ライフサイエンスコーパス: respiratory muscle

1 r atrophy in limb muscles when compared with respiratory muscle.

2 ic pressure and it increases the load on the respiratory muscles.

3 with activity related to the contractions of respiratory muscles.

4 ry control system or due to paralysis of the respiratory muscles.

5 rons, leading to atrophy of limb, axial, and respiratory muscles.

6 erception of increased work performed by the respiratory muscles.

7 itional/motor control, including that of the respiratory muscles.

8 ts primarily from the loss of innervation of respiratory muscles.

9 tory complications secondary to paralysis of respiratory muscles.

10 in the work output and metabolic rate of the respiratory muscles.

11 ary component in addition to the weakness of respiratory muscles.

12 ventilator imposes too little stress on the respiratory muscles.

13 consideration when setting PSV to unload the respiratory muscles.

14 educing the metabolic demands of cardiac and respiratory muscles.

15 lve truncal, neck-flexor, facial, bulbar and respiratory muscles.

16 reflect in part structural attributes of the respiratory muscles; (2) that the variation of maximal t

17 Respiratory muscle activation was achieved by placing th

18 exercise and associated effects on dyspnea, respiratory muscle activation, and pulmonary gas exchang

19 flow or pressure at the airway opening or in respiratory muscle activation.

20 f tightness were unchanged by the absence of respiratory muscle activity (p = 0.12).

21 isease-specific differences in mechanics and respiratory muscle activity did not influence the key as

22 e vestibular system contributes to adjusting respiratory muscle activity during changes in posture, a

23 he vestibular system contributes to altering respiratory muscle activity during movement and changes

24 reathing, and thereby minimize activation of respiratory muscle afferents and motor command, subjects

25 lail chest, interfere with the action of the respiratory muscles-again in a manner unique to each dis

26 s using bioelectrical impedance, quadriceps, respiratory muscle and handgrip strength, and physical p

27 ter fraction of whole-body VO2 towards their respiratory muscles and develop EIAH.

28 %max), quantifies the mechanical load on the respiratory muscles and relates closely to breathlessnes

29 ed a suppression of tone in the postural and respiratory muscles and simultaneously caused a signific

30 quired weakness syndromes affecting both the respiratory muscles and the limb muscles.

31 ter fraction of whole-body VO2 towards their respiratory muscles, and demonstrate EIAH, suggesting wo

32 other tissues, including multiple accessory respiratory muscles, and of course the heart itself for

33 lated by means of the recipient's airway and respiratory muscles, and they provided gas exchange in v

34 rmal values and in 293 patients referred for respiratory muscle assessment to compare the two measure

35 te that C-26 cancer cachexia causes profound respiratory muscle atrophy and weakness and ventilatory

36 This study was conducted to stimulate respiratory muscles by functional magnetic stimulation (

37 ypothesis that reflexes arising from working respiratory muscle can elicit increases in sympathetic v

38 Weakness of the respiratory muscles can dominate the clinical manifestat

39 An imbalance between work of breathing and respiratory muscle capacity often results in rapid, shal

40 related to physiologic work of breathing and respiratory muscle capacity, should improve application

41 aled minute ventilation, and the duration of respiratory muscle contraction assessed by the ratio of

42 l, pleural pressures generated during active respiratory muscle contraction, lung resistance and dyna

43 Accessory respiratory muscle deoxygenation was present only in pat

44 reserving O(2) transport to locomotor and to respiratory muscles during exercise.

45 urons or in coordinating the contractions of respiratory muscles during nonrespiratory responses (e.g

46 of the lung to increase ventilation and not respiratory muscle dysfunction a more attractive explana

47 r, a highly significant relationship between respiratory muscle dysfunction and symptoms of dyspnea.

48 Evidence has accumulated that respiratory muscle dysfunction develops in critically il

49 develop respiratory muscle weakness and that respiratory muscle dysfunction may contribute to the une

50 umans with peritonitis may be predisposed to respiratory muscle dysfunction.

51 tient-ventilator asynchrony and do not allow respiratory muscle effort assessment.

52 Research on the respiratory muscles embraces techniques of molecular bio

53 pogonadal and eugonadal patients had similar respiratory muscle endurance times (302 +/- 29 and 313 +

54 Respiratory muscle endurance was measured with an increm

55 nce in support of one possible mechanism for respiratory muscle failure in emphysema.

56 erated morbidity and mortality consequent to respiratory muscle failure.

57 PV in children with acute severe asthma with respiratory muscle fatigue and failure of medical treatm

58 re overweight or obese, populations in which respiratory muscle fatigue can be limiting.

59 eakness (Pdi(sniff) < 30 cm H(2)O), abnormal respiratory muscle function (lesser rise in Pdi) and a l

60 These patients had pathologic respiratory muscle function (P=0.005) and decreased insp

61 virus (HIV), the effects of HIV infection on respiratory muscle function have not been well character

62 Indeed, most clinicians do not evaluate respiratory muscle function in critically ill patients a

63 rage clinicians from having a closer look at respiratory muscle function in critically ill patients.

64 ring and possible implications of monitoring respiratory muscle function in critically ill patients.

65 To test this postulate, we measured limb and respiratory muscle function in nine clinically stable lu

66 uggest a new therapeutic approach to improve respiratory muscle function in patients with respiratory

67 No drugs are available to improve respiratory muscle function in these patients.

68 nsation of dyspnea was related to indices of respiratory muscle function including respiratory rate a

69 Impaired respiratory muscle function is the most severe consequen

70 Pulmonary emphysema impairs lung and respiratory muscle function leading to restricted physic

71 This impairment in respiratory muscle function may contribute to the feelin

72 ormal subjects, however, these decrements in respiratory muscle function may result in symptoms of sh

73 s an intact cough reflex as well as adequate respiratory muscle function to generate elevated intrath

74 Respiratory muscle function was assessed by pulmonary fu

75 ance, reducing ventilatory demand, improving respiratory muscle function, and altering central percep

76 LVRS improves not only lung recoil, but also respiratory muscle function, and reduces dynamic hyperin

77 hyperinflation, air trapping, and improving respiratory muscle function, enables the lung and chest

78 Restoration of diaphragm and other respiratory muscle function, irrespective of the method

79 of the interaction between lung function and respiratory muscle function.

80 ce might result from altered IC and impaired respiratory muscle function.

81 nly at the beginning of routinely monitoring respiratory muscle function.

82 and imbalance within the diaphragm and other respiratory muscles in emphysema has been considered the

83 s of studying neuronal circuits that control respiratory muscles in humans with better spatial and te

84 enic strains of PRV were injected into these respiratory muscles in nine ferrets; the strain injected

85 , it seems highly appropriate to monitor the respiratory muscles in these patients.

86 Chronic neuromuscular diseases affect the respiratory muscles in varying patterns and degrees.

87 Owing to a greater relative VO2 of the respiratory muscles in women, less of a change in work o

88 ientific understanding of ventilator-induced respiratory muscle injury has not reached the stage wher

89 Research on ventilator-induced respiratory muscle injury is in its infancy and portends

90 two-step process: the time from diagnosis to respiratory muscle involvement, followed by the time fro

91 In mammals, a key respiratory muscle is the diaphragm, which is innervated

92 nderstanding of disease states affecting the respiratory muscles is necessary for every physician who

93 lusion, whilst the diaphragm is an important respiratory muscle, it is likely that dystrophin needs t

94 Session 2 (S2): 5 min NB, 5 min of respiratory muscle loading with inspiratory resistance,

95 s and administration of pharmacotherapy, the respiratory muscles may be rendered almost (or completel

96 ung injury, regional forces generated by the respiratory muscles may lead to injurious effects on a r

97 sed state-dependent changes in breathing and respiratory muscle modulation under urethane anesthesia

98 sses the latest developments in the field of respiratory muscle monitoring and possible implications

99 which include reflex responses recorded from respiratory muscle nerves of the thorax and abdomen.

100 of vestibular-evoked responses recorded from respiratory muscle nerves of the upper airway. as well a

101 ion of airflow (e.g. neuromuscular junction, respiratory muscles or respiratory mechanics) and is not

102 ion, albuterol relieves dyspnea and enhances respiratory muscle output in patients with COPD primaril

103 Accessory respiratory muscle oxygenation was assessed using near-i

104 nn disease) is associated with quadriplegia, respiratory muscle paralysis and death in infancy.

105 also led to morbidity and mortality owing to respiratory muscle paralysis and paralysis in the face o

106 a potential nanotherapy for the treatment of respiratory muscle paralysis resulted from cervical SCI.

107 ith muscle fatigue, n = 11) displayed weaker respiratory muscles (Pdi(max) 61 versus 115 cm H(2)O; p

108 y reported that hypogonadism does not affect respiratory muscle performance and exercise capacity in

109 was to determine if this agent also improves respiratory muscle performance.

110 ropositivity is associated with a decline in respiratory muscle performance.

111 hieved by other mechanisms, such as improved respiratory muscle perfusion.

112 tment algorithm was defined to target a peak respiratory muscle pressure between 5 and 10 cm H2O.

113 tion with load-adjustable gain factors, peak respiratory muscle pressure can be estimated from the pe

114 istance to maintain a predefined boundary of respiratory muscle pressure is feasible, simple, and oft

115 We conclude that respiratory muscle pressure production is the predominan

116 .7-1.8) times per day, according to the peak respiratory muscle pressure target range in 91% of cases

117 The peak respiratory muscle pressure, estimated in cm H2O as (pea

118 ning, forced expiratory volumes, and maximum respiratory muscle pressure, leisure-time physical activ

119 The pressure output of the respiratory muscles, quantified as pressure-time product

120 ion, exercise performance, gas exchange, and respiratory muscle recruitment (estimated by esophageal

121 comotion to minimize antagonistic loading of respiratory muscles, reduce work of breathing and minimi

122 ing factors such as recruitment of accessory respiratory muscles, reduction in REM sleep, and loss of

123 Mead-Whittenberger technique was used, with respiratory muscle relaxation provided by brief manual v

124 of slow fibers, but overall strength of the respiratory muscles remains well preserved.

125 in posture can affect the resting length of respiratory muscles, requiring alterations in the activi

126 gm function in determining the metabolic and respiratory muscle response to arm elevation.

127 both excessive patient effort and excessive respiratory muscle rest.

128 enly had paralysis of all four limbs and the respiratory muscles, resulting in death.

129 We assessed respiratory muscle (RM) and cardiopulmonary function dur

130 se capacity, we evaluated static and dynamic respiratory muscle (RM) function, and dyspnea.

131 with severe COPD occurs as a consequence of respiratory muscle (RM) weakness or fatigue, we would ex

132 pulmonary endpoints that allow assessment of respiratory muscle status, especially in nonambulatory s

133 Nearly 35% of children had diminished respiratory muscle strength (aPiMax </= 30 cm H2O) at th

134 27, 95% confidence interval 0.02, 0.52), and respiratory muscle strength (g = 0.51, 95% confidence in

135 Measurements of respiratory muscle strength (PImax, 74 +/- 28 versus 50

136 c lateral sclerosis (ALS) and measurement of respiratory muscle strength (RMS) has been shown to have

137 xercise-related symptoms while IMT increased respiratory muscle strength and endurance.

138 sts (PFTs), arterial blood gases (ABGs), and respiratory muscle strength as estimated by maximum stat

139 Each respiratory muscle strength assessment individually achi

140 predictive power of invasive and noninvasive respiratory muscle strength assessments for survival or

141 effect of oral magnesium supplementation on respiratory muscle strength by using manuvacuometry and

142 ith significant bulbar involvement, tests of respiratory muscle strength do not predict hypercapnia.

143 Respiratory muscle strength during acute upper respirato

144 Reduced respiratory muscle strength has been reported in chronic

145 at it could usefully be included in tests of respiratory muscle strength in ALS and will be helpful i

146 neuromuscular disease develop reductions in respiratory muscle strength in association with URI.

147 ntation helped improve both the SK score and respiratory muscle strength in pediatric patients with C

148 n COPD is multifactorial, changes in DCO and respiratory muscle strength may contribute to its intens

149 From a previously published report respiratory muscle strength measurements were available

150 In the critically ill, respiratory muscle strength usually has been assessed by

151 Respiratory muscle strength was tested by measuring maxi

152 entional spirometry, lung volumes, DLCO, and respiratory muscle strength were measured.

153 y, body plethysmography, diffusion capacity, respiratory muscle strength, 6-min walk test, and increm

154 ovement in lung function, exercise capacity, respiratory muscle strength, and ventilatory efficiency.

155 y of life, physical function, peripheral and respiratory muscle strength, increasing ventilator-free

156 owed a linear decline for direct measures of respiratory muscle strength, whereas VC showed little to

157 tubation failure, with specific attention to respiratory muscle strength.

158 cal evaluations of extremity, hand grip, and respiratory muscle strength; anthropometrics (height, we

159 ate O2 supply and O2 demand in locomotor and respiratory muscles, subjects performed both maximal con

160 nation, complete pulmonary function testing, respiratory muscle testing, cardiopulmonary exercise tes

161 These patterns include the respiratory muscles that, in both birdsong and speech, p

162 e airways interferes with the ability of the respiratory muscles to generate subatmospheric pressure

163 thing depends on coordinated activity of the respiratory muscles to generate subatmospheric pressure.

164 This novel mechanism of treating respiratory muscles to prevent cardiomyopathy in dystrop

165 ss of intercostal muscles, reorganization of respiratory muscles to the ventral side of the ribs, (su

166 rate and variability and with modulation of respiratory muscle tone.

167 ned but respond to aggressive whole-body and respiratory muscle training with an improvement in stren

168 ercise endurance improvements accompanied by respiratory muscle unloading and dyspnea reductions in p

169 monstrate improved locomotor blood flow with respiratory muscle unloading during activity.

170 (S1): 5 min of normal breathing (NB), 5 min respiratory muscle unloading with a ventilator, and 5 mi

171 vels ranging from 7 to 25 cm H2O in terms of respiratory muscle unloading.

172 mbered, nasal O(2), and NIOV+Air, signifying respiratory muscle unloading.

173 alpha causes atrophy and loss of function in respiratory muscle, we asked whether transgenic mice dev

174 Because these neurons innervate respiratory muscles, we hypothesized that respiratory de

175 2) 67 versus 104% predicted; p < 0.0001) and respiratory muscle weakness (PI(max) 77 versus 115% pred

176 Patient 1 had cervical, limb girdle, and respiratory muscle weakness and died of respiratory fail

177 pothesized that HIV+ individuals may develop respiratory muscle weakness and that respiratory muscle

178 ssociated with relevant axial, proximal, and respiratory muscle weakness but without vocal cord palsy

179 In motor neurone disease (MND), respiratory muscle weakness causes substantial morbidity

180 g function tests revealed progressive global respiratory muscle weakness detectable from the time of

181 Respiratory muscle weakness frequently develops during m

182 Although respiratory muscle weakness is a known predictor of poor

183 e and early treatment for cardiomyopathy and respiratory muscle weakness is advocated because early t

184 Respiratory muscle weakness is the primary cause of resp

185 agm atrophy and contractile dysfunction, and respiratory muscle weakness is thought to contribute sig

186 reduced chest wall motion in the presence of respiratory muscle weakness.

187 y representing the earliest manifestation of respiratory muscle weakness.

188 necrosis, leading in many patients to fatal respiratory muscle weakness.

189 ion included predominant distal, proximal or respiratory muscle weakness.

190 , young adult females, with bulbar, neck, or respiratory muscle weakness.

191 d near infrared spectroscopy of an accessory respiratory muscle were obtained during exercise.

192 ow, as well as electromyographic activity in respiratory muscles were recorded in combination with lo

193 heart failure, pulmonary factors, including respiratory muscle work and airflow turbulence, contribu

194 or [2] clinical signs suggestive of intense respiratory muscle work and/or labored breathing) if it

195 blood flow; and reducing the normal level of respiratory muscle work during heavier intensity exercis

196 part, due to the accompanying high levels of respiratory muscle work.