ライフサイエンスコーパス: resting

1 Height was directly measured, and a resting 12-lead ECG obtained under standardized conditio

2 ing and lengthening of the macromolecules at resting active zones.

3 re sleeping heart rate, activity levels, and resting, active and total energy expenditure.

4 single-channel electrophysiology to measure resting affinities (binding free energies) of these and

5 DNA isolated from resting and activated AC mimicked their respective immun

6 r of BCR single-molecule localizations, both resting and activated B-cells intrinsically maintain a h

7 bolic consequence of ablating TBC1D1 in both resting and contracting skeletal muscles, utilizing a ra

8 nergetic effects of palmitate and glucose on resting and energetically challenged mitochondria in DRG

9 s and whole brain tissue, as well as between resting and physiologically stimulated neurons.

10 d distinct LRET signals were obtained in the resting and slow inactivated states.

11 scillatory activity in the 20-40 Hz range in resting and walking states, and increased interhemispher

12 LFPs were recorded in resting and walking states, before and after unilateral

13 sis is a form of cell death that occurs in a resting (and thus latently infected) T cell when a produ

14 However, whether resting astrocyte Ca(2+) can adjust to a new steady-stat

15 We define three main behavioral modes: resting at the gel's surface, digging while feeding near

16 A resting atom excites if a sum of its excited hard neighb

17 ng tumor growth and metastasis by converting resting B and T cells into IL-10-producing and IL-35-pro

18 For the resting brain functional activity, significant group x t

19 naptic strength and arteriole tone via their resting Ca(2+) activity.

20 environment, we measured a relatively lower resting Ca(2+) level that occluded any further drop in C

21 for muscle relaxation and maintenance of low resting Ca(2+) levels in the myoplasm.

22 Global resting CBF and regional CBF of right superior frontal c

23 enhanced HIV-1 infection of SAMHD1-deficient resting CD4 T cells of a patient with Aicardi-Goutieres

24 monocytes, macrophages, dendritic cells, and resting CD4 T cells, HIV encounters a block, limiting re

25 Purification of resting CD4(+) T cells from whole PBMC is expedited and

26 ble, replication-competent latent HIV-1 from resting CD4(+) T cells is essential for HIV-1 eradicatio

27 A latent reservoir for HIV-1 in resting CD4(+) T lymphocytes precludes cure.

28 type 1 (HIV-1) persists in latently infected resting CD4+ T cells (rCD4 cells), posing a major barrie

29 require the reactivation of latent HIV-1 in resting CD4+ T cells (rCD4s).

30 n cell-associated HIV RNA within circulating resting CD4+ T cells.

31 in the frequency of latent infection within resting CD4+ T cells.

32 cells, effectively promoted reactivation of resting CD4+ T-cell, as indicated by an increased viral

33 conserved SIRT1-FoxO1 axis as a regulator of resting CD8(+) memory T cell metabolism and activity in

34 8(-) T cells, and inhibiting its activity in resting CD8(+)CD28(+) T cells enhanced glycolytic capaci

35 wide range of hypoxia at different levels of resting cell membrane potential (Em ).

36 PE is generally sequestered inside a normal resting cell, and the mechanism by which circulating ant

37 IgG BCRs are more clustered than IgM BCRs on resting cells and form larger protein islands after anti

38 ndoplasmic reticulum Ca(2+) concentration in resting cells, and for the refilling of Ca(2+) stores af

39 dies targeting circRNAs have been limited to resting cells, leaving their role in dynamic cellular re

40 etween AKAP79 and calcineurin, and increases resting cellular PKA phosphorylation.

41 ological indicators: (1) global and regional resting cerebral blood flow (CBF), (2) oxygen extraction

42 st-conventional individuals showed increased resting cerebral blood flow in the ventral striatum and

43 e, whereas F57Bpa interacts predominantly in resting channel conformations.

44 esting that this conductance is active under resting conditions in colonic muscles.

45 In resting conditions, estrogens have strong regulatory eff

46 oading, current therapy is mainly focused on resting conditions.

47 esting T cells cholesterol keeps TCRs in the resting conformation that otherwise would become spontan

48 In the resting conformation, the TCR is not phosphorylated and

49 hey have a higher affinity for active versus resting conformations of the protein.

50 Moreover, resting cord blood KIR3DL1 NK cells exhibited a basal al

51 approaches to exclude HFpEF on the basis of resting data alone and reinforce the value of exercise t

52 ary arteries, left ventricular function, and resting ECG.

53 with invasively proven HFpEF on the basis of resting echocardiographic data alone.

54 ains unclear.We studied the relation between resting energy expenditure (REE), the estimated energy b

55 index, body composition, hip circumference, resting energy expenditure, and respiratory quotient.

56 S patients are hypermetabolic with increased resting energy expenditure, but if and how hypermetaboli

57 ntake, greater lipid fuel preference and non-resting energy expenditure, one-half the body fat, and b

58 nd repeating network patterns in whole-brain resting fMRI data, where networks are defined as graphs

59 activity induced a long-lasting reduction in resting free astrocyte Ca(2+) and that this phenomenon c

60 ABSTRACT: Chronic hypoxia increases resting heart rate (HR), but the underlying mechanism re

61 Increased resting heart rate correlated with behavioural (Cambridg

62 Resting heart rate is a heritable trait, and an increase

63 Increased resting heart rate is associated with worse outcomes in

64 Ivabradine safely reduced the resting heart rate of children with chronic HF and dilat

65 udy analyses have found loci associated with resting heart rate, at the time of our study these loci

66 A lower achieved resting heart rate, irrespective of treatment, was assoc

67 es [S-nitrosothiol (SNO)] on 491 proteins in resting hearts from a mouse model of DMD (dmd(mdx):utrn(

68 In seven healthy lowlanders, resting HR was determined at sea level (SL) and after 15

69 lasses of drug transporters, SLC and ABC, in resting human blood neutrophils.

70 These states very slowly returned to resting (i.e. ferric) enzyme, indicating that they repre

71 Resting in vivo diaphragm function was also unaffected b

72 All diastolic resting indexes tested were identical to iFR, both numer

73 of this study was to compare other diastolic resting indexes to iFR.

74 ronary stenoses, which is more accurate than resting indices and does not require adenosine.

75 a novel patient subgroup was identified with resting invasive hemodynamics consistent with pulmonary

76 to the increased reliance on fatty acids in resting KO animals.

77 GABAergic system, it is conceivable that the resting level of cortical GABAergic tone directly relate

78 Finally, we report that resting levels of long-chain triacylglycerols in mitocho

79 Resting levels of O2 in the rodent brain varied between

80 ut not Ca(2+)-free CaM were preassociated in resting live cells, while capsaicin activation induced b

81 tructures that form in LPS-activated but not resting macrophages following silica bead phagocytosis.

82 effects of Acanthamoeba on the activation of resting macrophages.

83 erived neurons showed impaired maturation of resting membrane potential and action potential firing,

84 nctions, cardiac macrophages have a negative resting membrane potential and depolarize in synchrony w

85 nd L811P evoked large depolarizations of the resting membrane potential and impaired action potential

86 Human cardiomyocytes exhibit two levels of resting membrane potential at subphysiological extracell

87 ation currents, reconstituting two levels of resting membrane potential in cardiomyocytes.

88 Resting membrane potential measured using intracellular

89 Intracellular recording revealed a resting membrane potential of approximately -70 mV.

90 Kir2) channels primarily maintain the normal resting membrane potential of cardiomyocytes.

91 ily 2 (Kir2) channels primarily maintain the resting membrane potential of cardiomyocytes.

92 ation currents, accounting for two levels of resting membrane potential of human cardiomyocytes.

93 The pumps maintain ionic gradients and the resting membrane potential of neurons, but increasing ev

94 d current-voltage relationships, causing the resting membrane potential to spontaneously jump from hy

95 TN neurons, the Nalcn current influences the resting membrane potential, contributes to maintenance o

96 CXCL12 depolarized the resting membrane potential, decreased the rheobase, and

97 reveal ionic mechanisms of the two levels of resting membrane potential, demonstrating a previously u

98 isoform 1 (K2P1) recapitulate two levels of resting membrane potential, indicating the contributions

99 ynaptic activity may be secondary to altered resting membrane potential.

100 ation currents, reconstituting two levels of resting membrane potential.

101 otential, again recapitulating two levels of resting membrane potential.

102 of K2P1 channels recapitulate two levels of resting membrane potential.

103 o-current potentials match the two levels of resting membrane potential.

104 of Kir2.1 and K2P1 channels to two levels of resting membrane potential.

105 ' that rundown of inhibitory SK responses at resting membrane potentials (RMPs) reflects depletion of

106 ular calcium stores, and run down rapidly at resting membrane potentials when calcium stores become d

107 tes show both hyperpolarized and depolarized resting membrane potentials; these depolarized potential

108 racterized by loss of naive B cells, loss of resting memory B cells due to their redistribution to th

109 ter amounts of gp120-specific B cells in the resting memory subset, whereas HIV-specific B cells in p

110 on recently activated T cells, but absent on resting memory T cells.

111 involved in stress, to determine whether its resting metabolic activity predicts risk of subsequent c

112 anges (DeltaWG compared with DeltaRG) in the resting metabolic rate (RMR) (43 +/- 25 kcal/d; P = 0.04

113 variants were analyzed for association with resting metabolic rate (RMR) and 24-h EE assessed in a w

114 Leptin contributes to the control of resting metabolic rate (RMR) and blood pressure (BP) thr

115 Evidence suggests that fat-free mass and resting metabolic rate (RMR), but not fat mass, are stro

116 rved no significant extrinsic sialylation in resting mice, suggesting that extrinsic sialylation is n

117 Functional connectivity in the resting motor network between right and left supplementa

118 The primary efficacy end point was change in resting myocardial perfusion over 6 months.

119 , we performed epigenetic profiling of human resting naive, central and effector memory T cells using

120 Resting of extruded pasta at 35 degrees C significantly

121 of daylight and ambient temperature on human resting or sleeping patterns using mobile phone data of

122 rlying mechanisms of the interaction between resting or stimulated human NK cells with M1 or M2.

123 fraction, patients with hypocapnia had lower resting PaCO2 and lung diffusing capacity (P < 0.01).

124 th a sampling rate of 25kHz, and a 30-minute resting period was analyzed for each patient.

125 arly dynamics of the afternoon and nocturnal resting periods appear to be counterbalancing each other

126 ion, drying) but also those occurring during resting periods at 35 degrees C, applied in-between them

127 hen jumping from a pH 5 stimulus to a single resting pH of 8.

128 Given that the resting pH of the synaptic cleft is highly dynamic and d

129 es sleep during the active phase but reduces resting-phase sleep quality.

130 nism of platelet aggregation pertains to how resting platelets ignore soluble fibrinogen, the third m

131 rface, thus mediating further recruitment of resting platelets.

132 At resting potential (-63.7 +/- 0.6 mV), approximately 90%

133 ncrease excitability via a depolarisation of resting potential and increased evoked firing.

134 ne potential excursions, raising the average resting potential and producing oscillations.

135 ted that a U-shaped relationship between the resting potential and the neuronal action potential thre

136 ing in axons and contributing to setting the resting potential.

137 nsitive, peaking at membrane potentials near resting potential.

138 conductance contributes to the regulation of resting potentials and excitability of colonic muscles.

139 of Kir2 gene products, with this regulating resting potentials and the excitability of colonic muscl

140 We show that CLEC3A is present in resting, proliferating, and hypertrophic growth-plate ca

141 pproximately 9% to 10% of total variation in resting QTc in EA individuals and approximately 12% to 1

142 Sixteen probands had normal resting QTc values and only developed QT prolongation an

143 e and energetic costs three to six times the resting rate of energy expenditure) that rapidly deplete

144 depicting that majority of cells were in the resting rather than active phase.

145 esting receptors that then become active, or resting receptors activate and then bind agonists.

146 at connect to form a cycle: Agonists bind to resting receptors that then become active, or resting re

147 ension (AT (norm)) and active tension at the resting sarcomere length (T (req), reflecting required c

148 in 10% of the reference value (ie, patient's resting SBP) or standard management strategy of treating

149 transduction also play a role in setting the resting sensitivity in OSNs.

150 /-) mice or CNGB1(DeltaCaM) mice show normal resting sensitivity, as determined by their unchanged EO

151 blunt alpha1 -adrenergic vasoconstriction in resting skeletal muscle would be independent of KIR , NO

152 METHODS AND Resting SM high-energy phosphate concentrations and ATP

153 crease of GL transcription in developing and resting splenic B cells and altered CSR in activated B c

154 tional magnetic resonance imaging included a resting state and an experimental paradigm focusing on t

155 s shown to enhance mPFC/ACC activity even at resting state and improve cognitive function in patients

156 e that the global directionality patterns in resting state brain networks can be predicted solely by

157 We hypothesized that resting state brain oscillations will show unique defici

158 , functional clustering based on independent resting state data revealed that DMN and shift regions c

159 em cells out of the proliferative and into a resting state during muscle growth.

160 dren with ASD were abnormally organized with resting state EEG.

161 Resting state fMRI (rs-fMRI) is commonly used to study t

162 nt-related spectral decomposition (ERSP) and resting state functional connectivity (rsfcMRI) approach

163 Resting state functional connectivity is defined in term

164 connectome data (diffusion tractography and resting state functional connectivity) to identify conne

165 icate in humans (154 healthy controls) using resting state functional connectivity, tracing studies c

166 nted for measures of volume and as seeds for resting state functional connectivity.

167 igate the organization of the amygdala using resting state functional magnetic resonance imaging (rsf

168 nectivity between different brain areas with resting state functional magnetic resonance imaging in h

169 Here we assess LRTCs in resting state human EEG data during a 40-hour sleep depr

170 s assessed brain spontaneous activity in the resting state in chronic smokers.

171 a suggest that reversible protonation of the resting state is likely occurring, and we term this stat

172 inherent problem with side-by-side turnover/resting state measurements, i.e., the difficulty to desi

173 ent brain locations but localize to a unique resting state network, providing insight into the neurob

174 cific spatiotemporal patterns in the form of resting state networks (RSNs).

175 ted copper(I) dihydridoborate complex is the resting state of the catalyst in this case.

176 The resting state of the catalyst is an NHC-Ni(eta(6)-arene)

177 With DTBM-SEGPHOS as the ligand, the resting state of the catalyst, which is also a catalytic

178 (2+) affinity of the RyR (or "stabilize" the resting state of the channel) and suggest that the accum

179 ntipsychotic drugs (<2 years), who underwent resting state scanning while entering 12 weeks of prospe

180 t five functional magnetic resonance imaging resting state scans during treatment.

181 MecA converts this resting state to an active planar ring structure by bind

182 t as an additive upon the reaction, catalyst resting state, and turnover-rate limiting step has been

183 hich is considered the hallmark of the brain resting state, in photosensitive patients and in people

184 >10(4) faster than the unobserved IET in the resting state, showing that CuZ degrees is the catalytic

185 ls with leaky gates that conduct ions in the resting state.

186 at two oxidation levels above an all-Co(III) resting state.

187 working state and functional connectivity at resting state.

188 activating histone modifications even in the resting state.

189 two oxidation equivalents above the Fe(III) resting state.

190 , and bromide efficiently restore the ferric resting state.

191 rements during substrate turnover and in the resting state.

192 s with successive oxidations from the cubane resting state.

193 tial correlation analysis of cross-sectional resting-state (18)F-fluorodeoxyglucose positron emission

194 Resting-state (rs)-fMRI and diffusion weighted imaging (

195 Furthermore, the frontocentral resting-state activity predicted the individual magnitud

196 at cognitive dissonance is reflected in both resting-state and choice-related activity of the prefron

197 Here we combined resting-state and task-driven functional magnetic resona

198 n architecture reverted to a more segregated resting-state architecture both immediately before and a

199 Although changes in resting-state brain connectivity are a transdiagnostic k

200 The resting-state brain regional connectivity had no signifi

201 bipolar disorder was associated with reduced resting-state cohesiveness of the sensorimotor network i

202 Activity flow over resting-state connections thus provides a large-scale ne

203 or memories could be predicted from baseline resting-state connectivity in locomotor-related networks

204 ) in the same group of subjects, we analyzed resting-state data from the core of the default-mode net

205 In 65 unmedicated depressed patients 15-min resting-state EEGs were recorded off medication (baselin

206 ed a genome-wide association study (GWAS) on resting-state fast beta EEG power.

207 we investigate how the switching behavior of resting-state FC relates with cognitive performance in h

208 f the time-shift analysis (TSA) approach for resting-state fMRI (rs-fMRI) blood oxygenation level-dep

209 the brain's functional connectivity (FC) is resting-state fMRI (rs-fMRI).

210 As previously reported, seed-based resting-state fMRI analyses revealed that the LC was fun

211 Here we use resting-state fMRI data from 176 subjects to show that s

212 We analyse resting-state fMRI data from 98 healthy adults previousl

213 sing an energy-landscape analysis applied to resting-state fMRI data.

214 ration when studying brain organization with resting-state fMRI in the future.

215 d wandering, which occurs unavoidably during resting-state fMRI scans and may induce variability of t

216 All the subjects underwent resting-state fMRI scans and the data were analyzed by t

217 Resting-state fMRI was administered to 33 cirrhotic pati

218 ng functional connectivity (FC) derived from resting-state fMRI with gold standard structural connect

219 d cingulate network connectivity measured by resting-state fMRI, even in the absence of hypometabolis

220 ndering in particular thought domains during resting-state fMRI.

221 missing hand, as evidenced by task-based and resting-state fMRI.

222 in the context of locomotor adaptation using resting-state fMRI.

223 Published MRI evidence of structural and resting-state functional brain abnormalities in MDD has

224 ivoxel pattern analysis, and high-resolution resting-state functional connectivity (RSFC) analyses, w

225 METHOD: Functional MRI resting-state functional connectivity analyses using a b

226 I/II during semantic prediction and enhanced resting-state functional connectivity between hubs of th

227 ings was also correlated with an increase in resting-state functional connectivity between the mid-in

228 e-function relationship, suggesting that the resting-state functional connectivity depends on direct

229 kinson disease (PD) by analyzing whole-brain resting-state functional connectivity in PD patients wit

230 d the subgenual cingulate was assessed using resting-state functional connectivity magnetic resonance

231 Capitalizing on recent advances in resting-state functional connectivity magnetic resonance

232 AC4 expression, fear-potentiated startle and resting-state functional connectivity of the amygdala in

233 The resting-state functional connectivity of the following t

234 ortical thickness of the identified regions, resting-state functional connectivity of the identified

235 Here, we studied resting-state functional connectivity of the left IFG in

236 visceral interoceptive attention task and a resting-state functional connectivity scan.

237 cale of the Behavioral Activation Scale) and resting-state functional connectivity using multivariate

238 Resting-state functional connectivity was unrelated to m

239 s have described the spatial overlap between resting-state functional correlation (RSFC) subnetworks

240 Moreover, resting-state functional coupling was decreased during a

241 rpose To prospectively investigate, by using resting-state functional magnetic resonance (MR) imaging

242 Major advances in resting-state functional magnetic resonance imaging (fMR

243 Resting-state functional magnetic resonance imaging (fMR

244 er FC, estimates temporal correlation of the resting-state functional magnetic resonance imaging (rs-

245 tructure and functional connectivity through resting-state functional magnetic resonance imaging (rsf

246 functional and structural connectivity using resting-state functional magnetic resonance imaging and

247 We used resting-state functional magnetic resonance imaging and

248 (D2R) antagonist amisulpride] in humans with resting-state functional magnetic resonance imaging and

249 Subsequently, we collected resting-state functional magnetic resonance imaging data

250 n, graph theory-based method, was applied to resting-state functional magnetic resonance imaging data

251 rbitofrontal cortex of MAM rats showed lower resting-state functional magnetic resonance imaging func

252 vioral measures, psychiatric assessment, and resting-state functional magnetic resonance imaging in a

253 s used to disclose the effects of DBS during resting-state functional Magnetic Resonance Imaging in t

254 We performed resting-state functional magnetic resonance imaging of p

255 ctroscopy to quantify GABA levels as well as resting-state functional magnetic resonance imaging to a

256 activation by c-Fos immunohistochemistry and resting-state functional magnetic resonance imaging; and

257 20 healthy control subjects underwent a 3-T resting-state functional MR imaging and static posturogr

258 ating-current stimulation (tACS) [8-12] with resting-state functional MRI (fMRI) [13] to follow both

259 a distributed reward network, assessed using resting-state functional MRI (fMRI), to predict the onse

260 etworks by applying a clustering analysis on resting-state functional MRI (RSfMRI) data from white-ma

261 ow-up functional connectivity analyses using resting-state functional MRI collected in the same parti

262 Here, using a large multisite study, resting-state functional MRI data were examined in young

263 We used resting-state functional MRI recordings in 27 patients w

264 We applied novel fetal resting-state functional MRI to measure brain function i

265 typically developing (TD) controls underwent resting-state functional MRI, and functional connectivit

266 transfer mapping-to test the hypothesis that resting-state functional network topology describes the

267 signal changes during sematic processing and resting-state GABA concentrations in the ATL.

268 ficant, duration-dependent decrease in local resting-state GABAA inhibition, as quantified by short i

269 Individuals with stronger resting-state long-range temporal correlations demonstra

270 ional magnetic resonance imaging (fMRI) with resting-state magnetic resonance spectroscopy (MRS) to m

271 isorders [5], highlighting the importance of resting-state measurements for understanding brain funct

272 ted based on estimated activity flow through resting-state network connections.

273 in, demonstrating the cognitive relevance of resting-state network topology.

274 d was applied to identify the cerebellar DNs resting-state network; first-level and high-level analys

275 ehavioural impairment and the segregation of resting-state networks empirically measured.

276 sults in the net behavior of nodes composing resting-state networks identified using functional magne

277 Resting-state regional cerebral blood flow (CBF) can be

278 1 males and 28 females) underwent an initial resting-state scan, followed by a cognitive reasoning ta

279 t levels of complexity, followed by a second resting-state scan.

280 Resting-state signals in blood-oxygenation-level-depende

281 rrelated with tinnitus loudness, we assessed resting-state source-localized EEG before and after one

282 The typical resting-state structure consisted of a high-frequency oc

283 mode network connectivity during a separate resting-state task.

284 s demonstrate that intrinsic fluctuations in resting-state variability exhibit unique maturation traj

285 In two independent "resting-state" datasets (electroencephalography surface

286 rks are modest when compared with intrinsic "resting-state" network architecture.

287 Such chelates are the major catalyst resting states but are in rapid equilibrium with ethylen

288 By correlating spontaneous activity during "resting states" [1], studies of intrinsic brain networks

289 anization was stable across task-engaged and resting states, suggesting that abstract context represe

290 nd determined to be interconverting catalyst resting states.

291 a different aspect of astrocyte Ca(2+): the resting, steady-state free Ca(2+) of astrocytes, its mod

292 ines the size and proliferative state of the resting T cell pool.

293 In resting T cells cholesterol keeps TCRs in the resting co

294 d dendritic cells and macrophages as well as resting T-cells, SAMHD1 blocks HIV-1 infection through t

295 ed for proper movement of dynein, increasing resting time between movements.

296 They are also involved in maintaining the resting tone of the cerebral vessels by releasing ATP an

297 Moreover, severe motor impairment, resting tremor and abnormal gait and posture, phenotypes

298 egions of genes involved in TLR signaling in resting UCB monocytes.

299 on and activity of cathepsin K compared with resting unpolarized macrophages.

300 ressive immobilization of water during dough resting, which could be linked to changes in evolution o