ライフサイエンスコーパス: restless

1 rolled (includes children who are impulsive, restless and distractible) and inhibited (includes child

2 toms (that is, pain, tired, short of breath, restless, anxious, sad, hungry, scared, thirsty, confuse

3 Clinically, human RESTless breast tumors also displayed significantly enha

4 1) is the most highly upregulated protein in RESTless breast tumors.

5 Restless genital syndrome is a rare disorder that can be

6 In patients with PD, restless genital syndrome should be included in the diff

7 ed to motor fluctuations but compatible with restless genital syndrome.

8 a mutant that was well described previously (restless) is also highly sensitive to Mg2+.

9 enhanced malignancy in tumors lacking REST (RESTless) is only partially understood.

10 syndrome (OSAS) (0.06%), parasomnia (0.7%), restless leg syndrome (0.9%), or psychological insomnia

11 Restless leg syndrome (RLS) is a sensorimotor disorder.

12 diopathic generalized seizures, and atypical restless leg syndrome (RLS).

13 Restless leg syndrome is a relatively common problem in

14 gical diseases (Parkinson's disease (PD) and restless leg syndrome) is associated with impulse contro

15 ntial uses other than heart disease, such as restless leg syndrome, sudden deafness, hepatorenal synd

16 y to sleep disorders, such as narcolepsy and restless leg syndrome.

17 hat has been implicated as a risk factor for restless legs display important features of the syndrome

18 gand binding and RLS severity (international restless legs scale, IRLS) in areas serving the medial p

19 ted odds ratio =1.26; 95% CI = 1.02-1.54) or restless legs syndrome (n = 108, adjusted odds ratio = 1

20 med as the strongest genetic risk factor for restless legs syndrome (odds ratio 1.92, 95% CI 1.85-1.9

21 ies suggested a positive association between restless legs syndrome (RLS) and coronary heart disease

22 Most research on the association between restless legs syndrome (RLS) and depression has involved

23 minergic medications relieve symptoms of the restless legs syndrome (RLS) but have the potential to c

24 Restless legs syndrome (RLS) is a CNS disorder involving

25 Restless legs syndrome (RLS) is a common neurologic cond

26 Restless legs syndrome (RLS) is a common neurological di

27 Although restless legs syndrome (RLS) is a disorder recognized in

28 revious cross-sectional study, we found that restless legs syndrome (RLS) was associated with erectil

29 Restless legs syndrome (RLS), also known as Willis-Ekbom

30 Restless Legs Syndrome (RLS), first chronicled by Willis

31 spinal cord is implicated in the etiology of Restless Legs Syndrome (RLS), which is more prevalent in

32 1 dopaminergic systems in the development of restless legs syndrome (RLS)-like movements during sleep

33 uding 46 drug-naive patients with idiopathic restless legs syndrome (RLS).

34 on deficiency (BID), a pathogenetic model of restless legs syndrome (RLS).

35 ief from dysaesthesias and motor symptoms in restless legs syndrome (RLS).

36 ales (p = 0.007), had a higher proportion of restless legs syndrome (RLS; p < 0.001), had a higher bo

37 ls were isolated from the motor cortex of 11 restless legs syndrome and 14 control brains obtained at

38 entified and replicated 13 new risk loci for restless legs syndrome and confirmed the previously iden

39 small effects have been identified for both restless legs syndrome and narcolepsy with cataplexy.

40 and studied the genetic correlations between restless legs syndrome and traits of interest.

41 clinical characteristics of childhood-onset restless legs syndrome are described.

42 r, a significant percentage of patients with restless legs syndrome are responsive to intravenous iro

43 loss of iron regulatory protein activity in restless legs syndrome brain tissue further implicates t

44 derlying cause of the iron deficiency in the restless legs syndrome brain.

45 erlying the decreased iron concentrations in restless legs syndrome brains is unknown.

46 f relative brain iron deficiency reported in restless legs syndrome brains may underlie the problems

47 romelanin cells from the substantia nigra of restless legs syndrome brains.

48 ns in the iron management protein profile in restless legs syndrome compared with controls at the sit

49 on in the brain is lower in individuals with restless legs syndrome compared with neurologically norm

50 ng of the molecular mechanisms that underlie restless legs syndrome could lead to new treatment optio

51 Restless legs syndrome is a curious neurological disorde

52 Restless legs syndrome is a neurological disorder charac

53 Restless legs syndrome is a prevalent chronic neurologic

54 A family history of restless legs syndrome was present in 23 of 32 (72%) sub

55 in and its receptor in the microvessels from restless legs syndrome was significantly decreased compa

56 controls and 14 individuals who had primary restless legs syndrome was subjected to histochemical st

57 (parkinsonism, dystonia, tremor, chorea, and restless legs syndrome) were included.

58 on's disease, dystonia, Tourette's syndrome, restless legs syndrome, and akathisia, have traditionall

59 nized conditions (motor stereotypy disorder, restless legs syndrome, and infantile masturbation) as w

60 onia, chorea, tics, myoclonus, stereotypies, restless legs syndrome, and various other disorders with

61 id eye movement sleep behavior disorder, and restless legs syndrome, as well as circadian disorders,

62 This issue provides a clinical overview of restless legs syndrome, focusing on diagnosis, treatment

63 medical condition, obstructive sleep apnea, restless legs syndrome, idiopathic insomnia, and circadi

64 For restless legs syndrome, implicated variants are typicall

65 xt of another primary sleep disorder such as restless legs syndrome, or secondary to another underlyi

66 ms associated with brain iron acquisition in restless legs syndrome.

67 n brain iron acquisition in individuals with restless legs syndrome.

68 in the epithelial cells of choroid plexus in restless legs syndrome.

69 or were upregulated in the choroid plexus in restless legs syndrome.

70 sleep disorders center received diagnoses of restless legs syndrome.

71 istory are characteristic of childhood-onset restless legs syndrome.

72 the activity of this protein is decreased in restless legs syndrome; a finding similar to our earlier

73 reathlessness, feeling faint, dizziness, and restless legs, especially among men [for all listed symp

74 particular circadian disorders, narcolepsy, restless-legs syndrome, and OSAS.

75 ss or tiredness, nausea, sweating, and being restless or overactive) did not differ between the group

76 addition, about two-thirds were agitated or restless, or both, and about one-quarter were aggressive

77 ority of the patients (80%) were agitated or restless, or both, with the pain and 26% were aggressive

78 profile was characterized by disinhibition, restless overactivity, a fatuous affect, puerile behavio

79 Recently, restless rapid-eye-movement (REM) sleep emerged as a rob

80 ntation that is specifically associated with restless REM sleep (beta = 0.31, P < 10(-26)).

81 hat was validated to be a specific proxy for restless REM sleep (selective fragmentation: R = 0.57, P

82 in emotion regulation, we hypothesized that restless REM sleep could interfere with the overnight re

83 The findings suggest that restless REM sleep reflects a process that interferes wi

84 In our restless society, such ongoing replacement is a pressing

85 Although it is known that these RESTless tumors are highly aggressive and include all tu

86 sed lymph node involvement observed in human RESTless tumors.

87 ced LIN28A expression when compared with non-RESTless tumors.

88 chemical probes, and possible responses to a restless volcano.

89 are commensurate with monitoring signals at restless volcanoes, thus improving our ability to foreca

90 the last eruption, Yellowstone has remained restless, with high seismicity, continuing uplift/subsid