ライフサイエンスコーパス: retinoic acid

1 is counteracted in the presence of all-trans retinoic acid.

2 e generated in the presence of vitamin C and retinoic acid.

3 cell-surface receptor known as stimulated by retinoic acid 6 (STRA6), which transports retinol into c

4 r retinoid-binding protein and stimulated by retinoic acid 6 protein showed significantly lower level

5 that membrane protein, STRA6 (stimulated by retinoic acid 6), is the RBP4 receptor and vitamin A cha

6 nergistically activating RXRalpha with 9-cis-retinoic acid (9-cis-RA), a natural ligand binding to th

7 he effect of the endogenous metabolite 9-cis retinoic acid (9cRA) on allergic sensitization is unknow

8 the neurorestorative effect of delayed 9 cis retinoic acid (9cRA) treatment for stroke.

9 Some serve on-site for the synthesis of retinoic acid, a hormone-like compound, which exerts ple

10 Retinoic acid, a known inducer of the gut-homing phenoty

11 following search terms: isotretinoin, 13-cis-retinoic acid, Accutane, retinoids, acitretin, surgery,

12 Retinoic acid, an active metabolite of dietary vitamin A

13 uclear hormone receptor activity mediated by retinoic acid, an endogenous client lipid of CRABP2.

14 Retinoic acid and 1,25D3 have antagonistic effects on th

15 ESCs, we found that mouse iPSCs treated with retinoic acid and a smoothened agonist differentiated in

16 form beta-apo-10'-carotenal, a precursor of retinoic acid and a transcriptional regulator per se The

17 Upon treating APL with all-trans retinoic acid and achieving complete remission, the leve

18 ith APL homogeneously treated with all-trans retinoic acid and anthracycline-based chemotherapy, acco

19 idence that potentiation of ST8SIA2 by 9-cis-retinoic acid and artificial polysialylation of oligoden

20 Retinoic acid and its synthetic compound AM80 play roles

21 on in the caudal neural tube is dependent on retinoic acid and Pax6, and that it is restricted to p1

22 d that Lcn2 KO mice have decreased levels of retinoic acid and retinol in adipose tissue.

23 1.3 complex containing silencing mediator of retinoic acid and thyroid hormone receptor (SMRT) and nu

24 corepressor (NCoR) and silencing mediator of retinoic acid and thyroid hormone receptor (SMRT) corepr

25 tor corepressor (NCoR)/silencing mediator of retinoic acid and thyroid receptors (SMRT) corepressor c

26 demonstrate that TGF-beta itself, along with retinoic acid and TLR signaling, drives expression of al

27 strongly increased by the Treg cell inducer "retinoic acid", and the abundantly expressed miR-150 cou

28 ration-dependent, 2) selective for all-trans-retinoic acid, and 3) requires the presence of apolipopr

29 Treatment with all-trans retinoic acid antagonizes stress-induced activation of d

30 Functionally, Cyp1b1 generated retinoic acid as well as 20-hydroxyeicosatetraenoic acid

31 al showed that the combination of all- trans-retinoic acid (ATRA) and arsenic trioxide (ATO) is at le

32 until the recent identification of all-trans retinoic acid (ATRA) as a Pin1 inhibitor.

33 The identification of all-trans retinoic acid (ATRA) as a potent Pin1 inhibitor provides

34 arge clinical trials that included all-trans retinoic acid (ATRA) as part of induction, we assessed k

35 We show that treatment with all-trans retinoic acid (ATRA) at clinically achievable doses mark

36 CYP26 enzymes are responsible for all-trans-retinoic acid (atRA) clearance.

37 All patients received all-trans retinoic acid (ATRA) during induction, each consolidatio

38 All-trans retinoic acid (ATRA) has been used in several clinical t

39 acute promyelocytic patients with all-trans retinoic acid (ATRA) has improved the survival of these

40 stem cells through treatment with all-trans retinoic acid (ATRA) have yielded limited success, parti

41 All-trans retinoic acid (ATRA) increased IRF4 expression, restored

42 The vitamin A metabolite all-trans retinoic acid (ATRA) induces a gut-homing phenotype in a

43 es the first evidence showing that all-trans retinoic acid (ATRA) induces the interaction and chromat

44 Although all-trans-retinoic acid (atRA) is a key regulator of intestinal im

45 All-trans-retinoic acid (ATRA) is a natural compound proposed for

46 All trans retinoic acid (atRA) is one of the most potent therapeut

47 All-trans-retinoic acid (atRA) is the active metabolite of vitamin

48 mice exhibited a blunted effect of all-trans-retinoic acid (ATRA) on body weight and fat mass, lipid

49 The combination of all-trans-retinoic acid (ATRA) plus arsenic trioxide (ATO) has bee

50 cute promyelocytic leukemia (APL), all-trans retinoic acid (ATRA) treatment induces granulocytic matu

51 All-trans retinoic acid (ATRA), a derivative of vitamin A, is a co

52 ibrillar fibronectin and show that all trans-retinoic acid (ATRA), which induces PSC quiescence, down

53 based screening, here we find that all-trans retinoic acid (ATRA)--a therapy for acute promyelocytic

54 Despite the great success of all-trans retinoic acid (ATRA)-based therapy, which results in a c

55 n and leukemogenesis, and inhibits all-trans-retinoic acid (ATRA)-induced AML cell differentiation, t

56 udy, we present an effective model All-Trans Retinoic Acid (ATRA)-induced differentiation of HL-60 ce

57 developmental genes, including the all-trans-retinoic acid (ATRA)-responsive ones, through its modifi

58 ted the gene expression profile of all-trans retinoic acid (ATRA)-treated human CD4(+) T cells.

59 We determine that cellular retinoic acid binding protein 1 (Crabp1) mediates the no

60 nd in the retinoic acid-transporter cellular retinoic acid binding protein 1 (p < 0.05 vs. NC-HDL).

61 atment, we investigated the role of cellular retinoic acid binding protein 2 (CRABP2) in MPNST in vit

62 selectively and efficiently modify cellular retinoic acid binding protein 2 (CRABP2), both in vitro

63 stellate cells and elevated levels of serum retinoic acid-binding protein 4, indicating increased bi

64 The goal of this study was to identify retinoic acid-binding proteins secreted by the choroid a

65 hich have substantially decreased endogenous retinoic acid biosynthesis, Fgf21 expression was increas

66 We analyzed production of retinoic acid by DCs and their ability to induce develop

67 hat Lcn2 is a retinoic acid target gene, and retinoic acid concurrently stimulated UCP1 and Lcn2 expr

68 nteract with RARalpha, and failed to inhibit retinoic acid-dependent transcriptional activity upon ex

69 inactive cholera toxin (CT), LDCs stimulated retinoic acid-dependent up-regulation of alpha4beta7 and

70 The retinoic acid derivative fenretinide (FR) is capable of

71 t that mechanical reprogramming of PSCs with retinoic acid derivatives might be a viable alternative

72 in response to 5 toxic compounds (all-trans retinoic acid, dexamethasone, doxorubicin, 5'-fluorourac

73 sis pathway, are more rapid in hPSCs than in retinoic-acid-differentiated hPSCs.

74 sis induction than are lineage-non-specific, retinoic-acid-differentiated hPSCs.

75 e we measure the gene expression dynamics of retinoic acid driven mESC differentiation from pluripote

76 Addition of retinoic acid drove trunk-related markers and HOX genes

77 onstrated using both ibuprofen and all-trans retinoic acid; drugs with anti-inflammatory and anti-pro

78 Notably, we also showed that treatment with retinoic acid enhanced NRIP1 binding to RARalpha RNA in

79 9-cis-retinoic acid enhances ST8SIA2 expression, providing a m

80 ralphabeta(-/-) HSCs, where there is greater retinoic acid flux.

81 ous endocrine formation while treatment with retinoic acid followed by combined EGF/KGF efficiently g

82 Functionally, Cyp1b1 could generate retinoic acid from retinol leading to cell-autonomous in

83 tk), driven by the promoter of stimulated by retinoic acid gene 8 (Stra8), a germ cell-specific gene

84 lates illumination differences and generates retinoic acid gradients that underlie the generation of

85 lyceride content in BAT, as well as impaired retinoic acid homeostasis, associated with decreased BAT

86 sis, associated with decreased BAT levels of retinoic acid in alcohol-consuming mice.

87 XA5 in breast cancer, and the role played by retinoic acid in HOXA5 function.

88 response to targeted therapy with all-trans retinoic acid in vivo was dependent on NB integrity.

89 Stimulus-dependent expression of the retinoic acid-inactivating enzyme Cyp26B1 in olfactory s

90 All-trans retinoic acid increased CD38 levels and decreased CD55 a

91 These data suggest a retinoic acid-independent, non-tumor suppressor role of

92 Haploinsufficiency of Retinoic Acid Induced 1 (RAI1) causes Smith-Magenis synd

93 nique adaptor molecule that is essential for retinoic acid induced gene-I (RIG-I) and melanoma differ

94 e haploinsufficient for the SMS causal gene, Retinoic acid induced-1 (Rai1), were hypersensitive to l

95 cument that overexpression of EVI1 abrogates retinoic acid-induced maturation of EML cells into commi

96 delta signaling cascades in conjunction with retinoic acid-induced neuronal differentiation brings ab

97 nterferon regulatory factor 3 (IRF3) via the retinoic acid inducible gene (RIG)-I/mitochondrial antiv

98 ble interferon regulatory factors (IRFs) and retinoic acid inducible gene (RIG-I).

99 re, we report that the sensing of IAV RNA by retinoic acid inducible gene I (RIG-I) initiates ZBP1-me

100 sm principally by the innate immune receptor Retinoic Acid Inducible Gene-I (RIG-I), whose activation

101 RIG-I (Retinoic Acid Inducible Gene-I) is a cytosolic innate im

102 re, Dandenong, and Lujo viruses, can inhibit retinoic acid-inducible gene 1 (RIG-i) and Melanoma Diff

103 The retinoic acid-inducible gene 1 (RIG-I) signaling pathway

104 differentiation-associated protein 5 (MDA5), retinoic acid-inducible gene 1 (RIG-I), and mitochondria

105 hich is critical for sustained and amplified retinoic acid-inducible gene 1 (RIG-I)-induced type I in

106 ognition receptors (Toll-like receptor 7 and retinoic acid-inducible gene 1), HCV RNA induced consist

107 danger signals that are transmitted via the retinoic acid-inducible gene 1-like receptor (RLR), nucl

108 (IFN-beta) by interacting with and degrading retinoic acid-inducible gene I (RIG-I) and melanoma diff

109 e expression of Toll-like receptor 3 (TLR3), retinoic acid-inducible gene I (RIG-I) and several antiv

110 , and IRF7 inducibly binds to the endogenous retinoic acid-inducible gene I (RIG-I) promoter.

111 Retinoic acid-inducible gene I (RIG-I) receptor recogniz

112 In the cytoplasm, the retinoic acid-inducible gene I (RIG-I) senses the RNA ge

113 ll three IFIT genes following stimulation of retinoic acid-inducible gene I (RIG-I), it could selecti

114 oduction, most likely through suppression of retinoic acid-inducible gene I (RIG-I)-like receptor (RL

115 IFI16) as well as viral RNA receptors of the retinoic acid-inducible gene I (RIG-I)-like receptor (RL

116 ed by active infection trigger a coordinated retinoic acid-inducible gene I (RIG-I)-Toll-like recepto

117 zed after stimulation with TLR4, TLR7/8, and retinoic acid-inducible gene I agonists.

118 The cytosolic RIG-I (retinoic acid-inducible gene I) receptor plays a pivotal

119 red type I interferon (IFN) signaling in the retinoic acid-inducible gene I-like receptor (RLR) pathw

120 iviral gene against RNA viruses that uses an retinoic acid-inducible gene I-like receptor-independent

121 IFNs and other inflammatory cytokines after retinoic acid-inducible gene I-like receptors recognize

122 OV10 was unique and independent of the known retinoic acid-inducible gene I/mitochondrial antiviral-s

123 ng oligomerization domain-like receptor, and retinoic acid-inducible gene RIG-like receptor pathways

124 ng oligomerization domain-like receptor, and retinoic acid-inducible gene RIG-like receptor signaling

125 By identifying a novel role for retinoic acid-inducible gene-I (RIG-I) as a central regu

126 Gprc5a is known as retinoic acid-inducible protein 3, and its deficiency le

127 rization domain (NOD)-like receptors (NLRs), retinoic acid-inducible protein I (RIG-I)-like receptors

128 he intestine, it increased the expression of retinoic acid-inducible target genes such as Aldh1a2, Dh

129 s such as myxoma resistance gene 1 (Mx1) and retinoic acid-inducing gene-I (RIG-I).

130 ties were reversed following addition of the retinoic acid inhibitor LE540.

131 Retinoic acid is a known upstream regulator of HOXA5 exp

132 We demonstrate that retinoic acid is critical in establishing asymmetric pig

133 During vertebrate somitogenesis, retinoic acid is known to establish the position of the

134 use the molecular mechanism of regulation by retinoic acid is still not fully uncovered, we investiga

135 inoid (retinol, retinyl ester, and all-trans-retinoic acid) levels were observed.

136 ion of CYP26 in BM stromal cells maintains a retinoic acid-low (RA-low) microenvironment that prevent

137 All-trans-retinoic acid may be an important molecular signal in th

138 oid epidermis suggested that an imbalance of retinoic acid metabolism is likely associated with keloi

139 alcohol has a significant impact on cellular retinoic acid metabolism, with resultant effects on its

140 genous gels demonstrated a stiffness-driven, retinoic-acid-modulated upregulation of SIRPalpha and th

141 In the absence of retinoic acid, MPNST cells depleted of CRABP2 had reduce

142 been applied, consistent with the effects of retinoic acid on alternative pathways for ceramide gener

143 with retinoic acid to control the action of retinoic acid on ocular targets during postnatal ocular

144 -SY5Y human neuroblastoma cells by all-trans retinoic acid, or oxidative stress induced by mitochondr

145 classic Th1 cells because of the presence of retinoic acid orphan receptor (ROR)C2 and the surface ex

146 ILC2s expressed GATA-3, retinoic acid orphan receptor (RORC) 2, and RORalpha; we

147 human keratinocytes reproduced the abnormal retinoic acid pathway expression pattern we had identifi

148 ion, low Myc levels and high expression of a retinoic acid program are characteristic for dHSCs.

149 y the combination of two targeted therapies: retinoic acid (RA) and arsenic.

150 intenance of somitogenesis symmetry requires retinoic acid (RA) and its coactivator Rere/Atrophin2.

151 All-trans Retinoic acid (RA) and its derivatives are potent therap

152 scriptional network that integrates opposing retinoic acid (RA) and Wnt signals to determine the rate

153 netic and in vitro approaches, we identified retinoic acid (RA) as an important regulator of brain va

154 th the differentiation-promoting activity of retinoic acid (RA) could provide an alternative strategy

155 M) and phasor analysis to measure endogenous retinoic acid (RA) directly in vivo, we have investigate

156 territories in the non-axial mesoderm while retinoic acid (RA) functions later, but also across the

157 Retinoic acid (RA) has been used therapeutically to redu

158 e onset of meiosis, known to be regulated by retinoic acid (RA) in mammals.

159 Retinoic acid (RA) is a critical regulator of the intest

160 CYP26B1, CRABP1 and RALDH3 establish dynamic retinoic acid (RA) landscapes in feather mesenchyme, whi

161 have shown that TLX1-dependent regulation of retinoic acid (RA) metabolism is critical for spleen org

162 of active vitamin D3 (VD3) and/or all-trans retinoic acid (RA) on wild-type mouse skin induces a hum

163 Here we report that retinoic acid (RA) or retinol (vitamin A) and ascorbate

164 Here, we find that retinoic acid (RA) pathway is required for hair cell reg

165 All-trans retinoic acid (RA) plays crucial roles in embryogenesis.

166 s whereby the vitamin A metabolite all-trans retinoic acid (RA) promotes the formation of plasma cell

167 ular control of embryonic bilateral symmetry.Retinoic acid (RA) regulates the maintenance of somitoge

168 d homeostasis, as well as the involvement of retinoic acid (RA) signaling in the entire process.

169 pathway co-receptor Lrp6 is mutated or when retinoic acid (RA) signaling in the eye is compromised.

170 Retinoic acid (RA) signaling is crucial for spermatogoni

171 Retinoic acid (RA) signaling is essential for skin epide

172 Previous studies have suggested that retinoic acid (RA) signaling is involved in diencephalic

173 Wnt, BMP, fibroblast growth factor (FGF) and retinoic acid (RA) signaling to obtain lung and airway p

174 e and shape, as well as SNPs associated with retinoic acid (RA) signaling-associated genes, have been

175 show that gdf6a mutant eyes exhibit expanded retinoic acid (RA) signalling and demonstrate that exoge

176 g Ca(2+) levels in neurons, thereby inducing retinoic acid (RA) synthesis and RA-dependent homeostati

177 , we demonstrate that many genes involved in retinoic acid (RA) synthesis or regulated by RA are diff

178 Although retinoic acid (RA) teratogenicity has been investigated

179 Retinoic acid (RA) treatment blocked the P4 increase in

180 nases (ALDH1As) involved in the synthesis of retinoic acid (RA) were chosen as model proteins.

181 Thyroid hormone (TH) and retinoic acid (RA) within the tanycytes and ependymal ce

182 All-trans-retinoic acid (RA), a bioactive derivative of vitamin A,

183 Retinoic acid (RA), an essential active metabolite of vi

184 example, the active metabolite of vitamin A, retinoic acid (RA), has been described to maintain homeo

185 e ability to metabolize vitamin A to produce retinoic acid (RA), which drives regulatory T-cell respo

186 They produce retinoic acid (RA), which imprints a gut-homing phenotyp

187 s distal to their origin unless treated with retinoic acid (RA), which results in proximodistal (PD)

188 of vitamin A are exerted by its metabolite, retinoic acid (RA), which through ligation of nuclear re

189 Here, we describe light-inducible polymeric retinoic acid (RA)-containing nanoparticles (NPs) with t

190 In all-trans retinoic acid (RA)-induced differentiation of acute prom

191 or the resolution and activation of numerous retinoic acid (RA)-inducible bivalent genes during the R

192 Retinoic acid (RA)-inducible gene I (RIG-I, encoded by D

193 wn to be coregulated by an extrinsic signal, retinoic acid (RA).

194 to its transcriptionally active metabolite, retinoic acid (RA).

195 tic cells and the gut-specific tissue factor retinoic acid (RA).

196 which was regulated by enzymes that degrade retinoic acid (RA).

197 plexed with NAD(+) and the product all-trans retinoic acid (REA).

198 We identified several retinoic acid receptor (RAR) agonists that reduced secre

199 We have previously shown that retinoic acid receptor (RAR) gamma-deficient mice have h

200 sion of lamin-A is known to be controlled by retinoic acid receptor (RAR) transcription factors, but

201 -cells by overexpressing a dominant-negative retinoic acid receptor (RAR)-alpha mutant (RARdn) using

202 ially due to the epigenetic silencing of the retinoic acid receptor (RAR)-beta The histone deacetylas

203 While human ILCPs express low levels of retinoic acid receptor (RAR)-related orphan receptor C (

204 ediated suppression of the expression of the retinoic acid receptor (RAR)-related orphan receptor gam

205 Tconv and Treg express similar levels of the retinoic acid receptor (RAR).

206 Notably, the expression of retinoic acid receptor (RAR-beta) and retinoid X recepto

207 ML) nuclear bodies (NBs) mediated by the PML-retinoic acid receptor alpha (RARalpha) oncoprotein.

208 expression of a dominant-negative mutant of retinoic acid receptor alpha (RARalpha) specifically to

209 iescence in PSCs via a mechanism involving a retinoic acid receptor beta (RAR-beta)-dependent downreg

210 ical avulsion, we show that treatment with a retinoic acid receptor beta (RARbeta) agonist results in

211 Here we show that retinoic acid receptor beta (RARbeta) controls developme

212 Here, we identify a single target, neuronal retinoic acid receptor beta (RARbeta), which modulates t

213 authors present the crystal structure of the retinoic acid receptor beta-retinoic X receptor alpha (R

214 the quaternary architecture of multi-domain retinoic acid receptor beta-retinoic X receptor alpha (R

215 utagenesis of five cis-acting element types (retinoic acid receptor binding elements [RARE], cyclic A

216 We previously found that retinoic acid receptor gamma (RARgamma) agonist blocks h

217 Here we report that the cytoplasmic retinoic acid receptor gamma (RARgamma) controls recepto

218 , the authors show that the nuclear receptor retinoic acid receptor gamma is released from the nucleu

219 Retinoic acid receptor responder 2 (RARRES2) is transcri

220 The retinoid increase drives intrinsic retinoic acid receptor signaling, and activation occurs

221 ector responses through their binding to the retinoic acid receptor, a ligand-activated transcription

222 We also demonstrate that the PML retinoic acid receptor-alpha (PML-RARalpha) oncofusion p

223 a was reduced by pharmacological blockade of retinoic acid receptor-alpha (RARalpha) signaling, indic

224 rs of vitamin D receptor (VDR)/RXR-alpha and retinoic acid receptor-gamma (RAR-gamma)/RXR-beta are bo

225 gh small-molecule thymus-specific isoform of retinoic acid receptor-related orphan nuclear receptor g

226 nd blocking antibodies in mice, we show that retinoic acid receptor-related orphan nuclear receptor g

227 fy STAT3-induced transcription of IL-17A and retinoic acid receptor-related orphan nuclear receptor,

228 gival expressions of interleukin (IL)-17 and retinoic acid receptor-related orphan receptor (ROR) gam

229 Here we showed that retinoic acid receptor-related orphan receptor alpha (RO

230 on in CRP in EAs (P </= 6.8 x 10(-4)) and in retinoic acid receptor-related orphan receptor alpha (RO

231 Retinoic acid receptor-related orphan receptor C (RORc,

232 Here we show that retinoic acid receptor-related orphan receptor gamma (RO

233 s response is driven by the master regulator retinoic acid receptor-related orphan receptor gammat (R

234 In addition, the level of retinoic acid receptor-related orphan receptor gammat mR

235 The retinoic acid receptor-related orphan receptors RORalpha

236 tivation with minimal cross-signaling of the retinoic acid receptor.

237 sms by which progesterone receptors (PR) and retinoic acid receptors (RAR) regulate CK5 expression an

238 ge organ of vitamin A, but activation of the retinoic acid receptors (RARs) in mouse liver and in hum

239 In this study, we target specific retinoic acid receptors (RARs) to either PD duplicate (R

240 al cofactor that directly interacts with the retinoic acid receptors (RARs) to modulate retinoic acid

241 caused by an erroneous signaling mediated by retinoic acid receptors on the MMP-1 promoter and leads

242 enhancers, to gain mechanistic insight into retinoic acid regulation of T-cell fate.

243 HDAC3-cKO mice failed to downregulate retinoic acid-related orphan receptor (ROR) gammat durin

244 ation containing T-box transcription factor, retinoic acid-related orphan receptor (ROR) gammat, IFN-

245 PAS domain-containing protein 2 (Npas2), and retinoic acid-related orphan receptor (Ror)alpha/gamma w

246 CD8(+) T cells that express retinoic acid-related orphan receptor (ROR)gammat (TC17

247 Foxp3(+)retinoic acid-related orphan receptor (ROR)gammat(+) T c

248 pression in livers of mice by activating the retinoic acid-related orphan receptor alpha, and induced

249 nset of RA signaling might take place before retinoic acid-related orphan receptor gammat is expresse

250 expression of the TH17 transcription factor retinoic acid-related orphan receptor gammat or intracel

251 Type 3 immunity is mediated by retinoic acid-related orphan receptor gammat(+) ILC3s, T

252 r 4, B cell-activating transcription factor, retinoic acid-related orphan receptor gammat, and SMAD2

253 uence of ILC-intrinsic RA signaling, because retinoic acid-related orphan receptor gammat-Cre x RARal

254 of Th17-associated genes IL-17A, IL-22, and retinoic acid-related orphan receptor gammat.

255 ell type, Th17 cells, HIF1alpha acts via the retinoic acid-related orphan receptor-gammat (RORgammat)

256 The frequency of Retinoic Acid Response Element (RARE) sequences was incr

257 a luciferase reporter plasmid showed reduced retinoic acid response element activity, supporting the

258 which results in dissociation of TACC1 from retinoic acid response elements and leads to transcripti

259 a complex in the nucleus that binds specific retinoic acid response elements in the absence of RA.

260 in visual pigments, decreased expression of retinoic acid-responsive genes and photoreceptor cell lo

261 Short-term treatment of mice with all-trans retinoic acid resulted in increased PreB lymphopoiesis i

262 sis that chronic alcohol consumption impairs retinoic acid signaling in brown adipose tissue (BAT), l

263 Our findings indicate that retinoic acid signaling is related to the pathogenesis o

264 y weight and fat mass, lipid metabolism, and retinoic acid signaling pathway activation in adipose ti

265 osis point to a role for thyroid hormone and retinoic acid signaling, as well as phototransduction pa

266 aling, an innate adaptor with involvement in retinoic acid signaling, resulted in reduced infiltratio

267 ceptor coregulator that positively regulates retinoic acid signaling.

268 rown/beige adipocyte formation via elevating retinoic acid signaling.

269 CRABP2 is a transcriptional co-activator of retinoic acid signaling.

270 ipose tissue may be mediated through altered retinoic acid signaling.

271 epresses Bco1 gene expression in response to retinoic acid signaling.

272 Abrogation of retinoic acid signalling in Th1 cells resulted in loss o

273 ardial lineage by activation of WNT, BMP and retinoic acid signalling pathways.

274 We generated and characterized mature retinoic acid-skewed dendritic cells (DC-RAs) and assess

275 Signaling pathways (retinoic acid, sonic hedgehog, and Notch) that pattern t

276 lant cultures of embryonic kidney rudiments, retinoic acid stimulated Nrip1 expression, whereas a pan

277 Our study results suggest that insufficient retinoic acid synthesis by keloid epidermal keratinocyte

278 ement activity, supporting the hypothesis of retinoic acid synthesis deficiency in keloid epidermis.

279 failure to express a key enzyme involved in retinoic acid synthesis.

280 We found that Lcn2 is a retinoic acid target gene, and retinoic acid concurrentl

281 to retinaldehyde, which is then oxidized to retinoic acid, the biologically active form of vitamin A

282 affinity labeling with all-trans-[11,12-(3)H]retinoic acid, the most abundant labeled protein detecte

283 ing experiments demonstrated that binding of retinoic acid to apolipoprotein A-I is 1) concentration-

284 pate in a regulatory feedback mechanism with retinoic acid to control the action of retinoic acid on

285 Consistent with the ability of retinoic acid to foster inducible Tregs, miR-33-depleted

286 e retinoic acid receptors (RARs) to modulate retinoic acid transcriptional activity.

287 tations can cause CAKUT by interference with retinoic acid transcriptional signaling, shedding light

288 ng protein 4 and apolipoprotein M and in the retinoic acid-transporter cellular retinoic acid binding

289 Retinoic acid treatment of A(-) mice at the peak of the

290 he mutant phenotype was partially rescued by retinoic acid treatment of the pregnant females.

291 Within 2 h of retinoic acid treatment, Hoxa1 is rapidly recruited to t

292 ignificantly increased in choroids following retinoic acid treatment.

293 BMP) and WNT signaling combined with FGF and retinoic acid treatments over the course of 18 days gene

294 idation of retinal to the pleiotropic factor retinoic acid using NAD(+).

295 by cell sorting or culturing with all-trans retinoic acid, we measured chemotaxis, intracellular cal

296 Wnt5a and retinoic acid were found to exhibit differential effects

297 t5a and Raldh2, the synthesizing enzymes for retinoic acid, were further analyzed for their function

298 n 4, indicating increased bioavailability of retinoic acid which contributes to differentiation of MD

299 be reversed by the enteral administration of retinoic acid, which induced Tregs and decreased NEC sev

300 Retinoic acids, which are metabolites of vitamin A, have