ライフサイエンスコーパス: retinyl acetate

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1 ene), and M(retinol)+10 (from [(1)(3)C(1)(0)]retinyl acetate).

2 umber of cones that was ameliorated by 9-cis-retinyl acetate.

3 ne the affinity constants of fenretinide and retinyl acetate.

4 dependence and the differentiating power of retinyl acetate.

5 gredient, the 9-cis-retinal precursor, 9-cis-retinyl acetate.

6 cluding retinoic acid, retinol, retinal, and retinyl acetate.

7 A reference dose of [13C10]retinyl acetate (0.4-1.0 mg) in oil was given to each vo

8 g the response to the dose of [(1)(3)C(1)(0)]retinyl acetate (0.5 mg) as a reference, our results (wi

9 9 nmol . d, and the AUC of retinol from 1 mg retinyl acetate (13)C(10) was 161.1 nmol . d.

10 dioactive and nonradioactive form, 4-hydroxy-retinyl acetate (4-OH-RAc), and 5,6-epoxy-RA, all of whi

11 Two oral doses of [(2)H(4)]retinyl acetate [52.4 micromol retinol equivalent (RE)]

12 A reference dose of [13C8]retinyl acetate (8.9 micromol) in oil was given to each

13 Long-term effects of treatment with 9-cis-retinyl acetate (9-cis-R-Ac), an artificial retinoid pro

14 congenital amaurosis with 9-cis-BC and 9-cis-retinyl-acetate, a well established 9-cis-retinal precur

15 orally with 30 micromol hexadeuterated (D6) retinyl acetate (all-trans-19,19,19,20,20,20-[2H6]retiny

16 yl acetate (all-trans-19,19,19,20,20,20-[2H6]retinyl acetate) and then with 37 micromol D6 beta-carot

17 retinoids, including retinoic acid, retinol, retinyl acetate, and other retinyl esters, using postsou

18 hexadeuterated (D(6)) beta-carotene and D(6) retinyl acetate approach.

19 n (DRD) technique at a dose of 5 mg [(2)H(4)]retinyl acetate at baseline and 5 mg [(2)H(8)]retinyl ac

20 al of provitamin A carotenoids by using [2H4]retinyl acetate (d4-RA) as an extrinsic reference standa

21 Although retinol, retinal, and retinyl acetate did not ionize during negative ion elect

22 ctively, whereas retinaldehyde, retinol, and retinyl acetate did not.

23 etinyl acetate at baseline and 5 mg [(2)H(8)]retinyl acetate during the repeat test 1 y later.

24 ma retinol kinetics of an oral dose of [2H4] retinyl acetate in 4 healthy adults (2 men and 2 women)

25 drawn, and subjects consumed 1 mg [(13)C(10)]retinyl acetate in a 0.5-g corn oil capsule and 300 g wh

26 [(1)(3)C(1)(0)]Retinyl acetate in an oil capsule was administered as a

27 low (1.05 mmol), or control (0 mmol) dose of retinyl acetate in oil (n=3 sows per treatment).

28 retinal with oral QLT091001 (synthetic 9-cis-retinyl acetate) in these patients.

29 They received 0.753 mumol [2H4]retinyl acetate/kg body wt orally 9-11 d before surgery.

30 all-trans-retinoic acid (RA), 13-cis-RA, and retinyl acetate] markedly suppressed PMA-mediated increa

31 eriod, the women were given 30 micromol D(6) retinyl acetate orally, followed 1 wk later with 37 micr

32 h the artificial chromophore pro-drug, 9-cis-retinyl acetate, partially protected inferior retinal co

33 ough activation of c-Myc, and that all-trans-retinyl acetate (RAc) independently upregulates tumour n

34 Compared with that of the [13C10]retinyl acetate reference dose (84.7 +/- 34.6 microg x d

35 Compared with the [13C8]retinyl acetate reference dose, spinach tbeta-carotene c

36 from the protonated molecules of retinol or retinyl acetate, respectively.

37 employed for simultaneous quantification of retinyl acetate, retinyl palmitate, alpha-tocopherol and

38 20 microg RE (x+/-SEM) plus 800 microg RE as retinyl acetate supplements for 32 d.

39 cid (all-trans-RA), 9-cis-RA, 13-cis-RA, and retinyl acetate] suppressed both basal levels of Cox-2 a

40 e cleavage of the acetyl ester of vitamin A (retinyl acetate) to all-trans-retinal, also by isoform 1

41 r to that observed in antioxidant- and 9-cis-retinyl acetate-treated mice.

42 A 10-mg oral dose of [2H4]retinyl acetate was administered to 60-81-y-old Guatemal

43 After an oral dose of [2H4]retinyl acetate was administered, plasma samples were co

44 ion for retinoic acid, retinal, retinol, and retinyl acetate were 23 pg, 1.0 ng, 0.5 ng, and 10 ng, r

45 amine), and an artificial chromophore (9-cis-retinyl acetate) were evaluated side by side in a recent

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