
45  of exogenous cytochrome c markedly restored respiratory functions.
46 the expression of numerous genes involved in respiratory functions.
47 in lungs, participating in the regulation of respiratory functions.
48 ons, which support other respiratory and non-respiratory functions.
49 (mtErbB2) negatively regulates mitochondrial respiratory functions.
50 r discharge to induce appropriate changes in respiratory function (1).
51  at presentation and 20% routinely monitored respiratory function; 32% relied on symptoms as the only
52 to common allergens, and to undergo tests of respiratory function, 659 women and 500 men agreed to an
53 ty (4.2%; P<0.05), tolerance to calcium, and respiratory function (72% increase in state 3 and 23% in
54 hore crabs in maintaining osmoregulatory and respiratory function after acute exposure to both anthro
55 reported significant abnormalities in infant respiratory function after clinical recovery from bronch
56 epresent an important strategy for improving respiratory function after spinal cord injury.
57 IBG pretreatment also improved mitochondrial respiratory functions after cold preservation.
58 in neural centers critical for autonomic and respiratory function alleviates the lethality associated
59 ng was defined as good cognitive, motor, and respiratory functioning, along with absence of disabilit
60 osition may play a role in the impairment of respiratory function among the abdominally obese.
61 ial permeability transition, decline in both respiratory function and activity of cytochrome c oxidas
62 rectly affects acid-base and ion regulation, respiratory function and aerobic performance in aquatic
63 eased glycolysis, but impaired mitochondrial respiratory function and biogenesis.
64 egration of pathways directing mitochondrial respiratory function and cell growth.
65    Consequently, inhibition of HsPDF reduces respiratory function and cellular ATP levels, causing de
66 hyperaccumulation, and restore mitochondrial respiratory function and cytochrome c levels.
67 in (0.2%) in 2 month old mdx mice to improve respiratory function and end-point functional and histol
68 hmogenic networks, their respective roles in respiratory function and how they interact to constitute
69 sphate (ATP); they had reduced mitochondrial respiratory function and increased oxidative stress, com
70 owever, the impact of single LT on long-term respiratory function and nutritional status has not been
71                                         Poor respiratory function and obesity are associated with all
72 sts that loss of Lon1 significantly modifies respiratory function and plant performance by small but
73 es mitophagy and improves both mitochondrial respiratory function and proteostasis in aged flies.
74     Late diagnosis significantly worsens the respiratory function and reduces the chance for normal d
75                Idebenone reduced the loss of respiratory function and represents a new treatment opti
76 process, which is necessary to regain normal respiratory function and restore the lungs to homeostasi
77 increases survival, rescues abnormalities in respiratory function and social recognition, and improve
78 ay enable frataxin to simultaneously promote respiratory function and stress tolerance.
79              Effect estimates for changes in respiratory function and symptoms were pooled by using f
80 raction between the disruption of this novel respiratory function and the loss of wild-type mtDNA.
81 hysiological basis of these vocalizations in respiratory function and to additional developmental var
82 t time a physiological improvement of cardio-respiratory functions and a correction of behavioral fea
83         We examined the correlations between respiratory function, and ACT and AHQ-JAPAN.
84 ities of Fe-S cluster enzymes, a decrease in respiratory function, and an increase in oxidative stres
85 chondrial protein degradation, mitochondrial respiratory function, and cell viability are compromised
86 ochrome b, several mutations that impair the respiratory function, and reversions that correct the de
87             Motor and cognitive development, respiratory function, and safety were evaluated, as well
88  have a role in maintenance of mitochondrial respiratory function, and this function is analogous to
89 piratory components for different aspects of respiratory functions, and collectively for the integrit
90 tigated whether defects in the mitochondrial respiratory function are consequences of the expression
91        Strategies to preserve and/or restore respiratory function are critical for successful treatme
92 ams that govern mitochondrial biogenesis and respiratory function are well known, posttranscriptional
93 ntilation, reduced lung injury with improved respiratory function, as compared with protective contro
94  the nos mutant likely resulted from altered respiratory function, as inhibition of NADH dehydrogenas
95  of nuclear genes required for mitochondrial respiratory function, as well as for other fundamental c
96  status, Do-Not-Resuscitate status, impaired respiratory function, ascites, hypoalbuminema, elevated
97                       Our aim was to compare respiratory function at 1 yr of age in infants assigned
98  treated with neonatal caffeine had improved respiratory function at 11 years of age compared with ch
99 t improved diaphragm muscle regeneration and respiratory function at 14 wk but not at 6 mo.
100 vidually and in groups, and ALS function and respiratory function at diagnosis.
101      38% of responding neurologists assessed respiratory function at presentation and 20% routinely m
102  fusion and fission events that can maintain respiratory function at steady-state levels amid the exi
103 eption of feeling secure, and improvement of respiratory function at the end of an SBT (most performe
104 ia in the critically ill can help to improve respiratory function, bowel function, mental status and
105 ic administration of methylxantines improves respiratory function but also leads to the development o
106 B. cepacia can lead not only to a decline in respiratory function but also to an acute systemic infec
107 t was not due to deficiency of mitochondrial respiratory function but was rather caused directly by t
108 bstance P modulates the reflex regulation of respiratory function by its actions both peripherally an
109 group, patients underwent daily screening of respiratory function by physicians, respiratory therapis
110                          Bedside measures of respiratory function can predict extubation success and
111                      Bedside measurements of respiratory function can predict extubation success and
112                                    Decreased respiratory function, cardiac involvement, and intranucl
113 ransport measurement and clinical condition (respiratory function, chest radiograph score, or Shwachm
114 less secure and reported less improvement of respiratory function compared with nurses' and physician
115 s that TMZ-mediated alterations in mtDNA and respiratory function contribute to TMZ-dependent acquire
116 hortened spine and trunk can severely affect respiratory function during early childhood.
117 t age-dependent decline in the mitochondrial respiratory function, especially COX activity, may parti
118 nt effects on blood pressure, pulse rate, or respiratory function (FEV1).
119       To determine the effects of sleep upon respiratory function, five adult patients with cystic fi
120     Oral quercetin supplementation protected respiratory function for 4-6 months during a 12 month do
121 ded into those above and those below average respiratory function for their age.
122                 There was a wide spectrum of respiratory function, from normal to markedly abnormal.
123 s examining the effect of sternal closure on respiratory function have not been published, and curren
124 erosolized brevetoxins in sea spray (reduced respiratory function); however, the reason for brevetoxi
125 g Cox1 synthesis in coa2Delta cells restores respiratory function if Cox10 protein levels are elevate
126                                          The respiratory function improvement was found to be associa
127 een identified a gene, CCC1, that maintained respiratory function in a Deltayfh1 yeast strain regardl
128 mmatory protein-2 significantly improves the respiratory function in addition to decreasing the infil
129 ed for those interested in cardiovascular or respiratory function in addition to motor function and c
130   This modified CHCHD4 protein reestablished respiratory function in AIF-deficient cells and enabled
131 ficant role in mitochondrial positioning and respiratory function in cardiac and skeletal muscle.
132 poorer birth outcomes, neurodevelopment, and respiratory function in children.
133                                              Respiratory function in hem15Delta cells can also be res
134 onchopulmonary dysplasia, but its effects on respiratory function in later childhood are unknown.
135  of nuclear genes required for mitochondrial respiratory function in mammalian cells.
136 yperexpanded lung, has been shown to improve respiratory function in many patients with end-stage emp
137 d magnesium sulphate (MgSO(4)) might improve respiratory function in patients with acute asthma.
138 ruitment, is a major cause of the decline in respiratory function in patients with CF and is a leadin
139 ajor differences in objective assessments of respiratory function in patients with moderate or severe
140  to the last weeks of gestation by comparing respiratory function in preterm infants whose mothers ha
141 ion between abdominal pattern of obesity and respiratory function in the European Prospective Investi
142 excessive sedation, and maintaining adequate respiratory function in the face of compromise resulting
143  Nurr1 has a key function in sustaining high respiratory function in these cells, and that Nurr1 abla
144 tified target genes, and those that regulate respiratory functions in melanocytes are among them.
145 hese results suggest that BCL-2 can regulate respiratory functions in response to mitochondrial distr
146 infected cells were adjacent to neurons with respiratory functions in the medulla.
147  resD mutant phenotypes, directly related to respiratory function, include streptomycin resistance, l
148    With increasing mutant ND5 mtDNA content, respiratory function including oxygen consumption and AT
149 n favor of HFOV in several other measures of respiratory function, including forced expiratory volume
150  mitochondria tolerance to calcium, enhances respiratory function (increases of 90% state 4, 220% sta
151  anti-leukemia agent, inhibits mitochondrial respiratory function, increases free radical generation,
152 tegrates cardiovascular, musculoskeletal and respiratory function into a single index that is largely
153                               Thus, impaired respiratory function is evident in infants born on avera
154 ngth is only mildly affected and cardiac and respiratory function is normal.
155                   We show that as the cells' respiratory function is reduced or eliminated, the expre
156                       However, monitoring of respiratory function is suboptimal and uncontrolled oxyg
157                                              Respiratory function is the main cause of mortality in p
158  variation in metabolic, cardiovascular, and respiratory function is unknown.
159                  However, based on assays of respiratory function, it has been reported that the prim
160  with its limited effects on hemodynamic and respiratory function, it is widely used in pediatric int
161 .02), with all exhibiting a rapid decline in respiratory function leading to death.
162 ed ubiquitin and mitophagy markers, and lost respiratory function, leading to neurodegeneration.
163 lar disease, smoking, body mass index grade, respiratory function levels (forced expiratory volume in
164      This study supports the hypothesis that respiratory function may be compromised after delayed st
165 c attenuation of TGF-beta signaling improves respiratory function, mdx mice were treated from 2 weeks
166 ctional Rating Scale-Revised (ALSFRS-R), and respiratory function, measured using percentage of predi
167  illness who had complete anthropometric and respiratory function measures obtained at a health visit
168                                              Respiratory function, mitochondrial membrane potential,
169 tracted, and effect estimates for changes in respiratory function, MPDA and asthma morbidity were poo
170 e was, however, no significant impairment of respiratory function, no alteration to the structure or
171       These data indicate that mitochondrial respiratory function, not abnormal redox homeostasis, di
172                       Daily screening of the respiratory function of adults receiving mechanical vent
173                                 Although the respiratory function of blood has been studied intensive
174                                              Respiratory function of coa4Delta cells was restored by
175 the essential role of the ND6 subunit in the respiratory function of Complex I and give some insights
176 hed the complex I assembly and disrupted the respiratory function of complex I.
177  on the informatics results, we analyzed the respiratory function of mitochondria isolated from wild-
178 verexpression of Mrs1 partially restores the respiratory function of mne1Delta cells.
179  consumption of Ptpn11(E76K/+) cells and the respiratory function of Ptpn11(E76K/+) mitochondria were
180  fitness variance is not directly related to respiratory functions of Hb.
181 ystemic administration of drugs that improve respiratory function often cause deleterious side effect
182              No differences in mitochondrial respiratory function or content were observed between le
183 er skeletal muscle mitochondrial content and respiratory function or the size of ischemic lesion afte
184 es in surfactant surface activity, postnatal respiratory function, or survival.
185                   The effect of idebenone on respiratory function outcomes was similar between patien
186 022), associated with improved mitochondrial respiratory function (postischemic percent respiratory c
187 iration in darkness is maximal, beyond which respiratory function rapidly declines) in upper canopy l
188  in the aged rats; (iii) brain mitochondrial respiratory function related to coupled oxidation was de
189 ce mutations greatly accelerated the loss of respiratory function, resulting from enhanced oxidative
190 enal chromaffin cells, which regulate cardio-respiratory function, resulting in irregular breathing w
191 e-radical generation and exhibit declines in respiratory function(s).
192 g, reduced overall lung volume, and improved respiratory function safely and consistently.
193 th in the dark, with both photosynthetic and respiratory functions severely compromised; growth in th
194     Oxygen consumption, a sensitive index of respiratory function, showed that mtDNA from chimpanzee,
195                                              Respiratory function studies demonstrated a gradual dete
196 sm, such as thyroid and parathyroid; and for respiratory function, such as trachea and lung.
197 mphysematous changes and correlate them with respiratory function tests (RFTs).
198                                   ACT score, respiratory function tests and respiratory resistance we
199 killing of P. aeruginosa by IgG2 have poorer respiratory function than infected patients who do not p
200 espiratory neuroplasticity after declines in respiratory function that are related to neurological im
201  minimal disturbance of gastrointestinal and respiratory function, thereby reducing the length of int
202 ce of deterioration in nutritional state and respiratory function to prevent further decline.
203                                  In tests of respiratory function, Trpa1(-/-) mice displayed profound
204 atory symptoms, total IgE, specific IgE, and respiratory function was assessed by logistic and multip
205                                              Respiratory function was compared within pairs of previo
206 ered relationship between protein import and respiratory function was confirmed through the investiga
207                                              Respiratory function was measured continuously for 30 mi
208                                              Respiratory function was measured in 29 control and 29 a
209                                              Respiratory function was measured in nonanesthetized mic
210                                              Respiratory function was measured using partial and rais
211                                              Respiratory function was measured with a head-out plethy
212                                              Respiratory function was protected for the first 4-6 mon
213  Using barometric plethysmography to measure respiratory function, we found that the MD rat develops
214  understand the transient nature of improved respiratory function, we measured PGC-1alpha pathway act
215 cause cardiolipin plays an important role in respiratory function, we measured the energy transformat
216 Functional Rating Scale (ALS-FRS) scores and respiratory function were analysed.
217                     Indexes of autonomic and respiratory function were assessed in CB intact and CB d
218 atment, and mitochondrial ultrastructure and respiratory function were assessed.
219 ory symptoms, and laboratory measurements of respiratory function were made for respiratory rate, tid
220                    The clinical outcomes and respiratory function were retrospectively compared betwe
221      The mutation had a severe effect on the respiratory function, with the activity of the bc(1) com
222 levels of mutant huntingtin on mitochondrial respiratory function within an appropriate cellular cont

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