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1 in systolic and diastolic diameter (akin to 'restrictive cardiomyopathy').
2 nts with known constrictive pericarditis and restrictive cardiomyopathy.
3 ing comparable to that observed during human restrictive cardiomyopathy.
4 the 58 children with mixed hypertrophic and restrictive cardiomyopathy.
5 sm or with mixed hypertrophic and dilated or restrictive cardiomyopathy.
6 sease should be ongoing in all patients with restrictive cardiomyopathy.
7 TnI) mutation R145W has been associated with restrictive cardiomyopathy.
8 or which mutations have been associated with restrictive cardiomyopathy.
9 icit and possibly reduced cardiac output and restrictive cardiomyopathy.
10 ic pattern of impaired diastolic filling and restrictive cardiomyopathy.
11 differentiate constrictive pericarditis from restrictive cardiomyopathy.
12 cities are markedly reduced in patients with restrictive cardiomyopathy.
13 fferentiating constrictive pericarditis from restrictive cardiomyopathy.
14 fferentiating constrictive pericarditis from restrictive cardiomyopathy.
15 y diagnosed with dilated, arrhythmogenic, or restrictive cardiomyopathies.
17 ilial amyloidosis (11), hemochromatosis (1), restrictive cardiomyopathy and cardiac cirrhosis (1), pr
18 nction between constrictive pericarditis and restrictive cardiomyopathy and may prove to be valuable
19 eak early diastolic velocity is blunted with restrictive cardiomyopathy and preserved with constricti
20 r hypertrophic cardiomyopathy (HCM), 12% for restrictive cardiomyopathy, and 23% for left ventricular
21 tions within this region are associated with restrictive cardiomyopathy, and C-terminal deletion of c
24 although other forms of cardiomyopathy (ie, restrictive cardiomyopathy, arrhythmogenic right ventric
25 on, force transmission disease; hypertrophic-restrictive cardiomyopathies as sarcomeric, force genera
27 trospective study of pediatric patients with restrictive cardiomyopathy diagnosed between April 1994
30 al echocardiographic criteria for idiopathic restrictive cardiomyopathy, mainly dilated atria with no
31 Consistent with the pathophysiology, the restrictive cardiomyopathy mutation, troponin I R192H, a
32 nt number of suspected HFpEF patients have a restrictive cardiomyopathy or chronic pericardial diseas
34 ological factors responsible for causing the restrictive cardiomyopathy phenotype in some of the gene
35 LNC variants among patients with dilated and restrictive cardiomyopathies, pointed to this gene as an
38 ation of constrictive pericarditis (CP) from restrictive cardiomyopathy (RCM) is a complex and often
45 use hypertrophic (HCM), dilated (DCM) and/or restrictive cardiomyopathy (RCM), but disease-causing me
46 anics of constrictive pericarditis (CP) with restrictive cardiomyopathy (RCM), or healthy controls; t
51 on that is seen in patients suffering from a restrictive cardiomyopathy that is associated with the c
53 Heart failure associated with unexplained restrictive cardiomyopathy was the most common clinical
54 5W, A171T, K178E, and R192H) associated with restrictive cardiomyopathy were distinguishable from hyp
55 s with constrictive pericarditis and 44 with restrictive cardiomyopathy were used for developing an a
56 rther activation of PKCalpha caused a lethal restrictive cardiomyopathy with marked interstitial fibr
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