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1 of apoE4 domain interaction on mitochondrial respiratory function.
2 d hippocampus were isolated and assessed for respiratory function.
3 abile zinc and interferes with mitochondrial respiratory function.
4 itochondrial RNA stability, translation, and respiratory function.
5 ns in respiratory supercomplex structure and respiratory function.
6 of scapular winging, contractures and normal respiratory function.
7 he effects of their toxic exoproducts impede respiratory function.
8 n, this measurement is limited as a guide to respiratory function.
9 f pulmonary integrity and edema compromising respiratory function.
10 igh-amplitude swelling and exhibited altered respiratory function.
11 obtained by spirometry, were used to assess respiratory function.
12 y barrier cells that hinders regeneration of respiratory function.
13 mutations caused little or no effect on the respiratory function.
14 All presented with a deterioration in respiratory function.
15 ruction would not lead to a deterioration in respiratory function.
16 DNA and protein content, gene expression, or respiratory function.
17 consequent barotrauma, resulting in improved respiratory function.
18 t (LS) is an essential system supporting the respiratory function.
19 SH, but fully restored mitochondrial GSH and respiratory function.
20 ling extubation demonstrate abnormalities of respiratory function.
21 and chest wall are important determinants of respiratory function.
22 ial DNA maintenance, and it is essential for respiratory function.
23 severe respiratory diseases and compromised respiratory function.
24 rsible inhibition of cardiac contractile and respiratory function.
25 ving only a marginal effect on mitochondrial respiratory function.
26 ial protein involved in iron homeostasis and respiratory function.
27 re non-viable, emphasizing the importance of respiratory function.
28 unctional Rating Scale-Revised score and the respiratory function.
29 cal role for Oma1 protease in fine-tuning of respiratory function.
30 coactivator-1alpha (PGC-1alpha) and enhanced respiratory function.
31 ormal structure and displayed a 35-43% lower respiratory function.
32 were clinically satisfactory, with adequate respiratory function.
33 ed to immediate and sustained improvement of respiratory function.
34 sociations of exposure to air pollution with respiratory function.
35 e amount of contractile tissue available for respiratory function.
36 mpiled using simple measures of physical and respiratory function.
37 nal medulla, and for their control of cardio-respiratory function.
38 armacologic activation of TrkB would improve respiratory function.
39 thin the lung interstitium leads to impaired respiratory function.
40 e expertise in caring for patients with poor respiratory function.
41 many nuclear genes specifying mitochondrial respiratory function.
42 it robustly measures the brain, cardiac and respiratory functions.
43 tioning in the case of immune, hormonal, and respiratory functions.
44 ucial roles in modulating cardiovascular and respiratory functions.
45 of exogenous cytochrome c markedly restored respiratory functions.
46 the expression of numerous genes involved in respiratory functions.
47 in lungs, participating in the regulation of respiratory functions.
48 ons, which support other respiratory and non-respiratory functions.
49 (mtErbB2) negatively regulates mitochondrial respiratory functions.
51 at presentation and 20% routinely monitored respiratory function; 32% relied on symptoms as the only
52 to common allergens, and to undergo tests of respiratory function, 659 women and 500 men agreed to an
53 ty (4.2%; P<0.05), tolerance to calcium, and respiratory function (72% increase in state 3 and 23% in
54 hore crabs in maintaining osmoregulatory and respiratory function after acute exposure to both anthro
55 reported significant abnormalities in infant respiratory function after clinical recovery from bronch
58 in neural centers critical for autonomic and respiratory function alleviates the lethality associated
59 ng was defined as good cognitive, motor, and respiratory functioning, along with absence of disabilit
61 ial permeability transition, decline in both respiratory function and activity of cytochrome c oxidas
62 rectly affects acid-base and ion regulation, respiratory function and aerobic performance in aquatic
65 Consequently, inhibition of HsPDF reduces respiratory function and cellular ATP levels, causing de
67 in (0.2%) in 2 month old mdx mice to improve respiratory function and end-point functional and histol
68 hmogenic networks, their respective roles in respiratory function and how they interact to constitute
69 sphate (ATP); they had reduced mitochondrial respiratory function and increased oxidative stress, com
70 owever, the impact of single LT on long-term respiratory function and nutritional status has not been
72 sts that loss of Lon1 significantly modifies respiratory function and plant performance by small but
73 es mitophagy and improves both mitochondrial respiratory function and proteostasis in aged flies.
74 Late diagnosis significantly worsens the respiratory function and reduces the chance for normal d
76 process, which is necessary to regain normal respiratory function and restore the lungs to homeostasi
77 increases survival, rescues abnormalities in respiratory function and social recognition, and improve
80 raction between the disruption of this novel respiratory function and the loss of wild-type mtDNA.
81 hysiological basis of these vocalizations in respiratory function and to additional developmental var
82 t time a physiological improvement of cardio-respiratory functions and a correction of behavioral fea
84 ities of Fe-S cluster enzymes, a decrease in respiratory function, and an increase in oxidative stres
85 chondrial protein degradation, mitochondrial respiratory function, and cell viability are compromised
86 ochrome b, several mutations that impair the respiratory function, and reversions that correct the de
88 have a role in maintenance of mitochondrial respiratory function, and this function is analogous to
89 piratory components for different aspects of respiratory functions, and collectively for the integrit
90 tigated whether defects in the mitochondrial respiratory function are consequences of the expression
92 ams that govern mitochondrial biogenesis and respiratory function are well known, posttranscriptional
93 ntilation, reduced lung injury with improved respiratory function, as compared with protective contro
94 the nos mutant likely resulted from altered respiratory function, as inhibition of NADH dehydrogenas
95 of nuclear genes required for mitochondrial respiratory function, as well as for other fundamental c
96 status, Do-Not-Resuscitate status, impaired respiratory function, ascites, hypoalbuminema, elevated
98 treated with neonatal caffeine had improved respiratory function at 11 years of age compared with ch
101 38% of responding neurologists assessed respiratory function at presentation and 20% routinely m
102 fusion and fission events that can maintain respiratory function at steady-state levels amid the exi
103 eption of feeling secure, and improvement of respiratory function at the end of an SBT (most performe
104 ia in the critically ill can help to improve respiratory function, bowel function, mental status and
105 ic administration of methylxantines improves respiratory function but also leads to the development o
106 B. cepacia can lead not only to a decline in respiratory function but also to an acute systemic infec
107 t was not due to deficiency of mitochondrial respiratory function but was rather caused directly by t
108 bstance P modulates the reflex regulation of respiratory function by its actions both peripherally an
109 group, patients underwent daily screening of respiratory function by physicians, respiratory therapis
113 ransport measurement and clinical condition (respiratory function, chest radiograph score, or Shwachm
114 less secure and reported less improvement of respiratory function compared with nurses' and physician
115 s that TMZ-mediated alterations in mtDNA and respiratory function contribute to TMZ-dependent acquire
117 t age-dependent decline in the mitochondrial respiratory function, especially COX activity, may parti
120 Oral quercetin supplementation protected respiratory function for 4-6 months during a 12 month do
123 s examining the effect of sternal closure on respiratory function have not been published, and curren
124 erosolized brevetoxins in sea spray (reduced respiratory function); however, the reason for brevetoxi
125 g Cox1 synthesis in coa2Delta cells restores respiratory function if Cox10 protein levels are elevate
127 een identified a gene, CCC1, that maintained respiratory function in a Deltayfh1 yeast strain regardl
128 mmatory protein-2 significantly improves the respiratory function in addition to decreasing the infil
129 ed for those interested in cardiovascular or respiratory function in addition to motor function and c
130 This modified CHCHD4 protein reestablished respiratory function in AIF-deficient cells and enabled
131 ficant role in mitochondrial positioning and respiratory function in cardiac and skeletal muscle.
134 onchopulmonary dysplasia, but its effects on respiratory function in later childhood are unknown.
136 yperexpanded lung, has been shown to improve respiratory function in many patients with end-stage emp
137 d magnesium sulphate (MgSO(4)) might improve respiratory function in patients with acute asthma.
138 ruitment, is a major cause of the decline in respiratory function in patients with CF and is a leadin
139 ajor differences in objective assessments of respiratory function in patients with moderate or severe
140 to the last weeks of gestation by comparing respiratory function in preterm infants whose mothers ha
141 ion between abdominal pattern of obesity and respiratory function in the European Prospective Investi
142 excessive sedation, and maintaining adequate respiratory function in the face of compromise resulting
143 Nurr1 has a key function in sustaining high respiratory function in these cells, and that Nurr1 abla
144 tified target genes, and those that regulate respiratory functions in melanocytes are among them.
145 hese results suggest that BCL-2 can regulate respiratory functions in response to mitochondrial distr
147 resD mutant phenotypes, directly related to respiratory function, include streptomycin resistance, l
148 With increasing mutant ND5 mtDNA content, respiratory function including oxygen consumption and AT
149 n favor of HFOV in several other measures of respiratory function, including forced expiratory volume
150 mitochondria tolerance to calcium, enhances respiratory function (increases of 90% state 4, 220% sta
151 anti-leukemia agent, inhibits mitochondrial respiratory function, increases free radical generation,
152 tegrates cardiovascular, musculoskeletal and respiratory function into a single index that is largely
160 with its limited effects on hemodynamic and respiratory function, it is widely used in pediatric int
162 ed ubiquitin and mitophagy markers, and lost respiratory function, leading to neurodegeneration.
163 lar disease, smoking, body mass index grade, respiratory function levels (forced expiratory volume in
164 This study supports the hypothesis that respiratory function may be compromised after delayed st
165 c attenuation of TGF-beta signaling improves respiratory function, mdx mice were treated from 2 weeks
166 ctional Rating Scale-Revised (ALSFRS-R), and respiratory function, measured using percentage of predi
167 illness who had complete anthropometric and respiratory function measures obtained at a health visit
169 tracted, and effect estimates for changes in respiratory function, MPDA and asthma morbidity were poo
170 e was, however, no significant impairment of respiratory function, no alteration to the structure or
175 the essential role of the ND6 subunit in the respiratory function of Complex I and give some insights
177 on the informatics results, we analyzed the respiratory function of mitochondria isolated from wild-
179 consumption of Ptpn11(E76K/+) cells and the respiratory function of Ptpn11(E76K/+) mitochondria were
181 ystemic administration of drugs that improve respiratory function often cause deleterious side effect
183 er skeletal muscle mitochondrial content and respiratory function or the size of ischemic lesion afte
186 022), associated with improved mitochondrial respiratory function (postischemic percent respiratory c
187 iration in darkness is maximal, beyond which respiratory function rapidly declines) in upper canopy l
188 in the aged rats; (iii) brain mitochondrial respiratory function related to coupled oxidation was de
189 ce mutations greatly accelerated the loss of respiratory function, resulting from enhanced oxidative
190 enal chromaffin cells, which regulate cardio-respiratory function, resulting in irregular breathing w
193 th in the dark, with both photosynthetic and respiratory functions severely compromised; growth in th
194 Oxygen consumption, a sensitive index of respiratory function, showed that mtDNA from chimpanzee,
199 killing of P. aeruginosa by IgG2 have poorer respiratory function than infected patients who do not p
200 espiratory neuroplasticity after declines in respiratory function that are related to neurological im
201 minimal disturbance of gastrointestinal and respiratory function, thereby reducing the length of int
204 atory symptoms, total IgE, specific IgE, and respiratory function was assessed by logistic and multip
206 ered relationship between protein import and respiratory function was confirmed through the investiga
213 Using barometric plethysmography to measure respiratory function, we found that the MD rat develops
214 understand the transient nature of improved respiratory function, we measured PGC-1alpha pathway act
215 cause cardiolipin plays an important role in respiratory function, we measured the energy transformat
219 ory symptoms, and laboratory measurements of respiratory function were made for respiratory rate, tid
221 The mutation had a severe effect on the respiratory function, with the activity of the bc(1) com
222 levels of mutant huntingtin on mitochondrial respiratory function within an appropriate cellular cont
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