ライフサイエンスコーパス: retinal dehydrogenase

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1 ssed in reporter cells with any one of three retinal dehydrogenases.

2 e retinal used for RA synthesis by cytosolic retinal dehydrogenases.

3 Inhibition of retinal dehydrogenase 1 restored the asthma phenotype of

4 Crbp), retinol dehydrogenase (Dhrs9/eRoldh), retinal dehydrogenase 1-3 (Aldh1a1-3), and cellular RA-b

5 lammatory cytokines and higher expression of retinal dehydrogenase 1.

6 an irreversible reaction mainly catalyzed by retinal dehydrogenase 2 (aldh1a2), whereas atRAL is conv

7 ogenase family 1, subfamily A2 (ALDH1A2) and retinal dehydrogenase activity both in vitro and in a mo

8 acterized aldehyde dehydrogenase (ALDH) with retinal dehydrogenase activity from rat kidney and the c

9 s two previously identified ALDHs with 9-cis-retinal dehydrogenase activity, rat retinal dehydrogenas

10 Retinal dehydrogenases (Aldh1a1, 2, and 3, formerly Rald

11 hesis, we characterized its effects on human retinal dehydrogenase ALDH1A2 in vitro as well as its ef

12 Multiple retinal dehydrogenases also were observed and were inhib

13 LSECs expressed functional retinal dehydrogenases and could convert vitamin A to RA

14 th a TLR1/2 agonist was sufficient to induce retinal dehydrogenases and to confer these DC with the c

15 Retinal dehydrogenase appears to utilize a disordered lo

16 ck most TLR signals, expressed low levels of retinal dehydrogenases (critical enzymes for all-trans r

17 this study suggest that RALDH2 is the major retinal dehydrogenase in the chick choroid and is respon

18 cotransfected with RDH1 and any one of three retinal dehydrogenase isozymes synthesize all-trans-reti

19 uenced from the purified rat liver cytosolic retinal dehydrogenase P1 were used to design oligonucleo

20 min A) via sequential actions of retinol and retinal dehydrogenases produces the active metabolite al

21 th 9-cis-retinal dehydrogenase activity, rat retinal dehydrogenase (RALDH) 1 and RALDH2.

22 e gut promote immune tolerance by expressing retinal dehydrogenase (RALDH), an enzyme that promotes r

23 glandin E2 (PGE2) as a negative regulator of retinal dehydrogenases (RALDH), the enzymes responsible

24 g-resident tissue MOs coexpress TGF-beta and retinal dehydrogenases (RALDH1 and RALDH 2) under steady

25 rough a cascade of metabolic reactions where retinal dehydrogenases (RALDHs) catalyze the terminal re

26 eps, catalyzed by retinol dehydrogenases and retinal dehydrogenases, respectively.

27 Eta-crystallin is a retinal dehydrogenase that has acquired a role as a stru

28 crophages and dendritic cells, which express retinal dehydrogenases that convert vitamin A to its mai

29 act cells, in conjunction with each of three retinal dehydrogenases that recognize 9-cis-retinal (RAL

30 ed amino acid sequence of P1, now designated retinal dehydrogenase type I or RalDH(I), has close simi

31 Retinal dehydrogenase type II (RalDH2) catalyzes this la

32 The crystal structure of retinal dehydrogenase type II cocrystallized with nicoti

33 icroM) for retinal: human ALDH1 (72.2%), rat retinal dehydrogenase, type I (71.5%), bovine retina (72

34 This enzyme has been designated retinal dehydrogenase, type II, RalDH(II).

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