ライフサイエンスコーパス: reversal potential

1 l conductance and ion selectivity (i.e., the reversal potential).

2 ated channels rather than on a change in Cl- reversal potential.

3 esulted from a positive shift of the GABA(A) reversal potential.

4 lutions that unpredictably display a nonzero reversal potential.

5 IL-2 did not alter the reversal potential.

6 7.0 s, n= 18) were recorded, with a similar reversal potential.

7 also be recorded as a leftward shift in the reversal potential.

8 same ionic dependence, inhibitor profile and reversal potential.

9 to 10 mM, and this caused a +30 mV shift in reversal potential.

10 of neuronal depolarization above the GABA(A) reversal potential.

11 tage-independent, and IL-2 did not alter the reversal potential.

12 flow of I:(Ca) when V(m) exceeded the I:(Ca) reversal potential.

13 a transient collapse in the chloride (Cl(-)) reversal potential.

14 and a depolarization of the stimulus-evoked reversal potential.

15 exchange function and abnormal translocator reversal potential.

16 II boundary toward more hyperpolarized GABAR reversal potentials.

17 on GABA-mediated hyperpolarization and GABA reversal potentials.

18 oaded electrodes (predicted GABA(A) receptor reversal potential: 0 mV) at -15 mV revealed the unexpec

19 hloride pipettes (predicted GABA(A) receptor reversal potentials: 0 mV and -80 mV, respectively) dire

20 e in mean open time, unitary conductance, or reversal potential; (2) an increase in charge transfer i

21 body of Up state activity exhibited a steady reversal potential (-37 mV on average) for hundreds of m

22 s readily detected by DeltaFd beyond the ICa reversal potential (+65 to +100 mV) and was not abolishe

23 The current-voltage relationships and the reversal potential (about +10 mV) of the Na3VO4-, pp60c-

24 all drop in input resistance and an apparent reversal potential above spike threshold, facilitating i

25 sms (shifted gamma-aminobutyric acid [GABA]A reversal potential, altered synaptic transmission), ther

26 nduced by odorants, including onset latency, reversal potential and adaptation to repeated stimulatio

27 We determined the reversal potential and conductance change over time duri

28 unted for by changes to the GABA(A) receptor reversal potential and demonstrates an important differe

29 cause dramatic depolarization of the GABA(A) reversal potential and dominating bicarbonate currents t

30 cellular chloride concentration and chloride reversal potential and how these are affected by changes

31 The unitary conductance, reversal potential and mean open time of the single-chan

32 These currents exhibited the reversal potential and pharmacology of a GABAA receptor-

33 inward currents; the currents had a similar reversal potential and slope conductance to their sponta

34 n amplitude between E14 and E18, whereas Cl- reversal potential and synaptic conductances remained re

35 hat are characterized by a leftward shift in reversal potential and the emergence of large outward cu

36 ergic, because they reversed at the chloride reversal potential and were blocked by bicuculline.

37 y down-state events reversed at the chloride reversal potential and were blocked by GABA(A) antagonis

38 racterized by significantly more depolarized reversal potentials and concomitant increases in excitat

39 The reversal potentials and conductances of IPSPs in granule

40 PNP calculations reproduce also experimental reversal potentials and permeability rations in asymmetr

41 ys, for instance, by dictating the GABAergic reversal potential, and thereby influencing neuronal exc

42 ntial ( approximately 8 mV) to GABA receptor reversal potential ( approximately -81 mV), and dampened

43 e relationship between -80 and +60 mV with a reversal potential around 0 mV, a mean open time of 2.6

44 al nonselective cation current of ChR-2 with reversal potential around zero in both mouse OHCs and HE

45 y NA was not accompanied by a change in EPSC reversal potential (around +5 mV), nor were inward curre

46 Ca2+ permeant and external Ca2+ shifted the reversal potential as expected for a channel exhibiting

47 d that of the NMDAsEPSCs was N-shaped with a reversal potential at -5.8 mV.

48 ore is also confirmed by measurements of the reversal potential at oppositely directed salt gradients

49 endritic compartments indicate that the GABA reversal potential at the distal dendrite is more hyperp

50 and sAMPAsEPSCs were outward rectified with reversal potentials at -12.2 mV and -10.8 mV, and that o

51 s was concentration independent, and for the reversal potential-based approaches were of comparable m

52 decrease in input resistance and an apparent reversal potential below spike threshold; consequently,

53 s, either an increase in conductance, with a reversal potential between -58 and +10 mV, or a parallel

54 rent was associated with a positive shift in reversal potential but no change in the kinetics or volt

55 ar charges has little effect on the chloride reversal potential, but greatly affects the effective in

56 on cell holding current, or on outward IPSC reversal potential, but it increased paired-pulse IPSC f

57 eplacement of external Cl- by I- shifted the reversal potential by about -30 mV and lengthened the lo

58 ctification of the ionic current and shifted reversal potential by approximately +10 mV, indicating i

59 The stoichiometry was calculated from the reversal potential by measuring the current-voltage rela

60 oichiometry of kNBC1 was calculated from its reversal potential by measuring the current-voltage rela

61 membrane conductance, and this current had a reversal potential close to the K(+) equilibrium potenti

62 TRH-induced inward current had a reversal potential close to the K+ reversal potential su

63 rizing or hyperpolarizing voltage ramps, had reversal potentials close to 0 mV, exhibited substantial

64 increased slope conductance but no change in reversal potential compared with uninjured neurons.

65 (+) (130 mM) revealed an inward current with reversal potential consistent with the Na(+)/Ca(2+) exch

66 ow a biphasic increase/decrease in Cm with a reversal potential corresponding to the voltage at peak

67 The reversal potential data, obtained from these current-vol

68 Based on reversal potential determinations and recordings with th

69 The reversal potential, determined with asymmetrical hydroge

70 with a reversal potential near the chloride reversal potential E(Cl).

71 re high in TRN neurons, resulting in a Cl(-) reversal potential (E(Cl)) significantly depolarized fro

72 Ps, driven by an extremely negative chloride reversal potential (E(Cl)), combined with a large hyperp

73 neocortical neurons, in order to compare the reversal potential (E(GABA)) and relative density of GAB

74 Here we present novel evidence that the GABA reversal potential (E(GABA)) of PVN presympathetic neuro

75 The GABA reversal potential (E(GABA)) was positive to resting pot

76 oduces a depolarizing shift in the GABAergic reversal potential (E(GABA)).

77 es, activated whole-cell currents that had a reversal potential (E(r)) of about +50 mV in 1.5 mM exte

78 When the mean reversal potential (E(r)) of I(Cl(Ca)) was shifted to ne

79 carbonate cotransporter kNBC1 determines the reversal potential (E(rev)) and thus the net direction o

80 rmeable cation current, which shifted oocyte reversal potential (E(rev)) by +33 mV.

81 Protons shifted the reversal potential (E(rev)) of I(ClC-3) between pH 8.2 a

82 Isoproterenol and forskolin shifted the reversal potential (E(rev)) of I(Na--Ca) by approximatel

83 The alpha1H reversal potential (E(rev)) shifts from +49 mV at pH(o)

84 The lack of change in the reversal potential (E(rev)) upon exposure to Al(3+) sugg

85 onic current associated with this g(m) had a reversal potential (E(rev)) value of -87 +/- 1.1 mV (n=

86 ed age-related alternations in its kinetics, reversal potentials (E(Gly)) and sensitivity to antagoni

87 resting potentials (E(rest)) and junctional reversal potentials (E(rev)).

88 ional modeling, a depolarizing shift in GABA reversal potential (EGABA) and increased intrinsic excit

89 nsequent depolarization of the neuronal GABA reversal potential (EGABA) selectively impairs cortical

90 ted patch recordings, we found that the GABA reversal potential (EGABA) was -73.6 +/- 1.2 mV when ind

91 tsynaptic cells, BDNF induced a shift in the reversal potential (EIPSC) toward more positive levels,

92 ions, there was a spontaneous current with a reversal potential (Er) that was altered by replacement

93 Reversal potential (Erev) ranged from -0.6 to +18 mV.

94 ure by measuring the amplitudes and apparent reversal potentials (Erevs) of inhibitory responses evok

95 to K(+), as judged by the 51-55 mV shifts in reversal potential following a 10-fold change in [K(+)](

96 Furthermore, the GABA(A) channel reversal potential for chloride ions was positive to the

97 In this scenario, the reversal potential for Cl(-) does not closely follow tha

98 hat at any value of these fixed charges, the reversal potential for Cl(-) equates that of K(+).

99 eases KCC2 expression and hyperpolarizes the reversal potential for Cl(-).

100 r was calculated in experiments in which the reversal potential for Cl- (ECl) was measured from the G

101 ronal K+/Cl- cotransporter KCC2 to shift the reversal potential for Cl- and thus alters the effective

102 induce persistent changes in neuronal E(Cl) (reversal potential for Cl-) did not alter vmax or Km of

103 ane translates to a hyperpolarization of the reversal potential for GABA (EGABA).

104 orated patch recordings demonstrate that the reversal potential for GABA is more depolarized in mutan

105 e resting membrane potential relative to the reversal potential for GABA(A) receptors, the hyperpolar

106 Seizures induce excitatory shifts in the reversal potential for GABA(A)-receptor-mediated respons

107 chloride homeostasis mechanisms that set the reversal potential for GABA(A)Rs, or by a change in the

108 ellular concentration of Cl- and, hence, the reversal potential for GABA.

109 CO3(-) measurements from the damselfish, the reversal potential for GABAA (EGABA) was calculated, ill

110 The reversal potential for glycine (E(gly)) can be hyperpola

111 blockers, by recording gating current at the reversal potential for ionic current (+50 mV).

112 ivation also induced a negative shift of the reversal potential for ionic currents suggesting that in

113 ulate neurons, this effect reversed near the reversal potential for K+, suggesting that it is mediate

114 nactivation kinetics of Na+ currents nor the reversal potential for Na+.

115 tage-clamp hyperpolarization negative to the reversal potential for NBCe failed, however, to terminat

116 odulation of KCC2 function will regulate the reversal potential for synaptic GABAergic inputs, thus s

117 (k and E1/2, respectively), and extrapolated reversal potential for the chord conductance (Erev).

118 r that alters intracellular chloride and the reversal potential for the GABAAR.

119 The reversal potential for the outward current measured usin

120 In addition, the reversal potential for these responses closely approxima

121 The reversal potentials for IH were -39 +/- 4 mV for A-type

122 The similarities in reversal potentials for the synaptic hyperpolarization e

123 sformation was accompanied by a shift of the reversal potential from that of chloride toward that of

124 than 15-fold under physiological conditions, reversal potential further decreased by another approxim

125 orated patch-clamp techniques to measure Cl- reversal potential, GABAergic synaptic responses, and vo

126 PAR GluA2 content and others in the chloride reversal potential, human stem-cell-derived neurons repr

127 Lastly, the calculated reversal potential in a tenfold salt gradient (0.1:1M KC

128 Finally, measurements of the GABA reversal potential in different starburst dendritic comp

129 al for Cl- (ECl) was measured from the GABAA reversal potential in low-HCO3- media during Cl- loading

130 The lysine mutant had a 25-mV reversal potential in solutions with symmetrical Cl(-) a

131 The reversal potential in the PP is only slightly more posit

132 discernable effect on the Cl(-) current, the reversal potential in the presence or absence of Cl(-)(o

133 tial decrease in MRC size and a shift in the reversal potential in vivo.

134 kedly different rectification properties and reversal potentials in coronary compared to mesenteric a

135 This was deduced from measurements of reversal potentials in pH gradients across planar lipid

136 Neither a significant shift in reversal potential (in voltage clamp mode) nor a change

137 determined by the permeability ratios (i.e., reversal potentials) in biionic conditions.

138 Measurement of reversal potentials indicated that the channel has a sim

139 e in mean open time, unitary conductance, or reversal potential, indicating an increase in n and/or P

140 GABA with currents which have unusually high reversal potentials, indicating that GABA may be excitat

141 ard citrate current had a markedly different reversal potential, ionic characteristics, inhibitor pro

142 by the fact that inward Ca2+ flux at the ICa reversal potential is exactly balanced by outward Cs+ cu

143 3) The reversal potential is more positive with Ca(2+) than wit

144 y under certain conditions in which the GABA reversal potential is shifted positive due to intracellu

145 of the resting potential and the inhibitory reversal potentials, is regulated together with extracel

146 parameters, including maximal conductances, reversal potentials, kinetics of ionic currents, measure

147 dence, strong inward rectification, positive reversal potential, limited cesium permeability, and sen

148 to Na+ permeability ratio (PCl/PNa) from the reversal potential measured in a 10-fold NaCl gradient.

149 of K(+) to Cl(-) was determined as 0.33 from reversal potential measurements in KCl gradients.

150 using fractional Ca(2+) currents (P(f)) and reversal potential measurements over a wide voltage and

151 Reversal potential measurements showed that the human P2

152 Ion substitution experiments and reversal potential measurements suggested that the carba

153 Based on single-channel reversal potential measurements, NMCCs are slightly more

154 The IPSP had a reversal potential near -70 mV and was blocked by the GA

155 uisqualate-induced current was linear with a reversal potential near 0 mV suggesting involvement of n

156 ceptors, that are strongly rectifying with a reversal potential near 0 mV.

157 of their flash responses was linear, with a reversal potential near 0 mV.

158 0 mV, activated on depolarization, and had a reversal potential near 0 mV.

159 5 microM) or cadmium (100 microM), and had a reversal potential near E(Cl), indicating that they were

160 e I(RC)-V(Cone) relations are linear, with a reversal potential near the chloride reversal potential

161 e potassium (K(ATP)) channels since it had a reversal potential near the equilibrium potential for K(

162 tions of 5-HT, is inwardly rectifying with a reversal potential near the equilibrium potential for K+

163 sociated with a decreased conductance with a reversal potential near the K(+) equilibrium potential.

164 0.10 +/- 0.03 pA pF(-1) (rabbit, n= 9), with reversal potentials near -100 mV, consistent with N= 2.

165 ion, since neither NMDA current magnitude or reversal potential, nor the levels of NR1-NR2A-D subunit

166 slope conductance of 17.0 +/- 3.2 pS, and a reversal potential of +7 +/- 4 mV (n = 9).

167 smic N-terminal domain (Nt) of NBCn1-B had a reversal potential of -156.3 mV (compared with a membran

168 nward at membrane potentials negative to its reversal potential of -30 mV, in 10 of 24 cells tested,

169 vating in the diastolic potential range with reversal potential of -37.5+/-1.0 mV, confirming the exp

170 ship was slightly inwardly rectifying with a reversal potential of -52 +/- 2 mV, and the P(K)/P(Na) r

171 s a slow membrane hyperpolarization toward a reversal potential of -73 mV through a relatively small

172 P) conductance was 14.0 +/- 1.5 nS and had a reversal potential of -91.4 +/- 0.9 mV that shifted by 5

173 HT activated a 4-AP-sensitive current with a reversal potential of -95 mV in these axons.

174 ates of about 18, 34 and 51 and 68 pS, and a reversal potential of 0 mV.

175 l conductance, maximum open probability, and reversal potential of AMPA receptors and did not find an

176 linear steady-state I-V relationship with a reversal potential of approximately -110 mV.

177 amate-gated anion conductance in cones has a reversal potential of approximately -30 mV compared with

178 ed a significant quasi-linear current with a reversal potential of approximately -40 mV.

179 P2X receptors was 12.3 as estimated from the reversal potential of ATP-induced current measured at di

180 amplifier, is accompanied by a shift of the reversal potential of BAS-CA1 postsynaptic potentials, a

181 versed at approximately -20 mV, close to the reversal potential of chloride, but treatment with dithi

182 pH 5/pH 8 gradient across the membrane, the reversal potential of colicin A is -21 mV, rather than 0

183 A inward/outward currents decreased, and the reversal potential of composite NMDA currents recorded i

184 tion and caused a depolarizing shift in GABA reversal potential of dorsal horn neurons.

185 potential of improved ChloC (iChloC) to the reversal potential of endogenous GABAA receptors.

186 ely followed the time course of the combined reversal potential of excitation and inhibition.

187 While the reversal potential of GABA(A) receptor-mediated currents

188 rization in 40% of neurons at P0-P2, but the reversal potential of GABA-evoked currents (E(GABA)) was

189 of hippocampal neurons led to a shift in the reversal potential of GABA-induced Cl- currents (E(Cl))

190 ter KCC2, as confirmed by the changes in the reversal potential of GABA-induced currents and the rest

191 tracellular Cl- levels, which determines the reversal potential of GABAAR-mediated currents and is in

192 The correlation between reversal potential of GABAergic currents (EGABA) and NH4

193 served to have a linear correlation with the reversal potential of GABAergic currents.

194 erologous expression system to show that the reversal potential of GAT-1 under physiologically releva

195 tion and induces a depolarizing shift in the reversal potential of glycine-mediated currents (E(glyci

196 The reversal potential of I(Cl(Ca)) was close to the theoret

197 The reversal potential of I(h) was -29 mV.

198 hyperkalemia-mediated depolarization of the reversal potential of I(K1) (E(K1)) would reduce excitab

199 d patch clamp and there was no change in the reversal potential of IKr in the presence of isoprenalin

200 d substitutions that significantly shift the reversal potential of improved ChloC (iChloC) to the rev

201 2A receptors to serotonin hyperpolarizes the reversal potential of inhibitory postsynaptic potentials

202 Furthermore, the reversal potential of IPSCs, which was not significantly

203 ociated with a negative or positive shift of reversal potential of IPSPs (E(IPSP)).

204 e cell resting potentials estimated from the reversal potential of K+ currents through a cell-attache

205 The reversal potential of OPC GABA(A) currents was -43 mV, a

206 e amplitude, kinetics, slope conductance and reversal potential of synaptic inputs in a dendritic dis

207 The reversal potential of the 20 pS channel was estimated to

208 The reversal potential of the ACh-activated inward current w

209 The reversal potential of the Bk-induced inward current was

210 ternal K+ concentration led to shifts in the reversal potential of the Ca2+-dependent current as pred

211 a time- and frequency-dependent shift in the reversal potential of the composite postsynaptic current

212 The reversal potential of the compound postsynaptic currents

213 at this function can be achieved only if the reversal potential of the cotransporter is negative to t

214 The reversal potential of the current, estimated by a novel

215 rding in bicarbonate-free buffer changed the reversal potential of the GABAd response significantly,

216 rent are Na+ and Cl- dependent; however, the reversal potential of the induced current suggests a Na+

217 The mean reversal potential of the inhibitory current was -78 +/-

218 A negative shift in the reversal potential of the instantaneous current under hi

219 5 mm instead of 5 mm Cl- failed to shift the reversal potential of the inward current, indicating tha

220 The reversal potential of the MOD-1 channel is dependent on

221 performed experiments to assess whether the reversal potential of the Na+-Ca2+ exchanger (ENa-Ca) wa

222 The reversal potential of the slow AHP was sensitive to chan

223 The mean reversal potential of the steady-state current was -21.2

224 In neurons that could be clamped at the reversal potential of their outward currents, the model

225 However, contrary to expectations, the reversal potential of this swelling-activated current sh

226 gradients, lowered pH(o) largely shifts the reversal potential of TWIK-1, TASK-1, and TASK-3 K(+) ch

227 The reversal potential of type I I5-HT,inward shifted to abo

228 r Cl(-) concentration was estimated from the reversal potential of whole-cell currents evoked by loca

229 r isotonic conditions positively shifted the reversal potential of whole-cell currents.

230 lative Ca(2+) permeability measured from the reversal potentials of ATP-gated currents was unaffected

231 Reversal potentials of currents elicited by gamma-aminob

232 Reversal potentials of GABA responses (E(GABA)) were det

233 at a holding potential intermediate for the reversal potentials of GABA(A) and P2X receptors, little

234 It is shown here that hyperpolarizing reversal potentials of GABA(A)ergic postsynaptic current

235 The reversal potentials of inward and outward currents were

236 The reversal potentials of the spontaneous and ATP-generated

237 The reversal potentials of these conductance changes ranged

238 ion independent when derived from changes in reversal potentials on going from a Na(+) reference solu

239 when P(Ca)/P(Na) was derived from changes in reversal potentials on going from a Na(+) reference solu

240 zed gamma-Aminobutyric acid receptor (GABAR) reversal potential or co-activation of alpha-amino-3-hyd

241 ith no change in single-channel conductance, reversal potential or mean open time.

242 r, there are no data available on either the reversal potential or the HCO3-:Na+ stoichiometry of pNB

243 hair cells were depolarized near the Ca(2+) reversal potential or their hair bundles were exposed to

244 Rs, with little or no change in conductance, reversal potential, or mean open time.

245 nd outside the cell greatly affect the Cl(-) reversal potential, particularly when osmolar transmembr

246 nine, and currents showed a 58.5 mV shift in reversal potential per 10-fold change in [Cl-], consiste

247 The method is insensitive to changes in the reversal potential, pipette capacitance, or widely varyi

248 n perforated-patch recording revealed a GABA reversal potential positive to both the resting membrane

249 These data indicate that IPSPs with reversal potentials positive to spike threshold may have

250 active during interspike intervals and have reversal potentials positive to threshold.

251 on and deactivation kinetics) or permeation (reversal potential) properties.

252 l phase, whereas the muscimol-evoked GABA(A) reversal potential remained unchanged.

253 , as resting membrane potential and the IPSC reversal potential remained within a few millivolts (1-4

254 Ca2+ influx through NMDARs as determined by reversal potential shift analysis and by a combination o

255 The reversal potential shifted 26 mV in response to a 10-fol

256 mV; the response was further reduced and the reversal potential shifted to -90 mV in a low-Na+, high-

257 was substituted with glutamate or I(-), the reversal potential shifted to more positive or more nega

258 Reversal potential shifts in response to changing [NH3]o

259 Measurement of current reversal potentials showed the relative BzATP-induced pe

260 ent had a reversal potential close to the K+ reversal potential suggesting that TRH inhibits resting

261 daily changes in K(+) currents and the GABA reversal potential, suggesting a role in modifying membr

262 high correlation between up- and down-state reversal potential suggests that despite these drastic c

263 The more negative reversal potential than E(K)(+) (-90 mV) was caused by t

264 These glycinergic IPSCs showed a reversal potential that varied with changes in [Cl-]i, a

265 tward-rectifying total leak conductance with reversal potential that was depolarized by approximately

266 substantial positive shifts in the GABAergic reversal potential that were proportional to the charge

267 Based on measurement of reversal potential the selectivity of SLC26A7 is NO(3)(-

268 either the membrane resistance or the Na(+) reversal potential, the conductance and the permeability

269 sociated with KORC (analysis of tail current reversal potentials), there is no correlation between Ca

270 nt and spatial spread of changes in the GABA reversal potential thereby altering homosynaptic as well

271 evoked GABA-A IPSPs and hyperpolarizes their reversal potential through a postsynaptic change in Cl(-

272 ased the amplitude of I(LVA) and shifted the reversal potential to +22 mV.

273 D-glucamine inhibited I(LVA) and shifted the reversal potential to -7 mV.

274 bicarbonate exposure produced a shift in the reversal potential to more negative potentials, consiste

275 attenuated Ca2+ permeability measured using reversal potentials under biionic conditions and fractio

276 Na(i,o)(+), long depolarizations shifted the reversal potential (V(R)) toward E(Na).

277 g Na(+) with K(+) caused a leftward shift in reversal potential (V(Rev)) that correlated with the cor

278 idin-perforated-patch method and found their reversal potential (V(rev)) to be depolarized relative t

279 ated strongly with the positive shift of the reversal potential (V(rev)) upon switching to a sodium-f

280 Tail current reversal potentials varied with extracellular K+ concent

281 ctivation significantly without changing its reversal potential, voltage dependence of activation, or

282 The PA current reversal potential (Vr) did not follow the equilibrium K

283 currents had a unitary conductance of 23 pS, reversal potential (Vr) of +10 mV and a low open probabi

284 The reversal potential (Vrev) for OCs shifted with changes i

285 gave a positive (depolarizing) shift in the reversal potential (Vrev, equivalent to the membrane pot

286 The reversal potential was +11 mV which was shifted to more

287 synaptic terminal; V(1/2) was -94 mV and the reversal potential was -33 mV.

288 ctance was 10.4 +/- 0.4 picosiemens (pS) and reversal potential was 0.2 +/- 1.7 mV.

289 uced current was inwardly rectified, and the reversal potential was dependent on external potassium c

290 The IPSC reversal potential was determined by gramicidin perforat

291 occurred regardless of whether the chloride reversal potential was hyperpolarizing (ECl-=-70 mV) or

292 ard current at potentials positive to the K+ reversal potential was observed through Kir3.1/Kir3.4, b

293 d with Cs+-filled pipettes, the outward IPSC reversal potential was shifted to -76 mV, closer to the

294 GABAA receptor reversal potential was unaffected by time of day or estr

295 type channels, while activation kinetics and reversal potential were not critical parameters.

296 Maximum conductance and reversal potential were unchanged.

297 For both neuronal groups, the sEPSCs reversal potentials were around 0 mV and there were no s

298 r K+ was replaced with Cs+, IPO tail current reversal potentials were dependent upon the extracellula

299 cs of current activation, rectification, and reversal potentials were unaltered.

300 influx by depolarization to above the I(Ca) reversal potential, with high intracellular Ca(2)(+) buf