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1 eased incidences of sinonasal infections and rhinosinusitis.
2 iseases, such as cystic fibrosis and chronic rhinosinusitis.
3 onization, fungus balls, and allergic fungal rhinosinusitis.
4 seases ranging from colonization to invasive rhinosinusitis.
5 fungal sinusitis, and acute fulminant fungal rhinosinusitis.
6 review the diagnosis and treatment of acute rhinosinusitis.
7 r of spontaneously resolving acute bacterial rhinosinusitis.
8 sinusitis who present for treatment of acute rhinosinusitis.
9 reating patients with upper airway bacterial rhinosinusitis.
10 ntly been associated with asthma and chronic rhinosinusitis.
11 in particular those of patients with chronic rhinosinusitis.
12 was a risk factor for noninfectious rhinitis/rhinosinusitis.
13 can arbitrarily be divided into rhinitis and rhinosinusitis.
14 characteristics very similar to eosinophilic rhinosinusitis.
15 the relationship between work exposures and rhinosinusitis.
18 e report herein two cases of allergic fungal rhinosinusitis accompanied by bone destruction of the ad
20 ts with CRS had a higher prevalence of acute rhinosinusitis, allergic rhinitis, chronic rhinitis, ast
22 or the clinical diagnosis of acute bacterial rhinosinusitis and for those with severe rhinosinusitis
23 nasal polyps (NPs) of patients with chronic rhinosinusitis and might play a significant role in type
24 nasal polyps (NPs) of patients with chronic rhinosinusitis and might play a significant role in type
26 n accordance with the European guidelines on rhinosinusitis and nasal polyps (EPOS 2012), CT is used
28 exact role of microbial biofilms in chronic rhinosinusitis and orbital cellulitis were not elucidate
29 ability of the clinical isolates in chronic rhinosinusitis and orbital cellulitis, and to look for t
34 act diseases including otitis media, chronic rhinosinusitis, and exacerbations of both cystic fibrosi
35 tissue homogenates in patients with chronic rhinosinusitis, and this effect was most prominent in CR
36 espiratory tract infection," "pharyngitis," "rhinosinusitis," and "the common cold." HIGH-VALUE CARE
37 opriate antibiotic use for adults with acute rhinosinusitis apply to the diagnosis and treatment of a
39 features mimic essential aspects of chronic rhinosinusitis-associated olfactory loss, and illuminate
42 usitis recommend that the diagnosis of acute rhinosinusitis be based on the presence of "cardinal sym
44 ad prevalence of allergic, viral and chronic rhinosinusitis, but how the brain encodes and maintains
46 l reflux disease, postnasal drip syndrome or rhinosinusitis, chronic obstructive pulmonary disease, p
54 It is increasingly recognized that chronic rhinosinusitis (CRS) comprises a spectrum of different d
55 ovide composite criteria to evaluate chronic rhinosinusitis (CRS) control, taking into consideration
61 e of asthma and its association with chronic rhinosinusitis (CRS) have not been widely studied in pop
62 Current clinical classifications of chronic rhinosinusitis (CRS) have weak prognostic utility regard
64 robial interactions in patients with chronic rhinosinusitis (CRS) in hopes of elucidating mechanisms
80 of the underlying cause or causes of chronic rhinosinusitis (CRS) over the past 20 or more years have
81 ently, ILC2s enrichment was noted in chronic rhinosinusitis (CRS) patients; however, the role of ILC2
84 s, were collected from patients with chronic rhinosinusitis (CRS) with nasal polyps (CRSwNP) or witho
87 ronchodilator lung function, asthma, chronic rhinosinusitis (CRS), and atopy with age using a large E
89 nasal polyps (NPs) of patients with chronic rhinosinusitis (CRS), as well as in bronchoalveolar lava
90 the high prevalence and morbidity of chronic rhinosinusitis (CRS), little is known about the mechanis
91 tems, we identified respondents with chronic rhinosinusitis (CRS), migraine headache, and fatigue sym
92 ed outcomes during the management of chronic rhinosinusitis (CRS), PROMs will play an essential role
100 plays a role in the exacerbation of chronic rhinosinusitis (CRS); however, the mechanism for this is
101 -to-treat CRS was defined as noncontrollable rhinosinusitis despite successful sinus surgery and appr
103 study to use the European Position Paper on Rhinosinusitis (EPOS) criteria to study the prevalence o
104 ifficult to distinguish from acute bacterial rhinosinusitis, especially during the first 10 days of s
105 s with CRS, which was defined as symptomatic rhinosinusitis for more than 12 weeks and documented imm
106 hould reserve antibiotic treatment for acute rhinosinusitis for patients with persistent symptoms for
107 rs may impact upon airway disease, including rhinosinusitis, gastroesophageal reflux disease, obesity
108 morbidities are discussed: rhinitis, chronic rhinosinusitis, gastroesophageal reflux, obstructive sle
109 Invasive disease includes indolent chronic rhinosinusitis, granulomatous fungal sinusitis, and acut
111 onship between allergic rhinitis and chronic rhinosinusitis has been assessed in a number of observat
113 tial management of suspected acute bacterial rhinosinusitis in adults and children were prepared by a
114 and treatment of acute maxillary and ethmoid rhinosinusitis in adults who are not immunocompromised.
116 orticosteroids are beneficial to treat acute rhinosinusitis in patients with a history of chronic or
117 hould reserve antibiotic treatment for acute rhinosinusitis in patients with persistent symptoms for
118 robiota: Staphylococcus aureus infection and rhinosinusitis in the nasal-sinus mucosa, as well as cys
120 r 10 days, the likelihood of acute bacterial rhinosinusitis increases, and initiation of antibiotic t
121 smitted infections, cystic fibrosis, chronic rhinosinusitis, inflammatory bowel disease, and glaucoma
126 ce to support antibiotic treatment for acute rhinosinusitis is limited, yet antibiotics are commonly
127 e included acute inflammation (n=6), chronic rhinosinusitis (n=2), and allergic rhinitis (n=20) and u
131 etions in pathologic states, such as chronic rhinosinusitis or hyperglycemia, promotes tonic activati
132 e cases, given the likelihood of acute viral rhinosinusitis or of spontaneously resolving acute bacte
133 ing disease (asthma, nasal polyps or chronic rhinosinusitis, or both), as well as on the methodology
134 es (Short-Form Health Questionnaire, 31-item Rhinosinusitis Outcome Measuring Instrument, and Asthma
135 onic obstructive pulmonary disease, acquired rhinosinusitis, pancreatitis, and lethal secretory diarr
137 s flavus which is a common cause of allergic rhinosinusitis, postoperative aspergillosis and fungal k
138 gh rarely some patients with acute bacterial rhinosinusitis present with dramatic symptoms of severe
139 s and treatment of acute viral and bacterial rhinosinusitis recommend that the diagnosis of acute rhi
141 The clinical diagnosis of acute bacterial rhinosinusitis should be reserved for patients with rhin
142 mptoms of asthma, allergic rhinitis, chronic rhinosinusitis, smoking status, and history of NSAID-ind
143 ed from malignant disease or invasive fungal rhinosinusitis, so an understanding of the clinical feat
144 AIDs was higher in participants with chronic rhinosinusitis symptoms (Odds Ratio 2.12; 95%CI 1.78-2.7
145 nusitis should be reserved for patients with rhinosinusitis symptoms lasting 7 days or more who have
147 ial rhinosinusitis and for those with severe rhinosinusitis symptoms-especially those with unilateral
149 Among patients with nasal polyps and chronic rhinosinusitis, the prevalence was 9.69% (95% CI, 2.16%
150 rflow is also a contributing factor in human rhinosinusitis, this in vivo model demonstrates for the
151 edius range in severity from bite wounds and rhinosinusitis to endocarditis; historically, these infe
152 mechanism in patients with aspirin-sensitive rhinosinusitis to the bronchial mucosa in patients with
153 pecimens from patients with AERD and chronic rhinosinusitis were analyzed by using quantitative PCR,
154 ) patients with clinical features of chronic rhinosinusitis were examined; patients with first-onset
155 ed trial of adults with uncomplicated, acute rhinosinusitis were recruited from 10 community practice
157 role for mast cells is suggested in chronic rhinosinusitis with nasal polyposis (CRSwNP), which is c
158 ncinate tissue (UT) of patients with chronic rhinosinusitis with nasal polyps (CRSwNP) and those with
164 1 and its ligands PD-L1 and PD-L2 in chronic rhinosinusitis with nasal polyps (CRSwNP) is poorly stud
165 is without nasal polyps (CRSsNP) and chronic rhinosinusitis with nasal polyps (CRSwNP) using real-wor
166 sitis without nasal polyps (CRSsNP), chronic rhinosinusitis with nasal polyps (CRSwNP), and aspirin-e
169 irway inflammatory diseases, such as chronic rhinosinusitis with nasal polyps and asthma, show increa
170 piratory disease is a severe form of chronic rhinosinusitis with nasal polyps in which nearly all pat
174 of premorbid medical conditions for chronic rhinosinusitis without nasal polyps (CRSsNP) and chronic
175 9, at lower extent, in patients with chronic rhinosinusitis without nasal polyps (CRSsNP) in comparis
176 LFs) from controls and patients with chronic rhinosinusitis without nasal polyps (CRSsNP), chronic rh
177 nasal polyps (CRSwNP) and those with chronic rhinosinusitis without nasal polyps (CRSsNP; P < .01).
178 from control subjects, patients with chronic rhinosinusitis without nasal polyps, and patients with c
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