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1 rcumference, resting energy expenditure, and respiratory quotient.
2 mine systemic resting energy expenditure and respiratory quotient.
3 olic rate, 24-h energy expenditure, and 24-h respiratory quotient.
4 = 0.03), which resulted in a lower mean 24-h respiratory quotient (0.845 +/- 0.01 vs. 0.850 +/- 0.01
5 r dynamic controlled atmosphere monitored by respiratory quotient 1.3 (DCA-RQ 1.3) showed lower ethyl
6 e dynamic controlled atmosphere monitored by respiratory quotient 1.5 (DCA-RQ 1.5) increased the acet
7 also led to reductions in REE (-266 kcal/d), respiratory quotient (-15%), heart rate (-14%), blood pr
8                                     The 24-h respiratory quotient (24-h RQ) and 24-h carbohydrate bal
9    The 24-h energy expenditure (24-EE), 24-h respiratory quotient (24-RQ), and the oxidation rates of
10 olic rate, sleeping metabolic rate, and 24-h respiratory quotient (24RQ), an indicator of the ratio o
11                                  The cardiac respiratory quotient also increased significantly by 28%
12                                     Elevated respiratory quotient and carbohydrate utilization during
13 ilizing lipid fuels, as evidenced by a lower respiratory quotient and increased clearance of lipids f
14                                      Greater respiratory quotient and leaf carbohydrate content at el
15      Indirect calorimetry was used to assess respiratory quotient and resting energy expenditure.
16                                              Respiratory quotient and substrate macronutrient oxidati
17 ese results indicate that the postabsorptive respiratory quotients and insulin-mediated glucose stora
18 to the concomitant PPAR-alpha agonism, lower respiratory quotient, and less fat accumulation, despite
19 sorptiometry; sleeping metabolic rate (SMR), respiratory quotient, and substrate oxidation rates were
20                                          The respiratory quotient at peak exercise was lower with the
21  activity, or systemic oxygen consumption or respiratory quotient at rest or during exercise.
22       We assessed REE, body composition, and respiratory quotient before and after weight loss in obe
23 cted no difference in energy expenditure and respiratory quotient between apoE(+/+) and apoE(-/-) mic
24  Neither plasma palmitate concentrations nor respiratory quotient by indirect calorimetry differed be
25 arily oxidized glucose, as demonstrated by a respiratory quotient close to 1.0 (higher than SM, P < 0
26 sed on chlorophyll fluorescence (DCA-CF) and respiratory quotient (DCA-RQ) on the quality and volatil
27 d dynamic controlled atmosphere monitored by respiratory quotient (DCA-RQ) with three fruit maturity
28                                          The respiratory quotient decreased by a mean of 0.12 (CI, 0.
29                                     The 24-h respiratory quotient decreased more rapidly and to a gre
30                   In nonlactating women, the respiratory quotient decreased over time, carbohydrate o
31         The sleeping metabolic rate and 24-h respiratory quotient did not differ significantly betwee
32  During exercise, leg substrate utilization (respiratory quotient) did not differ between groups or l
33 esting VCO(2) and consequently, an increased respiratory quotient during the resting phase, indicatin
34 ion and age did not affect substrate choice (respiratory quotient) during moderate exercise, but the
35 uivalents of carbon dioxide (a surrogate for respiratory quotient), energy expenditure was determined
36 ed with MGF exhibited a substantial shift in respiratory quotient from fatty acid toward carbohydrate
37 ients exercised to a satisfactory end point (respiratory quotient &gt;1.1).
38 a substantial effect; PCO2, base excess, and respiratory quotient have small effects.
39                           BMR, SMR, 24-h EE, respiratory quotient, heart rate, and activity levels we
40 MR), sleeping metabolic rate (SMR), 24-h EE, respiratory quotient, heart rate, and activity were meas
41                 On univariate analysis, BMI, respiratory quotient, high-density lipoprotein, triglyce
42 y metabolism, resting energy expenditure and respiratory quotient in ten chronic hemodialysis patient
43                                          The respiratory quotient in the Surf1(-/-) mice was signific
44                                      Reduced respiratory quotients in Pctp(-/-) mice were indicative
45                   D1/4KO mice showed reduced respiratory quotient, indicating increased use of lipids
46                                     The 24-h respiratory quotient is significantly higher in late pre
47 CP1-deficient mice by 0.1-0.3 degrees C, and respiratory quotient is slightly reduced.
48 of achievement of anaerobic metabolism (peak respiratory quotient &lt;/=1.05).
49                                              Respiratory quotient measurements in both transgenic (MC
50 nts leucine incorporation into fat), and the respiratory quotient obtained from indirect calorimetry
51  is inversely correlated with postabsorptive respiratory quotient of the muscle donors (r = -0.66, P
52                                     The 24-h respiratory quotient on the first day after treatment wa
53 ut altering body weight, energy expenditure, respiratory quotient, or adiposity.
54 , free T(3) was a negative predictor of 24-h respiratory quotient (P < 0.05) and a positive predictor
55 (P-time x treatment = 0.03) and postprandial respiratory quotient (P-time x treatment = 0.01) compare
56 riglycerides, free fatty acids, and insulin; respiratory quotient; percentage of body fat; liver volu
57 free mass, visceral fat, energy expenditure, respiratory quotient, physical fitness, and energy intak
58  to carbohydrate oxidation rate (an elevated respiratory quotient) predicts the development of obesit
59 t plasma ketones (r = 0.755, P = 0.006), and respiratory quotient (r = -0.797, P < 0.001) were relate
60 d not improve after adjusting for changes in respiratory quotient (r2 = 0.28).
61         The DeltaP < 0 reflected an apparent respiratory quotient (RQ) < 1.
62                                              Respiratory quotient (RQ) and resting metabolic rate (RM
63                        In obese adolescents, respiratory quotient (RQ) and substrate oxidation also d
64 as traditionally involved measurement of the respiratory quotient (RQ) by indirect calorimetry during
65  effects of sleep curtailment on 24-h EE and respiratory quotient (RQ) by using whole-room indirect c
66 influencing resting metabolic rate (RMR) and respiratory quotient (RQ) represent candidate genes for
67         Estimates based on the nitrogen-free respiratory quotient (RQ) revealed fat oxidation to be s
68 present work was to evaluate the appropriate respiratory quotient (RQ) value to achieve a safe lowest
69                                              Respiratory quotient (RQ) was calculated as V(CO2)/V(O2)
70                                  REE and the respiratory quotient (RQ) were measured by indirect calo
71 POR1 and ADIPOR2) on resting metabolic rate, respiratory quotient (RQ), and adiposity-related phenoty
72  diet (KD) is associated with changes in EE, respiratory quotient (RQ), and body composition.
73 xpenditure corrected for body mass (AEE/BM), respiratory quotient (RQ), and carbohydrate oxidation wi
74 variables [resting energy expenditure (REE), respiratory quotient (RQ), glucose/carbohydrate oxidatio
75  dramatically influenced by BMI, the resting respiratory quotient (RQ), T2DM, and sex.
76 y composition, resting metabolic rate (RMR), respiratory quotient (RQ), temperature, fasting serum gl
77  fat oxidation as reflected in reductions in respiratory quotient (RQ).
78 expenditure (EE) and substrate oxidation-ie, respiratory quotient (RQ).
79  on NPY stimulated eating and alterations in respiratory quotient (RQ).
80 ctedly lowers the respiratory exchange rate (respiratory quotient [RQ]) and decreases food intake.
81  Metabolic flexibility to glucose (change in respiratory quotient [RQ]) was mainly related to insulin
82               Significant changes in resting respiratory quotients (RQs) in normal (Baseline: 0.78+/-
83      Measures of energy expenditure (EE) and respiratory quotients (RQs) in the absence of food were
84 ion as estimated by fasting and postprandial respiratory quotients (RQs), or rate of lipolysis.
85 d carbon dioxide expired in order to compute respiratory quotients (RQs).
86                                              Respiratory quotient significantly increased 0.12 with o
87 ndividual variability in the response of the respiratory quotient to a high-fat diet with increased e
88                                    A reduced respiratory quotient, together with elevated beta-hydrox
89 ssed by combustion of 100% ethanol; the mean respiratory quotient was 0.667 +/- 0.001 (SEM).
90                                              Respiratory quotient was a process measure.
91                        A 14% decrease in the respiratory quotient was also observed.
92                        In the fed state, the respiratory quotient was lower (P = 0.01) with the high
93 oxidation rates were reduced by 50%, and the respiratory quotient was markedly increased compared wit
94                                              Respiratory quotient was not significantly affected by h
95                           Using the measured respiratory quotient, we found that the mean (+/-SD) EE(
96       Neither resting energy expenditure nor respiratory quotient were different according to UCP2 ex
97 sumption, carbon dioxide production, and the respiratory quotient were measured by indirect calorimet
98 , the thermic effect of food (TEF), and 24-h respiratory quotient were measured by using a respirator
99 Insulin concentration and the postabsorptive respiratory quotient were positively correlated with the
100               Resting energy expenditure and respiratory quotient were similar between patient groups
101 ges in substrate utilization as reflected by respiratory quotient, which is increased with chronic Mc

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