ライフサイエンスコーパス: rest tremor

1 nge in phase difference between postural and rest tremor.

2 c component, whereas 57 patients (40.7%) had rest tremor.

3 reduced neuromuscular strength, dystonia and resting tremor.

4 0.574 for spiral drawing, 0.441 to 0.488 for rest tremor, 0.266-0.577 for postural tremor and 0.306 f

5 Usp14-deficient axJ mice display a resting tremor, a reduction in muscle mass, and notable

6 symptoms of Parkinson's disease (PD) include resting tremor, akinesia, bradykinesia, and rigidity, an

7 kinson's disease cohort were associated with rest tremor amplitude (beta = -0.106, P < 0.05), rest tr

8 can achieve almost 50% average reduction in resting tremor amplitude and in so doing form the basis

9 fewer limb segments than Parkinson's disease rest tremor and essential tremor, and its frequency was

10 Moreover, severe motor impairment, resting tremor and abnormal gait and posture, phenotypes

11 Two mutant mice were identified with marked resting tremor and further characterized using the SHIRP

12 frequency of perinatal mortality, scoliosis, resting tremors and ptosis.

13 to those of previous studies of parkinsonian rest tremor, and highlight an important difference in th

14 ve parkinsonism with bradykinesia, rigidity, resting tremor, and impaired postural reflexes was seen

15 , these animals have severe limb weakness, a resting tremor, and notable neuroanatomical malformation

16 r symptoms including bradykinesia, rigidity, resting tremor, and postural instability.

17 is characterized by rigidity, bradykinesia, resting tremor, and postural instability.

18 n of clinical symptoms such as bradykinesia, resting tremor, and rigidity.

19 , decreased overall activity, acquisition of resting tremors, and increased susceptibility to pentile

20 Egr3-/- mice develop gait ataxia, scoliosis, resting tremors, and ptosis, suggesting a defect in prop

21 lowing inclusion criteria: the presence of a rest tremor, bradykinesia, and rigidity; a modified Hoeh

22 disease include motor impairments involving resting tremor, bradykinesia, postural instability, gait

23 ilability is associated with the severity of resting tremor but not non-motor symptoms.

24 tremor amplitude (beta = -0.106, P < 0.05), rest tremor constancy (beta = -0.109, P < 0.05), and ind

25 porter availability was also associated with rest tremor constancy (beta = -0.380, P < 0.05) and inde

26 P0-GGFbeta3 mice develop resting tremors, gait abnormalities, decreased hindlimb

27 Monosymptomatic rest tremor has recently been shown to be associated wit

28 Parkinson's resting tremor has been linked to pathophysiological cha

29 ing of cortical oscillations responsible for rest tremor in the periphery by delivering tremor-freque

30 e GPe-STN loop, which may be relevant to the resting tremor in Parkinson's disease.

31 p-brain stimulation of the STN can alleviate resting tremor in Parkinson's disease.

32 Whereas the rest tremor is characteristic, action tremor, re-emergen

33 these results we conclude that parkinsonian rest tremor is driven by a neural network, which include

34 characterized by bradykinesia, rigidity, and resting tremor, is the most common neurodegenerative mov

35 tion of postural and kinetic components with resting tremors less frequently seen.

36 or frequency that dictates that parkinsonian rest tremor may be significantly entrained by low freque

37 tive disorder characterized by bradykinesia, resting tremor, muscular rigidity, and postural instabil

38 directly linked to the characteristic 3-6 Hz rest tremor of this disease.

39 Physiological evidence indicates that the resting tremor of Parkinson's disease originates in osci

40 compared to akinetic-rigid patients with no resting tremor (P < 0.05).

41 was assessed in a test cohort comprising 16 rest tremor recordings in tremor-dominant Parkinson's di

42 Specifically, the amplitude of parkinsonian rest tremor remained unchanged despite spontaneous chang

43 nical features of bradykinesia, rigidity and resting tremor resulting from the deficiency of dopamine

44 r cortex to induce phase cancellation of the rest tremor rhythm.

45 It is characterized by resting tremor, rigidity, bradykinesia or slowness, gait

46 eurodegenerative disorders, characterized by resting tremor, rigidity, bradykinesia, and postural ins

47 minating in severe motor symptoms, including resting tremor, rigidity, bradykinesia, and postural ins

48 ied Parkinson's Disease Rating Scale (UPDRS) rest tremor score of 3 or greater for any limb, a Mini-M

49 ancy (beta = -0.109, P < 0.05), and index of rest tremor severity (beta = -0.104, P < 0.05).

50 tancy (beta = -0.380, P < 0.05) and index of rest tremor severity (beta = -0.322, P < 0.05).

51 ns predicted that without a voluntary drive (rest tremor) the neural drives would be more likely in p

52 Furthermore, patients exhibit resting tremor, unstable gait, and impaired balance, whi

53 Rest tremor was unilateral/asymmetric in up to 92.9% of