ライフサイエンスコーパス: retinitis

1 ients may suggest a rise in incidence of CMV retinitis.

2 stis carinii pneumonia and 1 cytomegalovirus retinitis.

3 ar degeneration, glaucoma, cataract, and CMV retinitis.

4 val of 27.0 years after the diagnosis of CMV retinitis.

5 remote graders evaluated each image for CMV retinitis.

6 uded in the study, 20 had improvement of CMV retinitis.

7 t, age, sex, and prior cytomegalovirus (CMV) retinitis.

8 ere ocular toxoplasmosis and cytomegalovirus retinitis.

9 ompartmental analysis of 1 patient with HCMV retinitis.

10 Among patients without CMV retinitis, 1 of 75 patients with immune recovery develop

11 Of the 21 eyes (10.2%) with CMV retinitis, 7 (33%) had visual symptoms.

12 ating cART and suggest that "immune recovery retinitis," a proposed immune recovery inflammatory synd

13 sed each image as CMV retinitis present, CMV retinitis absent, or unknown.

14 Among persons with CMV retinitis and AIDS, if there is immune recovery, long-te

15 indirect ophthalmoscopy for diagnosis of CMV retinitis and clinical features of CMV retinitis lesions

16 ceived FDA approval to treat cytomegalovirus retinitis and high blood cholesterol, respectively.

17 Bilateral CMV retinitis and larger lesion sizes, each of which is a ma

18 to >/=100 cells/muL; rates of new-onset CMV retinitis and of worsening of CMV retinitis (either incr

19 al prognosis was good in patients with focal retinitis and poor in patients with occlusive vasculitis

20 Sixty-four patients with CMV retinitis and retinal detachment were identified from th

21 monary infarcts were found in patient 3, and retinitis and subcutaneous lesions developed in patient

22 the rates of new-onset cytomegalovirus (CMV) retinitis and worsening existing CMV retinitis in patien

23 atic tumors of the retina are rare, simulate retinitis, and are associated with poor patient survival

24 ), glaucoma, cataract, cytomegalovirus (CMV) retinitis, and low vision.

25 fectious corneal ulceration, cytomegalovirus retinitis, and retinopathy of prematurity.

26 patients who develop active cytomegalovirus retinitis as an immune reconstitution inflammatory syndr

27 ecords of 116 HIV-infected patients with CMV retinitis attending our institution during January 2003-

28 ates of retinitis progression and increasing retinitis border activity among patients during the firs

29 dy was not highly sensitive in detecting CMV retinitis but may identify disease with an immediate thr

30 Three control patients with CMV retinitis but no retinal detachment were selected for ea

31 crease the number of people screened for CMV retinitis, but it is unclear whether nonophthalmologists

32 gible studies assessed the occurrence of CMV retinitis by funduscopic examination within a cohort of

33 y and specificity of remote diagnosis of CMV retinitis by nonexpert graders was variable, although se

34 terized further by whether it is primarily a retinitis, choroiditis, or retinal vasculitis; by whethe

35 efinition of cytomegalovirus-immune recovery retinitis (CMV-IRR) is proposed here.

36 yndrome (AIDS) patients with Cytomegalovirus retinitis (CMVR) -related retinal detachments(RD) in an

37 Although cytomegalovirus (CMV) retinitis (CMVR) is a well-recognized complication after

38 Cytomegalovirus (CMV) retinitis continues to be a leading cause of blindness i

39 reduce the likelihood that patients with CMV retinitis develop a retinal detachment.

40 t a cluster of 5 pediatric patients with CMV retinitis diagnosed in a 12-month period and compare thi

41 There were 5 cases of CMV retinitis diagnosed in those transplanted in 2014, a sta

42 hthalmologist, the sensitivity of remote CMV retinitis diagnosis by nonexpert graders ranged from 64.

43 -onset CMV retinitis and of worsening of CMV retinitis (either increasing border activity or retiniti

44 the consensus grade given by a panel of CMV retinitis experts.

45 level of accuracy comparable to that of CMV retinitis experts.

46 Cytomegalovirus (CMV) retinitis has been extensively described in patients wit

47 of symptoms ranging from febrile illness to retinitis, hepatic necrosis, hemorrhagic fever, and deat

48 ompare the proportion of CMV viremia and CMV retinitis in patients transplanted between January 2010

49 s (CMV) retinitis and worsening existing CMV retinitis in patients with AIDS after initiating combina

50 Our recent cluster of CMV retinitis in pediatric allogeneic HSCT patients may sugg

51 an ophthalmic screening protocol to diagnose retinitis in pediatric HSCT patients in the early, often

52 Prevalence of CMV retinitis in resource low- and middle-income countries,

53 compare this to the rate of CMV viremia and retinitis in the 4 years prior.

54 patients with immune recovery developed CMV retinitis in the first 6 months after initiating cART vs

55 CMV retinitis nor worsening of existing CMV retinitis in the first 6 months after initiating cART vs

56 The incidence of cytomegalovirus (CMV) retinitis in the pediatric allogeneic hematopoietic stem

57 Telemedicine screening for CMV retinitis instituted at the point of care for human immu

58 Eyes with retinitis involving >/=25% of the retina at presentation

59 Cytomegalovirus (CMV) retinitis is a leading cause of blindness in many develo

60 The CMV retinitis lesions missed by the remote graders (false-ne

61 Retinitis lesions occupied less than 10% of the total re

62 f CMV retinitis and clinical features of CMV retinitis lesions.

63 Ocular features included focal hemorrhagic retinitis (n = 13) and peripheral retinal necrosis (n =

64 Initial misdiagnosis as retinitis (n = 5), hemangioma (n = 1), choroidal neovasc

65 ition to previously reported clinical signs, retinitis, neuroretinitis, and retinal vasculitis were s

66 s neither an increased rate of new-onset CMV retinitis nor worsening of existing CMV retinitis in the

67 atients (15.5%) were diagnosed as having CMV retinitis, of whom 5 (31%) had bilateral disease.

68 Retinal metastases resemble retinitis, often with delay in diagnosis and poor life p

69 ly studied HIV-infected patients who had CMV retinitis on ART initiation or during the subsequent 6 m

70 ty for the 3 remote graders in detecting CMV retinitis on fundus photography was 30.2% (95% CI, 10.5%

71 xperienced paradoxical clinical worsening of retinitis (paradoxical CMV-IRR).

72 A diagnosis of retinitis pigementosa was made in this patient's childho

73 provisional diagnosis of autosomal dominant retinitis pigmentosa (adRP) that have disease-causing mu

74 from families affected by autosomal-dominant retinitis pigmentosa (adRP), a rare disorder characteriz

75 n cause of human blinding autosomal dominant retinitis pigmentosa (adRP).

76 the most common cause of autosomal dominant retinitis pigmentosa (ADRP).

77 EYS are associated with autosomal recessive retinitis pigmentosa (arRP) and autosomal recessive cone

78 sin gene associated with autosomal recessive retinitis pigmentosa (arRP) has yet to be determined.

79 otype in 4 families with autosomal recessive retinitis pigmentosa (arRP) that can be associated with

80 Secondary VPT (n = 67) occurred in eyes with retinitis pigmentosa (n = 15, 22%), pars planitis (n = 1

81 and neurogenic muscle weakness, ataxia, and retinitis pigmentosa (NARP), in mammalian oocytes using

82 DNA, associated with neuropathy, ataxia, and retinitis pigmentosa (NARP).

83 Retinitis pigmentosa (RP) affects about 1.8 million indi

84 Retinitis pigmentosa (RP) and age-related macular degene

85 ness in a number of retinal diseases such as retinitis pigmentosa (RP) and atrophic age-related macul

86 ncurable blinding retinal diseases including retinitis pigmentosa (RP) and atrophic age-related macul

87 d and irreversible disease that manifests as retinitis pigmentosa (RP) and bilateral neurosensory hea

88 e Leber congenital amaurosis (LCA), juvenile retinitis pigmentosa (RP) and cone-rod dystrophy.

89 generation and clinical phenotypes including retinitis pigmentosa (RP) and congenital stationary nigh

90 For ill-defined reasons, CS degenerate in retinitis pigmentosa (RP) and in the transitional zone (

91 d to various retinal degenerations including retinitis pigmentosa (RP) and macular/pattern dystrophy

92 etal developmental defects and non-syndromic retinitis pigmentosa (RP) are frequent findings.

93 Stargardt's disease (STGD) and Retinitis Pigmentosa (RP) are inherited retinal degenera

94 reatments for cystoid macular edema (CME) in retinitis pigmentosa (RP) are not always effective, may

95 eber congenital amaurosis (LCA) and juvenile retinitis pigmentosa (RP) are severe hereditary diseases

96 oherence tomography (SDOCT) in patients with retinitis pigmentosa (RP) associated with retinitis pigm

97 Retinitis pigmentosa (RP) encompasses a diverse group of

98 Cystoid macular edema (CME) in retinitis pigmentosa (RP) has been managed in several wa

99 Retinitis pigmentosa (RP) is a blinding disease often as

100 Retinitis pigmentosa (RP) is a clinically and geneticall

101 Retinitis pigmentosa (RP) is a group of genetically and

102 Retinitis pigmentosa (RP) is a group of inherited retina

103 Retinitis pigmentosa (RP) is a group of inherited retina

104 Retinitis Pigmentosa (RP) is a hereditary genetic diseas

105 Retinitis pigmentosa (RP) is a heterogeneous group of in

106 Retinitis pigmentosa (RP) is a highly heterogeneous grou

107 Retinitis pigmentosa (RP) is a major cause of blindness

108 Photoreceptor degeneration due to retinitis pigmentosa (RP) is a primary cause of inherite

109 Retinitis pigmentosa (RP) is an incurable neurodegenerat

110 Retinitis pigmentosa (RP) is an inherited neurodegenerat

111 Retinitis pigmentosa (RP) is an inherited neurodegenerat

112 Retinitis pigmentosa (RP) is an inherited photoreceptor

113 Retinitis pigmentosa (RP) is an inherited photoreceptor-

114 Retinitis pigmentosa (RP) is an inherited retinal degene

115 ted for some RP cases.SIGNIFICANCE STATEMENT Retinitis pigmentosa (RP) is an inherited, degenerative

116 associated with the various genetic forms of retinitis pigmentosa (RP) is currently untreatable and l

117 ird-most common cause of autosomal recessive retinitis pigmentosa (RP) is due to defective cGMP phosp

118 Retinitis pigmentosa (RP) is the leading cause of incura

119 Retinitis pigmentosa (RP) is the most common form of inh

120 Retinitis pigmentosa (RP) is the most frequent form of i

121 Because the secondary loss of cones in retinitis pigmentosa (RP) leads to blindness, the admini

122 some vision to patients blind as a result of retinitis pigmentosa (RP) or outer retinal degeneration.

123 t, we assess the natural progression rate of retinitis pigmentosa (RP) over an average of three years

124 analyze the genetic and clinical findings in retinitis pigmentosa (RP) patients of Ashkenazi Jewish (

125 cing (NGS) based molecular diagnosis for 105 Retinitis Pigmentosa (RP) patients randomly selected fro

126 ) II Retinal Prosthesis System (Argus II) in Retinitis Pigmentosa (RP) patients.

127 vel the molecular pathogenesis of an unusual retinitis pigmentosa (RP) phenotype observed in a Turkis

128 A retinitis pigmentosa (RP) phenotype was present in 50 pa

129 e limited published data on the phenotype of retinitis pigmentosa (RP) related to CNGB1 variants.

130 Retinitis pigmentosa (RP) shows progressive loss of phot

131 USH2A mutations are an important cause of retinitis pigmentosa (RP) with or without congenital sen

132 In humans, Rh mutations cause retinitis pigmentosa (RP), a degenerative disease that u

133 the rhodopsin gene have been associated with retinitis pigmentosa (RP), a family of inherited visual

134 Retinitis pigmentosa (RP), a genetically heterogeneous g

135 Retinitis pigmentosa (RP), a heterogeneous group of inhe

136 cells, are the most common cause of dominant retinitis pigmentosa (RP), a type of inherited blindness

137 viously reported to cause autosomal dominant retinitis pigmentosa (RP), and described their detailed

138 mans, such as Leber congenital amaurosis and retinitis pigmentosa (RP), are attributed to either homo

139 itary retinal degenerative diseases, such as retinitis pigmentosa (RP), are characterized by the prog

140 unrelated families with autosomal recessive retinitis pigmentosa (RP), but without extraocular invol

141 In retinitis pigmentosa (RP), cone cell death precedes rod

142 indings in patients with autosomal recessive retinitis pigmentosa (RP), cone-rod dystrophy (CRD) or c

143 individuals, it was found to segregate with retinitis pigmentosa (RP), goiter, primary ovarian insuf

144 y photoreceptor cells and cause nonsyndromic retinitis pigmentosa (RP), raising the issue of why cert

145 composed of IRD two with autosomal dominant retinitis pigmentosa (RP), two with autosomal recessive

146 enotype of the Rh1(G69D) Drosophila model of retinitis pigmentosa (RP).

147 rlying cause of many ciliopathies, including Retinitis Pigmentosa (RP).

148 t surgery accelerates disease progression in retinitis pigmentosa (RP).

149 r partial restoration of vision in end-stage retinitis pigmentosa (RP).

150 is a well-established animal model for human retinitis pigmentosa (RP).

151 dominant P23H rhodopsin mutation that causes retinitis pigmentosa (RP).

152 to treat retinal degenerative diseases like retinitis pigmentosa (RP).

153 alth, and employment among young adults with retinitis pigmentosa (RP).

154 ith unknown function that is associated with retinitis pigmentosa (RP).

155 orm of degenerative human blindness known as retinitis pigmentosa (RP).

156 S), Bardet-Biedl Syndrome, and some forms of retinitis pigmentosa (RP).

157 he change in the outer retinal structures in retinitis pigmentosa (RP).

158 in the Tulp1 gene cause severe, early-onset retinitis pigmentosa (RP14) in humans.

159 tor (RPGR) gene account for >70% of X-linked retinitis pigmentosa (XLRP) and 15-20% of all inherited

160 tations in the human RP2 gene cause X-linked retinitis pigmentosa (XLRP) and cone-rod dystrophy (XL-C

161 X-linked forms of retinitis pigmentosa (XLRP) are relatively severe blindi

162 ges of disease in a canine model of X-linked retinitis pigmentosa (XLRP) caused by a mutation in the

163 X-linked retinitis pigmentosa (XLRP) is one of the most severe fo

164 in the pathogenesis associated with X-linked retinitis pigmentosa (XLRP) resulting from mutations in

165 an RPGR point mutation that causes X-linked retinitis pigmentosa (XLRP).

166 ies thought to have adRP truly have X-linked retinitis pigmentosa (XLRP).

167 sure for future treatment trials in X-linked retinitis pigmentosa (XLRP).

168 RPGR genes are responsible for the X-linked retinitis pigmentosa (XLRP).

169 al vein occlusion 0.50%, macular hole 0.20%, retinitis pigmentosa 0.12%. and retinal detachment 0.10%

170 ases are caused by mutations in the X-linked retinitis pigmentosa 2 (RP2) and RPGR.

171 gmentosa GTPase regulator (RPGR) gene or the retinitis pigmentosa 2 (RP2) gene.

172 tions in the ARL3 GTPase activating protein, retinitis pigmentosa 2 (RP2).

173 s regulatory GTPase activating protein (GAP) Retinitis Pigmentosa 2 (RP2).

174 The retinitis pigmentosa 2 polypeptide (RP2) functions as a

175 ilies diagnosed as having autosomal dominant retinitis pigmentosa and 10% in families with variable c

176 ere omitted for 2 patients with non-X-linked retinitis pigmentosa and 16 patients who were unable to

177 itry, degenerate in retinal diseases such as retinitis pigmentosa and age related macular degeneratio

178 al degeneration and visual disorders such as retinitis pigmentosa and congenital stationary night bli

179 Family history was negative for retinitis pigmentosa and haemoglobinopathies.

180 s that BBS2 mutations can cause nonsyndromic retinitis pigmentosa and highlights yet another candidat

181 lly used during neurodegeneration arising in retinitis pigmentosa and prion infection.

182 We further found that PRPF8 mutants causing Retinitis pigmentosa assemble less efficiently with the

183 lium (RPE) from an individual suffering from retinitis pigmentosa associated with biallelic variants

184 Here, using a murine model of severe human retinitis pigmentosa at a stage when no host rod cells r

185 dopsin gene cause approximately one-tenth of retinitis pigmentosa cases worldwide, and most result in

186 ncharacterized cohort of autosomal recessive retinitis pigmentosa cases.

187 photoreceptor degeneration in patients with retinitis pigmentosa caused by IRBP mutation.

188 omitant loss of retinal function that mimics retinitis pigmentosa due to mutations in the CRB1 gene.

189 tified homozygous REEP6-E75K mutation in two retinitis pigmentosa families of different ethnicities.

190 etinal degeneration in XLRP.Mutations in the Retinitis Pigmentosa GTPase Regulator (RPGR) cause retin

191 Mutations in the Retinitis Pigmentosa GTPase Regulator (RPGR) cause X-lin

192 Mutations in the retinitis pigmentosa GTPase regulator (RPGR) gene accoun

193 Mutations in the X-linked retinitis pigmentosa GTPase regulator (RPGR) gene are a

194 Mutations in the retinitis pigmentosa GTPase regulator (RPGR) gene cause

195 ve disease-causing mutations in the X-linked retinitis pigmentosa GTPase regulator (RPGR) gene or the

196 igmentosa (XLRP) caused by a mutation in the Retinitis Pigmentosa GTPase Regulator (RPGR) gene.

197 th retinitis pigmentosa (RP) associated with retinitis pigmentosa GTPase regulator gene (RPGR) mutati

198 addition, SPATA7 directly interacts with the retinitis pigmentosa GTPase regulator interacting protei

199 Mutations in the ciliary protein RPGR (retinitis pigmentosa GTPase regulator) are a prominent c

200 Mutations in the ciliary protein RPGR (retinitis pigmentosa GTPase regulator) are common causes

201 Mutations in RPGR (retinitis pigmentosa GTPase regulator) are the most comm

202 y leading to decreased expression of FTO and retinitis pigmentosa GTPase regulator-interacting protei

203 It is unclear how genes, such as RPGR (retinitis pigmentosa guanine triphosphatase regulator) t

204 horoidal neovascularization in 2.3% of eyes; retinitis pigmentosa in 1.9% of eyes; severe cough in 1.

205 PF8, and PRPF31) cause nonsyndromic dominant retinitis pigmentosa in humans, an inherited retinal deg

206 The mutations cosegregated with retinitis pigmentosa in the studied families, and the af

207 Retinitis Pigmentosa is a group of hereditary retinal dy

208 Retinitis pigmentosa is a leading cause of inherited bli

209 Retinitis pigmentosa is a progressive retinal dystrophy

210 Retinitis pigmentosa is a rare disease, affecting only a

211 X-linked retinitis pigmentosa is a severe inherited retinal degen

212 rong light responses when used in retinas of retinitis pigmentosa model mice.

213 d CRB1 and CRB2 gene therapy vectors in Crb1-retinitis pigmentosa mouse models at mid-stage disease.

214 Several human diseases, such as retinitis pigmentosa or congenital night blindness, are

215 deficiency is linked to human diseases like retinitis pigmentosa or myeloid neoplasia, but its genom

216 specific visual cortical gray matter loss in Retinitis Pigmentosa patients associated with their visu

217 is likely the cause of phenotype observed in retinitis pigmentosa patients carrying T17M mutation.

218 We also find that MG from retinitis pigmentosa patients display an increase in Bre

219 Retinitis pigmentosa patients from 230 families of AJ or

220 tter volume has not been addressed before in Retinitis Pigmentosa patients with low vision.

221 whole brain gray matter volume changes in 27 Retinitis Pigmentosa patients with partially preserved v

222 PRPF31-associated retinitis pigmentosa presents a fascinating enigma: some

223 Retinitis pigmentosa refers to a family of inherited pho

224 Vision impairments and blindness caused by retinitis pigmentosa result from severe neurodegeneratio

225 Retinitis pigmentosa results in blindness due to degener

226 ere autosomal recessive retinal dystrophies (retinitis pigmentosa RP64 and cone-rod dystrophy CORD16)

227 risons with published studies of ungenotyped retinitis pigmentosa showed that the RPE65-LCA patients

228 Patient phenotypes range from retinitis pigmentosa to various forms of macular and pat

229 higher photosynthetic organisms, as well as Retinitis Pigmentosa Type 2-Clathrin Light Chain, a memb

230 tegies to optimize outcomes in patients with retinitis pigmentosa undergoing retinal prosthesis impla

231 udinal imaging follow-up in 71 patients with retinitis pigmentosa was studied using the main outcome

232 light perception or worse in both eyes) with retinitis pigmentosa were implanted with the Argus II pr

233 t male patients diagnosed as having X-linked retinitis pigmentosa were randomized to DHA or placebo.

234 Retinitis pigmentosa with or without CME.

235 understanding other dominant diseases (e.g., retinitis pigmentosa) caused by missense mutations in me

236 ilies with a diagnosis of autosomal dominant retinitis pigmentosa, 35 families with unspecified macul

237 e regulator (RPGR) gene are a major cause of retinitis pigmentosa, a blinding retinal disease resulti

238 mutation in IRBP was found in patients with retinitis pigmentosa, a frequent cause of retinal degene

239 utation in the human IRBP has been linked to retinitis pigmentosa, a progressive retinal degenerative

240 ular age-related macular degeneration (AMD), retinitis pigmentosa, and diabetic retinopathy are assoc

241 nvolved in age-related macular degeneration, retinitis pigmentosa, and Leber's congenital amaurosis m

242 sing CRISPR/Cas9 to model the human disorder retinitis pigmentosa, and to introduce point mutations o

243 such as age-related macular degeneration and retinitis pigmentosa, are the leading cause of blindness

244 ominant diseases, such as autosomal dominant retinitis pigmentosa, are thought to arise due to haploi

245 e limited visual perception to patients with retinitis pigmentosa, however loss of retinal ganglion c

246 Rod-cone dystrophy (RCD), also known as retinitis pigmentosa, is a progressive inherited retinal

247 In retinitis pigmentosa, loss of cone photoreceptors leads

248 tained families with a clinical diagnosis of retinitis pigmentosa, macular dystrophy, and/or pattern

249 d deletion of codon 153 (K153Delta) leads to retinitis pigmentosa, pattern dystrophy, and fundus flav

250 is a rare disorder characterized by obesity, retinitis pigmentosa, polydactyly, mental retardation an

251 Retinitis pigmentosa, which affects one in 3000 people,

252 he RPGR gene cause a common form of X-linked retinitis pigmentosa, which often results in severe loss

253 We found that neurofibromatosis- or retinitis pigmentosa-causing mutations in the 5' regions

254 t use, to our knowledge, of human iPSCs with retinitis pigmentosa-causing mutations to look at pathop

255 uccessfully promotes splicing of a defective retinitis pigmentosa-causing transcript.

256 ents with retinal degenerative diseases like retinitis pigmentosa.

257 in a rhodopsin knockout (RKO) mouse model of retinitis pigmentosa.

258 s in retinal wholemounts in a mouse model of retinitis pigmentosa.

259 model of the retinal degeneration condition retinitis pigmentosa.

260 spicule-like pigmented deposits, typical for retinitis pigmentosa.

261 (BBS), Leber congenital amaurosis (LCA), and retinitis pigmentosa.

262 macular degeneration, Stargardt disease, and retinitis pigmentosa.

263 2 missense mutations that cause nonsyndromic retinitis pigmentosa.

264 acular degeneration, retinal detachment, and retinitis pigmentosa.

265 promising treatment option for patients with retinitis pigmentosa.

266 e RP2 gene lead to a severe form of X-linked retinitis pigmentosa.

267 of a small protein therapy for some forms of retinitis pigmentosa.

268 t-Biedl syndrome usually develop early-onset retinitis pigmentosa.

269 electroretinogram confirmed the diagnosis of retinitis pigmentosa.

270 the most common cause of autosomal dominant retinitis pigmentosa.

271 r-old P23H rhodopsin transgenic rat model of retinitis pigmentosa.

272 or rod ERG function associated with X-linked retinitis pigmentosa.

273 e Leber congenital amaurosis and early-onset retinitis pigmentosa.

274 s or pathways pathologically associated with retinitis pigmentosa.

275 ses and visual behaviors in rodent models of Retinitis pigmentosa.

276 toreceptor death found in autosomal dominant retinitis pigmentosa.

277 dt disease and the Mertk(-/-) mouse model of retinitis pigmentosa.

278 members of a family with autosomal recessive retinitis pigmentosa.

279 n RPE65-LCA fell within reported results for retinitis pigmentosa.

280 ne of the most common causes of all forms of retinitis pigmentosa.

281 ntal retardation, and one subject exhibiting retinitis pigmentosa.

282 st 1 eye), and a negative family history for retinitis pigmentosa.

283 resembling a dry desert land and ends with a retinitis pigmentosa.

284 nts from the Trial of Oral Valproic Acid for Retinitis Pigmentosa.

285 henotype in rd10 mice, a model for inherited retinitis pigmentosa.

286 Graders diagnosed each image as CMV retinitis present, CMV retinitis absent, or unknown.

287 cute retinal necrosis (ARN) is an infectious retinitis primarily caused by the herpesviruses.

288 ng patients with CMV retinitis, the rates of retinitis progression and increasing retinitis border ac

289 initis (either increasing border activity or retinitis progression) were compared between those with

290 clinic for HIV treatment had less extensive retinitis than patients in recent reports from an ophtha

291 at the time of the initial diagnosis of CMV retinitis that predicted subsequent retinal detachment i

292 Among patients with CMV retinitis, the rates of retinitis progression and increa

293 where reported CD4 count at diagnosis of CMV retinitis was <50 cells/microL in 73.4% of cases.

294 so for the initial clinic visit at which CMV retinitis was diagnosed.

295 characteristics of patients with active CMV retinitis were described.

296 ent repair, endophthalmitis, and necrotizing retinitis were excluded, as were those with intraocular

297 No cases of CMV retinitis were identified.

298 al of 13.5 months after the diagnosis of CMV retinitis, whereas those with immune recovery had a mort

299 CMV retinitis without HIV infection was often aggressive at

300 Consecutive patients with CMV retinitis without HIV infection were reviewed.