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1 duction in dynamic hyperinflation at a lower respiratory rate).
2 ef changes in blood pressure, heart rate, or respiratory rate.
3 HB renal and total clearance, also improving respiratory rate.
4 no effect on blood pressure, heart rate, or respiratory rate.
5 rterial blood pressure, body temperature and respiratory rate.
6 te the trigeminal nerve and evoke changes in respiratory rate.
7 lter basal forebrain ACh release, sleep, and respiratory rate.
8 Dialysis administration of NLA did not alter respiratory rate.
9 id not differ in end-tidal carbon dioxide or respiratory rate.
10 ack of any increase in minute ventilation or respiratory rate.
11 adaptation of stimulation to alterations in respiratory rate.
12 gnals, skin temperature, skin hydration, and respiratory rate.
13 inergic synaptosomes from striatum had lower respiratory rates.
14 lved in the control of maximal mitochondrial respiratory rates.
15 ptoms and panic attacks, as well as elevated respiratory rates.
16 s remained viable and maintained significant respiratory rates.
17 ervation were evaluated by measuring ATP and respiratory rates.
18 e and age-matched WT littermates had similar respiratory rates.
19 eases in arousal, temperature, and heart and respiratory rates.
20 ilated with 12 mL/kg tidal volume, 28% FIO2, respiratory rate = 12 breaths/min) were hemorrhaged to <
22 -2.2 L/min vs. 10.1+/-2.9 L/min, p<0.01) and respiratory rate (16+/-5 bpm vs. 19+/-6 bpm, p<0.01) tha
23 s. 16%, p = .003) and had a higher intrinsic respiratory rate (22 breaths/min vs. 18, p = .03), but t
24 ion period, averages were pH, 7.29 +/- 0.05; respiratory rate, 22.4 +/- 3.7 breaths/min; PetCO2, 35.3
25 ventilation, 12.7+/-1.4 to 6.2+/-0.8 L/min; respiratory rate, 25.4+/-1.3 to 18.4+/-1.8 breaths/min;
27 d) initial values for pH were 7.08 +/- 0.18; respiratory rate, 35.1 +/- 9.1 breaths/min; PetCO2, 18.6
28 was independently associated with increased respiratory rate (4%; 95% confidence interval [CI], 1.01
29 eak airway pressure was similar at the three respiratory rates (66.8 +/- 8.7 vs. 66.4 +/- 9.5 vs. 67.
30 in measuring temperature, 85% for measuring respiratory rates, 98% for diagnosis, 98% for classifica
31 hours preceding ICU admission, with a higher respiratory rate, a more frequent acute kidney injury, a
32 6 +/- 41 ml/kg/min, p < 0.01), a decrease in respiratory rate, a prolongation of inspiratory time, an
33 athing 15 L/min of oxygen, plus either [1] a respiratory rate above 30/min or [2] clinical signs sugg
34 respiration was a dose-dependent decrease in respiratory rate, accompanied by an increase in tidal vo
35 matically increased and decreased changes in respiratory rate according to a set protocol: +2, -4, +6
36 lly ventilated (tidal volume V(T) = 8 mL/kg, respiratory rate adjusted to normocapnia) at low (n = 2,
38 ant peripheral oximetry, blood pressure, and respiratory rate alerts from artifacts in an online moni
39 hted average heart rate, blood pressure, and respiratory rate, along with changes-over-time for each.
41 ts have pulmonary hypertension and increased respiratory rates, although the pathophysiological basis
42 und a linear relationship between changes in respiratory rate and big up tri, openPetCO2 as well as b
45 afil, subjects had a significantly decreased respiratory rate and decreased minute ventilation at pea
47 ither abnormal lung compliance nor increased respiratory rate and displayed no markers of lung injury
48 i, openPetCO2) as well as between changes in respiratory rate and equilibration time (teq) for mechan
50 ication included higher body temperature and respiratory rate and higher percentage of immature neutr
51 al and hippocampal activation, increased the respiratory rate and hypoglossal nerve activity, induced
52 ome measures were evaluated as a function of respiratory rate and included tidal volume, maximal alve
54 guinea pigs while having no effect on basal respiratory rate and little or no effect on reflexes att
57 d respiratory rate (r = -.79, p =.0001), and respiratory rate and pH (r = -.80, p =.0001) were statis
58 ondrial Ca(2+) handling, membrane potential, respiratory rate and production of reactive oxygen speci
59 uring CPR was obtained by recording observed respiratory rate and relative tidal volume during treatm
60 eptor-responsive PPTn neurons also increased respiratory rate and respiratory-related genioglossus ac
61 lationship has been found between changes in respiratory rate and resulting changes in end-tidal cO2
67 respiration (brief episodes of increases in respiratory rate and tidal phrenic nerve activity) while
68 pproximate entropy (ApEn) was calculated for respiratory rate and tidal volume series of the terminal
69 ous ventilation, each breath's instantaneous respiratory rate and tidal volume were recorded for late
71 reased in parabolic fashion as a function of respiratory rate and was maximal at rates of 4.3-6.8 bre
74 ted from the brains of R6/2 mice had similar respiratory rates and Ca(2+) uptake capacity compared wi
76 in lung lavage fluids of infected mice with respiratory rates and measures of outflow obstruction an
77 hat combines the low tidal volume with lower respiratory rates and minimally invasive CO2 removal.
79 ain Assessment Card) and 60-second heart and respiratory rates and sustained change in quality of lif
80 trienes were correlated with both increasing respiratory rates and the degree of prolongation of expi
81 n saturation, end-tidal CO2, heart rate, and respiratory rate) and comfort (electroencephalogram [EEG
82 with monitoring of jugular venous pressure, respiratory rate, and arterial oxygen saturation and tre
84 an arterial pressure, rate-pressure product, respiratory rate, and catecholamine levels were all sign
85 splayed markedly reduced locomotor activity, respiratory rate, and energy expenditure, which were not
87 anxiolysis: reduced risk-avoidance, reduced respiratory rate, and increased positive valence, respec
88 ncluding mean arterial pressure, heart rate, respiratory rate, and oxygen saturation) were collected
91 high inspiratory/expiratory ratios and high respiratory rate apparently due to "auto-positive end-ex
92 phase during total liquid ventilation at low respiratory rates, apparently due to increased tidal vol
94 creased to 0.87 (95% CI, 0.71-0.98), whereas respiratory rate area under the curve started at 0.85 (9
95 n each case, indicators of pulmonary damage (respiratory rates, arterial oxygen partial pressures, an
96 ch intestinal ROI was sent for mitochondrial respiratory rate assessment and for metabolites quantifi
103 sed using clinical parameters such as pulse, respiratory rate, blood pressure (BP), and biochemical p
104 modified SOFA (mSOFA), the Confusion, Urea, Respiratory Rate, Blood Pressure and Age (CURB-65) score
105 nuous single-channel monitoring (heart rate, respiratory rate, blood pressure, and peripheral oxygen
106 ninvasive monitoring parameters (heart rate, respiratory rate, blood pressure, and peripheral oxygen
107 and followed oxygen saturation, spirometry, respiratory rate, blood pressure, heart rate, and expire
108 rome (SIRS), includes changes in heart rate, respiratory rate, body temperature, and circulating whit
109 glucose stimulation of insulin secretion and respiratory rate but demonstrated two different patterns
110 /kg SCH50911 plus l-lactate further improved respiratory rate compared with the same dose of either a
113 ecord oxygen requirement, oxygen saturation, respiratory rate, consciousness level, and other evidenc
115 morphology, as evidenced by measurements of respiratory rates, cytochrome contents, and also clearly
117 +/- 3.2 to 102 +/- 3.2 beats/min (p < .001), respiratory rate decreased from 39 +/- 3 to 25 +/- 1 bre
119 RNA or protein, indicating that the enhanced respiratory rate did not require a general increase in m
121 02-0.66 +/- 0.03 ml; p < 0.05) and increased respiratory rate (f;91 +/- 3.7-132 +/- 5.7 breaths/min;
126 tions to control heart rate, blood pressure, respiratory rate, gastrointestinal motility, hormone rel
127 e less than 20 mEq/L, lactate concentration, respiratory rate greater than or equal to 24 breaths/min
128 in vital signs (heart rate > 100 beats/min, respiratory rate > 24 breaths/min, and oral body tempera
129 essment criteria (Glasgow Coma Scale </= 14, respiratory rate >/= 22 breaths/min, or systolic blood p
130 </= 7.35, and at least one of the following: respiratory rate >/= 25/min, PaO2 </= 50 mm Hg, and oxyg
131 r 2 points for > 6 L/min; 1 point each for a respiratory rate >/= 30 and immune suppression) accurate
132 30 with arterial PCO2 > 20% of baseline, and respiratory rate >/= 30 breaths/min or use of accessory
133 ial hypertension (OR, 1.5; 95% CI, 1.1-2.1), respiratory rate >/=30 breaths per minute (OR, 1.6; 95%
134 Age >/= 80 years, heart rate >90/minute, respiratory rate >20/minute, white cell count <4 x 10(9)
135 eta-blockers during hospitalization included respiratory rate >24 breaths/min (30.8% vs. 16.9%; p = 0
136 her hospital (ORadj 2.08, 95% CI 1.33-3.25), respiratory rate >33 breaths/min (ORadj 2.39, 95% CI 1.5
137 0%-92%; specificity, 47%-54%) and tachypnea (respiratory rate >40 breaths/min; LR, 1.5 [95% CI, 1.3-1
138 hese effects were minimized in this model at respiratory rates >/=5.7 and </=6.8 breaths/min and insp
139 ed as >/=2 of 1) heart rate>90 beats/min, 2) respiratory rate>20 breaths/min, 3) body temperature>38
140 ecome increasingly irregular, but increasing respiratory rate has no effect on respiratory rate patte
145 Glasgow coma score, systolic blood pressure, respiratory rate, heart rate, and type of injury (blunt
147 dified early warning score was assessed from respiratory rate, heart rate, systolic blood pressure, b
148 eft ventricular ejection fraction, increased respiratory rate, high GRACE score, or presence of diabe
149 nal bleeding, and also decreased with higher respiratory rate, higher heart rate, longer time from tr
150 ventilation circuit dead space, increases in respiratory rate, higher positive end-expiratory pressur
151 expiratory time will depend on the baseline respiratory rate (i.e., less reduction in dynamic hyperi
152 rus (RSV) infection in the neonate can alter respiratory rates, i.e., lead to episodes of apnea.
154 -nitroquinoxaline-2,3-dione (CNQX) decreased respiratory rate in a dose dependent manner by lengtheni
160 cin-sensitive oxygen consumption and maximal respiratory rates in cells derived from wild type, but n
161 Among the 488 treated neonates, the mean respiratory rates in the first 24 h were 51 (SD 8) breat
162 attack is triggered, minute ventilation and respiratory rate increase regardless of whether the subj
164 idal volume did not vary between groups, but respiratory rate increased progressively from the contro
165 ptom ratings were positively correlated with respiratory rate increases, as well as with levels of ti
166 CH50911 completely prevented the decrease in respiratory rate, indicating agonism at GABA(B) receptor
167 ) nerve fibers, causing an alteration of the respiratory rate indicative of trigeminal activation.
168 vity, the faster enhancement of the cellular respiratory rate is due to intrinsic factors within the
169 ed, demonstrating that the inhibition of the respiratory rate is not due to loss of cytochrome c.
171 ial blood pressure, an increase in pulse and respiratory rate, lactic acidosis, and renal failure.
173 age, lower systolic blood pressure, abnormal respiratory rate, lower Glasgow Coma Scale score, lower
175 r=90 mm Hg, Glasgow Coma Scale score <or=12, respiratory rate <10 or >29 per minute, advanced airway
176 nclusion in the AMPT score included GCS <14, respiratory rate <10 or >29, flail chest, hemo/pneumotho
177 od pressure (> or =90 mm Hg), and 3 days for respiratory rate (< or =24 breaths/min), oxygen saturati
178 Changes in PaO2/Fio2 ratio, tidal volume, respiratory rate, mean airway pressure, plateau pressure
180 n between PetCO2 equilibria after changes in respiratory rate might not be dependent on moderate lung
182 Secondary outcomes included tidal volume, respiratory rate, minute volume, dynamic lung compliance
184 espiratory acidosis, encephalopathy, and the respiratory rate more quickly than air/O2 but does not p
185 .8 g/dL (odds ratio, 3.6; 95% CI, 1.1-11.9), respiratory rate more than or equal to 32 cycles/min (od
186 dal volume and its coefficient of variation, respiratory rate, neural timing components, and calculat
187 age of >65 yrs, b) altered mental status, c) respiratory rate of >30 breaths/min, d) low oxygen satur
188 rmalisation of patients' respiratory status (respiratory rate of </=20 breaths per min for adults or
189 iggers (heart rate of <40 or >140 beats/min, respiratory rate of <8 or >36 breaths/min, systolic bloo
190 ide clinical score termed quickSOFA (qSOFA): respiratory rate of 22/min or greater, altered mentation
192 vious 24 hours with persisting dyspnea and a respiratory rate of 24/min or greater were eligible prov
193 a peak inspiratory pressure of 50 cm H2O, a respiratory rate of 4 breaths x min(-1), and an inspirat
194 revealed a temperature of 38.1 degrees C, a respiratory rate of 48 breaths per minute, a heart rate
195 fill volume, 17.5-20 mL.kg(-1) tidal volume, respiratory rate of 5 breaths/min, inspiratory/expirator
196 an inspiratory/expiratory ratio of 1:2.5 and respiratory rate of 6.8 breaths/min appeared to provide
197 peak inspiratory pressure of 50 cm H2O, at a respiratory rate of 8 breaths.min, with an inspiratory t
198 ists for studies that reported heart rate or respiratory rate of healthy children between birth and 1
199 children aged from 1 month to 5 years with a respiratory rate of more than 50 breaths per min in chil
202 .6, 38.6 +/- 4.5, and 23.1 +/- 5.8 mL/sec at respiratory rates of 18, 12, and 6 breaths/min, respecti
205 aths 1-5 [mean+/-SD], 49+/-41% baseline) and respiratory rate often sufficient to sustain minute vent
206 therefore also be used for the titration of respiratory rate on the PetCO2 for a wider range of path
209 44, 95% CI .17-1.07, p = .076), and baseline respiratory rate (OR 2.03 per sd, 95% CI 1.38-3.08, p <
210 s in the mean number of days of fever, rapid respiratory rate, or hypoxia, whether these endpoints we
212 al volume, positive end-expiratory pressure, respiratory rate, oxygen administration, and head-of-bed
214 condary end points including height, weight, respiratory rate, oxygen saturation, cough, or respirato
216 001), lower PaCO2 (p < .05), and a lower set respiratory rate (p < .0001) as compared with the SIMV-t
218 improved oxygenation (P < 0.001) and lowered respiratory rate (P < 0.01), DeltaPes (P < 0.01), and pr
219 Low body mass index (P = .002) and elevated respiratory rate (P = .01) at tuberculosis diagnosis ind
220 r operating characteristic curve [AUC] 0.7), respiratory rate (p<.001, AUC 0.71), and oxygen saturati
223 sive vital sign monitoring data (heart rate, respiratory rate, peripheral oximetry) recorded on all a
225 istress Syndrome Network protocol plus lower respiratory rate plus minimally invasive Co2 removal.
227 riables: pH, Glasgow Coma Scale, spontaneous respiratory rate, positive end-expiratory pressure, and
228 spiratory rates, whereas increasingly higher respiratory rates progressively and significantly impair
229 PetCO2 and pH (r =.88, p =.0001), Petco2 and respiratory rate (r = -.79, p =.0001), and respiratory r
231 was in direct proportion to the increase in respiratory rate reflecting the level of conditioned fea
233 vate and lactate, with similar state 3 and 4 respiratory rates, respiratory control (state 3/state 4)
234 ands exhibited larger minute ventilation and respiratory rate responses relative to comparisons.
238 was a statistically significant decrease in respiratory rate (RR) at normocapnia, an elevated RR dur
239 of positive end-expiratory pressure (PEEP), respiratory rate (RR), and plateau pressure minus PEEP (
240 athing parameters showed significantly lower respiratory rates (RR) (36.6 +/- 17.9 versus 52.8 +/- 23
241 ublished reference ranges for heart rate and respiratory rate show striking disagreement, with limits
242 gic states, but also that "normal" or higher respiratory rates significantly compromise hemodynamics,
243 of spontaneous breathing was assessed by the respiratory rate standardized to age, the presence of re
244 n model are PaO2/FiO2 ratio, platelet count, respiratory rate, systolic blood pressure, and days of h
246 SV-infected mice showed significantly higher respiratory rates than did controls (409.2 vs. 305.2 bre
247 ntilation for patients with COPD such as low respiratory rates that maximize expiratory time and care
248 nd slow (60/min) representing the underlying respiratory rate, the other fast (> 300/min) and limited
249 tion coinciding with the increase in overall respiratory rate; this acquired capability was accompani
250 ysis was performed on each of the factors of respiratory rate, tidal volume, and inspiratory/expirato
252 ements of respiratory function were made for respiratory rate, tidal volume, lung volume, airway cond
253 g reported as work per liter of ventilation, respiratory rate, tidal volume, negative change in esoph
254 t rate-systolic blood pressure products, and respiratory rate/tidal volume ratios were obtained for p
255 iratory chain activity by calcium allows the respiratory rate to change severalfold with only small o
256 to 91 (range, 62-114; P <.001) and the mean respiratory rate to increase an average of 12 breaths pe
257 ncentration), others influence VRG to adjust respiratory rate to optimize partial pressure CO(2), and
259 es , 30 degrees , 40 degrees ) elevation and respiratory rate (to achieve a DeltaETco2 = +/-3-4 mm Hg
260 correlation between H(2)O(2) production and respiratory rates under different conditions of NO(radic
261 ment for age, degree of hypercarbia, maximal respiratory rate, use of an arterial catheter, and Pedia
262 0% of eupneic VT) with and without increased respiratory rate, using controlled and assist control me
264 arly, sedation interruption led to increased respiratory rate variability for low multiple organ dysf
265 sedation reduces heart rate variability and respiratory rate variability in critically ill patients
266 nuously monitored heart rate variability and respiratory rate variability in critically ill patients.
267 er restoration of heart rate variability and respiratory rate variability in patients with low organ
268 suggest that both heart rate variability and respiratory rate variability increased during sedation i
270 and quantified by an apnoea-hypopnoea index, respiratory rate variability index and the coefficient o
272 se to injury were explored by heart rate and respiratory rate variability measured with non-invasive
273 but in contrast, a further deterioration in respiratory rate variability occurred in the high multip
279 /- 2.6 vs. 21.3 +/- 2.9 cm H2O, p <.01) when respiratory rate was decreased from 12 to 6 breaths/min
280 /- 2.8 vs. 23.3 +/- 2.6 cm H2O, p <.01) when respiratory rate was decreased from 18 to 12 breaths/min
281 1) during maximum exercise increased whereas respiratory rate was lower (28 +/- 6 versus 32 +/- 7 bre
282 ar, but refusal rate was lower, reduction in respiratory rate was more rapid, and there were fewer co
283 During the lower respiratory rate condition, respiratory rate was reduced from 30.5 +/- 3.8 to 14.2 +
284 ation, minute ventilation, tidal volume, and respiratory rate were calculated by indirect calorimetry
285 ically significant and the correlations with respiratory rate were inversely related to pH and PetCO2
290 tochondria from Gq versus wild-type mice and respiratory rates were lower; these changes in mitochond
291 n contrast to WT hearts, complex I-dependent respiratory rates were protected against ischemic damage
295 r stimulation (i.e., increase in BP, HR, and respiratory rate) were located in a midline region just
296 on and acid-base status with lower-frequency respiratory rates, whereas increasingly higher respirato
297 ulla-spinal cord preparation revealed a high respiratory rate with short inspiratory duration and fre
298 ding pressure, small tidal volumes and rapid respiratory rates with the intent to recruit atelectatic
300 (VEI = 3800/[peak airway pressure - PEEP] x respiratory rate x PacO2), or static compliance as compa
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