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1 erial infection than febrile infants without respiratory syncytial virus infection.
2 e is no effective vaccine to protect against respiratory syncytial virus infection.
3 eported cases of myocarditis in infants with respiratory syncytial virus infection.
4 ocytes into the bronchoalveolar lumen during respiratory syncytial virus infection.
5 There were 794 viral culture-confirmed respiratory syncytial virus infections.
6 ategies to prevent group B streptococcus and respiratory syncytial virus infections, among others.
7 D1 were genotyped in 465 infants with severe respiratory syncytial virus infection and 930 control su
8 had lower rates of detectable rhinovirus or respiratory syncytial virus infection and higher rates o
9 eral months after resolution of influenza or respiratory syncytial virus infection and is associated
10 ection, and infection history on the rate of respiratory syncytial virus infection and the effect of
12 proaches to the development of a vaccine for respiratory syncytial virus infection are promising.
13 ndependently noted that febrile infants with respiratory syncytial virus infections are at significan
14 2.4; 95% confidence interval [CI], 1.4-3.9), respiratory syncytial virus infection (aRR, 1.9; 95% CI,
15 ate immune regulatory networks active during respiratory syncytial virus infection, asthma, and bronc
16 nalyzed independent cohorts of children with respiratory syncytial virus infection, asthma, and bronc
17 prevalent childhood lung diseases, including respiratory syncytial virus infection, asthma, and bronc
18 e and mucus production in the airways during respiratory syncytial virus infection but failed to modu
19 on for HMPV infection was less than that for respiratory syncytial virus infection but similar to tha
21 children in Kilifi, Kenya, was monitored for respiratory syncytial virus infections from January 31,
22 2 in regulating the immune response to acute respiratory syncytial virus infection, IL-12p40 gene-tar
24 l dysfunction and dysrhythmias may accompany respiratory syncytial virus infection in some infants an
25 with immunomodulators) for the treatment of respiratory syncytial virus infections in HM patients an
27 n of IFNG (but not CCL5) mRNA in response to respiratory syncytial virus infection of monocytes was a
28 erimental asthma in which AHR was induced by respiratory syncytial virus infection or ozone exposure
29 nce of preexisting lung disease, symptomatic respiratory syncytial virus infection, or presence of cy
31 gger CF bacterial infections, such as viral (respiratory syncytial virus) infections, which up-regula
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