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1 age or a less severe syndrome, such as upper respiratory infection).
2 ontrol IAV replication and succumb to lethal respiratory infection.
3 pes and organ systems dictate the outcome of respiratory infection.
4 erate or severe exacerbation and first upper respiratory infection.
5 tinopathy, migraine disorder, and post-upper respiratory infection.
6 a portal of entry for the establishment of a respiratory infection.
7 t important challenge to understanding lower respiratory infection.
8 8(+) Trm cells in the lung airways following respiratory infection.
9 rsening disease outcome is a new paradigm in respiratory infection.
10 lung tissue of alpaca that died of a severe respiratory infection.
11 e immune response during Pseudomonas-induced respiratory infection.
12 r antiviral treatment of patients with viral respiratory infection.
13 essential role in both persistence and acute respiratory infection.
14 opulations that are generated in response to respiratory infection.
15 er respiratory tract, increasing the risk of respiratory infection.
16 thology following intranasal inoculation and respiratory infection.
17 naling axis they activate to protect against respiratory infection.
18 urine host's gene expression and response to respiratory infection.
19 CD11b(+) macrophages in the lungs following respiratory infection.
20 aryngeal niche, a reservoir for ME and upper respiratory infections.
21 tant feature of diabetes that predisposes to respiratory infections.
22 ng antibiotic therapy in patients with acute respiratory infections.
23 uals and may help reduce symptoms related to respiratory infections.
24 us (RSV) is associated with severe childhood respiratory infections.
25 erate or severe COPD exacerbations and upper respiratory infections.
26 ial to revolutionize vaccine development for respiratory infections.
27 sorders, increased susceptibly to cancer and respiratory infections.
28 host protective against bacterial and viral respiratory infections.
29 associated with susceptibility to asthma and respiratory infections.
30 of asthma exacerbations are related to viral respiratory infections.
31 set inflammatory bowel disease and recurrent respiratory infections.
32 anisms and are therefore equipped to control respiratory infections.
33 ndings for early diagnosis and prevention of respiratory infections.
34 s in families remain a valuable way to study respiratory infections.
35 have a significant impact on the outcome of respiratory infections.
36 ly to the battle against influenza and other respiratory infections.
37 ir pollution exposure and the development of respiratory infections.
38 uary 2011 from patients with suspected acute respiratory infections.
39 fluid specimens from subjects with suspected respiratory infections.
40 n reduced the incidence of diarrhea or lower respiratory infections.
41 responses as a therapeutic modality to treat respiratory infections.
42 sed guidelines can reduce antibiotic use for respiratory infections.
43 l of individuals with high susceptibility to respiratory infections.
44 entification of these causative pathogens in respiratory infections.
45 Disruption of ciliary flow can lead to respiratory infections.
46 monologist views the conceptual framework of respiratory infections.
47 phages are the first line of defense against respiratory infections.
48 B) may confer nonspecific protection against respiratory infections.
50 ng ever-smokers associated with infant lower respiratory infection (-108.2 ml; P = 0.001) and home ov
51 sh (23/3), decreased appetite (20/15), upper respiratory infection (20/0), pneumonia (13/10), and ala
52 mission diagnoses of patients with AIDS were respiratory infection (28.6%) and sepsis (16.9%), which
55 had multiple diagnoses), 62.2% had an acute respiratory infection; 5.0% of these infections were rad
57 iated with increased susceptibility to upper respiratory infection-a major precipitant of exacerbatio
59 escribe the molecular epidemiology of HAdV21 respiratory infections across the country, 150 clinical
60 iddle East respiratory syndrome severe acute respiratory infection (adjusted odds ratio, 5.87; 95% CI
61 ction (admission HRR, 0 [95% CI, 0-.24]) and respiratory infections (admission HRR, 0.37 [95% CI, .16
62 jection, lymphocytic bronchiolitis (LB), and respiratory infection after lung transplantation (LTx).
65 The timing of a child's first acute lower respiratory infection (ALRI) is important, because the y
66 22% of all episodes of (severe) acute lower respiratory infection (ALRI) resulting in 55 000 to 199
67 in-hospital deaths due to severe acute lower respiratory infections (ALRI) in young children worldwid
68 h chronic rejection, LB, acute rejection, or respiratory infections, although significantly less pati
69 species, are the causative agents of severe respiratory infections among children and are linked to
71 ry syncytial virus (RSV) is a major cause of respiratory infections among young children and can lead
72 ate genes and 10 phenotypes related to viral respiratory infection and asthma control were examined i
75 ignificant morbidity and mortality are acute respiratory infection and dehydration from severe diarrh
78 hR) correlates with poorer ability to combat respiratory infection and lower antibody levels in the o
79 with reduced time in the hospital with acute respiratory infection and reduced diastolic blood pressu
81 The early phenotype was more likely to have respiratory infections and acute exacerbations at baseli
86 eutrophil migratory accuracy with age during respiratory infections and determine if and how a statin
88 pah virus emerged a few years later, causing respiratory infections and encephalitis in Southeast Asi
93 lated inflammasome activation contributes to respiratory infections and pathologic airway inflammatio
94 is a potential therapeutic target for upper respiratory infections and that flavones may have clinic
95 er, excessive mucus contributes to transient respiratory infections and to the pathogenesis of numero
96 ovide insight into the pathogenesis of viral respiratory infections and virus-induced exacerbations o
97 to the protective effect of breastfeeding on respiratory infections and wheezing in early infancy.
99 ion related to autoimmune diseases, smoking, respiratory infection, and pollution exposure, and possi
100 separately, duration of gastrointestinal and respiratory infections, and duration of hospitalization)
101 were collected during maternal acute febrile respiratory infections, and from infants with any respir
103 [95% confidence interval {CI}, .76-.95]) and respiratory infection (aOR, 0.82 [95% CI, .75-.90]) and
111 Previous studies have suggested that acute respiratory infection (ARI) and nonsteroidal anti-inflam
113 viral pathogens, causing epidemics of acute respiratory infection (ARI), especially bronchiolitis an
114 ce Project (IISP) monitored outpatient acute respiratory infection (ARI; defined as the presence of >
115 iratory visits, including specifically acute respiratory infections (ARI), asthma, pneumonia, chronic
117 s to reduce antibiotic prescribing for acute respiratory infections (ARIs), information on factors th
119 clinical trials, using an established mouse respiratory infection as a model and starting treatment
121 BALT) is associated with immune responses to respiratory infections as well as with local pathology d
122 n inherited disease characterised by chronic respiratory infections associated with bronchiectasis.
124 s of day care attendance and number of lower respiratory infections at 12 months were associated with
126 unced associations were seen for acute upper respiratory infections at multiple and unspecified sites
127 ing treatment for asthma, and without recent respiratory infections (baseline COPD was not an exclusi
128 chronic obstructive pulmonary disease, lower respiratory infections, breast cancer, other cardiovascu
129 ibutes to TCD8 impairment during viral lower respiratory infection, but how it regulates TCD8 impairm
130 1%) samples submitted for viral diagnosis of respiratory infection, but not in normal adult control s
132 tibody protects against RSV-associated lower respiratory infections, but placental malaria (PM) and m
133 oap could substantially reduce diarrhoea and respiratory infections, but prevalence of adequate handw
134 ed the importance of the Bvg(+) phase during respiratory infection, Bvg-regulated gene activation in
135 (CD8)) are impaired during acute viral lower respiratory infection by the inhibitory receptor program
136 mbers of the microbiota that protect against respiratory infection by the major human pathogens Strep
141 causative agent of whooping cough, a serious respiratory infection causing hundreds of thousands of d
144 n particular smokers are more susceptible to respiratory infections contributing to acute exacerbatio
145 to be due to a cold, otitis media, an upper respiratory infection, croup, asthma, bronchitis, bronch
147 icas in the mid-1990s as the cause of severe respiratory infections, designated hantavirus pulmonary
150 of 135 previously healthy infants with acute respiratory infection due to human rhinovirus (HRV; n =
152 lesser extent, among children who had had a respiratory infection during the first year of life (haz
153 oantibody seroconversion was associated with respiratory infections during the first 6 months of life
154 alth-care workers (HCWs) during outbreaks of respiratory infections (e.g. Influenza A H1N1 (2009)) is
155 y ventilated patients with severe sepsis and respiratory infection, early treatment with a neuromuscu
156 valid exposure route for a potentially fatal respiratory infection, even for viruses that do not demo
157 that acute otitis media occurs during upper respiratory infection, even in the absence of nasopharyn
162 iddle East respiratory syndrome severe acute respiratory infection from other etiologies; therefore,
163 of critically ill patients with severe acute respiratory infection from the Middle East respiratory s
164 onin-guided treatment in patients with acute respiratory infections from different clinical settings.
165 ve pulmonary disease, lung cancer, and lower respiratory infections from epidemiological studies usin
166 iddle East respiratory syndrome severe acute respiratory infection had more severe hypoxemic respirat
167 -infected participants in the HAART era with respiratory infections had an increased risk of death co
168 roportion to global estimates of acute lower respiratory infection hospitalizations among children ag
169 n lead to adverse health outcomes, including respiratory infections, impaired lung function, cardiac
173 RSV) is the most common cause of acute lower respiratory infection in infants and young children and
174 virus (RSV) is a major cause of severe lower respiratory infection in infants and young children and
175 revalent, parvovirus that is associated with respiratory infection in infants and young children.
176 eumovirus (HMPV) is a leading cause of acute respiratory infection in infants, children, and the elde
177 NALE: Bronchiolitis is the most common lower respiratory infection in infants; however, it remains un
178 ional antibiotic use for patients with acute respiratory infection in low-income and middle-income co
179 sol delivery of B. pseudomallei to establish respiratory infection in mice and studied CPS I in the c
180 different oxygen saturation targets in acute respiratory infection in older children, particularly in
181 ion for prevention of exacerbation and upper respiratory infection in patients with COPD are lacking.
182 was considered to be related to study drug (respiratory infection in the placebo group, traffic acci
183 veral virulence factors to establish chronic respiratory infections in bronchiectasis, chronic obstru
185 um is occasionally used to investigate lower respiratory infections in children but has not been wide
186 pectively assessed viral etiologies of acute respiratory infections in community-based elderly indivi
187 nza virus and other respiratory pathogens to respiratory infections in community-dwelling older adult
190 fraction of PM2.5 exacerbate upper and lower respiratory infections in early life, and that the carbo
192 sis supports an inverse relationship between respiratory infections in early-life and atopic diseases
193 h human and avian viruses mounted productive respiratory infections in ferrets following ocular-only
194 viruses are one of the most common causes of respiratory infections in humans, causing 1 billion infe
195 (IAVs) are one of the most common causes of respiratory infections in humans, resulting in thousands
196 nza viruses of the H5N1 subtype cause severe respiratory infections in humans, which have resulted in
202 t has previously been shown to reduce lethal respiratory infections in mice to undetectable levels wh
204 logical studies have observed an increase in respiratory infections in subjects and populations expos
205 e was a lower incidence of exacerbations and respiratory infections in subjects treated with OC000459
207 tory evidence on the role of early childhood respiratory infections in the development of asthma and
208 ht be enormous, given the high prevalence of respiratory infections in the first year of life and the
211 derate or severe exacerbation, but not upper respiratory infection, in patients with COPD with baseli
212 Mycoplasma pneumoniae is a leading cause of respiratory infections, including community-acquired pne
213 n for skin and soft-tissue infection (SSTI), respiratory infection, intra-abdominal infection, or uri
214 sting is essential component of severe acute respiratory infection investigation for at-risk patients
216 Overall, these data demonstrate that viral respiratory infection is associated with a marked increa
219 Rapid and definitive diagnosis of viral respiratory infections is imperative in patient triage a
221 Peninsula with a clinical syndrome of acute respiratory infections, later designated as Middle East
222 issue Infections of the Head and Neck, Upper Respiratory Infections, Lower Respiratory Tract infectio
225 s between early-life exposures (infant lower respiratory infection, manual social class, home overcro
227 iddle East respiratory syndrome severe acute respiratory infection (median [quartile 1, quartile 3] 5
228 ed mouse models of Chlamydia and Haemophilus respiratory infection-mediated, ovalbumin-induced severe
230 enza death across countries by comparing GHE respiratory infection mortality rates from countries wit
232 ntly by cause of death and was strongest for respiratory infections (MRR, 0.20 [95% CI, .07-.55]).
233 iddle East respiratory syndrome severe acute respiratory infection (n = 330) admitted between Septemb
238 atients with community-acquired severe acute respiratory infection of non-Middle East respiratory syn
239 n of MeV-specific T cells for 6 months after respiratory infection of rhesus macaques with wild type
240 During Mycobacterium tuberculosis and other respiratory infections, optimal T cell activation requir
243 Adjusted odds or hazards ratios for incident respiratory infections or non-infectious respiratory dia
244 disease-specific or other entities, such as respiratory infections or pollution, are responsible.
246 (OR = 1.013; 95% CI: 1.003, 1.023) and upper respiratory infections (OR = 1.015; 95% CI: 1.008, 1.022
247 with the following acute health conditions: respiratory infections, otitis media, gastroenteritis, n
248 ergistic interactions of smoking with infant respiratory infection (P = 0.04) and early-life home ove
249 iddle East respiratory syndrome severe acute respiratory infection patients were younger than those w
251 the typical clinical syndrome of viral upper respiratory infection progressing to the lower respirato
253 s were randomized if they exhibited signs of respiratory infection (purulent secretions and Clinical
255 antibiotic treatment in patients with acute respiratory infections reduces antibiotic exposure and s
256 e of YLDs in 1990, 2005, and 2010, and lower respiratory infections remained the leading cause of YLL
257 ociation between household air pollution and respiratory infections, respiratory tract cancers, and c
258 e animals after intravenous infection, while respiratory infection results in virus replication in th
259 rotective effects of BCG vaccination against respiratory infection (RI) and sepsis not attributable t
260 ism from IL4 gene was significant for pooled respiratory infections (rs2070874; 1.66 [1.29-2.14]).
262 included postextubation respiratory failure, respiratory infection, sepsis and multiorgan failure, IC
265 f pulmonary host defense mechanisms to viral respiratory infection susceptibility in very young child
266 n to communities around the world by causing respiratory infections that can be highly contagious and
267 ocalcitonin protocols in patients with acute respiratory infections thus has the potential to improve
272 = 80,399), pneumonia (n = 63,359), and upper respiratory infection (URI) (n = 359,246) among children
274 om immunocompetent subjects during 146 upper respiratory infections (URIs); the sensitivities for rev
276 /182 prescriptions), while it was non-severe respiratory infections using the control algorithm (ALMA
278 Index score, chronic kidney disease, cancer, respiratory infection, vasoactive agent use, and receipt
281 or in vivo 25(OH)D status to increased viral respiratory infections, we poorly understand how vitamin
282 pediatric and adult civilian cases of acute respiratory infection were characterized to compile mole
283 ED visits for asthma or wheeze and for upper respiratory infections were associated with PM2.5 concen
284 ient data from trials in which patients with respiratory infections were randomly assigned to receive
286 with oral amoxicillin) of suspected neonatal respiratory infections, were linked with traditional bir
287 ibuting to 14.3% of deaths), replacing lower respiratory infections, which were the leading cause of
288 Bvg(+) phase is necessary and sufficient for respiratory infection while the Bvg(-) phase is required
290 en-specific antibodies (Abs) protect against respiratory infection with influenza A virus (IAV) and S
291 esponses with desialylation may occur during respiratory infection with NA-expressing microbes and co
292 onses as well as enhanced protection against respiratory infection with P. aeruginosa compared to imm
295 ed significantly increased susceptibility to respiratory infection with serotype 3 S. pneumoniae rela
298 lmonary disease are susceptible to recurrent respiratory infections with pathogens, including nontype
299 imal models involved in vaccine research for respiratory infections, with advantages and disadvantage
300 rainfluenza viruses (HPIVs) cause widespread respiratory infections, with no vaccines or effective tr
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