ライフサイエンスコーパス: retinal toxicity

コーパス検索結果 (１語後でソート)

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1 creased ERG response, which is indicative of retinal toxicity.

2 choroidal and retinal NV, and did not cause retinal toxicity.

3 ight exposure exacerbates vigabatrin-induced retinal toxicity.

4 uroprotective agent against rotenone-induced retinal toxicity.

5 aminoglycosides is associated with cases of retinal toxicity.

6 l optic disc edema, and cyclosporine-related retinal toxicity.

7 imal studies need to be performed to examine retinal toxicity and sustained delivery mechanisms.

8 those affected by hydroxychloroquine-induced retinal toxicity and those unaffected.

9 Retinal toxicity as determined by characteristic visual

10 necroses were observed, suggesting isolated retinal toxicity at this concentration of moxifloxacin.

11 g exposure of at least 6 months, 2 developed retinal toxicity (at 11 and 17 months of exposure).

12 based largely on short-term users or severe retinal toxicity (bull's eye maculopathy).

13 gression, preserving vision while minimizing retinal toxicity complications.

14 Dose- related retinal toxicity could occur in pre-treated eyes even wh

15 are no prior reports of erlotinib-associated retinal toxicity despite over a decade of use in oncolog

16 in angiography, and there was no evidence of retinal toxicity due to BDNF treatment.

17 omplete release within 2 weeks and localized retinal toxicity due to high daunorubicin concentration.

18 Both drugs however can cause retinal toxicity eventually leading to irreversible macu

19 The highest AAV-RP2 dose group demonstrated retinal toxicity, highlighting the importance of careful

20 ry has previously documented formate-induced retinal toxicity in a rodent model of methanol intoxicat

21 The incidence of definite HCQ retinal toxicity in patients treated with HCQ at <6.5 mg

22 logic examination were used to determine the retinal toxicity of dasatinib.

23 Retinal toxicity of ISIS 2922 and ISIS 4015, phosphoroth

24 d to suggest immediate or delayed widespread retinal toxicity of IVTK.

25 nflammatory response, and 1 microM caused no retinal toxicity or inflammation.

26 modulator and a drug previously linked with retinal toxicity, paradoxically provided potent neuropro

27 ption of 4.0 to 5.0 mg/kg, the prevalence of retinal toxicity remained less than 2% within the first

28 In rabbits, retinal toxicity resulted when intravitreal injections o

29 topathological evaluation showed no signs of retinal toxicity to anecortave acetate delivery alone or

30 ide (AIP) was used in a rat in vivo model of retinal toxicity to compare the effects of on NMDA-induc

31 hange chromatography and found that in vitro retinal toxicity was also associated with phosphatidylch

32 Manganese retinal toxicity was assessed by comparing full-field, w

33 In this study, retinal toxicity was determined after intravitreal injec

34 Retinal toxicity was observed at high vector doses, high

35 idence of corneal, lenticular, choroidal, or retinal toxicity was observed by histopathologic evaluat

36 and previously linked to a low incidence of retinal toxicity, was unexpectedly found to exert marked

37 Clinical features indicative of retinal toxicity were scored for the 2 groups and were d

38 cal posterior subcapsular cataract and local retinal toxicity with high doses.

39 ychloroquine can initiate the development of retinal toxicity within 1-2 years.

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