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1 chemic by laser occlusion of the main branch retinal veins.
2 etinal hemorrhages, and dilated and tortuous retinal veins.
4 arterial occlusion (5/16), combined central retinal vein and cilioretinal artery occlusion (4/16), a
6 th, elevation of the pressure in the central retinal vein, and impaired perfusion of the neurons as t
7 was used to measure P(O2) separately in the retinal veins, arteries, and capillaries and in the chor
9 and hemodynamic changes in a major temporal retinal vein by laser Doppler, before and after plasmaph
10 SPCA), central retinal artery (CRA), central retinal vein (CRV), peripapillary choroid and sclera, an
11 e vision loss due to blockage of the central retinal vein (CRVO) or a branch retinal vein (BRVO).
14 al retinal artery equivalent (CRAE), central retinal vein equivalent (CRVE), and arteriole-to-venule
15 al retinal artery equivalent (CRAE), central retinal vein equivalent (CRVE), and arteriole-to-venule
16 entral retinal artery equivalent and central retinal vein equivalent from baseline to 1 week (from 17
20 were measured in the major superior temporal retinal vein in each subject by using a laser Doppler in
21 d by a robot micromanipulator, while a major retinal vein near the optic disc was occluded by argon l
22 hile 7 eyes demonstrated obvious findings of retinal vein obstruction (5 with central and 2 with hemi
24 ondary to outflow obstruction from a central retinal vein obstruction appears to be the most common c
25 bstruction (typically central or hemicentral retinal vein obstruction) using en face optical coherenc
27 pathy; (3) laser photocoagulation for branch retinal vein obstruction; (4) diabetic vitrectomy; (5) t
30 tral retinal vein occlusion (CRVO) or branch retinal vein occlusion (BRVO) causes hypoperfusion, high
31 ral retinal vein occlusion (CRVO) and branch retinal vein occlusion (BRVO) complicated by macular ede
32 olume distensibility in patients with branch retinal vein occlusion (BRVO) in comparison with normal
33 natural history of visual outcome in branch retinal vein occlusion (BRVO) is fundamental to its mana
35 optic nerve head (ONH) parameters and branch retinal vein occlusion (BRVO) using spectral domain opti
36 venous crossings are risk factors for branch retinal vein occlusion (BRVO), an eye disease in which c
42 aphy angiography (OCTA) in eyes with central retinal vein occlusion (CRVO) and branch retinal vein oc
43 o review the definition of ischaemic central retinal vein occlusion (CRVO) and stratify the risk of n
46 ents with macular edema secondary to central retinal vein occlusion (CRVO) or hemiretinal vein occlus
47 ble to macular edema associated with central retinal vein occlusion (CRVO) or hemiretinal vein occlus
48 fty-five patients with a nonischemic central retinal vein occlusion (CRVO) who were randomized to rec
49 To review the clinical picture of central retinal vein occlusion (CRVO), with an emphasis on recen
54 etic macular edema (DME), central and branch retinal vein occlusion (CRVO/BRVO), central serous chori
55 eneration (HR = 1.29; 95% CI: 1.08-1.54) and retinal vein occlusion (HR = 3.94; 95% CI: 3.11-4.99).
57 h retinal arterial occlusion (n = 3), branch retinal vein occlusion (n = 1), vitamin A deficiency (n
58 E (RR 19.5), diabetic retinopathy (RR 13.1), retinal vein occlusion (RR 12.9), macular hole (RR 7.7),
59 .07), uveitis (RR, 2.88; 95% CI, 1.50-5.51), retinal vein occlusion (RR, 4.47; 95% CI, 2.56-5.92), or
60 R), retinopathie of prematurity (ROP) or the retinal vein occlusion (RVO) are caused through a hypoxi
62 Results of ocular biometric measurements in retinal vein occlusion (RVO) eyes are still inconclusive
63 the prevalence, pattern and risk factors of retinal vein occlusion (RVO) in an elderly population of
64 the treatment of macular edema secondary to retinal vein occlusion (RVO) in treatment-naive patients
66 ion (AMD), diabetic macular edema (DME), and retinal vein occlusion (RVO) were evaluated by Pearson c
67 lopathy including diabetic retinopathy (DR), retinal vein occlusion (RVO), and neovascular-age relate
68 ents with diabetic retinopathy (DR), AMD and retinal vein occlusion (RVO), and to identify potential
74 ), diabetic macular edema (DME, n = 400), or retinal vein occlusion (RVO, n = 400) acquired with Zeis
75 The Standard of Care vs Corticosteroid for Retinal Vein Occlusion (SCORE) Study showed that intravi
76 the Standard Care versus COrticosteroid for REtinal Vein Occlusion (SCORE)-CRVO trial, CRUISE Study,
77 ercept in Subjects with Macular Edema Due to Retinal Vein Occlusion (TANZANITE) study who received ei
78 nsive retinopathy 0.88%, macular scar 0.37%, retinal vein occlusion 0.50%, macular hole 0.20%, retini
79 s in the Study of COmparative Treatments for REtinal Vein Occlusion 2 (SCORE2) and to compare with co
81 ) in the Study of COmparative Treatments for REtinal Vein Occlusion 2 (SCORE2); evaluate the baseline
83 at 6 and 12 months was 15.0 to 16.5 (central retinal vein occlusion [CRVO]) and 17.4 to 19.1 (branch
84 derived anatomic measurements between branch retinal vein occlusion and central retinal vein occlusio
87 thickness in patients suffering from central retinal vein occlusion and low visual acuity (<0.1) in c
88 sive patient, who developed bilateral branch retinal vein occlusion at 6T, the therapy was discontinu
90 uently developed retinal artery occlusion or retinal vein occlusion compared with patients undergoing
93 and the absence of such a difference in the retinal vein occlusion group could be explained by edema
94 = 0.024); however, the same analysis in the retinal vein occlusion group revealed no significant dif
95 en branch retinal vein occlusion and central retinal vein occlusion groups (all P >/= 0.058); therefo
97 to 28 % of patients presenting with central retinal vein occlusion have a baseline BCVA of less than
98 ged 18 years with macular edema secondary to retinal vein occlusion in the branch vein occlusion (BRA
100 Retinal ischemic damage associated with retinal vein occlusion is exacerbated by fluid extravasa
102 pressure in the cat after acute experimental retinal vein occlusion may define the role of intravascu
104 retinal vein occlusions (both <0.1 per 1000 retinal vein occlusion patients in 2011, 5.6 and 140.2 i
106 2 patients, one each with Eales' disease and retinal vein occlusion the procedure was unsuccessful, n
111 tinal artery occlusion and 463 patients with retinal vein occlusion were enrolled and matched for age
113 Treatment of macular edema secondary to retinal vein occlusion with ranibizumab has been shown t
114 rysm with subretinal hemorrhage (1), central retinal vein occlusion with vitreous hemorrhage (1), and
115 ction (5 with central and 2 with hemicentral retinal vein occlusion), 4 eyes were unremarkable at pre
116 tinal artery occlusion and 463 patients with retinal vein occlusion, 66 (58.4%) and 245 (52.9%) were
117 contributor to macular edema resulting from retinal vein occlusion, also confirmed in multicenter tr
119 , in models of retinal degeneration, central retinal vein occlusion, and oxygen-induced retinopathy.
120 such as proliferative diabetic retinopathy, retinal vein occlusion, and retinal detachment, than in
121 omplication of retinal ischemia in diabetes, retinal vein occlusion, and retinopathy of prematurity.
122 nal diseases including diabetic retinopathy, retinal vein occlusion, and retinopathy of prematurity.
124 ab and steroid implants can be considered in retinal vein occlusion, but trials are awaited to determ
125 ration (nAMD), diabetic macular edema (DME), retinal vein occlusion, choroidal neovascularization (CN
126 ular diseases (e.g., diabetic macular edema, retinal vein occlusion, choroidal neovascularization) ha
127 the amount of neovascularization induced by retinal vein occlusion, corneal growth factor pellets, a
128 cation of diabetes mellitus, prematurity, or retinal vein occlusion, is a major cause of blindness wo
130 g, contralateral PCME, diabetic retinopathy, retinal vein occlusion, macular hole, epiretinal membran
131 , diarrhoea, myocardial infarction, pyrexia, retinal vein occlusion, n=1 each; placebo: vomiting, whi
132 , leakage such as macular edema secondary to retinal vein occlusion, or a combination of proliferatio
133 This suggests that prompt treatment for retinal vein occlusion, particularly BRVO, may be associ
134 agent ranibizumab in diabetic macular edema, retinal vein occlusion, pseudophakic macular edema, and
135 diverse conditions as diabetic retinopathy, retinal vein occlusion, retinopathy of prematurity, exud
136 condary to AMD or Macular Edema Secondary to Retinal Vein Occlusion, Safety Assessment of Intravitrea
137 conditions, including diabetic retinopathy, retinal vein occlusion, sickle cell retinopathy, uveitis
138 te to progression of retinal nonperfusion in retinal vein occlusion, the same is true in patients wit
139 mic diseases including diabetic retinopathy, retinal vein occlusion, uveitis, and HIV-related retinit
141 e patients, 3 with branch and 9 with central retinal vein occlusion, were imaged in 27 sessions with
142 tic neuropathy in contrast to what occurs in retinal vein occlusion, where edema affects all retinal
143 eyes with macular edema secondary to central retinal vein occlusion, which were treated with a dexame
158 patients with diabetic retinal diseases and retinal vein occlusions (both <0.1 per 1000 retinal vein
159 pth (VCD) and both central (CRVO) and branch retinal vein occlusions (BRVO) using optical low coheren
160 sc were made for 13 patients with DE (7 with retinal vein occlusions and 6 with inflammatory optic ne
161 ckings (AVNs) in the retina are the cause of retinal vein occlusions and are also surrogates of cereb
162 ortant implications for the understanding of retinal vein occlusions and of cerebrovascular aging.
164 caused by inflammatory optic neuropathies or retinal vein occlusions on optical coherence tomography
165 CE PATTERN(R) GUIDELINES: New evidence-based Retinal Vein Occlusions Preferred Practice Pattern(R) (P
169 erative diabetic retinopathy), 8.3% to treat retinal vein occlusions, and 12.9% for all other uses.
174 horoidal sinus communicates with the central retinal vein, the choriocapillaris, and with large veins
175 ue plasminogen activator, 3) cannulating the retinal vein transvitreally, or 4) transecting the poste
176 inogen activator (rt-PA), 3) cannulating the retinal vein transvitreally, or 4) transecting the poste
177 ere used to create a vascular overlay of the retinal veins vs the retinal arteries to map the distrib
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