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1 attacks, strokes, and the need for arterial revascularisation).
2 urrent ischaemia, or the need for myocardial revascularisation).
3 nfarction, or ischaemia-driven target lesion revascularisation).
4 witch controlling both neuronal survival and revascularisation.
5 roke during the past two decades has been on revascularisation.
6 ifedipine, despite an increase in peripheral revascularisation.
7 failure, debilitating stroke, and peripheral revascularisation.
8 rction, or recurrent ischaemia necessitating revascularisation.
9 ociated with off-pump and on-pump myocardial revascularisation.
10 myocardial infarction, and any target-vessel revascularisation.
11 myocardial infarction, and any target-vessel revascularisation.
12 c events, especially the need for myocardial revascularisation.
13 farction, cardiac surgery, and target-vessel revascularisation.
14 ients needed clinically driven target lesion revascularisation.
15 rtery disease, not suitable for conventional revascularisation.
16 ently recover, either spontaneously or after revascularisation.
17 use death, any myocardial infarction, or any revascularisation.
18 or ischaemia-driven hospitalisation without revascularisation.
19 nt, reinfarction, or unplanned target lesion revascularisation.
20 onary angiography, 58.5% underwent inpatient revascularisation.
21 account when selecting patients for carotid revascularisation.
22 l admission for unstable angina, or coronary revascularisation.
23 efinition that did not include target lesion revascularisation.
24 fit from early and frequent monitoring after revascularisation.
25 history of myocardial infarction or coronary revascularisation.
26 , myocardial infarction, stroke, or arterial revascularisation.
27 lesion or vessel revascularisation, and any revascularisation.
28 rasound follow-up is necessary after carotid revascularisation.
29 sis, myocardial infarction, or target-lesion revascularisation]).
30 0 [0.10-0.84], p=0.004) and those undergoing revascularisation (0.37 [0.15-0.93] p=0.02) after 48 h i
32 .64, 95% CI 0.49-0.84, p=0.001) and coronary revascularisation (0.70, 95% CI 0.52-0.93, p=0.016).
34 1.23) and seemingly also by ischaemia-driven revascularisation (1.16, 0.997-1.34) with bivalirudin co
36 p=0.0003) and ischaemia-driven target lesion revascularisation (5.3% [169 of 3217] vs 3.9% [90 of 230
37 e of coronary artery disease (61%), coronary revascularisation (55%), or a positive stress test only
39 ower rates of ischaemia-driven target lesion revascularisation (9.4%vs 15.1%, -5.7% [-8.6 to -2.7], 0
40 a history of chronic angina with incomplete revascularisation after percutaneous coronary interventi
41 history of chronic angina who had incomplete revascularisation after percutaneous coronary interventi
42 ve the prognosis of patients with incomplete revascularisation after percutaneous coronary interventi
43 99, overall 0.87 0.79-0.96) and for coronary revascularisation alone (0.84, 0.75-0.94) and unstable a
45 se alone, and there was less need for urgent revascularisation and fewer major non-fatal ischaemic co
47 sed in patients undergoing elective coronary revascularisation and reperfusion after acute myocardial
49 days, six patients had undergone peripheral revascularisation and were excluded, and ten withdrew or
50 tion for unstable angina requiring unplanned revascularisation) and in sensitivity analyses alternati
51 mortality, all myocardial infarction, or all revascularisation) and the device-oriented composite end
52 nfarction, or ischaemia-driven target lesion revascularisation) and the primary safety outcome measur
53 ion, 65 (9%) had recurrent ischaemia needing revascularisation, and 100 (14%) had one or more of thes
54 ersus 10% (1.50, 1.04-2.17, p=0.032) for any revascularisation, and 5% versus 2% (2.25, 0.93-5.48, p=
55 l infarction, ischaemia-driven target lesion revascularisation, and all revascularisation did not dif
59 ding cardiovascular mortality and morbidity, revascularisation, and non-traumatic amputation within 5
60 uctions in the incidence of heart attack, of revascularisation, and of ischaemic stroke, with each 1.
63 for combined myocardial infarction, coronary revascularisation, and unstable angina (active treatment
64 failure as the first step towards validating revascularisation as a therapeutic option in heart failu
65 farction, or clinically driven target lesion revascularisation at 4 months FINDINGS: 71 stents, 10-15
66 , myocardial infarction, or ischaemia-driven revascularisation at 48 h) by 19% (3.6% vs 4.4%, OR 0.81
69 ction, or clinically indicated target lesion revascularisation-between the groups at 12 months after
70 imus-Eluting Stent for Percutaneous Coronary Revascularisation (BIOSCIENCE) trial to compare the perf
71 lead not only to recurrent angina and repeat revascularisation but also to acute coronary syndromes.
73 ts were reduced by 36% (-55 to -9), coronary revascularisations by 31% (-59 to 16), and rate of strok
74 tients without heart failure (19%) underwent revascularisation compared with 47 with heart failure (3
75 heart failure, the adjusted HR for death in revascularisation compared with receiving medical therap
76 n death, myocardial infarction, and coronary revascularisation; device and procedural success; and an
77 ven target lesion revascularisation, and all revascularisation did not differ between BVS and CoCr-EE
78 cardial infarction, non-fatal stroke, urgent revascularisation due to unstable angina, and hospital a
80 Study (COSS), a randomised trial of surgical revascularisation for complete carotid artery occlusion
82 (death, myocardial infarction, or unplanned revascularisation for ischaemia), major bleeding, and ne
84 ht be an important adjunct or alternative to revascularisation for patients with hibernating myocardi
85 gy (including early coronary angiography and revascularisation) for non-ST-elevation acute coronary s
86 vely) and clinically indicated target-lesion revascularisation (four cases [1%] vs three cases [2%],
88 f incident myocardial infarction or coronary revascularisation, hospital admission with congestive he
90 ic afterloader can be used to reduce overall revascularisation in patients undergoing treatment for d
91 revascularisation or hospitalisation without revascularisation in patients with a history of chronic
92 percutaneous coronary intervention (PCI) for revascularisation in patients with diabetes and multives
93 rafting (CABG) is the standard treatment for revascularisation in patients with left main coronary ar
95 ischaemic stroke, and the need for coronary revascularisation in people without kidney disease, but
103 th, myocardial infarction, and target-vessel revascularisation occurred in 356 (14.8%) patients who r
104 of 13% (95% CI 7-19; p<0.0001), in coronary revascularisation of 19% (95% CI 15-24; p<0.0001), and i
106 era of stenting and optimum medical therapy, revascularisation of patients with diabetes and multives
108 he carotid arteries (previous carotid artery revascularisation or asymptomatic carotid artery stenosi
109 educe the composite rate of ischaemia-driven revascularisation or hospitalisation without revasculari
110 time to first occurrence of ischaemia-driven revascularisation or ischaemia-driven hospitalisation wi
111 rventional strategy (angiography followed by revascularisation) or a conservative strategy (ischaemia
112 site of death, myocardial infarction, urgent revascularisation, or bailout glycoprotein IIb/IIIa inhi
113 oke, admission for unstable angina, arterial revascularisation, or cardiovascular death (prespecified
114 on to hospital for unstable angina, arterial revascularisation, or cardiovascular death) and the prot
118 ath, myocardial infarction, stroke, coronary revascularisation, or unstable angina; key secondary end
119 ocardial infarction, or repeat target-lesion revascularisation over 290 days compared with 51 [correc
120 t, or stroke or death within 30 days after a revascularisation procedure of the qualifying lesion dur
122 early follow-up MRI scan (within 12 h of the revascularisation procedure) and defined as a more than
125 0.75, 95% CI 0.60-0.94; p=0.01) and arterial revascularisation procedures (284 [6.1%] vs 352 [7.6%];
126 r defibrillator, and who were ineligible for revascularisation procedures were randomly assigned (1:1
127 yocardial infarctions, strokes, and coronary revascularisation procedures) by about one-quarter for e
129 s can be treated successfully using coronary revascularisation procedures, re-occlusion of the treate
134 6-2.51), and had higher urgent target vessel revascularisation rates (66 [4%] vs 21 [1%]; 2.39, 1.23-
135 balloon angioplasty, stenting, and surgical revascularisation should be considered in these patients
136 ifferences in ischaemia-driven target vessel revascularisation, stent thrombosis, or composite advers
137 ntion (PCI) has replaced thrombolysis as the revascularisation strategy for many patients presenting
138 care delivery systems prioritising immediate revascularisation through percutaneous coronary interven
141 longer lesions must be treated with surgical revascularisation to achieve acceptable long-term outcom
142 STICH) trial, PPAR-2 trial and Heart Failure Revascularisation Trial have all reported their results
143 or non-fatal myocardial infarction, coronary revascularisation, unstable angina, and new angina durin
145 opment of a randomised trial of renal artery revascularisation versus medical therapy in heart failur
147 After adjusting for co-variables, inpatient revascularisation was associated with approximately a 30
148 al strategy (routine angiography followed by revascularisation) was better than a conservative strate
149 ry or recurrent, and the need for myocardial revascularisation, was always based on objective electro
150 with critical limb ischaemia unsuitable for revascularisation were enrolled from 171 sites in 30 cou
151 selected for management without [corrected] revascularisation were randomly assigned to clopidogrel
152 lls (MSCs) improves islet graft function and revascularisation, which was associated with the mainten
153 coronary syndromes, managed medically and by revascularisation, who would benefit from tirofiban.
154 ficant reductions in target lesion or vessel revascularisation with BMS compared with PTCA (RR 0.68 [
155 ists in the setting of percutaneous coronary revascularisation with intracoronary stents have shown a
156 o compare clinical outcomes for renal artery revascularisation with medical therapy for renal artery
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