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1 ctivating structures including the ascending reticular activating system (ARAS), contributing to neoc
2 running and is associated with the ascending reticular activating system.
3 ive regions, with its peak in the brain stem reticular activating system.
4  located at the confluence of the limbic and reticular activating systems.
5 bservations that behavioral state influences reticular activity.
6 ith OLP from saliva samples of two subtypes (reticular and erosive) of OLP patients and healthy contr
7 f substrates across and into the endoplasmic reticular and mitochondrial membranes of eukaryotes.
8  more precisely the segmental imprint in the reticular and motor basal hindbrain of adult goldfish.
9 lexes at discrete plasmalemmal, sarcoplasmic reticular and myofilament sites, reveals differential ki
10                                        These reticular and vesicular structures are more abundant in
11   Notably, pex3 mutant cells already contain reticular and vesicular structures that harbor key prote
12 opodia to spread along the cell base and are reticular and/or fibrous in character.
13 ways compartment: epithelium (basal region), reticular basement membrane (Rbm) and underlying lamina
14             We assessed airway inflammation, reticular basement membrane (RBM) thickness, airway smoo
15 n BAL and assessed smooth muscle area (SMA), reticular basement membrane (RBM) thickness, and epithel
16 but not IL-13, was associated with increased reticular basement membrane thickness in EB specimens fr
17 modeling using image analysis, together with reticular basement membrane thickness, mucus gland area,
18 n of extracellular matrix, collagen markers, reticular basement membrane thickness, or glandular perc
19 n many cell types by sensing low endoplasmic reticular Ca(2+) levels and then coupling to plasma memb
20  addition, we identify the sarco/endoplasmic reticular Ca(2+)-ATPase 1A and the alpha1S subunit of th
21 sitive HCM, whereas sarcoplasmic endoplasmic reticular calcium ATPase 2 abundance and sarcoplasmic re
22 of genotype, as was sarcoplasmic endoplasmic reticular calcium ATPase 2/phospholamban protein ratio (
23              (45)Ca sarcoplasmic endoplasmic reticular calcium ATPaseuptake assay showed reduced upta
24    Down regulation of dStim, the endoplasmic reticular calcium sensor and a principal component of SO
25 rteriolar endothelial cells, CXCL12-abundant reticular (CAR) cells, and cells of the osteoblastic lin
26 ch as CXC chemokine ligand (CXCL)12-abundant reticular (CAR) cells, stem cell factor-expressing cells
27 at lymphotoxin signaling in the fibroblastic reticular cell (FRC) stromal subset was required for pro
28        However, the mechanisms that regulate reticular cell function are not well understood.
29 and pathological alterations of fibroblastic reticular cell networks in the draining lymph nodes.
30                    Mice lacking fibroblastic reticular cell PDPN or platelet CLEC-2 exhibited signifi
31  lymph nodes, dendritic cells (DCs) maintain reticular cell survival in multiple compartments.
32 in (PDPN) signalling in stromal fibroblastic reticular cells (FRCs) and its modulation by CLEC-2 expr
33                                 Fibroblastic reticular cells (FRCs) are known to inhabit T cell-rich
34                 In lymph nodes, fibroblastic reticular cells (FRCs) form a collagen-based reticular n
35                                 Fibroblastic reticular cells (FRCs) form the cellular scaffold of lym
36                                 Fibroblastic reticular cells (FRCs) in the T cell zone of lymph nodes
37 anized in a rigid 3D network of fibroblastic reticular cells (FRCs) that are a rich cytokine source.
38                 gp38(+) stromal fibroblastic reticular cells (FRCs) that express VEGF are enriched fo
39 LN) stromal cells, particularly fibroblastic reticular cells (FRCs), provide critical structural supp
40                                 Fibroblastic reticular cells (FRCs), through their expression of CC c
41 al expansion and differentiation of marginal reticular cells (MRCs), a population of lymphoid stromal
42 xpressing host cells, including fibroblastic reticular cells and follicular dendritic cells.
43 romal organizers give rise to adult marginal reticular cells and form a dedicated stromal niche for i
44 stromal cells, which include CXCL12-abundant reticular cells and osteoblasts, results in constitutive
45 icroscopy showed that SFTSV colocalized with reticular cells but did not colocalize with dendritic ce
46 ectional crosstalk with MAdCAM-1(+) marginal reticular cells by providing tumor-necrosis factor (TNF)
47 The major target cells of SFTSV appear to be reticular cells in lymphoid tissues of intestine and spl
48 s support lymphocyte function, and targeting reticular cells is a potential strategy for controlling
49  component of the CHT niche, and mature into reticular cells lining and interconnecting sinusoids.
50 critical mediators, and LTbetaR signaling on reticular cells mediated cell survival by modulating pod
51                                 Fibroblastic reticular cells responded rapidly to DST by transcribing
52   Within secondary lymphoid tissues, stromal reticular cells support lymphocyte function, and targeti
53 occurs in the lymph node, where fibroblastic reticular cells support the maintenance of naive T cells
54 rents in the postsynaptic thalamic relay and reticular cells were dramatically elevated, favoring reb
55                                 Fibroblastic reticular cells were flow-sorted at different timepoints
56 s, uncontrolled proliferation of bone marrow reticular cells, and fibrosis of the marrow compartment.
57 including endothelial cells, CXCL12-abundant reticular cells, leptin-receptor-positive stromal cells,
58  immunoregulatory properties of fibroblastic reticular cells, we reviewed the most recent advances in
59 hat gives rise exclusively to adult marginal reticular cells.
60 follicular dendritic cells, and fibroblastic reticular cells.
61 ells, a profile associated with fibroblastic reticular cells.
62                                              Reticular chemistry approach was successfully employed t
63   Here, we demonstrate the successful use of reticular chemistry as an appropriate strategy for the d
64                         We report the use of reticular chemistry for the fabrication of a chemically
65 selective catalysts, based on the premise of reticular chemistry in MOFs and the ability to incorpora
66                 Successful implementation of reticular chemistry using a judiciously designed rigid o
67      This work illustrates two principles of reticular chemistry: first, selectivity for helical over
68 ectivity-based optogenetic tagging, we found reticular circuits to be composed of distinct subnetwork
69 However, when Hh signalling is inhibited the reticular dermis does not respond to epidermal beta-cate
70  to the transition between the papillary and reticular dermis.
71  basaloid cells present in the papillary and reticular dermis.
72 ceous unit but also within the papillary and reticular dermis.
73 adipogenic differentiation in the developing reticular dermis.
74  score was significantly worse in those with reticular drusen (mean score +/- standard deviation [SD,
75 unctional analysis in patients with AMD with reticular drusen (RDR) have focused on photopic sensitiv
76                                              Reticular drusen identified from photographs were confir
77 Current smoking at baseline predicted higher reticular drusen incidence (OR 2.1, 95% CI 1.0-4.5) afte
78 95% CI, 2.1-4.4) were associated with higher reticular drusen incidence.
79             A higher proportion of eyes with reticular drusen located outside versus within the macul
80   Neither total area nor central location of reticular drusen predicted 5-year progression to late AM
81 sured were significantly lower in those with reticular drusen than in those without.
82 ith decreased likelihood of progression from reticular drusen to late AMD (adjusted OR, 0.5; 95% CI,
83 ese risk factors and 5-year progression from reticular drusen to late AMD.
84          The 15-year cumulative incidence of reticular drusen was 4.0% (n = 95).
85 SNP rs10490924, and the 15-year incidence of reticular drusen were analyzed in discrete logistic regr
86                             Of 118 eyes with reticular drusen, 40 (33.9%) developed late AMD over 5 y
87 re associated with greater long-term risk of reticular drusen.
88 ndrial intermembrane space and is mutated in reticular dysgenesis (RD), a rare form of severe combine
89 D and 32 with leaky SCID, Omenn syndrome, or reticular dysgenesis.
90 ied as having leaky SCID, Omenn syndrome, or reticular dysgenesis; and 3% had a history of enzyme rep
91 ot affect TGF-beta target gene expression in reticular fibroblasts, and TGF-beta inhibition does not
92 ation, differentiation and ECM production by reticular fibroblasts.
93 ive cell cluster is centered on a tegmental (reticular) field traversed by fibers of the superior cer
94 ulomotor area from the central mesencephalic reticular formation (cMRF), a region implicated in horiz
95                            The mesencephalic reticular formation (MRF) is formed by the pedunculopont
96 he two major components of the mesencephalic reticular formation (MRF), namely the pedunculopontine a
97 lycinergic fibers ascending from the pontine reticular formation (PRF) of the brainstem evoked fast a
98  characteristic locations within the pontine reticular formation (PRF).
99 ting with the parabrachial nucleus (PBN) and reticular formation (RF), and those interconnecting NST
100  vibrissa-related region of the intermediate reticular formation (vIRt).
101 of startle response neurons of the mammalian reticular formation [4], and studies of this circuit hav
102 lon (pretectum and thalamus), mesencephalon (reticular formation and nucleus ruber), rhombencephalon
103 of Darkschewitsch, mesencephalic and pontine reticular formation and pontine nuclei.
104 estigated the convergence of inputs from the reticular formation and sensory afferents on presynaptic
105 he PCC to the ventral tegmental area/pontine reticular formation and thalamus, in addition to the LC,
106  formed a column of scattered neurons in the reticular formation and were found in the octavolateral
107  projections, several parts of the medullary reticular formation as well as the spinally projecting r
108 he lateral part of the SC and the underlying reticular formation corresponding to locations where rea
109                                          The reticular formation in the brainstem controls motor outp
110 ated by a central amygdala projection to the reticular formation in the brainstem.
111  superior colliculus (SC) and the underlying reticular formation is correlated with the initiation an
112 cingulate cortex (1.6-fold increase) and the reticular formation of the medulla (6.5-fold increase).
113 ells localized in the hindbrain intermediate reticular formation were noncholinergic in nature (nonmo
114  cMRF input by injecting this portion of the reticular formation with anterograde tracers in combinat
115 ncerta (ZI), anterior pretectum, and pontine reticular formation) provides temporally precise and foc
116 cularis, octavolateralis area, parvocellular reticular formation), many of the ASP-immunonegative neu
117 ruber), rhombencephalon (cerebellar nucleus, reticular formation, and inferior olive), and spinal cor
118 AG, the cuneiform nucleus, the mesencephalic reticular formation, and the superior colliculus.
119 al midbrain tegmentum, posterior tuberculum, reticular formation, and viscerosensory lobe.
120 hetized animals by stimulating the brainstem reticular formation, basal forebrain, or thalamus.
121  to a broad network of regions including the reticular formation, basal ganglia, thalamus, posterior
122 em in the vicinity of the raphe nucleus, and reticular formation, hypothalamus, and septum/striatum o
123  neocortex (middle frontal gyrus), brainstem reticular formation, precerebellar nuclei, and the red n
124 ensory afferents and premotor neurons of the reticular formation, where central pattern generator cir
125 deep layers of the lateral SC and underlying reticular formation.
126 us, solitary tract nucleus, motoneurons, and reticular formation.
127  lung occupied by ground glass, ground glass-reticular (GGR), honeycombing, emphysema, and normal lun
128 mphadenopathy (16%), pleural effusion (12%), reticular infiltration (4%), and pericardial effusion (4
129 tic input to thalamocortical relay cells and reticular interneurons and activate intrathalamic circui
130  in the ER but changes its distribution from reticular into punctate spots.
131 a subpopulation of Dbx1-derived intermediate reticular (IRt) neurons are rhythmically active during i
132  The magnitude and phase differences between reticular lamina and basilar membrane vibrations are abs
133 ell into a narrow fluid-filled space between reticular lamina and tectorial membrane.
134 r flow that arises from shearing between the reticular lamina and the tectorial membrane.
135 ng subnanometer vibrations directly from the reticular lamina at the apical ends of outer hair cells
136 s travelling wave vibrates in phase with the reticular lamina at the best frequency, and results in m
137 expected; instead, they actively vibrate the reticular lamina over a broad frequency range.
138 s can generate sufficient force to drive the reticular lamina over all audible frequencies in living
139                        The phase relation of reticular lamina to basilar membrane vibration changes w
140                   The outer hair cell-driven reticular lamina vibration collaboratively interacts wit
141 living mouse cochleae that the sound-induced reticular lamina vibration is substantially larger than
142 ial vibrations of the tectorial membrane and reticular lamina were tuned.
143 band and slow sharply tuned responses of the reticular lamina, but only a slow tuned response of the
144 t the OHC bundle, the tectorial membrane and reticular lamina, to the transverse motion of the basila
145 cell stereocilia, the tectorial membrane and reticular lamina, were sharply tuned in the radial direc
146 t junctions that outline the scala media and reticular lamina.
147 ere rather located in both the papillary and reticular layers of the LP.
148                                         This reticular-limbic pathway may thus function in processing
149  and generate osteoblasts, chondrocytes, and reticular marrow stromal cells, but not adipocytes.
150 CM/adhesion coupling, whereas highly pliable reticular matrices promote adhesion retraction.
151 71(-) infected red blood cell devoid of host reticular matter, a process that normally spans 24 hours
152  the trans side of the plasma or endoplasmic reticular membrane, respectively.
153 s of Parkin did produce abnormal tubular and reticular mitochondria restricted to the motor cell bodi
154 talization to the mitochondria and preserves reticular mitochondrial morphology in response to sI.
155 neonatal rat cardiomyocytes (NRCMs) retain a reticular mitochondrial phenotype after simulated ischem
156 s and T cells, which resulted in an expanded reticular network and enhanced immunity.
157 e vasculature, expansion of the fibroblastic reticular network and maintenance of lymphoid stromal ph
158                            The cardiomyocyte reticular network is the ideal location of sensors for b
159  alveoli into contiguous alveoli, creating a reticular network that leads to death by asphyxiation.
160 reticular cells (FRCs) form a collagen-based reticular network that supports migratory dendritic cell
161   Abnormal subepithelial cells, which formed reticular networks deep to the basal epithelial cells, w
162 s partially resemble the cells that form the reticular networks in organized lymphoid tissues, potent
163                                     Thalamic reticular neurons are inhibitory cells interconnected by
164 thors show that the receptive field sizes of reticular neurons are small enough to provide localized
165  biophysical properties interconnecting some reticular neurons in mice lacking Cx36.
166                        Our results show that reticular neurons in the cat operate over discrete spati
167 ence revealed spinal-projecting Galphat-S-ir reticular neurons in the caudal hindbrain.
168              Characteristically, the largest reticular neurons of the lamprey brainstem (Muller cells
169 rents within postsynaptic thalamic relay and reticular neurons.
170 AA+ ATPases that localize to the endoplasmic reticular/nuclear envelope (ER/NE) lumen.
171 ase resident in the lumen of the endoplasmic reticular/nuclear envelope.
172 r cortex (M1) strongly innervates brain stem reticular nuclei containing whisker premotor neurons, wh
173 interpeduncular nucleus, superior and middle reticular nuclei, magnocellular vestibular nucleus, soli
174  ZI has strong connections between brainstem reticular nuclei, sensory nuclei, and nonspecific thalam
175  by cholinergic neurons in several motor and reticular nuclei.
176 d the rostral parvicellular and intermediate reticular nuclei.
177 us (mRN, rostral hindbrain) and the inferior reticular nucleus (iRN, caudal hindbrain).
178 re input system originating from the lateral reticular nucleus (LRN) can represent sensory event timi
179 LR sends bilateral projections to the middle reticular nucleus (mRN, rostral hindbrain) and the infer
180 d responses to GABA uncaging in the thalamic reticular nucleus (nRT) that is absent in both nm1054 mi
181 y GABA released from neurons of the thalamic reticular nucleus (nRT), acting predominantly via synapt
182 ffect on synaptic inhibition in the thalamic reticular nucleus (nRT).
183  restricted prefrontal areas to the thalamic reticular nucleus (RE), consistent with recent anatomica
184  down-regulation of GABA inhibition from the reticular nucleus (RTN).
185 y two major inhibitory systems: the thalamic reticular nucleus (TRN) and extrathalamic inhibitory (ET
186 tiple single-unit recordings in the thalamic reticular nucleus (TRN) and medial prefrontal cortex (mP
187 s for a specific involvement of the thalamic reticular nucleus (TRN) come from its unique neuronal ch
188 nerally thought that neurons in the thalamic reticular nucleus (TRN) form GABAergic synapses with oth
189                It is known that the thalamic reticular nucleus (TRN) gates sensory information en rou
190                      The inhibitory thalamic reticular nucleus (TRN) is a hub of the attentional syst
191                                 The thalamic reticular nucleus (TRN) is a unique brain structure at t
192                                 The thalamic reticular nucleus (TRN) is hypothesized to regulate thal
193 agation in thalamocortical (TC) and thalamic reticular nucleus (TRN) neurons remains unknown.
194 liminates rebound bursting in model thalamic reticular nucleus (TRN) neurons.
195  show that GABAergic neurons in the thalamic reticular nucleus (TRN) of mice and rats form two types
196           Neurons within the mature thalamic reticular nucleus (TRN) powerfully inhibit ventrobasal (
197 show that local tonic activation of thalamic reticular nucleus (TRN) rapidly induces slow wave activi
198 olinergic transmission in the mouse thalamic reticular nucleus (TRN), a brain structure essential for
199 nergic synaptic transmission in the thalamic reticular nucleus (TRN), a brain structure intimately in
200 hd1 is selectively expressed in the thalamic reticular nucleus (TRN), a group of GABAergic neurons th
201 s (VB) and GABAergic neurons in the thalamic reticular nucleus (TRN).
202 ion, we found neurons of the visual thalamic reticular nucleus (visTRN) to exhibit PFC-dependent chan
203 icity at electrical synapses in the thalamic reticular nucleus - paired burst spiking in coupled neur
204 on evoked responses from inhibitory thalamic reticular nucleus and excitatory tectothalamic terminals
205 which was associated with decreased thalamic reticular nucleus and primary somatosensory cortex activ
206  unreported retinotopic maps in the thalamic reticular nucleus and the substantia nigra.
207 s between inhibitory neurons of the thalamic reticular nucleus are bidirectionally modulated by chang
208 s been proposed that neurons in the thalamic reticular nucleus are interconnected through GABAergic s
209                                 The thalamic reticular nucleus has a critical role in modulating info
210 address the question of whether cells in the reticular nucleus have receptive fields small enough to
211 ection from the ipsilesional gigantocellular reticular nucleus in response to the injury.
212                                 The thalamic reticular nucleus is an important structure governing th
213 alamocortical neurons and GABAergic thalamic reticular nucleus neurons and that these properties are
214 major synaptic input from GABAergic thalamic reticular nucleus neurons, as well as neurons and astroc
215              In thalamocortical and thalamic reticular nucleus neurons, the site of AP generation and
216  the first 2 weeks after birth, the thalamic reticular nucleus of the mouse lacks intrinsic GABAergic
217                                  Because the reticular nucleus of the thalamus (RtN) provides tonic f
218  The visual sector of the overlying thalamic reticular nucleus receives input from relay cells and su
219 hlight hypothesis proposes that the thalamic reticular nucleus regulates thalamic relay activity thro
220 tions from the visual sector of the thalamic reticular nucleus to the lateral geniculate nucleus comp
221 al nucleus, somatosensory thalamus, thalamic reticular nucleus, and primary somatosensory cortex.
222 ers of the superior colliculus, the thalamic reticular nucleus, and the caudate nucleus.
223 the cochlear nucleus, and via caudal pontine reticular nucleus, pontine central gray, and MS, reached
224 her FEF connections were with the claustrum, reticular nucleus, zona incerta, lateral posterior and m
225 uence thalamic relay nuclei via the thalamic reticular nucleus.
226  by driving inhibitory cells of the thalamic reticular nucleus.
227 the GABAergic neurons of the rodent thalamic reticular nucleus.
228 ons and precerebellar neurons of the lateral reticular nucleus.
229 ypotheses about the function of the thalamic reticular nucleus.
230 ules (n = 14), followed by cysts (n = 6) and reticular opacities (n = 4).
231                    Consecutive patients with reticular pattern dystrophy of the retina underwent a co
232                             In this study on reticular pattern dystrophy of the retina, SDOCT provide
233 along the interlobular septa, causing a fine reticular pattern on CT images.
234  bands of yellowish-white material forming a reticular pattern, called ribbon pseudodrusen, which wer
235 ethods of detection and only 1 that formed a reticular pattern.
236 yloid deposits were usually distributed in a reticular/pericellular pattern, whereas transthyretin am
237                                       Senile reticular pigmentary change was the predominant peripher
238 tary changes, reticular pseudodrusen, senile reticular pigmentary changes, cobblestone degeneration,
239 ctaval nuclei; the viscerosensory lobes; and reticular populations distributed from trigeminal to vag
240 f the ARMS2 risk allele (P = .4) between the reticular pseudodrusen (homozygous = 20.0%; heterozygous
241  of the CHF risk allele (P = .7) between the reticular pseudodrusen (homozygous = 26.7%; heterozygous
242 en (n = 30) or (2) large soft drusen without reticular pseudodrusen (n = 43).
243                                              Reticular pseudodrusen (RPD) are frequently associated w
244 s were graded for the presence or absence of reticular pseudodrusen (RPD) using a multimodal approach
245 rrelation to visual acuity (VA) in eyes with reticular pseudodrusen (RPD) vs those with drusen withou
246                                  Presence of reticular pseudodrusen (RPD) was assessed by masked grad
247 zed as soft drusen, cuticular drusen, and/or reticular pseudodrusen (RPD).
248                        The ability to detect reticular pseudodrusen (RPD)/subretinal drusenoid deposi
249                                              Reticular pseudodrusen appeared as yellowish round to ov
250                                              Reticular pseudodrusen are a frequent finding in patient
251                                              Reticular pseudodrusen are associated more strongly with
252                                              Reticular pseudodrusen are highly concurrent with AMD an
253                                          The reticular pseudodrusen group was older (median age 87 vs
254                                          The reticular pseudodrusen phenotype was associated with inc
255                                              Reticular pseudodrusen represent subretinal deposits tha
256                                              Reticular pseudodrusen seem to confer an increased risk
257                              The presence of reticular pseudodrusen was an independent risk factor fo
258                                              Reticular pseudodrusen was found in 43 participants (41%
259                                              Reticular pseudodrusen were deposits juxtaposed to photo
260                                              Reticular pseudodrusen were hyporeflective on NIR reflec
261                                              Reticular pseudodrusen were identified frequently in SFD
262                                   Peripheral reticular pseudodrusen were seen in 15%.
263 subjects (n = 73) with the phenotypes of (1) reticular pseudodrusen without large soft drusen (n = 30
264 ation levels (C3d:C3 ratio), the presence of reticular pseudodrusen, and AMD phenotype.
265 sen, hypopigmentary/hyperpigmentary changes, reticular pseudodrusen, senile reticular pigmentary chan
266 SupV) and the parvicellular and intermediate reticular regions dorsal to the facial motor nucleus.
267 essive excitatory cortical drive to thalamic reticular (RT) neurons or heightened inhibition of thala
268  Grem1 expression also identifies intestinal reticular stem cells (iRSCs) that are cells of origin fo
269 r), local caspase-3/7, and local endoplasmic reticular stress-related genes.
270 - common progenitors can give rise to marrow reticular stromal cells and perivascular mesenchymal pro
271 al motif that likely drives the formation of reticular structures in living cells.
272 equired to form three-way junctions found in reticular subdomains of the endoplasmic reticulum.
273  selection, in which the PFC biases thalamic reticular subnetworks to control thalamic sensory gain,
274   Recently, significant progress was made by reticular synthesis of related organic solid-state mater
275  applying the 'node and strut' principles of reticular synthesis to molecular crystals.
276 allization, and there are fewer examples of 'reticular synthesis', in which multiple building blocks
277 etal-organic frameworks (MOFs) are formed by reticular synthesis, which creates strong bonds between
278 gely cortical in origin and suggest that the reticular system contributed, at least in part, to these
279  are largely cortical in origin and that the reticular system contributed, at least in part, to these
280   An acoustic startle cue, which engages the reticular system, suppressed MEP size during power grip
281 tic startle cue, a stimulus that engages the reticular system, suppressed MEP size during power grip
282 hibition we assessed the contribution of the reticular system, which projects to cortical neurons, an
283 naptic linkage between anterogradely labeled reticular terminals and retrogradely labeled medial rect
284  thalamocortical cells when the presynaptic, reticular thalamic (nRT) neurons fired in tonic mode.
285 ortant regulators of [Cl(-)]i Neurons of the reticular thalamic (RT) nucleus express reduced levels o
286 r KCC2 is an important Cl(-) extruder in the reticular thalamic (RT) nucleus, despite this nucleus ha
287  activity of reciprocally coupled inhibitory reticular thalamic cells (nRTs) and quantified cycle-by-
288 Gd) of the medial geniculate body (MGB), the reticular thalamic nucleus and dorsal nucleus of the lat
289 eniculate body, suprageniculate nucleus, and reticular thalamic nucleus, as well as of the inferior c
290 ets them apart from GABAergic neurons of the reticular thalamic nucleus.
291 ons in BGMT and CbMT and with neurons in the reticular thalamic nucleus.
292 men, aged 18 to 69 years, who had at least 1 reticular vein with a minimum length of 10 cm in 1 of th
293                                              Reticular veins are subdermal veins located in the lower
294 ective than with 75% HG alone in eliminating reticular veins from the treated area (95.17% vs 85.40%;
295 sclerotherapy is the treatment of choice for reticular veins in the lower limbs, no consensus has bee
296                                          The reticular veins were measured on images obtained before
297 icacy end point was the disappearance of the reticular veins within 60 days after treatment with scle
298 G was superior to 75% HG alone in sclerosing reticular veins, with no statistical difference for comp
299 nol plus 70% HG or 75% HG alone to eliminate reticular veins.
300 cy and safety of 2 sclerosants used to treat reticular veins: 0.2% polidocanol diluted in 70% hyperto

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