ライフサイエンスコーパス: rheumatic fever

1 a tuberculosis clinic for reasons other than rheumatic fever.

2 complicated pharyngitis, impetigo, and acute rheumatic fever.

3 f carditis as a major manifestation of acute rheumatic fever.

4 sses and 2 (0.28%) were diagnosed with acute rheumatic fever.

5 tries with the added factors of SH, C'D, and rheumatic fever.

6 skin infection and pharyngitis-induced acute rheumatic fever.

7 logic manifestation of streptococcal-induced rheumatic fever.

8 ly with a decrease in the incidence of acute rheumatic fever.

9 in timely fashion in order to prevent acute rheumatic fever.

10 at which group A streptococcus causes acute rheumatic fever.

11 st-infection sequelae glomerulonephritis and rheumatic fever.

12 of group A streptococcal infection in acute rheumatic fever.

13 ll the Jones Criteria for diagnosis of acute rheumatic fever.

14 . pyogenes, including glomerulonephritis and rheumatic fever.

15 hronic lung or heart disease, and history of rheumatic fever.

16 They had had a total of 20 attacks of acute rheumatic fever.

17 s with the increased frequency of DRB1*16 in rheumatic fever.

18 sence of other major manifestations of acute rheumatic fever.

19 e absence of documented Sydenham's chorea or rheumatic fever.

20 ccal throat culture who are at risk of acute rheumatic fever.

21 tein types epidemiologically associated with rheumatic fever.

22 e are likely involved in the pathogenesis of rheumatic fever.

23 atient-years), 19 (0.6%) had recurrent acute rheumatic fever (3.49/1000 patient-years), and 20 (0.7%)

24 djusted odds ratio, 74.6 [CI, 12.5 to 447]), rheumatic fever (adjusted odds ratio, 13.4 [CI, 4.5 to 3

25 e pathogenic mechanisms for developing acute rheumatic fever after group A streptococcal pharyngitis

26 The authors compared the prevalence of rheumatic fever among the relatives of 33 children admit

27 were compared with those in 33 patients with rheumatic fever and 190 normal individuals.

28 tives of 33 children admitted for "incident" rheumatic fever and 33 control children admitted in a tu

29 nical entity, with a group looking more like rheumatic fever and a group with spondyloarthropathy tra

30 iated with serious sequelae, including acute rheumatic fever and acute glomerulonephritis.

31 fluoride on group A streptococci that causes rheumatic fever and acute poststreptococcal glomerulonep

32 he current hypotheses of the pathogenesis of rheumatic fever and group A streptococcal autoimmune seq

33 anded expression in nearly all patients with rheumatic fever and is thought to be a trait marker for

34 e intervals between the last attack of acute rheumatic fever and operation ranged from 2 to 8 years.

35 rategies for the primary prevention of acute rheumatic fever and rheumatic heart disease in children

36 Acute rheumatic fever and rheumatic heart disease remain major

37 iitis, as well as the postinfection sequelae rheumatic fever and rheumatic heart disease.

38 itis, destruction of tissues, and subsequent rheumatic fever and rheumatic heart disease.

39 Acute rheumatic fever and subsequent rheumatic heart disease r

40 lly respiratory infections at older ages and rheumatic fever and syphilis at younger ages, predicted

41 ints and cardiac valves in disorders such as rheumatic fever and systemic lupus erythematosus remain

42 d aging, whereas systemic hypertension (SH), rheumatic fever, and Chagas' disease (C'D) are higher in

43 r transient ischemic attack, recurrent acute rheumatic fever, and infective endocarditis.

44 ocumented GAS infections (pharyngitis, acute rheumatic fever, and severe invasive disease) also had s

45 Although acute rheumatic fever (ARF) and its sequel, rheumatic heart di

46 gated adverse outcomes for people with acute rheumatic fever (ARF) and rheumatic heart disease (RHD)

47 Acute rheumatic fever (ARF) and rheumatic heart disease (RHD)

48 Acute rheumatic fever (ARF) and subsequent rheumatic heart dis

49 Archived sera (collected in 1946) from acute rheumatic fever (ARF) and untreated scarlet fever and/or

50 Streptococcus pyogenes-induced acute rheumatic fever (ARF) is one of the best examples of pos

51 The pathogenesis of acute rheumatic fever (ARF) is poorly understood.

52 Acute rheumatic fever (ARF), a sequelae of group A Streptococc

53 otein is an epidemiological marker for acute rheumatic fever (ARF)-associated serotypes of group A st

54 es, Kawasaki disease has now surpassed acute rheumatic fever as the leading cause of acquired heart d

55 strain Manfredo and MGAS8232, another acute rheumatic fever-associated strain.

56 utoimmunity: beta-hemolytic streptococci and rheumatic fever; B3 Coxsackieviruses and myocarditis; Tr

57 presence of an "inherited predisposition" to rheumatic fever because the disease was more prevalent a

58 or group A streptococcal infection and acute rheumatic fever between 1948 and 1954.

59 f 16 subjects 7-21 years old who had not had rheumatic fever but who had possible PANDAS anorexia ner

60 tional guidelines for the diagnosis of acute rheumatic fever by defining high-risk populations, recog

61 was considered part of the spectrum of acute rheumatic fever by some, whereas others stressed the dif

62 Both rheumatic fever (cases) and tuberculosis (controls) were

63 have the diagnostic Jones criteria for acute rheumatic fever changed, but substantial advances have b

64 fections, myringotomy, measles, hepatitis A, rheumatic fever, common colds, rubella and chronic sinus

65 Sera from patients with acute rheumatic fever contained antibodies against recombinant

66 The incidence of acute rheumatic fever declined from 6.1 to 3.7 cases/100,000 (

67 Cardiovascular Disease in the Young and its Rheumatic Fever, Endocarditis, and Kawasaki Disease Comm

68 ation under the auspices of the Committee on Rheumatic Fever, Endocarditis, and Kawasaki Disease, Cou

69 ans to diagnose cardiac involvement in acute rheumatic fever, even when overt clinical findings are n

70 Rheumatic fever has become uncommon in the United States

71 that serum samples from patients with acute rheumatic fever have higher levels of antibodies that re

72 a recent increase in the incidence of acute rheumatic fever in North America, rheumatic heart diseas

73 storic Jones criteria used to diagnose acute rheumatic fever in the context of the current epidemiolo

74 t-streptococcal glomerulonephritis and acute rheumatic fever indicated that FBP54 is expressed in viv

75 ive soft tissue infections, pharyngitis, and rheumatic fever indicated that Slr is produced in vivo.

76 ive soft tissue infections, pharyngitis, and rheumatic fever indicated that these four proteins are e

77 Rheumatic fever is a significant public health problem p

78 Diseases such as tuberculosis, rheumatic fever, leprosy, and brucellosis, although more

79 Sydenham's chorea, a major manifestation of rheumatic fever, may provide a medical model for obsessi

80 cry" of host characteristics and involved in rheumatic fever or acute glomerulonephritis.

81 None of the patients had rheumatic fever or Sydenham's chorea.

82 None had rheumatic fever or Sydenham's chorea.

83 disorder and Tourette's syndrome, as well as rheumatic fever or Sydenham's chorea.

84 while DRB1*16 was increased in patients with rheumatic fever (OR 4.3, P=0.028).

85 of population-specific differences in acute rheumatic fever presentation and changes in presentation

86 t of GAS pharyngitis are cornerstones of the Rheumatic Fever Prevention Programme, but these are hind

87 As distinct from acute rheumatic fever, PSReA is characterized by a shorter lat

88 l on the hereditary versus social origins of rheumatic fever, Read, Ciocco, and Taussig, from Johns H

89 Acute rheumatic fever remains a serious healthcare concern for

90 of New Zealand where the incidence of acute rheumatic fever remains unacceptably high.

91 Primary prevention of acute rheumatic fever requires antibiotic treatment of acute s

92 vular heart lesions similar to those seen in rheumatic fever (RF).

93 sin are present in the sera of patients with rheumatic fever (RF).

94 of 13 KS sera, as compared with 5 of 8 acute rheumatic fever sera, contained Ab titers to human cardi

95 a different pattern of reactivity than acute rheumatic fever sera, further supporting the association

96 homologue in certain diseases such as acute rheumatic fever, suggest that SOK plays an important rol

97 l was to determine whether a trait marker of rheumatic fever susceptibility (labeled D8/17) could ide

98 hors' goal was to determine whether D8/17, a rheumatic fever susceptibility trait marker, identifies

99 mphocyte antigen with expanded expression in rheumatic fever, Sydenham's chorea, and subgroups of obs

100 pathogenesis may be more similar to that of rheumatic fever than to that of ReA associated with ente

101 Once primarily caused by rheumatic fever, the most common pathogenesis today is a

102 othesis is proven in a few illnesses such as rheumatic fever, there is no definitive evidence of an i