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1 vivo, whereas knockdown of GPC5 was able to reverse the effect.
2 of the Notch Intracellular Domain (NICD) can reverse the effect.
3 dition of high molecular weight HA failed to reverse this effect.
4 cocaine-seeking behavior induced by OrxA but reversed DynA effect.
5 n fluvastatin sensitivity; knocking out ZEB1 reversed this effect.
6 bsequent addition of ketotifen significantly reversed this effect.
7 irus production, while CCL2 immuno-depletion reversed this effect.
8 erior Insular Cortex, while deep hypothermia reversed this effect.
9 e, whereas inhibition of miR-301a completely reverses the effects.
10 le's growth; interfering with Yap's activity reverses this effect.
11 ial function, as inhibition of NOS partially reverses this effect.
12 T1AR internalization and that MDM2 knockdown reverses this effect.
13 in phosphatases PP-1 and PP-2B (calcineurin) reverses this effect.
14 emia and identify dietary interventions that reverse these effects.
15 that treatment of edema has the potential to reverse these effects.
16 85.9%), accounting for more than half of the reverse triage effect.
17 be blocked by a small molecule predicted to reverse the tau effect.
18 in UM-UC-3 or ASS1 silencing in RT112 cells reversed these effects.
19 ChemR23 downregulation reversed these effects.
20 caveolin-1 scaffolding domain peptide (CSP) reversed these effects.
21 AGEs or RAGE deletion in hepatocytes in vivo reversed these effects.
22 uated, and anti-heparanase or PI3K activator reversed these effects.
23 at 22d after HI, while wortmannin partially reversed these effects.
24 K27 methylation homeostasis in hypoxic cells reversed these effects.
25 on of Plin5 in the livers of Plin5(LKO) mice reversed these effects.
26 saggregases, or overexpression of kinesin-1, reverses these effects.
27 tests, and genetic deletion of hepatic FoxO1 reverses these effects.
28 COS women and oestradiol (EE) administration reverses these effects.
29 s, and restoration of mitochondrial function reverses these effects.
30 ond NMR coupling constants in ethers and the reverse anomeric effect.
31 th reduction is normally expected due to the reverse Hall-Petch effect.
32 , whereas damage to structure B produces the reverse pattern of effects.
33 f the active vitamin D metabolite calcitriol reversed all these effects.
34 t with mannitol after traumatic brain injury reversed all these effects.
35 a well-described KLF4 target, did not fully reverse KLF4-mediated effects.
36 her pharmacological upregulation of KLF2 can reverse the radiation effects.
37 All of these changes were reversed by blocking TSP1 effects.
38 h VKAs and LMWH, there are no antidotes that reverse their anticoagulant effect.
39 ht loss via dietary modification effectively reverses these deleterious effects.
40 Knockdown of Nrf2 expression reversed these IER3-dependent effects.
41 o identify brain changes that do not reflect reverse causation or treatment effects.
42 es and trial lenses induce indistinguishable reverse Pulfrich effects, (2) anti-Pulfrich corrections
43 drenoceptor antagonist (BRL 44408) partially reversed the oxytocin effect, a descending noradrenergic
47 n immunosuppressive cytokine, TGFbeta, might reverse these effects and thereby potentiate oHSV effica
48 and programmed cell death 1 (PD-1) partially reverse this effect and are becoming standard of care in
50 SH-6 restored splicing factor expression and reversed both hormone staining effects and patterns of g
51 lenishing infected cells with exogenous heme reversed these effects and restored NAD(P)H oxidase acti
52 at the naturally occurring statin mevastatin reverses FPP's effects and promotes healing by using in
53 n of RASSF1A or inhibition of P4HA2 activity reverses these effects and increases markers of lung dif
54 netic inactivation of IMPDH2 in glioblastoma reverses these effects and inhibits cell proliferation,
56 lipoxin A4 receptor with the antagonist Boc2 reversed this effect, and treatments were ineffective in
58 expression in the NAc shell (NAcsh) not only reversed these cocaine-induced effects but also downregu
61 of the quantum anomalous Hall effect can be reversed via field-effect control of the chemical potent
62 o fluorite-structure binary oxides-in which 'reverse' size effects counterintuitively stabilize polar
65 ntly marketed KV 7 channel opener, partially reversed these effects for the majority of analyzed muta
66 y is suppressed, and this inhibition must be reversed to engender tissue effects; however, the mechan
67 overexpression in the PFC of these HET mice reversed the antidepressant-like effect in SPT and TST.
68 ade of the synthesis of 20-HETE with HET0016 reversed the renoprotective effects in SS.5(BN) rats.
70 d chains on the islands, adding both species reversed this effect-in part because the apex predators
71 ersely proportional, whereas NOS1 deficiency reverses this effect, increasing leak and elevating reac
72 oncogenic kinase that stimulates glycolysis, reversed these effects, indicating that metabolism of py
73 rial treatment using BRAF and MEK inhibitors reversed the developmental effects induced by BRAF(V600E
78 was confirmed by depletion experiments that reversed the tumor inhibitory effects observed in C3aR-d
79 mpact of these other factors likely reflects reverse causation-namely, downstream effects of early ad
80 ith the alpha2delta-1 receptor, and thus may reverse or ameliorate the effects of AIE on hippocampal
81 ged animal to young blood can counteract and reverse pre-existing effects of brain aging at the molec
82 nt to alleviate depression-like behavior and reverse the adverse effects of unpredictable chronic mil
83 as mDia1-interacting domains of Prohibitin2 reverse the anti-myogenic effects of mDia1DeltaN3, while
84 of intravenous idarucizumab would be able to reverse the anticoagulant effect of dabigatran in patien
86 med to using vitamin K if there is a need to reverse the anticoagulant effect of vitamin K antagonist
87 intravenous idarucizumab and its capacity to reverse the anticoagulant effects of dabigatran in patie
91 onclusion of carotid endarterectomy (CEA) to reverse the anticoagulant effects of heparin and to limi
92 aspartate supplementation was sufficient to reverse the antihypertrophic effect of ACC2 deletion dem
93 at cranial transplantation of stem cells can reverse the deleterious effects of chemobrain, through a
94 udy, we determined ifmaternal exercise could reverse the detrimental effects of maternal high-fat fee
95 receptor antagonists can partially or fully reverse the detrimental effects of Tat, glutamate recept
97 RNA-induced cell-cell adhesion but failed to reverse the effect of Galpha13 siRNA on proteolytic inva
98 tricted oxytocin receptor antagonist did not reverse the effect of intranasal oxytocin on alcohol dri
102 contra-lesional and sham cerebellar rTMS to reverse the effects of a 'virtual-lesion' in health.
103 icated on identifying molecular targets that reverse the effects of aging in vulnerable regions of th
104 ly, irrigation can regionally cancel or even reverse the effects of all other forcings combined.
106 ated platelets was sufficient to efficiently reverse the effects of ibrutinib, and platelet functions
107 witching offspring to a healthy diet did not reverse the effects of maternal WSD on muscle insulin ac
108 afe and effective opiate antagonist that can reverse the effects of overdose and minimizing the delay
110 hrine uptake inhibitor desipramine failed to reverse the effects of TBZ, and higher doses of these dr
112 xanet and ciraparantag have been reported to reverse the effects of the anti-Xa NOACs (rivaroxaban, a
114 natives during invasion could weaken or even reverse the negative effects of exotic-native phylogenet
115 deficiency in IL-4 and IL-10 was required to reverse the negative effects of helminth coinfection on
116 LP diets with branched-chain AA (BCAA) would reverse the negative effects of these diets on growth an
118 g SMCs with rexinoids would more effectively reverse the pathophysiologic effects of angiotensin II t
119 leeding when it does occur with NOACs and to reverse the pharmacological effect of these agents if ne
121 IL-6, IL-8, and CXCR1 signaling pathways to reverse the proapoptotic effect of the IL-32gamma isofor
122 important given the potential to mitigate or reverse the side effects of immunosuppressive medication
123 d activation of mPT were found to mirror and reverse, respectively, the antiseizure effects of the KD
125 y, we also determined that PLN-alen not only reversed protumoral effects of the PLN carrier, but also
126 Importantly, maternal ADN supplementation reversed the adverse effects of maternal obesity on plac
129 atus, and that suppression of AhR expression reversed the anti-proliferative effects of flutamide.
133 rs of phagosome and autophagosome maturation reversed the beneficial effect of statins on bacterial g
134 ic oxide synthase inhibitor to FoxO4 KO mice reversed the beneficial effects of FoxO4 deletion on pos
135 Additionally, cell-autonomous PDF signaling reversed the circadian behavioral effects of lowered Ral
136 cetyl cysteine attenuated ROS production and reversed the cytotoxic effects of K-Ras(G12V) in the TKO
138 malized monocyte/macrophage polarization and reversed the detrimental effects of hyperglycemia, sugge
145 nhibitors of ERK and AKT pathways completely reversed the effect of GHRH-R agonists on CSC proliferat
150 but not beta2, subunit knockdown effectively reversed the effect of increased ACh signaling in a mous
151 sion of a catalytically inactive Shp2 mutant reversed the effect of ITSN1 on Spry2 dephosphorylation
152 cells expressing mutant NRas with trametinib reversed the effect of mutant NRas expression by restori
153 ogical blockade of CRF1 receptors in the VTA reversed the effect of nicotine on GABAergic input to do
155 verexpression of several proteasome subunits reversed the effect of UBE3A(T485A) on Wnt signaling.
156 effect of AC whereas remethylation of AC DNA reversed the effects of activation and restored the abil
157 s Tap1 and Tap2 The PSMB8 inhibitor ONX-0914 reversed the effects of BPTF ablation, consistent with a
159 complex or inhibition of Fyn only partially reversed the effects of CD36 on hepatic insulin signalin
160 Moreover, we showed that a CD27 agonist Ab reversed the effects of CD70 ablation, indicating that t
164 which can function downstream of DDR1, also reversed the effects of Galpha13 knockdown on cell-cell
165 at also co-localizes to cell-cell junctions, reversed the effects of Galpha13 knockdown on cell-cell
166 pharmacologic manipulation of AC reduced or reversed the effects of HC on AC activity (THAM 103%, HC
167 nd the SK2 specific blocker apamin partially reversed the effects of increased NeuroD2 expression on
168 ion of KCNQ channels altered cell firing and reversed the effects of M1R compounds, suggesting that K
169 tumor formation, and ectopic MYCN partially reversed the effects of MDM2 depletion, indicating that
173 of ULK1 (by MRT68921) or autophagy (by 3-MA) reversed the effects of mTOR inhibition, leading to incr
178 res induced expression of ductal markers and reversed the effects of Slug by inducing ductal structur
181 Inhibition of STAT1, STAT3, STAT5, or AKT reversed the effects of TC-PTP overexpression on epiderm
182 ated with pharmacologic interventions, which reversed the effects of the extravasated vasopressors: i
183 of an oncogenic Kras p.G12D mutation in mice reversed the effects of the MSC2504877/palbociclib combi
187 that both IFNalpha and IFNbeta (type I IFNs) reversed the immunosuppressive effect of MSCs on splenoc
188 nted 5-HT-induced phosphorylation of Mypt-1, reversed the inhibitory effect of 5-HT on efferocytosis,
190 analogue 1-oleoyl-2-acetyl-sn-glycerol (OAG) reversed the inhibitory effect of candesartan, while thi
192 In addition, siRNA interference of FBXW7 reversed the inhibitory effect of MAGEA1 on migration an
193 tative KChIP2-interacting residues of DPP6-L reversed the inhibitory effect of NS5806 into potentiati
194 ns, increased Fos in CeM output neurons, and reversed the inhibitory effect of social choice-induced
195 ly, acetylation at K6, but not at K9 or K15, reversed the inhibitory effect of T3 phosphorylation.
196 cytes, and the addition of a STAT5 inhibitor reversed the inhibitory effect of the CB2 agonist on IL-
197 luc cells delivered with miR-135 and miR-203 reversed the inhibitory effect of the miRNAs on tumor gr
198 rofen arginate but not ibuprofen sodium also reversed the inhibitory effects of ADMA and N(G)-nitro-l
200 diate alpha-ketoglutarate to the Rb TKO MEFs reversed the inhibitory effects of glutamine deprivation
201 OCK1/2 activity in Slug-expressing Kras mice reversed the inhibitory effects of Slug on ADM, ERK1/2 p
202 P = 0.011) or contra-lesionally (P = 0.005), reversed the inhibitory effects of the cortical 'virtual
203 ed drug delivery in various tumor models and reversed the negative effect of VEGF ablation on drug de
204 sion in the brain through carbogen breathing reversed the neuroprotective effects of FLASH, while rad
207 4, a LXA4 receptor antagonist, significantly reversed the protective effect of MSCs on both survival
208 Targeting the ErbB3/ErbB2 pathway partially reversed the protective effects of fibroblast/CAF-derive
209 itor or Zileuton, a 5-LOX inhibitor with VPC reversed the protective effects of VPC against neuroinfl
210 Moreover, TNPO1 overexpression partially reversed the repressive effect of miR-128 on L1 retrotra
211 ROCK target, and inhibitors of RhoA and ROCK reversed the suppressive effect of 5-HT on efferocytosis
213 all molecule agonist directed against TRPML1 reversed the toxic effects of VacA on endolysosomal traf
218 of GLUD2 into murine glioma progenitor cells reverses deleterious effects of IDH1 mutation on metabol
221 n of CD47 by TSP1 or loss of CD47 expression reverses some inhibitory effects of VEGF on proliferatio
226 ild-type and ob/ob mice, and also completely reverses the deleterious effects of the Cdk5 ablation.
228 Addition of heme to NSCLC cells partially reverses the effect of CycT on oxygen consumption, proli
231 , has identified mechanisms whereby ketamine reverses the effect of stress, but little is known regar
234 s, whereas warming to 39 degrees C partially reverses the effects of hypothermia on monocyte function
235 Suppression of PGC-1beta and PRC with siRNA reverses the effects of IGF-1 and disrupts mitochondrial
236 s a naturally occurring NMDAR modulator that reverses the effects of NMDAR antagonists in animal mode
238 (1)-pyrroline-5-carboxylate (P5C) or proline reverses the effects of P5C synthase knockdown but not P
239 inhibition of calcineurin and NFAT signaling reverses the effects of phenformin on keratinocyte diffe
241 -1 receptor (Sig-1R), a molecular chaperone, reverses the pathological effects of (G4C2)-RNA repeats
242 JAK/STAT3 pathway by a JAK inhibitor AZD1480 reverses the pro-metastatic effect of sevoflurane and th
247 part human disorder but more importantly for reversing the adverse effects of the mutant gene using g
248 n glucocorticoid-treated macrophages without reversing the anti-inflammatory effects of glucocorticoi
249 changes in neurons, a potential strategy for reversing the deleterious effects of DOX treatment.
250 tatory neurostimulation protocols capable of reversing the disruptive effects of focal cortical inhib
251 onoclonal antibody fragment idarucizumab for reversing the effects of dabigatran, the investigational
254 rformance in humans, perhaps forestalling or reversing the effects of neurodegeneration in aging.
255 ine transmission are relatively effective at reversing the effort-related effects of TBZ, and are con
256 d PMEPs bilaterally (F1,14 = 7.4, P = 0.017) reversing the inhibitory effects of 1 Hz rTMS in the pre
257 obin, improving systemic oxygen delivery and reversing the inhibitory effects of CO on mitochondria.
259 recurrent mitral regurgitation, and improved reverse remodeling without adverse effect on left ventri
261 ter in development, CD40-Fc, which activates reverse signalling, had no effect on early sensory axon
262 o the direct activator of AMPK, ZMP, did not reverse the effects on TNF-alpha-induced CFB expression,
265 tration of edoxaban (60 mg) dose-dependently reversed edoxaban's effects on bleeding duration and end
269 pretreatment with clopidogrel (n = 3) partly reversed the detrimental effects on SEC injury and there
271 ed by peripheral stimulation and LHb lesions reversed the inhibitory effects on cocaine locomotion pr
272 full leptin-receptor antagonist, completely reversed the leptin-mediated effects on cell growth and
273 vation of PPARgamma by Rosiglitazone (10 uM) reversed the LPS-mediated effects on inflammatory cytoki
275 olinesterase inhibitor, donepezil (Aricept), reverses AIE effects on dendritic spines, possibly by in
276 indicated that TGF-beta pathway attenuation reverses carbofuran's inhibitory effects on neurogenesis
277 al administration of ketamine (2.5-20 mg/kg) reverses CSDS-induced effects on reward or, in separate
278 e in Ras GTPase activating protein activity, reverses miR-431's effect on promoting invasion, Erk pho
279 XE991 or the KCNQ channel opener retigabine reverses the effects on consolidation caused by manipula
280 tophagy and induces neuronal damage, and NAC reverses these deleterious effects on mitochondrial func
281 nduces candidal resistance to fluconazole by reversing the antifungal's effect on the ergosterol bios
282 even 24 h after acute stress, is capable of reversing the delayed enhancing effects on BLA synaptic
284 sion; however, this treatment demonstrated a Reverse Warburg effect phenotype observed in cancer-asso
286 d viability of Huh7.5A2 cells, PD-1 blockade reversed this effect, producing enhanced cytolytic elimi
287 ly, combining antioxidant treatment and LDIR reverses this effect, promoting wild-type cell prolifera
288 trophils, whereas the presence of tryptophan reversed this effect, providing a possible mechanism for
290 ve, and enhancing B cell Ab responses, could reverse the immunosuppressive effects resulting from tob
292 the histone H3K4 methyltransferase Trithorax reverses these effects, suggesting that an Lsd1/CoRest c
293 with bevacizumab (BEV, anti-VEGF), it could reverse the adverse effects that precipitate fibrotic ch
294 nhibition of the protein kinases Akt or mTOR reversed the IL-7 effect, thereby restoring the function
295 knockdown of OGT by RNA interference (RNAi) reverses this effect, thereby opening up the opportunity
296 n and to determine whether it is possible to reverse these effects through nutritional interventions
299 tentially toxic chemosensitizing agents that reverse MDR effects, which has raised expectations in th