ライフサイエンスコーパス: resveratrol

1 vanillic acid, caffeic acid, epicatechin and resveratrol.

2 s, such as catechin, epicatechin, piceid and resveratrol.

3 removal as PVPP, but with lower affinity to resveratrol.

4 rly soluble natural polyphenols curcumin and resveratrol.

5 enol, is low and negligible when compared to resveratrol.

6 e consensus about the physiological roles of resveratrol.

7 dimension to the physiological mechanism of resveratrol.

8 magnitude smaller than those of curcumin and resveratrol.

9 lsions-based delivery systems to encapsulate resveratrol.

10 underlie the functional benefits mediated by resveratrol.

11 huic acid, (+)-catechin, quercetin and trans-resveratrol.

12 against SIRT1 (SIRT1-ASO), IL-1beta, and/or resveratrol.

13 uding the valuable compounds piceatannol and resveratrol.

14 during SIRT1 overexpression or activation by resveratrol.

15 330-400nm, respectively, for the analysis of resveratrol.

16 AhR3'DT-1, added the prenyl group to C-3' of resveratrol.

17 r growth in combination with the antioxidant resveratrol.

18 important role in the mechanism of action of resveratrol.

19 was determined in complex with its substrate resveratrol (1.89 A), its product vanillin (1.75 A), and

20 Here, we show that resveratrol (10 microM, 48 hr) induces both a cell growt

21 ere randomized to receive a daily capsule of resveratrol, 125 mg or 500 mg, or placebo for 6 months.

22 To determine whether resveratrol, 125 mg/d or 500 mg/d, improves the 6-minute

23 Administration of resveratrol, 125 or 500 mg/d, or placebo once daily.

24 hin (482 mug/g), gallic acid (319 mug/g) and resveratrol (29.8 mug/g) in skin of Ghara Shani, quercet

25 resveratrol, piceatannol, pterostilbene, and resveratrol-3-beta-glucoside (polydatin).

26 the content of stilbene represented by trans resveratrol-3-glucoside was only 18.5-70.5mg/100gdm.

27 However, resveratrol-3-O-sulfate was able to significantly protec

28 This microsomal fraction-derived resveratrol 4-dimethylallyl transferase utilizes 3,3-dim

29 When resveratrol, a pharmacological activator of SIRT1, was d

30 Resveratrol, a phytoalexin found in the skin of red grap

31 Resveratrol, a polyphenol found in various plant sources

32 enefits have been ascribed to consumption of resveratrol, a polyphenol that can be extracted from gra

33 Here we show that resveratrol, a polyphenol, significantly induces PP2A ac

34 Research shows that resveratrol, a sirtuin activator in red wine, improves e

35 process that is considered a key feature of resveratrol action is the activation of the nicotinamide

36 tidiabetic drug metformin or the antioxidant resveratrol activated AMPK signaling and inhibited mTORC

37 In this study, we report that resveratrol acts as a pathway-selective estrogen recepto

38 Maternal resveratrol administration recovers metabolic activity o

39 these data reveal a novel mechanism by which resveratrol alleviates NTHi-induced inflammation in airw

40 sent study is to investigate whether and how resveratrol alters basal inhibitory synaptic transmissio

41 e anti-neuroinflammatory activity of a novel resveratrol analog 4-(E)-{(p-tolylimino)-methylbenzene-1

42 m for the inhibition of Stat3 signaling by a resveratrol analog and suggest that the preferential gro

43 -dimethoxystyryl)phenyl acetate (Cmpd1) is a resveratrol analog that preferentially inhibits glioma,

44 olved the structure of SIRT1 in complex with resveratrol and a 7-amino-4-methylcoumarin (AMC)-contain

45 s suggest that the chemopreventive action of resveratrol and aspirin involves the elimination of tetr

46 between the ripening ratio and C6 compounds, resveratrol and carbonyl compounds.

47 ific to the peptide sequence, while those of resveratrol and curcumin are non-specific in that they s

48 Resveratrol and curcumin bind only to the hydrophobic re

49 pruning including a strong accumulation of E-resveratrol and E-piceatannol during the first six weeks

50 genes, followed by a rapid accumulation of E-resveratrol and E-piceatannol.

51 Furthermore, binding between resveratrol and gliadin increased at higher temperatures

52 ee had also both the highest levels of trans-resveratrol and piceid, and Muscat de Hambourg the highe

53 ble from weeks 0 to 5 did not differ between resveratrol and placebo groups.

54 for encapsulation, protection and release of resveratrol and potentially other bioactive compounds.

55 Resveratrol and quercetin are well-known polyphenolic co

56 Additionally, forced degradation studies of resveratrol and quercetin were established and the metho

57 for routine in vitro and in vivo analysis of resveratrol and quercetin.

58 yroxine preferential binding sites, by using resveratrol and radiolabeled T4 as probes.

59 acity was mostly affected by the presence of resveratrol and rutin, while total polyphenolic content

60 Both resveratrol and salicylate reduced the formation of tetr

61 did not exhibit significant competition for resveratrol and thyroxine preferential binding sites and

62 te the interactions of polyphenols with both resveratrol and thyroxine preferential binding sites, by

63 ation was the highly preferential binding of resveratrol and thyroxine, both characterized by negativ

64 Resveratrol and valproic acid treatment of one of the CS

65 gallate (EGCG), gallic acid, propyl gallate, resveratrol, and alpha-tocopherol) were investigated for

66 The multiple-ring compounds, EGCG, resveratrol, and curcumin, redirect Abeta(17-36) from a

67 BP1) was identified as a potential target of resveratrol, and in vitro binding assay results using re

68 Apigenin, resveratrol, and piceatannol all induced Nrf2 translatio

69 Nrf2, and this can be modulated by apigenin, resveratrol, and piceatannol.

70 bene to be a weaker inhibitor of CYP3A4 than resveratrol, and stronger than dimethoxy-nitrostilbene.

71 ests a potential anti-restenotic modality of resveratrol application suitable for open surgery.

72 F (% ejection fraction <45) was administered resveratrol ( approximately 320 mg/kg per day).

73 lying that compounds such as piceatannol and resveratrol are potentially available in what is now ess

74 c acid), piracetam, quercetin, vitamin D and resveratrol as potential longevity promoting compounds,

75 of a class of reported STACs (represented by resveratrol) as direct SIRT1 activators is under debate

76 ives and stilbenes, as trans-piceatannol and resveratrol, as main secondary metabolites.

77 The Nrf2 inducer resveratrol, as opposed to catalase, reversed oxidative

78 Concordantly, resveratrol attenuated Akt phosphorylation in injured ar

79 In addition, we found that resveratrol blocked endocannabinoid-mediated long-term s

80 e we present a 2.1 A co-crystal structure of resveratrol bound to the active site of TyrRS.

81 Nitrostilbene and resveratrol, but not dimethoxy-nitrostilbene, engage ele

82 , which strongly cautions against the use of resveratrol by pregnant women.

83 ication of catechin, epicatechin, quercetin, resveratrol, caffeic acid, gallic acid, p-coumaric acid,

84 out whether the antidiabetic effects of oral resveratrol can act directly on these tissues.

85 e beneficial cardiovascular effects, whether resveratrol can be used for the treatment and management

86 uation of the oxygen-labeling pattern of the resveratrol-cleaving CCO, NOV2, previously reported to b

87 ry skin anthocyanin and flavonol amount or t-resveratrol concentration in both skins and wines.

88 ol, and in vitro binding assay results using resveratrol-conjugated Sepharose 4B beads confirmed thei

89 A focussed chemical screen revealed that resveratrol could ameliorate dnj-14 mutant phenotypes, a

90 This study showed that resveratrol could be encapsulated within low-energy nano

91 performed to examine the effect of vanillin, resveratrol, curcumin, and epigallocatechin-3-gallate (E

92 ons were optimised to resolve cis- and trans-resveratrol, d4-resveratrol, dienestrol, hexestrol, oxyr

93 ntakes over the previous year, and an ad hoc resveratrol database.

94 Here we show for the first time that resveratrol decreases expression of pro-inflammatory med

95 Thus, the effects of resveratrol depend on its dose and degree of oxidative s

96 Resveratrol did not change KSRP expression, but immunopr

97 ed to resolve cis- and trans-resveratrol, d4-resveratrol, dienestrol, hexestrol, oxyresveratrol, pice

98 rnative technique to determine the amount of resveratrol dietary supplements, as a model for more com

99 se, and is specifically blocked in vivo by a resveratrol-displacing tyrosyl adenylate analogue.

100 In conclusion, low resveratrol doses promoted in vitro muscle regeneration

101 n of combined epigallocatechin-3-gallate and resveratrol (EGCG+RES) increased energy expenditure and

102 id injury model, periadventitial delivery of resveratrol either via Pluronic gel (2-week), or polymer

103 We report that resveratrol elevated cAMP levels by itself and further p

104 the potential of a formulation comprised of resveratrol, ellagic acid, genistein, curcumin and querc

105 Gravid female rats were fed with a resveratrol-enriched diet during gestational days 3-22.5

106 plemented with the SIRT1-activating molecule resveratrol exhibited increased hippocampal SIRT1 activi

107 Resveratrol exhibits a clear amyloid-solubilizing effect

108 ted the association between habitual dietary resveratrol exposure and the development of FS after 3-,

109 Higher habitual dietary resveratrol exposure was associated with lower risk of o

110 Moreover, fecal transplantation from healthy resveratrol-fed donor mice is sufficient to improve gluc

111 samples, the predominant stilbenoid was (E)-resveratrol, followed by (E)-epsilon-viniferin.

112 Hence, we examined the efficacy of resveratrol for counteracting age-related memory and moo

113 Our observations do not support the use of resveratrol for improving glycemic control.

114 optimize the bioavailability of encapsulated resveratrol for potential oral administration.

115 Treatment of MT-COMP mice with aspirin or resveratrol from birth to P28 decreased mutant COMP intr

116 ity of nanoemulsions in sustained release of resveratrol from dialysis bags compared to the unencapsu

117 d apigenin were able to effectively displace resveratrol from its preferential binding site, whereas

118 the thermodynamic parameters suggested that resveratrol-gliadin binding mainly occurs through hydrop

119 curonide, malonyl caffeoylquinic acid, trans-resveratrol glucoside and caffeoylglucaric isomer.

120 rol group vs placebo; P = .12 for the 500-mg resveratrol group vs placebo).

121 rol group vs placebo; P = .96 for the 500-mg resveratrol group vs placebo).

122 or the placebo group (P = .18 for the 125-mg resveratrol group vs placebo; P = .12 for the 500-mg res

123 or the placebo group (P = .07 for the 125-mg resveratrol group vs placebo; P = .96 for the 500-mg res

124 g time were 0.5 (2.3) minutes for the 125-mg resveratrol group, -0.6 (2.1) minutes for the 500-mg res

125 alk distance were 4.6 (8.1) m for the 125-mg resveratrol group, -12.8 (7.5) m for the 500-mg resverat

126 veratrol group, -12.8 (7.5) m for the 500-mg resveratrol group, and -12.3 (7.9) m for the placebo gro

127 rol group, -0.6 (2.1) minutes for the 500-mg resveratrol group, and 0.4 (2.1) minutes for the placebo

128 eta(17-36) aggregation is as follows: EGCG > resveratrol > curcumin > vanillin, consistent with exper

129 Blank formulations without resveratrol had no negative effect on cell viability or

130 The presence of trans-resveratrol has a special relevance at light toasting: 1

131 Finally, we show that resveratrol has anti-inflammatory effects post NTHi infe

132 activated toward some specific substrates by resveratrol has been poorly understood.

133 Resveratrol has been reported to lower glycemia in roden

134 The plant-derived polyphenol resveratrol has been shown to increase memory performanc

135 Resveratrol has long been thought as an interesting ther

136 Although resveratrol has multiple beneficial cardiovascular effec

137 otential biological activity of low doses of resveratrol has not been extensively studied and, thus,

138 rition timing and suggest that metformin and resveratrol have therapeutic potential to prevent PTB.

139 vators (CAY10591: IC50 approximately 10 muM, resveratrol: IC50 approximately 5 muM) or prostacyclin a

140 This binding was further increased by resveratrol, implicating SIRT1 as a feedback inhibitor r

141 Encapsulation of resveratrol improved its chemical stability after exposu

142 Resveratrol improves insulin sensitivity and lowers hepa

143 provides initial evidence that supplementary resveratrol improves memory performance in association w

144 TORE trial found no consistent evidence that resveratrol improves walking performance in patients 65

145 addition to the effects of duodenally acting resveratrol in an acute 3 d HFD-fed model of insulin res

146 methods were valid for the determination of resveratrol in dietary supplements.

147 entation with the anti-inflammatory compound resveratrol in pregnant dams on lipopolysaccharide (LPS)

148 Encapsulation of resveratrol in protein nanoparticles can be used to over

149 A rapid analytical approach for the assay of resveratrol in red wines, based on Paper Spray Mass Spec

150 y also give evidence of a promising role for resveratrol in the prophylaxis and therapy of AD.

151 ignificantly blocked by the SIRT1 activator, resveratrol, in osteoblastic UMR 106-01 cells.

152 Resveratrol increased to 7.19+/-0.07microg/g dry weight

153 variety of biochemical assays, we find that resveratrol indeed acts through the ICRF-187 binding loc

154 sm of resveratrol-induced p53 activation and resveratrol-induced apoptosis by direct targeting of G3B

155 mitochondrial calcium uniport, prevents the resveratrol-induced augmentation in oxidative capacities

156 downstream AMPK pathway partly abolished the resveratrol-induced increase of glucose oxidation.

157 Knockdown of KSRP expression prevented resveratrol-induced mRNA destabilization in human and mu

158 These findings disclose a novel mechanism of resveratrol-induced p53 activation and resveratrol-induc

159 molecular mechanism and direct target(s) of resveratrol-induced p53 activation remain elusive.

160 Depletion of G3BP1 significantly diminishes resveratrol-induced p53 expression and apoptosis.

161 Herein we show that resveratrol ingestion produces taxonomic and predicted f

162 Furthermore, resveratrol inhibits NTHi-induced ERK1/2 phosphorylation

163 induction or treatment with a SIRT1 agonist, resveratrol, inhibits AR-stimulated proliferation.

164 Resveratrol interacted with both proteins, but the bindi

165 Maternal resveratrol intervention protects offspring against high

166 e, we also found that short-term infusion of resveratrol into the duodenum lowered HGP in two other r

167 Resveratrol is a natural compound found in red wine that

168 Resveratrol is a natural phytoalexin synthesized by plan

169 Resveratrol is a natural polyphenol found in various pla

170 Resveratrol is a promising anti-restenotic natural drug

171 Resveratrol is a stilbene, which is one of a group of po

172 Oral administration of resveratrol is able to improve glucose homeostasis in ob

173 Interestingly, resveratrol is chemically similar to ICRF-187, a clinica

174 One suggested molecular target of resveratrol is eukaryotic topoisomerase II (topo II), an

175 ynthesis of prenylated stilbenoids, in which resveratrol is prenylated at its C-4 position to form ar

176 Resveratrol is reported to extend lifespan and provide c

177 sed on this similarity, we hypothesized that resveratrol may antagonize topo II by a similar mechanis

178 Resveratrol may play a protective role against the frail

179 meostasis in obese mice, suggesting that the resveratrol-mediated changes in the gut microbiome may p

180 Resveratrol might be an option for prevention of acute l

181 lity that the tyrosine-like phenolic ring of resveratrol might fit into the active site pocket to eff

182 This suggests that resveratrol might improve the oxidative capacities of ca

183 Resveratrol modifies the lipidomic profile, increases ox

184 ogether, these results provide evidence that resveratrol modulates basal inhibitory synaptic transmis

185 mulsion droplets, thus better protecting the resveratrol molecule.

186 The two NTD-bound resveratrol molecules are principally responsible for pr

187 The structure reveals the presence of three resveratrol molecules, two of which mediate the interact

188 an-3-ols, flavonols, anthocyanins, acids and resveratrol), nitrogen (TAC, TAN, YAN and TAS) and volat

189 avonols were lowest when compared to GBW and resveratrol not was found in sweet wines.

190 Resveratrol nullifies the catalytic activity and redirec

191 In this work, trans-resveratrol, oenin, malvin, catechin, epicatechin, querc

192 Resveratrol oligomers are biologically active polyphenol

193 In order to discover if the resveratrol oligomers can pass the intestinal barrier, t

194 An efficient synthetic route to the resveratrol oligomers quadrangularin A and pallidol is r

195 at the intestinal absorption rate of the two resveratrol oligomers, epsilon-viniferin and hopeaphenol

196 ested effects of periadventitial delivery of resveratrol on all three major pro-restenotic pathologie

197 ith a drink with omega-3s, antioxidants, and resveratrol on Mini-Mental State Examination (MMSE) scor

198 the biochemical effects of both ICRF-187 and resveratrol on the human isoforms of topo II, and reveal

199 ion effects of two polyphenols, curcumin and resveratrol, on the aggregation of islet amyloid polypep

200 Resveratrol, on the other hand, directly binds to G3BP1

201 Moreover, oral treatment with either resveratrol or aspirin, the prodrug of salicylate, repre

202 In contrast, the SIRT1 activator resveratrol or BMS-345541 (inhibitor of IKK) inhibited I

203 emory abilities were chosen and treated with resveratrol or vehicle for four weeks.

204 an M2 phenotype by either interleukin (IL)4, resveratrol, or adiponectin.

205 In the present work, the resveratrol photoreaction products were analyzed by HPLC

206 and quercetin 3-glucoside and the stilbenes resveratrol, piceatannol, astringin and isorhapontin wer

207 lationship with all the polyphenols studied (resveratrol, piceid, tyrosol, gallic, caffeic and feruli

208 ersies in the literature and elucidated that resveratrol plays an important activation role by stabil

209 combination of both measures [total dietary resveratrol plus total urinary resveratrol (TDR+TUR)] wa

210 ted for in the wine samples were found to be resveratrol (polyphenolic non-flavonoid) and rutin (flav

211 Resveratrol potentiated GABAA and GABAB-mediated inhibit

212 deed, when sirtuin 1 activity was rescued by resveratrol pretreatment in EtOH-treated hepatocytes, a

213 Resveratrol pretreatments attenuated cocaine-induced con

214 Taken together, periadventitial delivery of resveratrol produces durable inhibition of all three pro

215 Maternal resveratrol promotes beige adipocyte development in offs

216 Both metformin and resveratrol protected against spontaneous and inflammati

217 utions and structural information concerning resveratrol, pterostilbene, and piceids obtained by MSI.

218 Stilbene phytoalexins, namely resveratrol, pterostilbene, piceids and viniferins play

219 n vitro antioxidant activity of tyrosol (T), resveratrol (R) and their acetylated derivatives (AcD),

220 However, the low bioavailability of resveratrol raises questions about whether the antidiabe

221 Together with previous studies showing that resveratrol reduces beta-amyloid toxicity they also give

222 munoprecipitation experiments indicated that resveratrol reduces the p38 MAPK-related inhibitory KSRP

223 Recent studies have shown that resveratrol regulates dopaminergic transmission and beha

224 he glucoregulatory role of duodenally acting resveratrol required activation of Sirt1 and AMP-activat

225 trocytic cells were pretreated with TIMBD or resveratrol (RES) and then transfected with a plasmid en

226 Resveratrol (RES) has been studied extensively as an ant

227 We previously found that dietary resveratrol (RES) induces beige adipocyte formation in a

228 Resveratrol (RES) is a polyphenol phytoalexin with anti-

229 Resveratrol (RES), a polyphenol found in natural foods,

230 Natural products like resveratrol (RES), and quercetin (QUE) are known free ra

231 Resveratrol resulted in 30% maternal weight loss and imp

232 We investigated the efficacy of resveratrol (RESV) for preventing SE-induced neurodegene

233 udy, we investigated the potential impact of resveratrol (RESV) on EMT and the fibrotic process in cu

234 f this study is to investigate the impact of resveratrol (RESV) on progression of experimental period

235 igand-binding domain (LBD) as a complex with resveratrol revealed a unique perturbation of the coacti

236 ow here that acute intraduodenal infusion of resveratrol reversed a 3 d high fat diet (HFD)-induced r

237 Antioxidants, as resveratrol (RS), may reduce oxidative stress, restore m

238 Resveratrol (RSV) acts either as an antioxidant or a pro

239 Resveratrol (RSV) and SRT1720 (SRT) elicit beneficial me

240 Nanodesign of niosomes containing resveratrol (RSV) was carried out using food-grade surfa

241 oal of this study was to increase stilbenes (resveratrol (RV), pterostilbene (PT) and pinosylvin (PS)

242 In contrast, resveratrol showed a Papp of 11.9x10(-6)cms(-1).

243 Resveratrol shows beneficial effects in inflammation-bas

244 Resveratrol significantly counteracted the ROS generatio

245 the pharmacological activation of SIRT1 with resveratrol significantly reduces motor incoordination o

246 how (14% fat) to distinguish between WSD and resveratrol-specific effects in these animals.

247 Resveratrol, staurosporine, and TNF-alpha significantly

248 However, the rapid metabolism of resveratrol strongly limits its bioavailability.

249 onversions and use pharmacokinetic data from resveratrol studies to demonstrate how confusion between

250 Resveratrol supplement sales exceed $30 million annually

251 type 2 diabetes, 5 wk of twice-daily 500 mg-resveratrol supplementation had no effect on GLP-1 secre

252 Here, we evaluated further the effect of resveratrol supplementation of pregnant mice on offsprin

253 Antioxidant/omega-3/resveratrol supplementation was associated with favorabl

254 These data suggest that a resveratrol-supplemented diet may restore homeostasis of

255 e striata of pups from the dams fed with the resveratrol-supplemented diet.

256 ound to interact directly with NF-kappaB and resveratrol-suppressed IL-1beta and NAM but not SIRT1-AS

257 d genes like senescence-associated proteins, resveratrol synthase, 9s-lipoxygenase, pathogenesis-rela

258 Resveratrol targets the pyruvate dehydrogenase (PDH) com

259 Total dietary resveratrol (TDR) intake was estimated at baseline with

260 total dietary resveratrol plus total urinary resveratrol (TDR+TUR)] was computed with the use of the

261 ethide intermediates to the synthesis of the resveratrol tetramers nepalensinol B and vateriaphenol C

262 amples showed higher concentrations of trans-resveratrol than those observed in juices made from diff

263 The maximum amount of resveratrol that could be dissolved in the oil phase was

264 Natural polyphenols related to resveratrol that have been shown to impact topo II funct

265 d nanoemulsions which were able to transport resveratrol through cell monolayers in characteristicall

266 fed a WSD (36% fat) supplemented with 0.37% resveratrol throughout pregnancy.

267 tudied age-related factors (i.e., rapamycin, resveratrol, TNF-alpha, and staurosporine), quantitative

268 rophosphate as a prenyl donor and prenylates resveratrol to form arachidin-2.

269 ble-blind, randomized clinical trial, called Resveratrol to Improve Outcomes in Older People With PAD

270 lity experiments showed selective binding of resveratrol to the RNA-binding protein KSRP, a central p

271 n, explains the activity restoration role of resveratrol toward some "loose-binding" substrates of SI

272 Resveratrol (trans-3,5,4'-truhydroxystilbene) possesses

273 Resveratrol-treated animals also displayed increased net

274 proved learning, memory and mood function in resveratrol-treated animals but impairments in vehicle-t

275 f ALD or NAFLD further showed that BALB/c or resveratrol-treated mice fed alcohol or a high-fat diet

276 These results provide novel evidence that resveratrol treatment in late middle age is efficacious

277 Resveratrol treatment of mice with established HF also r

278 In the current study, we determined whether resveratrol treatment of mice with established HF could

279 Resveratrol treatment of mice with established HF lessen

280 We evaluated the effects of 5 wk of resveratrol treatment on GLP-1 secretion, gastric emptyi

281 IRT1 overexpression or chemical induction by resveratrol treatment reverses senescence phenotype, and

282 a lack of improvement in ejection fraction, resveratrol treatment significantly increased median sur

283 n, and physical activity, were improved with resveratrol treatment.

284 We found trans-resveratrol (tRES) and hesperetin (HESP), at concentrati

285 Total urinary resveratrol (TUR) was analyzed with the use of liquid ch

286 These results demonstrate that resveratrol use during pregnancy yields improvements in

287 ant industrial compounds (e.g., artemisinin, resveratrol, vanillin).

288 n and SMC de-differentiation were blocked by resveratrol via its inhibition of the Akt-mTOR pathway.

289 rescence spectroscopy studies confirmed that resveratrol was encapsulated in the inner core of the na

290 ze on the chemical stability of encapsulated resveratrol was examined by preparing systems with diffe

291 Resveratrol was identified in guava and surinam cherry b

292 The thermal behavior of resveratrol was investigated by using simultaneous therm

293 In addition, resveratrol was significantly reduced following heat tre

294 clearly demonstrated for p-coumaric acid and resveratrol, which is associated with many health benefi

295 complexing with mitoxantrone, in contrast to resveratrol, which shows the lowest affinity.

296 cient synthesis of higher-order oligomers of resveratrol will facilitate the biological studies neces

297 iments were used to study the interaction of resveratrol with gliadin and zein.

298 Piceatannol, a stilbene analogue to resveratrol with higher antioxidant activity, was firstl

299 on of procyanidin B1, caffeic acid and trans-resveratrol, with higher levels compared to those report

300 we have identified an effective analogue of resveratrol, (Z)3,4,5,4'-trans-tetramethoxystilbene (TMS