ライフサイエンスコーパス: reticulocyte

1 show for infecting immature red blood cells (reticulocytes).

2 the proerythroblast stage to the enucleated reticulocyte.

3 or kinase during hemoglobin synthesis in the reticulocyte.

4 that normally spans 24 hours for uninfected reticulocytes.

5 blood mononuclear cells and were shown to be reticulocytes.

6 e-stage nucleated erythroblasts to anucleate reticulocytes.

7 ed labeling of in vitro-differentiated human reticulocytes.

8 roblasts differentiate to produce enucleated reticulocytes.

9 egulate the co-ordinated daily production of reticulocytes.

10 erythroid cells undergo enucleation to form reticulocytes.

11 s red blood cell (RBC) volume, especially in reticulocytes.

12 mouse and human cells, with higher levels in reticulocytes.

13 owest in the orthochromatic erythroblast and reticulocytes.

14 mediate erythroblasts and reduced release of reticulocytes.

15 not essential for mitochondrial clearance in reticulocytes.

16 teins distribute between extruded nuclei and reticulocytes.

17 natal life, with transient production of HbF reticulocytes.

18 ndogenous lectin, has been pinpointed in rat reticulocytes.

19 are involved in mitochondrial clearance from reticulocytes.

20 r than a cause of autophagosome formation in reticulocytes.

21 in these progenitors and in marrow-resident reticulocytes.

22 ythropoiesis elevated hemoglobin using fewer reticulocytes.

23 evels of hemoglobin and increased numbers of reticulocytes.

24 ulocytes and produced higher CHCs than in AA reticulocytes.

25 Hematide showed a dose-dependent increase in reticulocytes.

26 concentration, number of RBCs and number of reticulocytes.

27 could explain the preference of P vivax for reticulocytes.

28 al hemoglobin, with decreased leukocytes and reticulocytes.

29 es malaria in humans and exclusively infects reticulocytes.

30 scicularis) normocytes with a preference for reticulocytes.

31 human cells that generates normal enucleated reticulocytes.

32 P. vivax and P. ovale, which develop inside reticulocytes.

33 1 fL and otherwise normal blood cell counts; reticulocytes, 0.98%; stable creatinine, 1.1 mg/dL; calc

34 oximately 74000 small molecules in search of reticulocyte 15-hLO-1 selective inhibitors.

35 actions of three human LOs (platelet 12-hLO, reticulocyte 15-hLO-1, and epithelial 15-hLO-2) with ara

36 ralogs, platelet-type 12-human lipoxygenase, reticulocyte 15-human lipoxygenase type-1, and epithelia

37 Human reticulocyte 15-lipoxygenase (15-hLO-1) and epithelial 1

38 Human reticulocyte 15-lipoxygenase-1 (15-hLO-1) and human plat

39 Human reticulocyte 15-lipoxygenase-1 (15-hLO-1), which catalyz

40 ge of dense RBCs (-2.41 vs -0.08, P < .001); reticulocytes (-4.12 vs -0.46, P < .001); lactate dehydr

41 sis of the catalytic action of RTA on rabbit reticulocyte 80S ribosomes establishes a catalytic effic

42 Plasmodium vivax merozoites only invade reticulocytes, a minor though heterogeneous population o

43 o exosomes may be the mechanism by which the reticulocyte adapts to environmental changes during its

44 We have shown previously that young sickle reticulocytes adhere to resting endothelial cells throug

45 well-described rapid decrease in circulating reticulocytes after birth suggests that they may have a

46 hemical analysis of the hemoglobin-producing reticulocyte (an erythrocyte precursor) revealed that th

47 actile actomyosin ring (CAR) between nascent reticulocyte and nucleus, in a population of enucleating

48 ring and coalescence of lipid rafts between reticulocyte and pyrenocyte, steps which reiterate the c

49 ring, and coalescence of lipid rafts between reticulocyte and pyrenocyte.

50 nsgenic mouse model, effects on steady-state reticulocyte and red blood cell (RBC) levels were limite

51 During 5-fluorouracil-induced anemia, both reticulocyte and red cell formation in DYRK3-/- mice wer

52 x invasion, a narrow preference for immature reticulocytes and a rapid remodeling of the host cell, p

53 t increase the frequencies of micronucleated reticulocytes and erythrocytes in the bone marrow and bl

54 fferentiation, we conducted RNA-seq in human reticulocytes and identified nuclear receptor coactivato

55 s not revealed any differences between these reticulocytes and in vitro-cultured adult reticulocytes

56 These mutants only complete development in reticulocytes and mature into both schizonts and gametoc

57 allel increases in circulating CD71-positive reticulocytes and other markers of young RBCs.

58 asonal pattern of counts of red blood cells, reticulocytes and platelets.

59 ions, volume reduction was exaggerated in SS reticulocytes and produced higher CHCs than in AA reticu

60 rther evidenced by the presence of primitive reticulocytes and pyrenocytes (ejected RBC nuclei) in th

61 ls and mature erythrocytes, as well as fewer reticulocytes and sickle cells, in the peripheral blood

62 for the altered membrane function of normal reticulocytes and sickle red cells.

63 s enucleate by nuclear extrusion, generating reticulocytes and small, nucleated cells with a thin rim

64 lly lost during in vitro maturation of mouse reticulocytes and that it is associated with exosomes, r

65 s in our understanding of these processes in reticulocytes and the role of these processes in erythro

66 concomitant decrease in the total number of reticulocytes and various markers of RBC destruction fol

67 associated with reduced NRF2 levels in HbSS reticulocytes and with decreased glutathione regeneratio

68 od revealed a modest neutrophilia, a loss of reticulocytes, and a massive lymphopenia.

69 mitogen-activated T and B cells, circulating reticulocytes, and all cell lines that we have studied.

70 ist of MIR122 increased blood levels of EPO, reticulocytes, and hemoglobin.

71 Aytl2 mRNA was detected in mouse reticulocytes, and the presence of the product of the hu

72 on between the extruding nucleus and nascent reticulocyte are critical steps in erythroblast enucleat

73 The autophagic vesicles found on reticulocytes are identical to those observed on red cel

74 ts low abundance cell types such as immature reticulocytes as well as high abundance cell types such

75 on width) and quantify similar parameters of reticulocytes as well.

76 he membrane-skeletal junctions are weaker in reticulocytes, as is the attachment of transmembrane pro

77 acteria, human cells and plasmodium-infected reticulocytes, as well as a viral RNA genome.

78 locyte-binding domains efficaciously blocked reticulocyte binding of native PvRBPs, suggesting that t

79 Members of the reticulocyte binding protein homolog (PfRh) family have

80 We recently showed that P. falciparum reticulocyte binding protein homolog 5 (PfRH5), a merozo

81 P. falciparum reticulocyte binding protein homologues (PfRhs) are expr

82 Previously we identified that P. falciparum reticulocyte binding protein-like homologue 4 (PfRh4) bi

83 yte binding antigen family and P. falciparum reticulocyte binding-like families.

84 al component of a ternary complex, including Reticulocyte binding-like Homologous protein 5 (PfRH5) a

85 icate process in which Plasmodium falciparum reticulocyte binding-like homologous protein 5 (PfRH5) i

86 by the erythrocyte binding antigen (eba) and reticulocyte binding-like homologue (Rh) gene families.

87 y, naturally acquired antibodies against the reticulocyte-binding domains efficaciously blocked retic

88 Reticulocyte-binding domains of PvRBP2c/PvRBP1a are targ

89 cyte-binding specificity, and their specific reticulocyte-binding domains were mapped within their N-

90 nvasion can be mediated by the P. falciparum reticulocyte-binding homologue protein 4 (PfRh4) on the

91 -based analysis was performed of the PvRBP reticulocyte-binding properties and binding-inhibitory a

92 y, the Duffy-binding protein (PvDBP) and the reticulocyte-binding protein (PvRBP) families.

93 human malaria parasite Plasmodium falciparum reticulocyte-binding protein homolog 5 (PfRH5) as a targ

94 The Plasmodium falciparum reticulocyte-binding protein homolog 5 (PfRH5) has recen

95 For instance, Plasmodium falciparum reticulocyte-binding protein homolog 5 (PfRH5) is essent

96 the contribution of the recently discovered Reticulocyte-binding protein Homolog 5 (RH5)-Basigin (BS

97 cies, are two parasite protein families, the reticulocyte-binding protein homologue (RH) and erythroc

98 Plasmodium falciparum reticulocyte-binding protein homologue 2b (PfRh2b) is an

99 Plasmodium falciparum reticulocyte-binding protein homologue 5 (PfRH5) is a bl

100 We show that the full-length P. falciparum reticulocyte-binding protein homologue 5 (PfRH5) is high

101 Members of the reticulocyte-binding protein-like (RBL) family of invasi

102 ticulocytes that is mediated by the P. vivax reticulocyte-binding proteins (PvRBPs) specifically PvRB

103 PvRBP2c and PvRBP1a displayed a distinct reticulocyte-binding specificity, and their specific ret

104 ifted our understanding of the P. falciparum reticulocyte-binding-like family to the level of individ

105 wn to invade and develop in erythrocytes and reticulocytes, but little is known about their infection

106 od-group antigen, which mediates invasion of reticulocytes by Plasmodium vivax.

107 Surprisingly, it was the immature reticulocytes (CD71(+)) that are generally restricted to

108 t were preferentially invaded, whereas older reticulocytes (CD71(-)), principally found in the periph

109 Our results show that the immature reticulocytes (CD71+) targeted by P. vivax invasion are

110 n a coupled transcription/translation rabbit reticulocyte cell-free expression system.

111 Red cell and reticulocyte cellular indices are widely used in the dia

112 the initially biomechanically rigid CD71(+) reticulocytes convert into a highly deformable CD71(-) i

113 resents a newly described mechanism by which reticulocytes could adapt to environmental modifications

114 n of DBP with a small population of immature reticulocytes could explain the preference of P vivax fo

115 Reversion, predicted by baseline reticulocyte count >/=400 x 10(9)/L (P< .001), occurred

116 Abnormal reticulocyte count (P<0.001, c-statistic 0.623) and hemo

117 markers of intravascular hemolysis, such as reticulocyte count (r = 0.44, P = .02).

118 al red cell indices, in particular increased reticulocyte count and decreased hemoglobin concentratio

119 nd-Stage Liver Disease (MELD) to incorporate reticulocyte count and hemoglobin concentration (MELD-re

120 fined as transfusion-dependent anemia with a reticulocyte count of 60 x 10(9) cells/L or less and bon

121 Mean absolute reticulocyte count peaked at day 11 or 15 in each group

122 Abnormal reticulocyte count was also found to predict mortality i

123 versus normal RBC, but the correlation with reticulocyte count was poor, with inter-individual varia

124 ilar in both msk(-/-) and msk(+/+) mice, but reticulocyte count was significantly increased in msk(-/

125 Serum erythropoietin concentration, absolute reticulocyte count, and adverse events.

126 d by transfusion independence, increments in reticulocyte count, and nontransfused hemoglobin.

127 Host ABO blood group, reticulocyte count, and parasitemia were not correlated

128 lobin is lowest in patients with the highest reticulocyte counts and concomitantly shortened RBC life

129 hrocyte and hemoglobin levels with increased reticulocyte counts and elevated plasma erythropoietin c

130 e antioxidant, tempol, resulted in decreased reticulocyte counts and improved erythrocyte survival.

131 y lower erythrocyte and significantly higher reticulocyte counts compared to patients with low biliru

132 Peak reticulocyte counts increased from a median of 10x10(9)

133 nia, lympocytosis, hyperglycemia, and higher reticulocyte counts, along with the activation of pro-in

134 m a significant decrease in serum bilirubin, reticulocyte counts, and serum erythropoietin following

135 ze rbcs in vivo as demonstrated by increased reticulocyte counts, plasma hemoglobin and bilirubin, an

136 al hemoglobin and higher white blood cell or reticulocyte counts, reinforcing the need for early diag

137 or near-normal hemoglobin values and normal reticulocyte counts.

138 arge amounts of undigested Hb remains in the reticulocyte cytoplasm and in vesicles in the parasite.

139 Furthermore, iron uptake studies in hem6 reticulocytes demonstrate defective incorporation of iro

140 Here, using a rabbit-reticulocyte-derived in vitro translation system, we fou

141 ticular, ribosome elimination, a hallmark of reticulocyte differentiation, was defective in Ube2o(-/-

142 n, which normally partition predominantly to reticulocytes, distribute to both nuclei and reticulocyt

143 ata1-DYRK3 mice, in contrast, produced fewer reticulocytes during hemolytic anemia, and pA2gata1-DYRK

144 othesized stromal niche and possibly promote reticulocyte egress to blood.

145 CD71/RNA double staining of reticulocytes enriched from adult peripheral blood revea

146 In addition, reticulocyte-enriched chorea-acanthocytosis red cell fra

147 Urea triggered volume reduction in SS and AA reticulocytes, establishing a higher CHC.

148 ge of 95% of the cord blood erythrocytes and reticulocytes expressed HbA and the adult beta-globin ge

149 in vitro, in humans, they are restricted to reticulocytes expressing both transferrin receptor 1 (Tr

150 phosphatidylserine-exposing erythrocytes are reticulocytes expressing high levels of CD47, a "do-not-

151 ce of increased levels of circulating mature reticulocytes expressing inside-out PS-exposed autophagi

152 sucrose gradients, following synthesis in a reticulocyte extract or as purified proteins.

153 for degradation by the N-end rule pathway in reticulocyte extracts and mouse NIH 3T3 cells and after

154 pe and mutant SOD1 in synchronized cell-free reticulocyte extracts replete with the full complement o

155 mitochondria and ribosomes, which occurs in reticulocytes following nuclear extrusion, depends on au

156 ith elevations of plasma EPO and circulating reticulocytes following single oral dose administration,

157 Invasion assays based on the CD71(+) reticulocyte fraction revealed substantial postinvasion

158 We measured micronuclei frequency in reticulocytes from peripheral blood and bone marrow and

159 quantities of functional mature human adult reticulocytes from peripheral blood CD34(+) cells.

160 oth lymphocytes and immature erythroid cells/reticulocytes from the BM and peripheral blood.

161 ron incorporation into heme was unaltered in reticulocytes from UCP2-deficient mice.

162 se reticulocytes and in vitro-cultured adult reticulocytes functionally or at the molecular level, an

163 proving the preferential infection of young reticulocytes (generally restricted to the bone marrow),

164 RECENT FINDINGS: Functional human reticulocytes have been generated from adult peripheral

165 Functional adult reticulocytes have been made in culture and shown to sur

166 Reticulocyte hemoglobin content fell only in control sub

167 on >11 g/dL, mean corpuscular volume >70 fL, reticulocyte hemoglobin equivalent >25 pg, serum ferriti

168 rrin saturation, transferrin receptor level, reticulocyte hemoglobin level, and mean cell volume) and

169 The P. falciparum protein, reticulocyte homology 4 (PfRH4), is uniquely up-regulate

170 We therefore adapted a cell-free rabbit reticulocyte in vitro transcription-translation system t

171 We found a higher percentage of reticulocytes in adult Klf1(wt/ko) mice and a mild compe

172 reticulocytes, distribute to both nuclei and reticulocytes in an ankyrin-1-deficient murine model of

173 e elimination, remain polarized in Atg7(-/-) reticulocytes in culture.

174 es (BIAbs) also inhibit P. vivax invasion of reticulocytes in vitro.

175 Reticulocytes increased more than 2-fold.

176 of hepcidin, non-transferrin-bound iron, and reticulocyte indexes is being explored in research setti

177 me also is detected in HeLa cells and rabbit reticulocytes, indicating that it exists in vivo.

178 f spleen cells are immature CD71(-)Ter119(+) reticulocytes, indicating that massive erythropoiesis oc

179 Reticulocyte infection is facilitated by P vivax Duffy b

180 evelopment of a protocol that allows sorting reticulocytes into defined developmental stages and a ro

181 The transition of reticulocytes into erythrocytes is accompanied by extens

182 ts in the release of 2 million new enucleate reticulocytes into your circulation and mine each second

183 aps in our understanding of Plasmodium vivax reticulocyte invasion and protective immunity have hampe

184 Plasmodium vivax invasion into human reticulocytes is a complex process.

185 bsence of Atg7, mitochondrial clearance from reticulocytes is diminished but not completely blocked.

186 In human reticulocytes, KCC3a mRNA levels were consistently the h

187 In adult reticulocytes, less than 5% expressed gamma-globin mRNA.

188 ted human malaria parasite, is restricted to reticulocytes, limiting its asexual proliferation.

189 AQP-1 in young reticulocytes localizes to the plasma membrane and also

190 d prey proteins (probes) in cell-free rabbit reticulocyte lysate (RRL) systems.

191 expressed in a mammalian cell lysate, rabbit reticulocyte lysate (RRL), was able to assemble into cap

192 ent in mock and Ric-8A-immunodepleted rabbit reticulocyte lysate (RRL).

193 ver, it did not affect translation in rabbit reticulocyte lysate (RRL).

194 e dimer is dependent upon factors present in reticulocyte lysate and other cytosols.

195 determined using both lens fiber lysate and reticulocyte lysate as sources of ubiquitinating and pro

196 determined using both lens fiber lysate and reticulocyte lysate as sources of ubiquitinating and pro

197 order Q70E/Q162E>Q162E> Q70E=WT betaB2 using reticulocyte lysate as the source of degradation machine

198 , by the endogenous ubiquitinating system in reticulocyte lysate fraction II, and by intact HEK293 ce

199 thase by the native ubiquitinating system of reticulocyte lysate is dependent upon both Hsp70 and the

200 The apo-nNOS activating activity of reticulocyte lysate is retained in a pool of fractions c

201 Furthermore, translation activity in rabbit reticulocyte lysate is strongly inhibited by RNAs exceed

202 An in vitro assay with reticulocyte lysate recapitulated the release of intact

203 pitation of proteins in vitro expressed in a reticulocyte lysate system showed an interaction between

204 lenocysteines can be reconstituted in rabbit reticulocyte lysate translation reactions.

205 tion substrates in fraction II of the rabbit reticulocyte lysate with an efficiency parallel to their

206 70, RPS3, and NF45) were expressed in rabbit reticulocyte lysate, bacteria, and MCF-7 cells.

207 ssa in vitro translation system or in rabbit reticulocyte lysate.

208 e potent inhibitors of translation in rabbit reticulocyte lysate.

209 h the RNA subunit hTR in two systems (rabbit reticulocyte lysates and human cell lines) with respect

210 sufficient to inhibit translation in rabbit reticulocyte lysates and sufficient to inhibit reporter

211 of translational repression, we used rabbit reticulocyte lysates as an in vitro translation system t

212 vitro degradation of a target mRNA in rabbit reticulocyte lysates containing in vitro-translated Vhs.

213 accumulation of heavy polymeric ribosomes in reticulocyte lysates in vitro.

214 0-2 AGG interruptions, both in vitro (rabbit reticulocyte lysates) and in cell culture (HEK-293 cells

215 s degraded moderately in both lens fiber and reticulocyte lysates, alpha A(1-168)-crystallin was resi

216 bition of eRF1 enhanced PRF in yeast, rabbit reticulocyte lysates, and mammalian cells.

217 fused to glutathione S-transferase in rabbit reticulocyte lysates, suggesting a role for the pU(L)34/

218 lation of this mRNA both in cells and rabbit reticulocyte lysates.

219 ranscription, translation and prenylation in reticulocyte lysates.

220 porter gene and assayed their translation in reticulocyte lysates.

221 tallin to degradation in both lens fiber and reticulocyte lysates.

222 by an endogenous methyltransferase in rabbit reticulocyte lysates.

223 degrees C than 30 degrees C, when tested in reticulocyte lysates.

224 Although genetic ablation of Rip3 normalizes reticulocyte maturation and prevents anemia, ROS accumul

225 Secretion of exosomes during reticulocyte maturation is an integral part of the red c

226 o the loss of all the internal compartments, reticulocyte maturation is characterized by an extensive

227 Early reticulocyte maturation is characterized by the selectiv

228 We show that the final stage of reticulocyte maturation occurs by a previously undescrib

229 restingly, the total DARC protein throughout reticulocyte maturation was constant.

230 erminal erythroid differentiation and during reticulocyte maturation was discerned.

231 also critically facilitates erythroblast and reticulocyte maturation, including hemoglobinization, ce

232 n the constitutive expression of DARC during reticulocyte maturation.

233 nistic basis of crucial changes accompanying reticulocyte maturation.

234 and their interactions change during murine reticulocyte maturation.

235 kle erythrocytes to a physiological event in reticulocyte maturation.

236 AQP9 levels rise as reticulocytes mature into erythrocytes and as neonatal p

237 owever, studies of the process whereby human reticulocytes mature to erythrocytes have been hampered

238 During maturation to an erythrocyte, a reticulocyte must eliminate any residual organelles and

239 e found that although glycophorin C sorts to reticulocytes normally, it distributes to nuclei in 4.1R

240 ice leads to severe anemia despite increased reticulocyte numbers.

241 atures of P. vivax, particularly invasion of reticulocytes, occurrence of dormant liver forms of the

242 ment rate and preference of the parasite for reticulocytes on four key outcome measures assessing ane

243 Reticulocyte parameters such as Hb content (CHr or ret-H

244 was significantly higher compared with other reticulocyte populations and erythrocytes.

245 om adult peripheral blood reveals 4 distinct reticulocyte populations: CD71(high)/RNA(high) ( approxi

246 In this article, we analyze the reticulocyte preference of a large number of Indian P. v

247 zonts in circulation correlating with a high reticulocyte preference.

248 solates significantly vary in their level of reticulocyte preference.

249 ature erythrocytes (normocytes), rather than reticulocytes, preferentially form rosetting complexes,

250 In response to hemolytic anemia, however, reticulocyte production increased severalfold due to DYR

251 globin levels (r = 0.241; P = 0.022) and the reticulocyte production index (RPI) (r = 0.280; P = 0.02

252 ed by further stimulating erythropoiesis and reticulocyte production.

253 g a cell-free translation system from rabbit reticulocytes programmed with mRNAs containing different

254 led to absence of lipid raft assembly in the reticulocyte-pyrenocyte border.

255 In contrast, CD71-positive reticulocytes rarely contained Babesia nuclei and failed

256 by increased hematocrit from 23% to 34% and reticulocyte reduction from 61% to 18%, indicating a sig

257 s, parasitemia preceded reticulocytosis, but reticulocytes remained refractory to B. microti.

258 vax for transferrin receptor (CD71)-positive reticulocytes remained unexplained, given the constituti

259 Reticulocyte remodeling has emerged as an important mode

260 We assess the effect of varying the reticulocyte replacement rate and preference of the para

261 ere, we show that mitochondrial clearance in reticulocytes requires the BCL2-related protein NIX (BNI

262 reased parasitemia in mice infected with the reticulocyte-restricted parasite Plasmodium berghei NK65

263 Reticulocyte RNA and functional minigene splicing assays

264 This screen revealed that reticulocytes show high levels of phosphorylated ribosom

265 obin increased by 1.2-1.9 g/dL (P = 0.01) as reticulocytes simultaneously decreased; that is, better

266 ace a large arc in red blood cells (RBCs) by reticulocyte space as compared to surviving mice.

267 ll hemoglobin occupy a small area of RBCs by reticulocyte space, suggesting this approach can be used

268 Several members of the PvRBP family bind reticulocytes, specifically suggesting a role in mediati

269 n also be expanded and differentiated to the reticulocyte stage.

270 proteome undergoes a rapid transition at the reticulocyte stage; however, the mechanisms driving prog

271 sophilic, polychromatic, orthochromatic, and reticulocyte stages to mature erythrocytes.

272 m underlying the preferential infection of a reticulocyte subset by P vivax.

273 ed the urea-stimulated volume decrease in SS reticulocytes, suggesting that the dysfunctional activit

274 ic alterations in P cynomolgi-infected human reticulocytes that are strikingly similar to those obser

275 rentiate efficiently into mature, functional reticulocytes that can be isolated by filtration.

276 ard expression of the WT allele in mRNA from reticulocytes that could be recapitulated in primary ery

277 P. vivax exclusively invades reticulocytes that is mediated by the P. vivax reticuloc

278 Thus, in reticulocytes, the induction of ubiquitinating factors m

279 In reticulocytes, this includes enucleation and the elimina

280 nd dramatic decrease from proerythroblast to reticulocyte; this enabled us to devise a new strategy f

281 associated with a rapid release of PODXL(+) reticulocytes to blood.

282 5'-monoribonucleotides during maturation of reticulocytes to erythrocytes.

283 Adhesion of sickle reticulocytes to HC-treated endothelial cells was decrea

284 Saporin-L1 inhibition of rabbit reticulocyte translation was protected by these inhibito

285 Recombinant 4E-BP1 inhibits capped mRNA reticulocyte translation, which is partially reversed by

286 During maturation of murine reticulocytes, tubulin and cytosolic actin were lost, wh

287 and recombinant DBP to CD71(high)/RNA(high) reticulocytes was significantly higher compared with oth

288 For a given level of parasite preference for reticulocytes we uncover an optimal erythropoietic respo

289 st increases in HbF-containing red cells and reticulocytes were demonstrated by flow cytometry, thoug

290 rd with this, erythroid progenitor cells and reticulocytes were substantially reduced in number in mi

291 In reticulocytes, where UBE2O is highly up-regulated, unass

292 Plasmodium vivax preferentially invades reticulocytes, which are immature red blood cells.

293 ed through an autophagy-related process, and reticulocytes, which completely eliminate their mitochon

294 er switching leads to a surge of E-Tmod41 in reticulocytes, which degrades quickly in the cytosol.

295 the preferential binding of DBP to immature reticulocytes, which is the potential mechanism underlyi

296 from the elevated phosphorylation of 4.1R in reticulocytes, which leads to a decrease in shear resist

297 ng, and ultimately shed their nuclei to form reticulocytes, which then become mature erythrocytes in

298 but poorly understood biologic process, and reticulocytes, which undergo programmed mitochondrial cl

299 roid cell lines and their differentiation to reticulocytes will be possible.

300 Survival in reticulocytes with reduced or absent Hb digestion may im