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1  from the recognition and elimination by the reticuloendothelial system.
2  is persistent bacterial colonization of the reticuloendothelial system.
3 cal and nonclassical pathways as well as the reticuloendothelial system.
4 that persists within phagocytic cells of the reticuloendothelial system.
5 ablishing persistent infection in the murine reticuloendothelial system.
6 s targeting platelets for destruction by the reticuloendothelial system.
7 uding dendritic cells (DCs) and cells of the reticuloendothelial system.
8 e biological particulate filter known as the reticuloendothelial system.
9 ism, such as the phagocytic functions of the reticuloendothelial system.
10 estration of iron in phagocytic cells of the reticuloendothelial system.
11  autocrine/paracrine mediator in the hepatic reticuloendothelial system.
12 infection and to clear the inoculum from the reticuloendothelial system.
13  tissue level, and systemic clearance by the reticuloendothelial system.
14 esistance to toxic oxidative products of the reticuloendothelial system.
15 f their rapid uptake from circulation by the reticuloendothelial system.
16 bacteria directly within the confines of the reticuloendothelial system.
17 nd provides a signal for cell removal by the reticuloendothelial system.
18 reatment of storage diseases that affect the reticuloendothelial system.
19  resulting in extravascular hemolysis by the reticuloendothelial system.
20  release of iron from the macrophages of the reticuloendothelial system.
21     CoF itself had no effect on mesangial or reticuloendothelial system [125I]AHIgG uptake.
22 lity to home to tissues rich in cells of the reticuloendothelial system after intravenous injection i
23 erebroside substrate in cells throughout the reticuloendothelial system and clinical manifestations i
24 nships between various cell types within the reticuloendothelial system and suggesting possible targe
25  gene expression in hepatocytes, the splenic reticuloendothelial system and the bronchiolar epitheliu
26 ivatized phospholipids are able to evade the reticuloendothelial system and thereby remain in circula
27  lost from the circulation, sequester in the reticuloendothelial system, and do not return to circula
28              Drug molecules carried into the reticuloendothelial system are released from SWNTs and e
29 on of glucocerebroside in macrophages of the reticuloendothelial system, as a consequence of a defici
30 stinal colonization and dissemination to the reticuloendothelial system, as well as lower levels of i
31 n transfused platelets, inducing a transient reticuloendothelial system blockade by infusions of spec
32 the disease is primarily at the level of the reticuloendothelial system, but few virulence factors ha
33 crophages allowing multiplication within the reticuloendothelial system, but this does not preclude t
34     Iron oxide loading of macrophages in the reticuloendothelial system by means of intravenous ferum
35 ide initial cellular entry points within the reticuloendothelial system by which Listeria establishes
36 nd undergoes substantial phagocytosis by the reticuloendothelial system, causing a short blood circul
37 ls were highest among patients with mixed HC/reticuloendothelial system cell (RES) iron deposition.
38 helial cell and a cytoplasmic compartment of reticuloendothelial system cells.
39                                        Rapid reticuloendothelial system clearance of QD will require
40 f histologic improvement seen throughout the reticuloendothelial system, even in animals that were en
41                                          The reticuloendothelial system has a central role in erythro
42 injury through the specialized organs of the reticuloendothelial system, including the lungs, liver,
43 f the resident macrophage populations of the reticuloendothelial system is a key component of the com
44 d slowly and are largely retained within the reticuloendothelial system, making clinical translation
45 -containing CPPs were rapidly cleared by the reticuloendothelial system, namely Kupffer cells of the
46 d that f-SWNT are not retained in any of the reticuloendothelial system organs (liver or spleen) and
47 d biochemical improvements were found in the reticuloendothelial system organs (livers, spleens, and
48 dye molecules without severe accumulation in reticuloendothelial system organs, making them very prom
49                Consistent with uptake by the reticuloendothelial system, R2* value increased in the l
50 lation of 64Cu-labeled SCKs in organs of the reticuloendothelial system (RES) (56.0 +/- 7.1 %ID/g and
51  polyethylene glycol (PEG) used to evade the reticuloendothelial system (RES) and anisamide (AA) for
52 orter, is also expressed in the cells of the reticuloendothelial system (RES) and is likely to be inv
53                                  Presence of reticuloendothelial system (RES) cell iron in the liver
54 patocellular (HC) pattern [63/849 (7.4%)], a reticuloendothelial system (RES) cell pattern [91/849 (1
55 iscrete components of posttransplant hepatic reticuloendothelial system (RES) function-phagocytosis a
56 60)[C(COOH)(2)](10) to possess the first non-reticuloendothelial system (RES) localizing behavior for
57 ly delayed clearance of nanomaterials by the reticuloendothelial system (RES) of mice, a highly desir
58 rial debris may accumulate in tissues of the reticuloendothelial system (RES) serving as an inflammat
59 the Fc-receptors of, cells of the phagocytic reticuloendothelial system (RES) using medronate liposom
60 on up to 1 day, relatively low uptake in the reticuloendothelial system (RES), and near-complete clea
61 umor penetration, and avoid clearance by the reticuloendothelial system (RES).
62                                              Reticuloendothelial system-specific agents improve lesio
63 nspecific extracellular gadolinium chelates, reticuloendothelial system-specific iron oxide particula
64 s iron to be sequestered within cells of the reticuloendothelial system, suppressing erythropoiesis a
65 eostasis including the developing and mature reticuloendothelial system, the duodenum, and the pregna
66 hat may allow interactions with cells of the reticuloendothelial system to be minimized, yet permit s
67 , HFE enables the intestinal crypt cells and reticuloendothelial system to interpret the body's iron
68 d extrinsic factors (eg, the capacity of the reticuloendothelial system to remove defective RBCs).
69 ense systems, particularly the organs of the reticuloendothelial system, to remove phage particles fr
70 oped a new strategy to temporarily blunt the reticuloendothelial system uptake of nanodrugs, a major
71 ated RGD-PASP-IO nanoparticles and prominent reticuloendothelial system uptake.
72 limits bacterial growth in the organs of the reticuloendothelial system very quickly after infection,
73 nt upon action potentials transmitted to the reticuloendothelial system via the vagus and splenic ner
74 cholinergic antiinflammatory pathway and the reticuloendothelial system was unknown.
75 umans is safe, and cells accumulating in the reticuloendothelial system were detectable on clinical m
76 f body weight) to preload macrophages of the reticuloendothelial system with iron oxide nanoparticles
77  syndromes of childhood are disorders of the reticuloendothelial system with variable clinical manife

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