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   1 halene carboxylic acid or N-(4-hydroxyphenyl)retinamide.                                             
     2 tic retinoid, fenretinide N-(4-hydroxyphenyl)retinamide.                                             
     3 tic retinoid, fenretinide N-(4-hydroxyphenyl)retinamide.                                             
  
     5  We explored efficacy of N-(4-hydroxyphenyl) retinamide (4-HPR) and (-)-epigallocatechin-3-gallate (E
     6 e synergistic actions of N-(4-hydroxyphenyl) retinamide (4-HPR) and paclitaxel (PTX) to control the g
  
     8 of breast cancer cells to N-(4-hydroxyphenyl)retinamide (4-HPR) by suppressing nitric oxide productio
     9 etic retinoid fenretinide N-(4-hydroxyphenyl)retinamide (4-HPR) has shown promise for treating breast
  
    11    The synthetic retinoid N-(4-hydroxyphenyl)retinamide (4-HPR) induces apoptosis in a variety of cel
  
  
  
  
    16 etic retinoid, N-(4-hydroxyphenyl)-all-trans-retinamide (4-HPR), on the induction of prostate cancer 
    17 romolar concentrations of N-(4-hydroxyphenyl)retinamide (4-HPR), which downmodulates cyclin D1, induc
    18 acological agents such as N-(4-hydroxyphenyl)retinamide (4-HPR, fenretinide), by treatment with sphin
    19 th the synthetic retinoid N-(4-hydroxyphenyl)retinamide (4-HPR; CON+/-4HPR group; 1,173 )microg of 4-
  
    21 h the cytotoxic retinoid N-(4-hydroxyphenyl) retinamide (4-HPR; fenretinide) may decrease relapse.   
  
    23  not the closely related N-(4-methoxyphenyl) retinamide (4-MPR) could reduce viral RNA levels and tit
    24 Mean +/- SD Cp of 4-HPR, N-(4-methoxyphenyl) retinamide (4-MPR), and retinol after 1 month of 4-HPR w
    25 tabolites of 4-HPR, 4-oxo-N-(4-hydroxyphenyl)retinamide (4-oxo-4-HPR) showed the highest inhibitory e
    26 inamide (4-MPR) and 4-Oxo-N-(4-methoxyphenyl)retinamide (4-oxo-4-MPR) had minimal effects on DES acti
  
  
    29 by the synthetic retinoid N-(4-hydroxyphenyl)retinamide (4HPR) has been documented in vitro in variou
    30    The synthetic retinoid N-(4-hydroxyphenyl)retinamide (4HPR) has been shown to induce apoptosis in 
    31    The synthetic retinoid N-(4-hydroxyphenyl)retinamide (4HPR) has shown potential as a chemopreventi
    32    The synthetic retinoid N-(4-hydroxyphenyl)retinamide (4HPR) is being examined in both chemoprevent
  
  
  
  
  
    38  another biologic agent, N-(4-hydroxyphenyl) retinamide (4HPR, fenretinide) with rituximab against a 
    39 e growth inhibited by RA, N-(4-hydroxyphenyl)retinamide, and 4-oxoretinol, only the 4-oxoretinol is g
  
    41  to its major metabolite, N-[4-methoxyphenyl]retinamide, as well as several other metabolites of 4-HP
    42 ltiplicity of PIN, whereas 4-(hydroxyphenyl) retinamide at 392 or 784 mg/kg diet, did not have an inh
  
  
    45  the synthetic retinoid, N-(4-hydroxyphenyl) retinamide (HPR), induces HL60 cells to undergo apoptosi
  
    47  derivative fenretinide (N-(4-hydroxyphenyl) retinamide; HPR) exerts therapeutic effects in mouse mod
    48 led retinoic acid (RA) or N-(4-hydroxyphenyl)retinamide, indicating that these drugs enhance [3H]reti
  
  
    51 rs activities of novel C-4 heteroaryl 13-cis-retinamides that modulate Mnk-eIF4E and AR signaling are
  
    53    The synthetic retinoid N-(4-hydroxyphenyl)retinamide, which does not bind effectively to RAR-beta,
    54 is-retinoic acid (9cRA) and 4-(hydroxyphenyl)retinamide, which in previous studies have shown promisi
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