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1 /absence of insertion sequences and a type I restriction modification system.
2 ular lactate, and modification of the type I restriction-modification system.
3 and C.BclI, a controller protein of the BclI restriction-modification system.
4  to be the endonuclease component of a novel restriction-modification system.
5 eria, it does not appear to be part of a DNA restriction-modification system.
6 , but it does not appear to be part of a DNA restriction-modification system.
7 ompetitive inhibitor of the bacterial type I restriction/modification system.
8  for a methyltransferase from a model type I restriction/modification system.
9 ases, those whose function lies outside of a restriction/modification system.
10  methyltransferase that is not part of a DNA restriction/modification system.
11 clude blocking phage attachment, CRISPR, and restriction modification systems.
12 at are now termed Type I, II, III and IV DNA restriction-modification systems.
13 CCWGG motifs as a marker of self DNA akin to restriction-modification systems.
14 tions such as those observed with the Type I Restriction-Modification systems.
15 ors, called C proteins, controls a subset of restriction-modification systems.
16 d disease processes, as well as in bacterial restriction-modification systems.
17 strains in nature vary dramatically in their restriction-modification systems.
18 in because of their sensitivity to bacterial restriction-modification systems.
19 15 nucleotides in the TfiTok6A1I and Tsp32IR restriction-modification systems.
20  which in turn, are rarely incorporated into restriction/modification systems.
21 is, cell-surface-associated proteins and DNA restriction/modification systems.
22         VPI-2 encodes a P4-like integrase, a restriction modification system, a Mu phage-like region,
23 ; these include a previously uncharacterized restriction-modification system, a nuclease-helicase com
24 ic DNA in cells carrying the wild-type EcoRI restriction-modification system: (a) binding to EcoRI* s
25    Correspondingly, two loci encoding Type I restriction-modification systems able to change their sp
26                                          The Restriction-modification system AhdI contains two conver
27                      For the hpyV and hpyAIV restriction-modification systems, an in-depth analysis o
28 ound on plasmids, including those encoding a restriction-modification system and arsenic resistance,
29 ned and expressed the ahdIC gene of the AhdI restriction-modification system and have purified the re
30 oth systems are closely related to the PvuII restriction-modification system and share its target spe
31 re are barriers to genetic transfer, such as restriction-modification systems and CRISPR loci, that l
32 may contribute to genetic variability, i.e., restriction-modification systems and integrases.
33 ches in DNA is key for maintaining bacterial restriction/modification systems and gene silencing in h
34 mology to DNA methyltransferases of type III restriction/modification systems and has 40 tetranucleot
35 rast to M. jannaschii, A. fulgidus has fewer restriction-modification systems, and none of its genes
36 lleviation may be a characteristic of Type I restriction-modification systems, and that it can be ach
37                               Phase-variable restriction-modification systems are a feature of a dive
38                                   Type I DNA restriction/modification systems are oligomeric enzymes
39 tes, SfiI displays all of the hallmarks of a restriction-modification system as opposed to a recombin
40      We also identified a number of putative restriction-modification systems, bacteriophage genes an
41 er of MTase genes, presumably with the whole restriction-modification systems, between Bacteria and A
42                           However, if type I restriction-modification systems bind to unmodified targ
43 tal role in shaping bacterial evolution, and restriction-modification systems can modulate this flow.
44            We conclude that PV of a Type IIG restriction-modification system causes changes in site-s
45                                      Type II restriction-modification systems cleave and methylate DN
46                                     The BcgI restriction-modification system consists of two subunits
47                Most bacterial genomes harbor restriction-modification systems, encoding a REase and i
48 striction (R) and methylase (M) genes of the Restriction-Modification system Esp1396I are tightly reg
49 sm is proposed for the evolution of the NaeI restriction-modification system from a topoisomerase/lig
50                                     The BslI restriction-modification system from Bacillus species wa
51 ave isolated and characterized the genes for restriction-modification systems from two species of Sal
52                DNA modification, mediated by restriction-modification systems, functions as an immune
53          The cloning and sequencing of BsoBI restriction-modification system has been described by Ru
54 sporters, cryptic phages, and three types of restriction-modification systems have been identified in
55                                              Restriction-modification systems have been identified in
56                                          Two restriction-modification systems have been previously di
57                                              Restriction-modification systems have to distinguish bet
58 ch systems, which include the CRISPR-Cas and restriction-modification systems, have proven to be inva
59                                   A type IIs restriction-modification system, hpyIIRM, was active in
60 ion efficiencies suggested the presence of a restriction-modification system in F. nucleatum.
61 which recognizes GAATTC and is a member of a restriction-modification system in Rhodobacter sphaeroid
62 i encodes a homologue of an unusual Type IIG restriction-modification system in which the endonucleas
63                 We conclude that the type II restriction-modification systems in H. pylori are highly
64  transformation are numerous strain-specific restriction-modification systems in H. pylori.
65 ng the inner barriers to transformation were restriction-modification systems in M. xanthus, which co
66                             We conclude that restriction-modification systems inhibit the genomic int
67                                    The PvuII restriction-modification system is a type II system, whi
68           Therefore, the thermophilic Tsp45I restriction-modification system is plasmid-borne within
69 ole of facilitated diffusion in this type II restriction-modification system is proposed.
70 ion sequence (IS) elements, that encodes the restriction/modification system LlaI and carries an abor
71 bits low %G+C and encodes proteins of phage, restriction modification systems, mobile elements, and o
72                              However, unlike restriction-modification systems, phage DNA does not app
73 rial genomes (Labrie et al, 2010), including restriction-modification systems (R-M) (Tock & Dryden, 2
74 n identified in bacterial genomes, including restriction-modification systems (R-M), abortive infecti
75                                    The PspGI restriction-modification system recognizes the sequence
76 e systems, in particular toxin-antitoxin and restriction-modification systems, show nonrandom cluster
77 nsists of genetic rearrangements in a Type I restriction-modification system (SpnD39III).
78 ldarius, the mode of GGCC methylation by its restriction-modification system, SuaI, was investigated.
79                                     Type IIB restriction-modification systems, such as BcgI, feature
80     An extensive analysis is included of the restriction-modification systems that are predicted to b
81     Finally, we describe examples of Type II restriction-modification systems that have features in c
82 toxA flanking DNA contained a homologue of a restriction/modification system that was shown to be fun
83 spite the pronounced similarity of the three restriction-modification systems, the flanking sequences
84 known about the distribution and movement of restriction-modification systems themselves.
85 lts support generalization of the concept of restriction-modification system to the concept of self-r
86 g frames with homology to enterotoxin genes, restriction-modification systems, transposases, and seve
87 cificity subunit of hetero-oligomeric type I restriction-modification systems) was significantly high
88 ains of Escherichia coli expressing the SfiI restriction-modification system were transformed with pl
89 nes encoding surface-associated proteins and restriction-modification systems were especially diverse
90                                 These type I restriction-modification systems were originally identif
91 epresents a minimal approach to assembling a restriction-modification system wherein a single DNA rec
92 involvement of PT modifications in a type of restriction-modification system with wide distribution i
93 ided the starting point for the evolution of restriction-modification systems with novel sequence spe

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