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1 halene carboxylic acid or N-(4-hydroxyphenyl)retinamide.
2 tic retinoid, fenretinide N-(4-hydroxyphenyl)retinamide.
3 tic retinoid, fenretinide N-(4-hydroxyphenyl)retinamide.
5 We explored efficacy of N-(4-hydroxyphenyl) retinamide (4-HPR) and (-)-epigallocatechin-3-gallate (E
6 e synergistic actions of N-(4-hydroxyphenyl) retinamide (4-HPR) and paclitaxel (PTX) to control the g
8 of breast cancer cells to N-(4-hydroxyphenyl)retinamide (4-HPR) by suppressing nitric oxide productio
9 etic retinoid fenretinide N-(4-hydroxyphenyl)retinamide (4-HPR) has shown promise for treating breast
11 The synthetic retinoid N-(4-hydroxyphenyl)retinamide (4-HPR) induces apoptosis in a variety of cel
16 etic retinoid, N-(4-hydroxyphenyl)-all-trans-retinamide (4-HPR), on the induction of prostate cancer
17 romolar concentrations of N-(4-hydroxyphenyl)retinamide (4-HPR), which downmodulates cyclin D1, induc
18 acological agents such as N-(4-hydroxyphenyl)retinamide (4-HPR, fenretinide), by treatment with sphin
19 th the synthetic retinoid N-(4-hydroxyphenyl)retinamide (4-HPR; CON+/-4HPR group; 1,173 )microg of 4-
21 h the cytotoxic retinoid N-(4-hydroxyphenyl) retinamide (4-HPR; fenretinide) may decrease relapse.
23 not the closely related N-(4-methoxyphenyl) retinamide (4-MPR) could reduce viral RNA levels and tit
24 Mean +/- SD Cp of 4-HPR, N-(4-methoxyphenyl) retinamide (4-MPR), and retinol after 1 month of 4-HPR w
25 tabolites of 4-HPR, 4-oxo-N-(4-hydroxyphenyl)retinamide (4-oxo-4-HPR) showed the highest inhibitory e
26 inamide (4-MPR) and 4-Oxo-N-(4-methoxyphenyl)retinamide (4-oxo-4-MPR) had minimal effects on DES acti
29 by the synthetic retinoid N-(4-hydroxyphenyl)retinamide (4HPR) has been documented in vitro in variou
30 The synthetic retinoid N-(4-hydroxyphenyl)retinamide (4HPR) has been shown to induce apoptosis in
31 The synthetic retinoid N-(4-hydroxyphenyl)retinamide (4HPR) has shown potential as a chemopreventi
32 The synthetic retinoid N-(4-hydroxyphenyl)retinamide (4HPR) is being examined in both chemoprevent
38 another biologic agent, N-(4-hydroxyphenyl) retinamide (4HPR, fenretinide) with rituximab against a
39 e growth inhibited by RA, N-(4-hydroxyphenyl)retinamide, and 4-oxoretinol, only the 4-oxoretinol is g
41 to its major metabolite, N-[4-methoxyphenyl]retinamide, as well as several other metabolites of 4-HP
42 ltiplicity of PIN, whereas 4-(hydroxyphenyl) retinamide at 392 or 784 mg/kg diet, did not have an inh
45 the synthetic retinoid, N-(4-hydroxyphenyl) retinamide (HPR), induces HL60 cells to undergo apoptosi
47 derivative fenretinide (N-(4-hydroxyphenyl) retinamide; HPR) exerts therapeutic effects in mouse mod
48 led retinoic acid (RA) or N-(4-hydroxyphenyl)retinamide, indicating that these drugs enhance [3H]reti
51 rs activities of novel C-4 heteroaryl 13-cis-retinamides that modulate Mnk-eIF4E and AR signaling are
53 The synthetic retinoid N-(4-hydroxyphenyl)retinamide, which does not bind effectively to RAR-beta,
54 is-retinoic acid (9cRA) and 4-(hydroxyphenyl)retinamide, which in previous studies have shown promisi
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