ライフサイエンスコーパス: respiratory insufficiency

1 pleural effusion and clinically as hypoxemic respiratory insufficiency.

2 bulbar palsy, sensorineural hearing loss and respiratory insufficiency.

3 ower limbs), hearing loss, optic atrophy and respiratory insufficiency.

4 and sensorimotor neuropathy with or without respiratory insufficiency.

5 th in severe cases as a result of associated respiratory insufficiency.

6 o WNV-infected or sham-infected mice without respiratory insufficiency.

7 equently, neonatal pups died at birth due to respiratory insufficiency.

8 zed by muscle weakness, spinal rigidity, and respiratory insufficiency.

9 tients had lethargy, 1 had ataxia, and 1 had respiratory insufficiency.

10 e oxygen/RS is a better indicator of chronic respiratory insufficiency.

11 d fever, myalgias, progressive weakness, and respiratory insufficiency.

12 is feasible for noninvasive therapy of acute respiratory insufficiency.

13 r only slowly progressive weakness and early respiratory insufficiency.

14 ss the protein and they die perinatally from respiratory insufficiency.

15 (4% versus 1%), pneumonitis (8% versus 4%), respiratory insufficiency (13% versus 8%), transient cen

16 deaths in the validation cohort were due to respiratory insufficiency, 14 percent to myocardial infa

17 , renal failure (0.44% versus 0.29%; P=0.6), respiratory insufficiency (3.5% versus 3.8%; P=0.7), and

18 Hypertrophic cardiomyopathy (9 [53%]) and respiratory insufficiency (8 [47%]) were also prominent

19 One patient died of respiratory insufficiency after a laparotomy.

20 sorders, such as obstructive sleep apnea, or respiratory insufficiency after spinal injury or during

21 ies for treating breathing disorders such as respiratory insufficiency after spinal injury.

22 The p.F93del mutation results in respiratory insufficiency and loss of complex I stabilit

23 resented with severe hypotonia, bradycardia, respiratory insufficiency, and heart failure; two sister

24 zed by accumulation of pulmonary surfactant, respiratory insufficiency, and increased infections.

25 rash, marked osteopenia, lytic bone lesions, respiratory insufficiency, and thrombosis.

26 infections contribute to lung tissue damage, respiratory insufficiency, and ultimately death in the p

27 ises arthrogryposis, spontaneously resolving respiratory insufficiency at birth, muscular atrophy pre

28 akness of proximal dominance, hypotonia, and respiratory insufficiency but typically not cardiac dysf

29 greater distally than proximally, as well as respiratory insufficiency, cardiomyopathy, and cervical

30 soon after birth, apparently as a result of respiratory insufficiency caused by rib and sternum defe

31 Egr2-null mice die perinatally of respiratory insufficiency characterized by subnormal res

32 ) is susceptible to fibrosis and the ensuing respiratory insufficiency contributes to significant mor

33 he presence of eosinophils in cases of acute respiratory insufficiency due to diffuse alveolar damage

34 Severely affected babies often die from respiratory insufficiency due to progressive chest defor

35 admission diagnoses were sepsis (21.5%) and respiratory insufficiency/failure (25.7%).

36 children who have varying degrees of chronic respiratory insufficiency from many causes.

37 ential sequelae to even mild or intermittent respiratory insufficiency have been identified.

38 nophilic pneumonia is characterized by acute respiratory insufficiency, hypoxemia, fever, diffuse rad

39 ic pleural effusion in 366 (30.1%) patients, respiratory insufficiency in 141 (11.6%), acute respirat

40 may inspire novel therapeutic strategies for respiratory insufficiency in diverse conditions, such as

41 get in developing therapeutic strategies for respiratory insufficiency in patients with, for example,

42 ced development of cardiomyopathy and severe respiratory insufficiency in their teens; two had rigid

43 To explore the possibility that neurological respiratory insufficiency is a broad mechanism of death

44 Respiratory insufficiency is the leading cause of death

45 ough, atelectasis and pneumonia, and chronic respiratory insufficiency leading to respiratory failure

46 We assessed in health centres respiratory insufficiency, low back pain, degree of phys

47 interventions were IV sodium chloride before respiratory insufficiency (n = 17), IV sodium chloride a

48 fficiency (n = 17), IV sodium chloride after respiratory insufficiency (n = 22), and fluid restrictio

49 Respiratory insufficiency occurred in two of these patie

50 opment or steroidogenesis) are not born with respiratory insufficiency or abnormal lung development,

51 al because they lacked significant weakness, respiratory insufficiency, or scoliosis.

52 Quadriplegia, quadriparesis, or respiratory insufficiency persisted after the first week

53 tients who received IV sodium chloride after respiratory insufficiency, plasma sodium levels were inc

54 ients who received IV sodium chloride before respiratory insufficiency, plasma sodium levels were inc

55 l course was characterized by progression of respiratory insufficiency, pleural effusions and pulmona

56 The predominant reasons for admission were respiratory insufficiency, postoperative care, and heart

57 els display similar perinatal lethality with respiratory insufficiency, reduced body weight and lengt

58 Thirty-four patients with 35 episodes of respiratory insufficiency requiring airway support or ox

59 of a severely constricted thoracic cage and respiratory insufficiency; retinal degeneration, cystic

60 Neurological respiratory insufficiency strongly correlates with morta

61 entilatory control disorders associated with respiratory insufficiency, such as spinal injury and mot

62 respiratory neuroplasticity in disorders of respiratory insufficiency suggests that membrane estroge

63 lowly declines, and skeletal deformities and respiratory insufficiency supervene.

64 produces a dramatic neuromotor syndrome and respiratory insufficiency that often necessitate intensi

65 ely study clinical and radiological PPCs and respiratory insufficiency therapies in a high-risk surgi

66 deformity develop before the children die of respiratory insufficiency, usually in the second year.

67 Adult patients with acute respiratory insufficiency were randomized to receive NPP

68 however, these died shortly after birth from respiratory insufficiency, without primary cardiopulmona