ライフサイエンスコーパス: restore

1 MnAs structural and magnetic properties are restored.

2 d substrate uptake by these DAT mutants were restored.

3 as suppressed when antioxidant defenses were restored.

4 rature allows the initial locked state to be restored.

5 al to recover if local reductions in CBF are restored.

6 us normal mineral volume is never completely restored.

7 rromagnetism observed in the bulk crystal is restored.

8 ucose intolerance, and suggest approaches to restore 17beta-estradiol levels as a novel treatment opt

9 ticipant, we modified the interface gains to restore a higher level of upper body mobility.

10 r eFSE prevented granule cell dysmaturation, restored a functional balance of gamma-band network osci

11 and 1 endodontically treated and fiber post-restored abutment.

12 rcinoma, Gadd45b inhibition in myeloid cells restored activation of proinflammatory tumor-associated

13 This abnormality was corrected by restoring ADAMTS13 activity in cTTP serum, which prevent

14 vidence that visually mediated behaviors are restored after regeneration, consistent with recovery of

15 Despite the ability of adoptive transfer to restore allergic airways inflammation in ROCK2-insuffici

16 rkinje neuron atrophy, repetitive spiking is restored, although at a greatly reduced firing frequency

17 uced amphetamine-stimulated hyperlocomotion, restored amphetamine-disrupted prepulse inhibition, impr

18 lucose uptake; activation of glucokinase was restored and insulin action was improved, stimulating mu

19 replaced by EDI, the fluorescence of QDs was restored and its intensity was proportional to the EDI c

20 Clinically relevant Ezh2 inhibitors restore androgen receptor expression and sensitivity to

21 onment triggers Treg instability locally and restores anti-tumor immunity.

22 from patients with ALF and might be used to restore antimicrobial responses to patients.

23 ble to suppress T-cell activation and showed restored antimycobacterial activity against Mycobacteriu

24 intact, and transfer of WT platelets did not restore arthritis susceptibility.

25 on), insulin secretion was almost completely restored at elevated glucose concentrations.

26 es to ameliorate pain and inflammation in RA restores autonomic balance and reduces cardiovascular ev

27 Blocking of FDC secretion of IFN-alpha restored B cell tolerance and reduced the amount of GCs

28 requency, increased stem cell polarity and a restored balance of lymphoid and myeloid cells in periph

29 ed HA accumulation, diminished swelling, and restored basal tissue stiffness.

30 Blocking IL-6 or downstream signaling restored Bcl-2/Bcl-xL dependence and may therefore repre

31 percutaneous coronary intervention (PCI) to restore blood flow in an infarct-related coronary artery

32 be reproduced or rescued by knocking out or restoring BMAL1 exclusively in skeletal muscle, respecti

33 r 7(th) rib with adjacent muscle and skin to restore bone defects, internal lining, and external cove

34 depletion of NK1.1(+) cells in IP(-/-) mice restored both the HDM-induced lung inflammation and ILC2

35 ge, suggesting greater potential to preserve/restore breathing capacity.

36 Plasticity onset was restored by a homeoprotein Otx2, which binds the major C

37 hile ovariectomy abolished pLTF, it could be restored by acute systemic E2, or by intraspinal applica

38 However, translocation could be restored by alpha-helical domains in a position- and org

39 This could be restored by blocking inhibitory pathways and, in particu

40 ibited impaired chemotaxis to CCL19 that was restored by exogenous leukotriene C4 .

41 eficit in (18)F-FDG concentration, which was restored by iMAR processing, indicating that metal artif

42 GLUT4 protein expression was only partially restored by increased NAD/P levels.

43 f heterochromatin mutants could be partially restored by inhibiting cep-1/p53, endogenous meiotic dou

44 ent cells but stability can be significantly restored by inhibiting mTORC1 or p70S6 kinase (p70S6K),

45 tenuated by knocking out WNT5A, but could be restored by MMP7 overexpression.

46 We show that the Esx-1 function was restored by nonsense suppression.

47 ssion in these hammerhead ribozymes could be restored by putative t2M/t4M refolding of stem secondary

48 erved in mouse prion disease were completely restored by treatment with benzyl quinolone carboxylic a

49 ursting and coordination during crawling and restoring cacophony selectively in two pairs of cells lo

50 rdiomyocytes represents a potential means of restoring cardiac function following myocardial injury.

51 duced Nef interactions with AP-2 and CD4 and restored CD4 expression to the surface of HIV-infected c

52 ized the DeltadacA mutant in rich medium and restored cefuroxime resistance.

53 e can reverse the process of necroptosis and restore cell viability.

54 Removal of blocking cargo also restores cell morphology and attenuates the ER-stress re

55 Further disruption of the gene for dDia2 restores cells' ability to initiate blebs and thus migra

56 ssion in these cells was sufficient to fully restore chemoresistance under low nutrient conditions.

57 entrosome-targeted Neurl-4 was sufficient to restore ciliogenesis in cells with manipulated daughter

58 We then restore cohesin and monitor the re-formation of each loo

59 y found linked to colistin resistance (dedA) restored colistin susceptibility by reducing the minimum

60 eceiving SRT but not ERT, BH4 deficiency was restored, concomitant with ameliorated cardiac and renal

61 A significant proportion of restored CpGs were associated with phenotypic outcome wi

62 fter lesions in the adult mouse motor cortex restored damaged cortical pathways.

63 l of patients with HCV-CV, DAA-based therapy restored disturbances in peripheral B- and T-cell homeos

64 new strategies for how PKR activity might be restored during infection to limit HCMV disease.

65 anine microdystrophin (cMD1) is effective in restoring dystrophin expression and stabilizing clinical

66 s offers a promising therapeutic approach in restoring dystrophin protein expression.

67 Our results suggest that recovering and restored ecosystems have less abundance, diversity and c

68 BB ligation demonstrates a potent ability to restore effector functions.

69 served master regulator of metabolism, which restores energy balance during metabolic stress both at

70 ous AMPK targets has helped explain how AMPK restores energy homeostasis.

71 ng CXCR3-replete monocytes was sufficient to restore engraftment.

72 Importantly, adoptive transfer of ILC2s restored eosinophil influx and IL-4, IL-5 and IL-13 prod

73 iven DSS protected them from weight loss and restored epithelial proliferation and STAT3 activation,

74 ly wound healing in response to lung injury, restoring epithelial integrity through spreading and mig

75 vates the unfolded protein response (UPR) to restore ER homeostasis.

76 Estrogen addition restored ERK phosphorylation in EVI1-silenced cells, sug

77 Moreover, the ICB approach partially restored expression of several derepressed miR-155 targe

78 nhibitors or knockdown of HDAC1 and/or HDAC2 restored FBP1 expression and inhibited HCC cell growth.

79 Over-expression of Rab1a restores ferritinophagy, suggesting that alpha-syn impai

80 Kidney transplantation (KT) may restore fertility in chronic kidney disease (CKD).

81 virus particles bud from infected cells and restoring filament formation at the infected-cell surfac

82 by spermine and spermidine, which results to restore fluorescence of Tyr on the surfaces of Au NPs.

83 ere asthmatics and PP5 knockdown using siRNA restored fluticasone repressive action on chemokine prod

84 Myocardial active relaxation and restoring forces are known determinants of left ventricu

85 that prevent worsening, reverse damage, and restore function are a major unmet need.

86 ent, designer chimeric BM protein in vivo to restore function of a polymerization-defective laminin,

87 olled prosthetic arms are being developed to restore function to people with upper-limb paralysis.

88 ulate the enteric neuromuscular syncytium to restore function, at the organ level, in a dysmotile gas

89 gral component of resolving inflammation and restoring function of damaged and infected tissues.

90 formed in selected patients can nearly fully restore functional and aesthetic effects in 1 single pro

91 How organs maintain and restore functional integrity during ordinary tissue turn

92 aling in CNS vessels during EAE/MS partially restores functional BBB integrity and limits immune cell

93 anipulations targeting the GABAergic network restored gathering behaviour.

94 tosolic oxidoreductase, could be involved in restoring GC1 basal activity and NO sensitivity because

95 These drugs could restore glial and glymphatic function, enabling efficien

96 rossed onto the mouse Ren1d-null background, restoring granulation in juxtaglomerular cells.

97 Restoring HapR expression in classical biotype V. choler

98 Consistently, restoring high levels of SOX5/6/21 in human primary glio

99 creased T cell exhaustion marker expression, restored HIV-specific CD8 T cell function, and led to de

100 Thus, HDAC inhibition restored HLA class-I surface expression in vitro and in

101 Finally, novel therapies that might restore homeostasis in the GI tract during GVHD are high

102 ancers and TFs that dynamically cooperate to restore homeostasis upon fasting.

103 vates the unfolded protein response (UPR) to restore homeostasis.

104 Expression of EEA1 restored homeostatic synaptic plasticity in Mecp2-defici

105 juvenating factors elaborated by BM ECs that restore HSC function and the immune repertoire in aged m

106 ed peptide (AgRP)) bypasses these signals to restore hunger-like response patterns in insular cortex.

107 helf elevation/reorientation, accompanied by restored hyaluronic acid accumulation in the palatal mes

108 t of the nuclear factor-kappaB pathway while restoring hypothalamic leptin sensitivity.

109 All cloned AHAs could restore immune effector functions to proteolytically gen

110 in TFR patients off-therapy, suggesting the restored immune control observed in MMR and MR(4.5) is n

111 phylaxis and allergic lung inflammation were restored in ADAM10(DC)(-/-) with the overexpression of t

112 -mediated repression at the GR locus, but is restored in advanced prostate cancers upon reversion of

113 ar perfusion often occurs even after flow is restored in an upstream artery.

114 Although AR activity is generally restored in CRPC despite the castrate level of androgens

115 otility and wild-type reversal frequency are restored in double mutants that are defective in both EP

116 is attenuated in vivo, and pathogenicity was restored in either RIPK3- or DAI-deficient mice.

117 survival of the DeltacpsA mutant was largely restored in macrophages missing LAP components (Nox2, Ru

118 and cone-bipolar connectivity patterns were restored in regenerated retinas, suggesting that regener

119 ompared with wild types, which was partially restored in Scn2a+/-; Kcna1-/- mice.

120 DC numbers were restored in the spleen of C57BL/6 and peripheral blood o

121 frame-shifting indel is followed by a frame-restoring indel.

122 represent a promising adjunctive therapy for restoring iodide avidity within the full spectrum from R

123 European Association of Nuclear Medicine to restore its surveys of reported adverse reactions.

124 riginal thickness upon humidity reduction to restore its warmth-preservation function.

125 utoactivity, but lowering the protein levels restored its specific activation response, including ext

126 inhibition of PPM1A activity by sanguinarine restored JNK activation, resulting in increased apoptosi

127 of KLF2 to the carotid bodies in CHF rabbits restored KLF2 expression, and reduced AHI (7 +/- 2/h), R

128 for alternatives to liver transplantation to restore liver function and bridge patients to transplant

129 ter Deadlock to simulans Rhino, by contrast, restores localization to clusters.

130 ble if we have to prevent their collapse and restore lost interactions.

131 keleton remodeling, induced by okadaic acid, restores lymphocyte migration in response to chemokines,

132 deliver active payloads, strategies aimed at restoring lysosomal functionality might overcome resista

133 ces protease transport to lysosomes, thereby restoring lysosomal proteolytic activity.

134 ral complementation with wild-type MDH2 cDNA restored MDH2 levels and mitochondrial MDH activity.

135 lication during flower development partially restored MdMYB39L expression, stamen development, and po

136 small molecules or drugs that can protect or restore mechanosensory hair cells has been hampered by l

137 y Anakinra corrects transcriptional changes, restores MeCP2 levels and spine plasticity and ameliorat

138 n could be a potential therapeutic target to restore memory deficits after TBI.SIGNIFICANCE STATEMENT

139 oth trazodone and dibenzoylmethane treatment restored memory deficits, abrogated development of neuro

140 Enriching HFD with inulin restored microbiota loads, interleukin-22 (IL-22) produc

141 Dietary cyclocreatine tempered autophagy, restored microglial clustering around plaques, and decre

142 tant mESCs with a phosphomutant DGCR8, which restored microRNA levels but did not rescue the exit fro

143 Pharmacological MAPK inhibition restores migration and apoptosis potential in a mouse LC

144 e with BRD4-mediated pathogenesis, either by restoring miR-29b levels via bortezomib treatment or by

145 tes and inducing mitochondrial biogenesis to restore mitochondrial function.

146 Nrf2 increased mitochondrial NADH levels and restored mitochondrial membrane potential in p62-deficie

147 death, and IL-18 secretion, suggesting that restoring mitophagy and inhibiting inflammasome activati

148 y work to correct these neuroadaptations and restore motivation for non-drug rewards.

149 t sites within the central nervous system to restore motor function following spinal cord injury, the

150 e upper limb, a cerebellar-dependent process restoring movement accuracy after introduction of a pert

151 members of the CCR4-Not deadenylase complex, restored mRNA levels for a class of downregulated, H3K36

152 rolonged life span of Tk2-deficient mice and restored mtDNA copy number as well as respiratory chain

153 nase inhibitor GNE-7915, either prevented or restored mtDNA damage to control levels.

154 reveal the potential to protect and possibly restore myelin elaborated by existent oligodendrocytes i

155 tiation of ASO treatment after disease onset restored myelination, MNCV, and CMAP almost to levels se

156 te that estrogen replacement might partially restore neurogenesis in human premature infants.SIGNIFIC

157 Restoring neuronal PINK1 function strikingly reduces amy

158 pression in CD56(dim) NK cells and partially restored NK cell cytotoxic function.

159 ve-modulator, when systemically administered restored NMNAT2 expression in rTg4510 tauopathy mice to

160 itrate therapies, an alternative strategy to restore NO-cGMP signaling is via inorganic nitrite.

161 an reverse this mechanism by recoupling NOS, restoring NO production and reducing oxidative and nitro

162 aneous administration of Cu to these animals restore normal ATP7A levels in these tissues.

163 AnkB variants that fail to restore normal lipid accumulation and GLUT4 localization

164 ation in response to hypercapnic acidosis to restore normal pH and PCO2Tac1-Pet1 axonal boutons were

165 unction following tissue loss is critical to restore normal physiology, yet few cases are documented

166 with Fluoxetine, a 5-HT reuptake inhibitor, restored normal breathing.

167 campus, and blocking IL-1 receptor signaling restored normal synaptic responses and reduced seizure s

168 d alteration of stress responses, ultimately restoring normal ATP production, macromolecule biosynthe

169 rising in the absence of Siw14p are cured by restoring normal levels of the protein.

170 ceiving insulin supplementation that did not restore normoglycemia.

171 gliptin in modulating alpha cell function to restore normoglycemia.

172 in vivo inhibition of STAT6 phosphorylation restores Notch1 expression in HR(+) ETPs, which regain T

173 Conversely, upregulation of Chd1 restores nucleosomal dynamics, promotes normal induction

174 This was restored once steady state was reached.

175 re created to disrupt two genes, and also to restore one gene by another allelic exchange mutation le

176 Resensitization to tamoxifen is restored only by reducing eIF4E expression or mTOR activ

177 of considerable interest for application in restoring organ function.

178 n with wild-type, but not mutagenized, C1qbp restored OXPHOS protein levels and mitochondrial enzyme

179 Attenuation of IP6K restored p53 expression but did not affect the hypermeta

180 Thus, SNAG-mGluR2 restores patterned vision and combinatorial light respon

181 letion of XPA or progerin each significantly restored PCNA at replication forks.

182 rming metabolic therapeutic interventions to restore perceptual abilities clinically.

183 2 inhibitor ruxolitinib in vitro and in vivo restored perforin expression in CD56(dim) NK cells and p

184 Donepezil, a cholinesterase inhibitor, restored performance in animals impaired by continuous s

185 Increased MEL-46 levels also restored perturbed microRNA (miR-2) function in smn-1(lf

186 nous application of bacterial peptidoglycans restored phagocytic uptake in the lysosomal degradation-

187 panin function, pseudorevertant alleles that restored plaque formation for lysis-defective mutants of

188 ddition of the third IN substitution (V165I) restored polyprotein processing, virus particle maturati

189 ific medical conditions hampering efforts to restore population levels.

190 undamentally different management actions to restore populations.

191 usly unknown native moth in experimental and restored populations suggests the potential for restorat

192 swimming preferentially when unstable, thus restoring preferred postures.

193 which reinstates metabolic flexibility with restored preprandial lipid oxidation and postprandial gl

194 of the C-terminal nucleotide binding domain restored proper protein trafficking and cell surface loc

195 oligosaccharide is functionally active, can restore protein binding, and allows activation of cell s

196 proved crucial in glutamine-deprived ECs to restore protein synthesis, suppress ER stress, and react

197 PR) increases ER-protein-folding capacity to restore protein-folding homeostasis.

198 CK2 inhibition restores PTEN nuclear distribution and DNA repair activi

199 nism through which myeloid bone marrow cells restore quiescence of myeloid-biased HSCs, with implicat

200 A non-ribosylatable version of Ras restores reactive oxygen species production and results

201 Furthermore, a selective Mcl-1 inhibitor restores regorafenib sensitivity in CRC cells with intri

202 ommunities [2], but ecological structure was restored relatively rapidly [1].

203 (MI) structural and functional outcomes via restored S1PR1 signaling, and sought to determine mechan

204 Inhibition of DDR restored satellite cell differentiation ability.

205 cking the aberrant activity of these targets restore sensitivity to chemotherapy.

206 Interventions to correct these phenotypes restore sensitivity to the mTORC1 signaling pathway and

207 eatment with both AtRALF1 and CML38 proteins restored sensitivity in these mutants.

208 ckdown of isoform 2 in BRAFi resistant cells restored sensitivity to BRAFi compared with controls.

209 Depletion of murine CAFs from PDX restored sensitivity to HT, with a concurrent reduction

210 otein or BRD4 in in vitro and in vivo models restores sensitivity to EGFR TKIs.

211 ctinomycin D, which represses transcription, restores SG assembly suppression and viral yield.

212 Overexpression of TRPM7 in TRPM7(-/-) cells restores SOCE.

213 but should eventually result in therapies to restore some degree of vision to the blind.

214 t amitosis as an unappreciated mechanism for restoring stem cell homeostasis, but one with some assoc

215 ns to restore the SLs, but not the sequence, restored strong miR159-mediated silencing.

216 Regeneration is a process that restores structure and function of tissues damaged by in

217 1-resistant M. tuberculosis was dominant and restored susceptibility to APYS1 to wildtype levels.

218 lasticity as a novel therapeutic strategy to restore synaptic strength.

219 ed mEPSC amplitudes to wild-type levels, and restored synaptic scaling down of mEPSC amplitudes after

220 both cases that incident X-ray fluences then restore the [Fe{H2 B(pz)2 }2 (bipy)] moiety to an electr

221 res, revealing that cytokine signaling could restore the acquisition of effector function in ITK-defi

222 efg1Delta/Delta cph1Delta/Delta mutant could restore the capacity to cause immunopathology during mur

223 thiocyanate, iodide, and bromide efficiently restore the ferric resting state.

224 zes processive repeat synthesis, which could restore the full 100 nucleotides of (T2AG3)n lost from

225 lobulin fragment complex (OPG-Fc) completely restore the function of fast-twitch extensor digitorum l

226 Hyperfusogenic gB could restore the fusion function with PILRalpha when a gD con

227 This role in plants may have evolved to restore the growth of embryos after the accumulation of

228 can be activated by bacterial phosphatase to restore the helical structure, thus contributing to stro

229 rowth; and (6) ethanol-adapted E. coli cells restore the majority of these reduced activities through

230 r rinse and backwash showed effectiveness to restore the membrane permeance for repetitive usage.

231 ansplantation non-cell-autonomous mechanisms restore the numbers of interstitial cells of Cajal that

232 as sufficient to reverse these events and to restore the sensitivity of ribociclib-resistant cells to

233 ther development of carbon offset schemes to restore the sequestration capacity and other ecosystem s

234 while introducing complementary mutations to restore the SLs, but not the sequence, restored strong m

235 ly localized to the inflamed mucosa but also restored the ability of the endothelium to resolve intes

236 C DNA reversed the effects of activation and restored the ability to inhibit inflammation.

237 complemented PF-positive T. denticola strain restored the activation.

238 Depletion of NK cells restored the allergen responsiveness in the lungs and wa

239 liminating the flow in the DFR significantly restored the antibiotic susceptibility.

240 Surprisingly, the presence of S. mutans restored the biofilm-forming ability of C. albicans bcr1

241 R signaling since inhibition of this pathway restored the Cx43 trafficking defect in PRB cells.

242 Accordingly, an exogenous source of alphaKG restored the DNA demethylation cycle by promoting TDG fu

243 Treatment also restored the duration of sensory nerve compound potentia

244 or mice into HFD Bnull recipients completely restored the effect of HFD to induce insulin resistance.

245 rupting mutation of W50R into residues 44-65 restored the immunoreactivity of mAbs whose epitope bein

246 Additionally, calorie restriction partially restored the impaired BAT glucose uptake.

247 GSNO and auxin restored the root hair phenotype of the hairless root ha

248 A mutation in SP restored the structure of vegetative SAMs in ltm sp doub

249 pha or anti-IL-4Ralpha monoclonal antibodies restored the TH1- and TH2-induced epithelial barrier dys

250 GSNO, but not auxin, restored the wild-type root glycome and transcriptome pr

251 ome because a proteasome inhibitor partially restores the alpha1 protein level.

252 tivation induces global DNA hypomethylation, restores the expression of tumor suppressor p15(INK4B) t

253 Exogenous IL-2 restores the phenotypic changes and overall gene-express

254 bona fide CHIs in the Arabidopsis chi mutant restores the seed coat transparent testa phenotype and t

255 itant inhibition of both Hh and MET pathways restores the sensitivity to anti-EGFR drugs, here we aim

256 Importantly, BRCA2 overexpression partially restores the USP21-associated survival defect.

257 to accumulate, a process rapidly reversed by restoring the abundance of NAD(+) Thus, NAD(+) directly

258 ional programs drives B-ALL and suggest that restoring the balance of these pathways might inhibit B-

259 and the JAK2/STAT5 pathway, and it helps in restoring the decreased RANKL/OPG ratio in adult mice.

260 the effects of EGFR inhibition by partially restoring the EGFR-promoted gene expression program, inc

261 survival of the inner matrix detached cells, restoring the normal hollow lumen morphology in Vitamin

262 Furthermore, partly restoring the PGE2 synthesizing capacity in the anterior

263 T-cell responses in HCCs and to find ways to restore their antitumor functions.

264 e of mushroom spines fraction and is able to restore their deficiency in hippocampal neurons obtained

265 ular morphology at the adult stage, but cold restored their beiging characteristics, a phenomenon ter

266 protein to naturally acquire mutations that restore this function.

267 into platelet-specific CLEC-2 knockout mice restored thrombosis.

268 tive transfer of in vitro differentiated MCs restored thrombosis.

269 obiota inhibits CS, but this response can be restored through oral transfer of control gut bacteria t

270 we suppressed the effects of BRAF(V600E) and restored thyroid morphology and hormone synthesis.

271 ution is an active process that functions to restore tissue homeostasis.

272 ve been implicated in repair mechanisms that restore tissue integrity after injury.

273 tial means of preventing self-reactivity and restoring tissue homeostasis.

274 t 270 DAT, was reduced in epileptic mice but restored to naive levels in epileptic mice receiving MGE

275 p-Raptor, and p-S6RP was observed, which was restored to normal by elevating ERK1/2 activity in these

276 While circulating B cells are virtually restored to preinfection levels during the chronic patho

277 gnitive flexibility in abstinent smokers was restored to the level of nonsmokers following stimulatio

278 vely reestablished when GPR88 expression was restored to the striatum.

279 [ATP] by 35%; both [ATP] and [5-InsP7] were restored to WT levels by overexpression of PPIP5K1, and

280 CS disaccharides partially restored total fecal short-chain fatty acids from the le

281 Previous results showed that IL-7 can restore TRAF1 expression in virus-specific CD8 T cells i

282 ting to the transmembrane helix of TatB that restored transport activity to Tat signal peptides with

283 e Ct strains utilize extracellular indole to restore tryptophan levels.

284 ild-type, but not 5-phosphatase-dead, INPP5E restored TZ molecular organization and Smoothened accumu

285 task requiring basic image recognition were restored up to 13 mo following injection.

286 its superhydrophilic surface properties are restored upon hydrolysis.

287 selective NPY2 and NPY5 receptor antagonists restored vascular integrity and limited HSPC mobilizatio

288 l cell response to tumor stiffening may help restore vessel structure, minimize metastasis, and aid i

289 se with reintroduced photoreceptors, thereby restoring vision in patients blinded by retinal degenera

290 s that have already been lost and thereby to restore visual function.

291 o Improve Outcomes in Older People With PAD (RESTORE), was conducted at Northwestern University.

292 T cell proliferation was restored when MDSC were treated with inhibitors of induc

293 firmed the variant DTT-IYG to be the best at restoring wild-type-like properties in prevention of lun

294 ssociated gene expression networks that were restored with ASO treatment, we also identified potentia

295 ssue loss on 7-y clinical performance of ETT restored with fiber posts.

296 not seen with the implant OFF was partially restored with the implant ON in all but 1 participant (P

297 When intelligibility was restored with the insertion of silence gaps, LRTC in the

298 Dkk inhibitors) into pregnant Pax9(+/-) mice restored Wnt signaling and led to the growth and fusion

299 erentiation into coronary smooth muscle, and restores Wt1 activity upon MI.

300 lochaperones (ZMCs) reactivate mutant p53 by restoring zinc binding to zinc-deficient p53 mutants.