ライフサイエンスコーパス: reticuloendothelial

1 pattern of marrow replacement and changes in reticuloendothelial activity after enzyme replacement th

2 Depressed reticuloendothelial cell function may have contributed t

3 no mosaic color was seen in regions lacking reticuloendothelial cells (e.g., tumors).

4 ity of focal hepatic lesions with negligible reticuloendothelial cells (eg, metastases).

5 ng impairment in the movement of iron out of reticuloendothelial cells and hepatocytes.

6 these mice shows abundant iron stores within reticuloendothelial cells and hepatocytes.

7 germinal centers of lymphoid tissue, and in reticuloendothelial cells in the spleen.

8 ysosomal storage is present primarily within reticuloendothelial cells such as Kupffer cells and cell

9 heir ability to be recognized and cleared by reticuloendothelial cells.

10 primarily due to rapid clearance by hepatic reticuloendothelial cells.

11 the apoB48R may provide essential lipids to reticuloendothelial cells.

12 ein, which resides on the plasma membrane of reticuloendothelial cells.

13 mately 3.8-kb mRNA is expressed primarily by reticuloendothelial cells: monocytes, macrophages, and e

14 extensive proliferation of inflammatory and reticuloendothelial cells; however, anti-HMGB1 treatment

15 ravenously as a biomarker for normal hepatic reticuloendothelial function and SPECT was repeated.

16 ited increased Fpn1-mediated iron uptake and reticuloendothelial iron overload as young adult mice.

17 oportin 1 (FPN1), lead to autosomal dominant reticuloendothelial iron overload in humans.

18 ring chronic inflammatory states, leading to reticuloendothelial iron sequestration and an associated

19 osomal storage is marked within hepatocytes, reticuloendothelial Kupffer cells, and cells of the sinu

20 An important function of reticuloendothelial macrophages is phagocytosis of senes

21 FPN1 is also abundantly expressed in reticuloendothelial macrophages of the liver, spleen, an

22 The expression of ferroportin1 (FPN1) in reticuloendothelial macrophages supports the hypothesis

23 ption in enterocytes, iron recycling through reticuloendothelial macrophages, and iron release from s

24 Together, our results suggest that murine reticuloendothelial macrophages, but not those in the bo

25 ropoiesis and profound iron sequestration in reticuloendothelial macrophages, duodenum, and other tis

26 creased bacterial burden was detected within reticuloendothelial organs of iNOS(-/)- mice only beyond

27 ysis revealed that the ASB activities in the reticuloendothelial organs of this animal, as well as tw

28 poor enzymatic stability, rapid clearance by reticuloendothelial organs, immunostimulation, and coagu

29 t concentrations of MAC were observed in the reticuloendothelial organs, with a maximum of 6.9 log10

30 ion and then spreads to the spleen and other reticuloendothelial organs.

31 (marrow iron absent) on the basis of visible reticuloendothelial (RE) marrow iron stores.

32 In several LSDs, cells of the reticuloendothelial (RE) system are the primary targets

33 USB was taken up primarily by tissues of the reticuloendothelial (RE) system.

34 ion from the intestine and iron release from reticuloendothelial stores.

35 lation of 64Cu-labeled SCKs in organs of the reticuloendothelial system (RES) (56.0 +/- 7.1 %ID/g and

36 polyethylene glycol (PEG) used to evade the reticuloendothelial system (RES) and anisamide (AA) for

37 orter, is also expressed in the cells of the reticuloendothelial system (RES) and is likely to be inv

38 Presence of reticuloendothelial system (RES) cell iron in the liver

39 patocellular (HC) pattern [63/849 (7.4%)], a reticuloendothelial system (RES) cell pattern [91/849 (1

40 iscrete components of posttransplant hepatic reticuloendothelial system (RES) function-phagocytosis a

41 60)[C(COOH)(2)](10) to possess the first non-reticuloendothelial system (RES) localizing behavior for

42 ly delayed clearance of nanomaterials by the reticuloendothelial system (RES) of mice, a highly desir

43 rial debris may accumulate in tissues of the reticuloendothelial system (RES) serving as an inflammat

44 the Fc-receptors of, cells of the phagocytic reticuloendothelial system (RES) using medronate liposom

45 on up to 1 day, relatively low uptake in the reticuloendothelial system (RES), and near-complete clea

46 umor penetration, and avoid clearance by the reticuloendothelial system (RES).

47 CoF itself had no effect on mesangial or reticuloendothelial system [125I]AHIgG uptake.

48 lity to home to tissues rich in cells of the reticuloendothelial system after intravenous injection i

49 erebroside substrate in cells throughout the reticuloendothelial system and clinical manifestations i

50 nships between various cell types within the reticuloendothelial system and suggesting possible targe

51 gene expression in hepatocytes, the splenic reticuloendothelial system and the bronchiolar epitheliu

52 ivatized phospholipids are able to evade the reticuloendothelial system and thereby remain in circula

53 Drug molecules carried into the reticuloendothelial system are released from SWNTs and e

54 n transfused platelets, inducing a transient reticuloendothelial system blockade by infusions of spec

55 Iron oxide loading of macrophages in the reticuloendothelial system by means of intravenous ferum

56 ide initial cellular entry points within the reticuloendothelial system by which Listeria establishes

57 ls were highest among patients with mixed HC/reticuloendothelial system cell (RES) iron deposition.

58 helial cell and a cytoplasmic compartment of reticuloendothelial system cells.

59 Rapid reticuloendothelial system clearance of QD will require

60 The reticuloendothelial system has a central role in erythro

61 f the resident macrophage populations of the reticuloendothelial system is a key component of the com

62 d that f-SWNT are not retained in any of the reticuloendothelial system organs (liver or spleen) and

63 d biochemical improvements were found in the reticuloendothelial system organs (livers, spleens, and

64 dye molecules without severe accumulation in reticuloendothelial system organs, making them very prom

65 hat may allow interactions with cells of the reticuloendothelial system to be minimized, yet permit s

66 , HFE enables the intestinal crypt cells and reticuloendothelial system to interpret the body's iron

67 d extrinsic factors (eg, the capacity of the reticuloendothelial system to remove defective RBCs).

68 oped a new strategy to temporarily blunt the reticuloendothelial system uptake of nanodrugs, a major

69 ated RGD-PASP-IO nanoparticles and prominent reticuloendothelial system uptake.

70 limits bacterial growth in the organs of the reticuloendothelial system very quickly after infection,

71 nt upon action potentials transmitted to the reticuloendothelial system via the vagus and splenic ner

72 cholinergic antiinflammatory pathway and the reticuloendothelial system was unknown.

73 umans is safe, and cells accumulating in the reticuloendothelial system were detectable on clinical m

74 f body weight) to preload macrophages of the reticuloendothelial system with iron oxide nanoparticles

75 syndromes of childhood are disorders of the reticuloendothelial system with variable clinical manife

76 lost from the circulation, sequester in the reticuloendothelial system, and do not return to circula

77 on of glucocerebroside in macrophages of the reticuloendothelial system, as a consequence of a defici

78 stinal colonization and dissemination to the reticuloendothelial system, as well as lower levels of i

79 the disease is primarily at the level of the reticuloendothelial system, but few virulence factors ha

80 crophages allowing multiplication within the reticuloendothelial system, but this does not preclude t

81 nd undergoes substantial phagocytosis by the reticuloendothelial system, causing a short blood circul

82 f histologic improvement seen throughout the reticuloendothelial system, even in animals that were en

83 injury through the specialized organs of the reticuloendothelial system, including the lungs, liver,

84 d slowly and are largely retained within the reticuloendothelial system, making clinical translation

85 -containing CPPs were rapidly cleared by the reticuloendothelial system, namely Kupffer cells of the

86 Consistent with uptake by the reticuloendothelial system, R2* value increased in the l

87 s iron to be sequestered within cells of the reticuloendothelial system, suppressing erythropoiesis a

88 eostasis including the developing and mature reticuloendothelial system, the duodenum, and the pregna

89 ense systems, particularly the organs of the reticuloendothelial system, to remove phage particles fr

90 Reticuloendothelial system-specific agents improve lesio

91 nspecific extracellular gadolinium chelates, reticuloendothelial system-specific iron oxide particula

92 from the recognition and elimination by the reticuloendothelial system.

93 release of iron from the macrophages of the reticuloendothelial system.

94 bacteria directly within the confines of the reticuloendothelial system.

95 nd provides a signal for cell removal by the reticuloendothelial system.

96 reatment of storage diseases that affect the reticuloendothelial system.

97 is persistent bacterial colonization of the reticuloendothelial system.

98 cal and nonclassical pathways as well as the reticuloendothelial system.

99 that persists within phagocytic cells of the reticuloendothelial system.

100 ablishing persistent infection in the murine reticuloendothelial system.

101 s targeting platelets for destruction by the reticuloendothelial system.

102 uding dendritic cells (DCs) and cells of the reticuloendothelial system.

103 tissue level, and systemic clearance by the reticuloendothelial system.

104 e biological particulate filter known as the reticuloendothelial system.

105 ism, such as the phagocytic functions of the reticuloendothelial system.

106 estration of iron in phagocytic cells of the reticuloendothelial system.

107 autocrine/paracrine mediator in the hepatic reticuloendothelial system.

108 resulting in extravascular hemolysis by the reticuloendothelial system.

109 infection and to clear the inoculum from the reticuloendothelial system.

110 esistance to toxic oxidative products of the reticuloendothelial system.

111 f their rapid uptake from circulation by the reticuloendothelial system.

112 cation of bacteria inside macrophages within reticuloendothelial tissues.

113 events nonselective accumulation of qdots in reticuloendothelial tissues.