ライフサイエンスコーパス: response element

1 the transcriptional control of an NF-kappaB response element.

2 ream of the transcription start as a key p53 response element.

3 lude bound small molecules and their hypoxia response element.

4 the core promoter and a region within p53's response element.

5 umber of genes by binding to the antioxidant response element.

6 nscription factors (TF) with their chromatin response elements.

7 ial proximity to the intron 7 glucocorticoid response elements.

8 was no decrease in HIF-1 binding to hypoxia response elements.

9 tes with a distal PAS domain to bind hypoxia response elements.

10 CpG sites were located within glucocorticoid response elements.

11 uadruplex based structures as ultrasensitive response elements.

12 h as estrogen response elements and androgen response elements.

13 gene transcription via interferon stimulated response elements.

14 d binding with SMAD family member(s) at SMAD-response elements.

15 ize these methyl groups within their cognate response elements.

16 ts, suggesting that they function as EWS/FLI-response elements.

17 cer (IGF-1 5'-upstream region growth hormone response element; 5URGHRE) as a GHRE specificity control

18 ound to three different DNA sequences: a p21 response element, a puma response element and a nonspeci

19 contain a cis-acting sequence called the A2 response element (A2RE), hnRNP A2 proteins that bind spe

20 es under the control of optimized amino acid response elements (AAREs) had low basal expression and h

21 e 1 expression, and increased glucocorticoid response element activation.

22 ement and nuclear factor of activated T-cell response element activities and ERK phosphorylation, ind

23 eporter plasmid showed reduced retinoic acid response element activity, supporting the hypothesis of

24 d recruitment of PPARalpha to this PPARalpha response element after bezafibrate treatment of human he

25 Dysregulation of cellular and humoral immune response elements, along with organ-defined molecular ab

26 NA sequences: a p21 response element, a puma response element and a nonspecific DNA sequence.

27 nduces activation of the intracellular serum-response element and NFkappaB signaling pathways and the

28 ry effect on kisspeptin stimulation of serum response element and nuclear factor of activated T-cell

29 ear export dynamics regulated by HIV-1's Rev response element and the viral Rev protein; transient ag

30 -bp 5' proximal MET promoter contains a PAX3 response element and two ETS1 consensus motifs.

31 lated genes of canary HVC, 20% lack estrogen response elements and 4 to 8% lack androgen response ele

32 imal promoter region containing the TGF-beta response elements and a combination of transient transfe

33 iption factor binding sites such as estrogen response elements and androgen response elements.

34 rr2 attenuates the binding of AR to androgen response elements and consequently decreases transcripti

35 a targets, many of them bearing both hypoxia response elements and estrogen response elements, are re

36 in dissociation of TACC1 from retinoic acid response elements and leads to transcriptional activatio

37 ediator binds to nuclear receptors at target response elements and recruits chromatin-modifying enzym

38 drugs increased AhR recruitment onto the AhR-response elements and significantly induced breast cance

39 s [CRE], NF-kappaB binding sites [kB], serum response element, and ETS/ELK-1 binding site) and multip

40 IF-1alpha gene itself also bears an estrogen response element, and its expression is directly regulat

41 The MtLAX2 promoter contains several auxin response elements, and treatment with indole-acetic acid

42 Finally, signal response elements are differentially localized to respon

43 Because estrogen response elements are known to regulate genes over long

44 t the pharmaceutically important antioxidant response element (ARE) activation, and the resultant str

45 We identify an androgen response element (ARE) in the Hand2 locus and present bi

46 The androgen response element (ARE) is a palindromic, dihexameric mot

47 increased expression of the NRF2-antioxidant response element (ARE) pathway.

48 ty and stimulate higher antioxidative stress response element (ARE) reporter activity.

49 2 transcription factor-dependent antioxidant response element (ARE) signaling is activated by selecti

50 one oxidoreductase 1 (NQO1) gene antioxidant response element (ARE).

51 both hypoxia response elements and estrogen response elements, are regulated by ERalpha in normoxia

52 ns by recognizing and binding to antioxidant response elements (AREs) in their gene promoter regions.

53 or the same canonical high-affinity androgen response elements (AREs) that are preferentially engaged

54 the expression of genes bearing antioxidant-response elements (AREs).

55 o the induction of genes through antioxidant response elements (AREs).

56 tor Ets-2 that binds to the antigen receptor response element (ARRE)-2 of the proximal IL-2 promoter.

57 TOE1 with the viral specific transactivator response element as part of the inhibitory mechanism.

58 uitment of ERalpha and SRC-1 to the estrogen response element at the apoA-V promoter, implying the pa

59 table TF-cofactors chromatin interactions at response elements at the single-molecule level.

60 creen potential toxicants is the antioxidant response element beta lactamase reporter gene assay (ARE

61 stitution analyses reveal that the Pparalpha response element between nucleotides -557 and -543 is re

62 expression after learning requires the cAMP-response element binding (CREB) interaction domain of th

63 expression and secretion by inhibiting cAMP response element binding (CREB) protein and AKT phosphor

64 ch decreased two transcription factors, cAMP response element binding and Nurr1, controlling synapsin

65 The transcription factors CREB (cAMP response element binding factor), SRF (serum response fa

66 ng protein of cyclic adenosine monophosphate response element binding protein (CBP), p300, and Cbp/p3

67 pletion reduces the activity of carbohydrate response element binding protein (ChREBP), a cellular he

68 ction via increasing phosphorylation of CAMP response element binding protein (CREB) and PSD95 after

69 ound that activated PERK phosphorylates CAMP response element binding protein (CREB) and PSD95 direct

70 ns transfected with a dominant-negative cAMP response element binding protein (CREB) and was eliminat

71 ates with the transcriptional regulator cAMP response element binding protein (CREB) in both mouse an

72 ellular cAMP and the phosphorylation of cAMP response element binding protein (CREB) in INS-1 cells.

73 cAMP response element binding protein (CREB) is a key regulat

74 cAMP-response element binding protein (CREB) is a nuclear tra

75 The cyclic AMP response element binding protein (CREB) is a primary hub

76 rthermore, TDCA significantly increased cAMP response element binding protein (CREB) phosphorylation

77 tion all impair efficient TSH-dependent cAMP response element binding protein (CREB) phosphorylation.

78 GMP) leading to increased levels of the cAMP response element binding protein (CREB), a transcription

79 novel intracellular signaling molecule, cAMP-response element binding protein (CREB), which serves as

80 homimetic mutant S386/396E bound to the cAMP response element binding protein (CREB)-binding protein

81 The starvation-inducible coactivator cAMP response element binding protein (CREB)-cAMP-regulated t

82 lation of the cyclic adenosine monophosphate response element binding protein (CREB)-regulated transc

83 t was mediated by inhibition of carbohydrate response element binding protein activity.

84 horylation of cyclic adenosine monophosphate response element binding protein and activating transcri

85 ling, leading to the phosphorylation of cAMP response element binding protein and, consequently, SMC

86 ption levels of specific members of the cAMP Response Element Binding protein gene family.

87 an enduring reduction in phosphorylated cAMP-response element binding protein levels in the NAcSh tha

88 with the magnitudes of hepatic carbohydrate response element binding protein signaling activation.

89 iding the first evidence that enhancing cAMP response element binding protein signaling can alleviate

90 veal a previously unappreciated role of cAMP response element binding protein signaling in RTT pathog

91 shows that signaling by hepatic carbohydrate response element binding protein that improves glucose t

92 ine suppressed binding of phospho-CREB (cAMP response element binding protein) to Bdnf promoters in V

93 ivity of the transcription factor CREB (cAMP-response element binding protein).

94 ween proinflammatory cytokines, carbohydrate response element binding protein-mediated transcription,

95 nase-3, ribosomal S6 kinase, c-Jun, and cAMP response element binding protein.

96 in decreased cAMP signaling and reduced cAMP-response-element binding protein (CREB) activation, a cr

97 0 production via the phosphorylation of cAMP response element-binding (CREB) protein on the IL-10 pro

98 ng and colleagues show that the carbohydrate response element-binding protein (ChREBP) coordinates an

99 1 (SP1)-regulated expression of carbohydrate response element-binding protein (ChREBP) in cells conta

100 The carbohydrate-response element-binding protein (ChREBP) is a glucose-r

101 ted receptor alpha (PPARalpha), carbohydrate response element-binding protein (ChREBP), and cAMP resp

102 ge-derived IL-10 resulted in epithelial cAMP response element-binding protein (CREB) activation and s

103 tivated PKA subsequently phosphorylated cAMP response element-binding protein (CREB) at Ser-133 to ac

104 gulating the phosphorylation of cAMP/calcium response element-binding protein (CREB) at serine 133 an

105 For example, deficits in cAMP response element-binding protein (CREB) binding protein

106 r (ATF)-adenosine 3',5'-monophosphate (cAMP) response element-binding protein (CREB) family transcrip

107 ng activity of the transcription factor cAMP response element-binding protein (CREB) in young adult r

108 transition under basal/nicotine-induced/cAMP-response element-binding protein (CREB) overexpressed co

109 Cyclic AMP-response element-binding protein (CREB) plays key transc

110 Cyclic-AMP response element-binding protein (CREB) signaling has a

111 t via the PKA-mediated induction of the cAMP response element-binding protein (CREB) signaling pathwa

112 overed that HDAC2 is a direct target of cAMP response element-binding protein (CREB) that is activate

113 t protein kinase II (CaMKII) and Ca(2+)/cAMP response element-binding protein (CREB) transcription fa

114 nse to mRNA for a transcription factor, cAMP response element-binding protein (CREB), and by an inhib

115 However, phosphorylation of cAMP-response element-binding protein (CREB), the cAMP-regula

116 the transcriptional coactivators cyclic-AMP response element-binding protein (CREB)-binding protein

117 Here, we show that high glucose induced cAMP response element-binding protein (CREB)-binding protein

118 tream cascade adenylyl cyclase-cAMP-PKA-cAMP response element-binding protein (CREB).

119 The transcription factor cyclic AMP-response element-binding protein 1 (CREB1) controls appr

120 reasing MCU's transcriptional regulator cAMP response element-binding protein 1.

121 carboxylase Acc1 phosphorylation, and sterol response element-binding protein 1c-dependent gene expre

122 bits proteolytic processing of CREB3L1 (cAMP response element-binding protein 3-like 1), a membrane-b

123 ivation of the transcription factor cellular response element-binding protein and an increase in brai

124 signaling to cyclic adenosine monophosphate response element-binding protein and the induction of CD

125 adenylate cyclase --> cAMP --> PKA --> cAMP response element-binding protein pathway mediating cell

126 enetic program, including Ras and cyclic AMP response element-binding protein pathways and other Gata

127 pEC50 of ACEA-induced Galphas-dependent cAMP response element-binding protein phosphorylation.

128 Consequently, the cAMP-->PKA-->cAMP response element-binding protein signaling axis is inhib

129 regulated by transcription factor CREB (cAMP-response element-binding protein) and silencing of CREB

130 d with a reduction in the activation of cAMP response element-binding protein, but not the activation

131 e element-binding protein (ChREBP), and cAMP response element-binding protein, hepatocyte specific (C

132 ion status of their respective targets, cAMP response element-binding protein, p38, and extracellular

133 ase activates the transcription factor, cAMP response element-binding protein, regulating miR-212, wh

134 ough several kinases to inhibit carbohydrate response element-binding protein-alpha and -beta.

135 mouse islet alpha cells by GS/cAMP/PKA/cAMP-response element-binding protein-dependent activation of

136 r outputs: p38-dependent growth arrest, cAMP response element-binding protein-dependent cell survival

137 phorylation of the transcription factor cAMP response element-binding protein.

138 on of a pathway involving Akt1/Akt2 and cAMP response element-binding protein.

139 by the transcription factors CREB/CREM (cAMP response element-binding protein/modulator) is linked to

140 substitutions that determined the receptors' response element-binding specificity were far from the p

141 tion of beta-catenin, even to functional Wnt-response elements, can no longer be considered a proxy f

142 transcription factor 4 (Atf4) via C/ebp-Atf-Response-Element (CARE) enhancers.

143 its recruitment to cis-regulatory C/EBP:ATF response elements (CAREs) together with a dimeric partne

144 ed concatenated tandem array of consensus TF response elements (catTFRE) approach to profile the acti

145 eased Nrf2 binding to a putative antioxidant response element consensus sequence in the sterol regula

146 lucose-stimulated expression of carbohydrate response element-containing genes and glucose-stimulated

147 nscriptional activation mediated by the cAMP response element (CRE) in reporter gene studies (10-fold

148 ress or ATF4 coexpression: the -267 ATF/cAMP response element (CRE) site and a novel -248 ATF/CRE mod

149 r binding elements [RARE], cyclic AMP [cAMP] response elements [CRE], NF-kappaB binding sites [kB], s

150 ent of dexamethasone-induced, glucocorticoid response element-dependent transcription by formoterol w

151 ous study demonstrated that distant estrogen response elements (DEREs) located on chromosome 20q13 ar

152 lated-tau levels compared to transactivation response element DNA-binding protein 43 (TDP-43) protein

153 odified with DNA sequences known as estrogen response elements (DNA-ERE), where ERalpha binds specifi

154 ultaneous attenuation of the synthetic auxin response element DR5.

155 r translocator), which recognizes the dioxin response element (DRE) in the promoter of downstream gen

156 21 promoter contains several putative dioxin response elements (DREs).

157 ility of GR and MR to cis-activate a hormone response element-driven reporter variably affected the s

158 nt Cp transcription is mediated by the EBNA2 response element (E2RE), a region that contains at least

159 hat recognizes two versions of a 7-base pair response element, either 5- GAG CA-3 or 5- GAG CA-3 (whe

160 Estrogen receptor alpha (ERalpha)-estrogen response element (ERE)-DNA pull-down assays using HeLa n

161 criptional level directly through a Forkhead response element (FHRE) in its promoter.

162 ed an lncRNA, which has acquired 16 microRNA response elements for miR-143-3p in the Catarrhini branc

163 erentially located in the cytoplasm, and the response elements for miRNAs they share with their targe

164 y growth hormone (GH) through growth hormone response elements (GHREs).

165 s were defined by direct GR binding via a GC response element (GRE) and exclusively increased reporte

166 through interaction with the glucocorticoid-response element (GRE) carrying rs6543115(C) variant.

167 Rbeta binds to the intergenic glucocorticoid response element (GRE) of the STAT1 gene.

168 ceptor (GR) binds to a single glucocorticoid response element (GRE), which drives the expression of p

169 The GR-ACTN4 complex enhances glucocorticoid response element (GRE)-driven reporter activity.

170 GR binding sites dominated by palindromic GC response elements (GRE), suggesting a non-redundant GRIP

171 mechanisms underlying GC-induced positive GC response element [(+)GRE]-mediated activation of transcr

172 n/rat CLDN1 promoters contain glucocorticoid response elements (GREs) and adjacent transcription repr

173 inity GR binding sites within glucocorticoid response elements (GREs) for PRODH and AASS that contrib

174 it contains two glucocorticoid receptor (GR) response elements (GREs).

175 and its paralogs evolved to bind activating response elements [(+)GREs] and negative glucocorticoid

176 mide designed to bind the consensus androgen response element half-site has antitumor activity agains

177 GCs activated consensus hypoxia response element (HRE) reporters in a glucocorticoid rec

178 iminates it from the closely related hypoxia response element (HRE), and is globally affected by the

179 ein construct under the control of a hypoxia response element (HRE)-containing promoter driven by HIF

180 and activation of the HIF-responsive hypoxia-response element (HRE)-luciferase reporter construct.

181 own as HIF-beta) heterodimer bind to hypoxia response elements (HREs) and regulate expression of targ

182 r (AR) on a tandem array of positive hormone response elements (HREs) in chromatin.

183 hypoxia-inducible factors (HIFs) to hypoxia response elements (HREs) of oxygen-regulated genes.

184 JQ1 prevented HIF binding to the hypoxia response element in CA9 promoter, but did not alter HIF

185 , also known as lipid rafts, are the primary response element in EF sensing.

186 1 mutation proteins 2 and 4) bind the stress-response element in gene promoters in the yeast Saccharo

187 he viral protein Rev, which binds to the Rev response element in stem IIB located on unspliced viral

188 eucine-rich protein 1 (PELP1) to an estrogen response element in the CTSD distal promoter; this compl

189 receptor binds directly to a glucocorticoid response element in the CXCR4 promoter and recruits the

190 reased the binding of Nrf2 to an antioxidant response element in the enhancer region of the EDNRB gen

191 criptional control by the c-Jun-binding cAMP response element in the GADD34 gene promoter and posttra

192 PPARgamma bound and activated a canonical response element in the miR-15a gene.

193 rectly to a transcriptionally active hypoxia-response element in the miR-210 proximal promoter.

194 2 (NRF2) by direct binding to an antioxidant response element in the p62 promoter.

195 actor kappa B (NF-kappaB) through a specific response element in the promoter of CD47, and the site o

196 ents confirmed PARP-1 recruitment at the LXR response element in the promoter of the ABCA1 gene.

197 r gene constructs revealed a functional cAMP response element in the proximal promoter of Lrrtm2, ind

198 (NFkappaB), which can stimulate a NF-kappaB response element in the Shh gene promoter.

199 ozygous mutant mice with deletion of hypoxia-response element in the vascular endothelial growth fact

200 ardation of migration of activator protein-1 response elements in EMSA.

201 response elements and 4 to 8% lack androgen response elements in orthologous promoters in the zebra

202 association of beta-catenin with Tcf1 on Wnt response elements in target gene enhancers.

203 Mechanistic investigations identified NFATc2 response elements in the 3' enhancer region of SOX2, and

204 he nucleus that binds specific retinoic acid response elements in the absence of RA.

205 ected mutagenesis revealed prominent hypoxia response elements in the CLDN1 promoter region.

206 ligand-activated AHR to the putative dioxin response elements in the EBF1 promoter was demonstrated

207 cells and CARNs and discovered new androgen response elements in the Nkx3.1 3' UTR.

208 c leukemia 1 (MKL1), which targets the serum response elements in the promoter of rat Slc1a2 gene enc

209 itamin D receptor binding with the vitamin D-response elements in the promoter of the gene encoding i

210 f GGAA-microsatellites as EWS/FLI activating response elements in vivo and reveal an unexpected role

211 ion of hepatic IGF-1 intron 2 growth hormone response element (IN2GHRE) histone methylation of key ly

212 Zta recognizes several methylated Zta-response elements, including meZRE1 (5- GAG C A-3) and m

213 nary conserved inverted repeated negative GC response element (IR nGRE)-mediated direct transrepressi

214 nary conserved inverted repeated negative GC response element (IR nGRE)-mediated direct transrepressi

215 a promoter containing interferon-stimulated response elements (ISRE).

216 d gene (ISG), and two adjacent IFN-sensitive response elements (ISREs) in the promoter region of cGAS

217 ription factors to loci with their consensus response elements led to the increased formation of an a

218 mobility shift assays identified a PPARalpha response element located at position -2109 base pair rel

219 FoxO1 binds to insulin response elements located in the promoters of insulin-li

220 85-5p expression via binding to the androgen response elements located on the promoter of miR-185-5p,

221 decoy oligonucleotide carrying wild-type p53-response element markedly attenuated OT-induced THTR1, P

222 nd utilization of regional-specific estrogen response elements may be associated with differential ac

223 as increased cyclic adenosine monophosphate response element-mediated transcriptional activity and p

224 t the critical role of ventral striatal cAMP-response element modulator (CREM) in mediating impulsivi

225 ranscriptional repressor isoform of the cAMP response element modulator (CREM).

226 ew methods exist for the prediction of miRNA response elements (MREs) in lncRNAs acting as ceRNAs (sp

227 inatorial interactions between sets of miRNA response elements (MREs), providing strong evidence for

228 ic yeast Pichia pastoris by binding to Mxr1p response elements (MXREs) present in their promoters.

229 lines driven by functional ROS (antioxidant response elements), NFkappaB, and mothers against decape

230 TGFbeta1 signaling through ROS (antioxidant response elements), NFkappaB, and SMAD3 in both cell lin

231 ements [(+)GREs] and negative glucocorticoid response elements (nGREs).

232 suppressor P53, and inhibit the antioxidant response element Nrf-2, resulting in depleted glutathion

233 PGR proteins were recruited on progesterone response element of Gpr64 gene in the uteri of wild-type

234 the increased binding to the glucocorticoid response element of histone deacetylase 2 (HDAC2) promot

235 chanism, predominantly at the proximal serum response element of the c-fos promoter.

236 d that more IRF8 bound to the IFN-stimulated response element of the Ifit1 gene than to those of the

237 its auxin inducibility and binding to auxin response elements of rtcs and rtcl promoters, ARF34 (AUX

238 adenovirus triggers the cellular DNA damage response, elements of which include cell death and cell

239 RORalpha directly bound to a specific ROR response element on the promoter of Sema3e and negativel

240 imeric partner RXR to the negative vitamin D response element on the promoter of Smad7, which leads t

241 xin transporters at a specific PIN CYTOKININ RESPONSE ELEMENT (PCRE) domain.

242 PPAR response element (PPRE) activity was measured in PPRE-lu

243 sis identified three peroxisome proliferator response elements (PPREs) within the 863-nt region and s

244 ent manner, a consequence of a Nanos/Pumilio response element (PRE) in its 3'UTR.

245 ivation and contains a putative progesterone response element (PRE).

246 Polycomb response elements (PREs) are specific DNA sequences that

247 xG) group proteins are assembled on Polycomb response elements (PREs) to maintain tissue and stage-sp

248 e short genomic fragments, known as Polycomb response elements (PREs), that direct Polycomb repressiv

249 ired for targeting PRC complexes to Polycomb Response Elements (PREs).

250 A via cis-acting elements known as "Polycomb response elements" (PREs).

251 ts in its decreased binding to the ER stress response element present in the promoter region of Grp78

252 Mxr1p binds to Mxr1p response elements present in the promoters of AAT2, MDH2

253 nes expressed in response to the antioxidant response element promoter.

254 The frequency of Retinoic Acid Response Element (RARE) sequences was increased in Brazi

255 130 bp that contains PRE half-sites and a RA response element (RARE).

256 nitiated via p53 binding to its specific DNA response elements (RE).

257 caused by oxidative stress using antioxidant response element reporter gene assay models and big data

258 creased transcription from NFAT or NF-kappaB response element reporters, respectively.

259 Canonical p53 response elements (REs) are made of two decameric half-s

260 nscription factor proteins (TFs) and the DNA response elements (REs) they recognize, control most bio

261 loop limiting excessive accumulation of iron-response element RNA-binding activity, whose disruption

262 Transactivation response element RNA-binding protein (TRBP; TARBP2) is k

263 also known as SRC-1 and SRC-3) to an AR-ROR response element (RORE) to stimulate AR gene transcripti

264 tor of expression of virion (Rev) to the Rev response element (RRE) and subsequent oligomerization in

265 The HIV-1 Rev response element (RRE) is a 351-base element in unsplice

266 ines in the stem loop II region of HIV-1 Rev response element (RRE) RNA enhanced binding of HIV-1 Rev

267 structured element within viral RNA, the Rev response element (RRE), and escorts RRE-containing RNAs

268 nome encapsidation or, unexpectedly, the Rev response element (RRE), which regulates the nuclear expo

269 , by binding to and oligomerizing on the Rev Response Element (RRE).

270 element and key GSR marker, the rapid stress response element (RSRE), in the promoters of robustly in

271 three proteins bind to the synaptic activity response element (SARE), an enhancer sequence found upst

272 chIFN-kappa regulated the IFN-stimulated response element signalling pathways and activated a pan

273 ession and binding to the p28 IFN-stimulated response element site.

274 ease was lost when estrogen-related receptor response element sites were mutated.

275 to create aGPCR probes, we developed a serum response element (SRE)-luciferase-based screening approa

276 omoter contains a putative conserved hypoxic response element suggesting that the transcription facto

277 th MYBS1 and MYBS2 could recognize the sugar response element, TA-box, in the promoter and induced pr

278 Transactivation response element (TAR) DNA-binding protein 43 (TDP-43) m

279 iption (Tat) and its cognate transactivation response element (TAR) RNA transactivates viral transcri

280 tem-loop constructs of HIV-1 transactivation response element (TAR) RNA, we achieved nucleotide-speci

281 pable of binding to an interferon-stimulated response element than do cells expressing T387A STAT2.

282 moter also contains a functional antioxidant response element that is directly bound by the NRF2 orth

283 th factor-beta (Smads), and hypoxia (hypoxia response element), that regulated levels of all pri-miR

284 rmined that the Adam10 promoter harbors PPAR response elements; that knockdown of PPARalpha, but not

285 ning in C. elegans, we uncover two metformin response elements: the nuclear pore complex (NPC) and ac

286 mVP24 leading to upregulation of antioxidant response element transcription, which is distinct from o

287 rs (TRs) alpha and beta act by binding to TH response elements (TREs) in regulatory regions of target

288 ta have consistently shown GR to occupy AP-1 response elements (TREs), even in the absence of AP-1.

289 aperones, and activated the unfolded protein response element (UPRE), suggesting that VPA induces the

290 d expression was dependent on glucocorticoid response elements upstream of annexins and was reinforce

291 ded DNA and function in trans as a vitamin D response element (VDRE)-binding protein.

292 We identified and characterized vitamin D response elements (VDREs) located in both genes and show

293 ER stress response elements were identified in the catalase gene a

294 tivate the FOXM1 promoter through a Forkhead response element, whereas FOXM1 can activate AURKA expre

295 F composite element or interferon-stimulated response element, whereas R291Q retained BATF/JUN intera

296 eceptor (GR) binds as a homodimer to genomic response elements, which have particular sequence and sh

297 Blimp-1 binds an IFN-stimulated response element within HIV-1 provirus, and it is displa

298 eta-catentin and TCF4 and interacts with Wnt response elements (WREs) and promoters of direct target

299 ity depends on its binding to the xenobiotic response element (XRE) in partnership with the AhR nucle

300 lysis revealed the presence of Zn-deficiency-response elements (ZDREs) in a number of the ZIPs.