ライフサイエンスコーパス: rhinovirus

1 primary leucocytes following treatment with rhinovirus.

2 ally prolonged, especially for bocavirus and rhinovirus.

3 FOXA3 was induced by either IL-13 or rhinovirus.

4 ere inoculated with sham HeLa cell lysate or rhinovirus.

5 actor kappaB did inhibit T-cell responses to rhinovirus.

6 duced in epithelial cells by IL-13 and human rhinovirus.

7 tory syndrome (SARS), coxsackie viruses, and rhinoviruses.

8 ion of human respiratory syncytial virus and rhinoviruses.

9 eroviruses, it seems to occur more rarely in rhinoviruses.

10 enteroviruses but appears to be sporadic in rhinoviruses.

11 and asthma exacerbations compared with other rhinoviruses.

12 ruses precipitating asthma exacerbations are rhinoviruses.

13 ement among ICAM-1-binding enteroviruses and rhinoviruses.

14 .7%) tested for other respiratory pathogens, rhinovirus (107 [12.8%]), adenovirus (64 [6.0%]), respir

15 Rhinovirus (15 cases, 58%) and adenovirus (4 cases, 15%)

16 n, suppression of ADAM15 expression enhanced rhinovirus 16 and vesicular stomatitis virus-mediated pr

17 sthmatics challenged intranasally with human rhinovirus 16 were monitored directly ex vivo or else in

18 diates the replication of enterovirus 68 and rhinovirus 16.

19 enterovirus 68 but disadvantageous to human rhinovirus 16.

20 use airway cells infected with mouse-adapted rhinovirus 1B exhibited a striking enrichment in express

21 ) beta was administered with fluticasone and rhinovirus 1B in both groups of mice.

22 IFN beta (10(4) units) with fluticasone and rhinovirus 1B led to upregulation of interferon-inducibl

23 Mice were also infected with rhinovirus 1B to exacerbate lung inflammation and therap

24 -inflammatory effects in allergen-driven and rhinovirus 1B-exacerbated allergic airways disease (AAD)

25 lfoxide, control), 1 h before infection with rhinovirus 1B.

26 ytic degradation of eIF4G alone by the human rhinovirus 2A protease abrogated this translation strate

27 Respiratory syncytial virus (83%) and human rhinovirus (34%) were most commonly detected.

28 ad life-threatening disease, 575 (24.8%) had rhinovirus, 347 (14.9%) had adenovirus (ADV), and 30 (1.

29 react with UbcH7, USP08, or GST-tagged human rhinovirus 3C protease.

30 < .01), Coxsackieviruses (aOR 5.1, P = .09), rhinovirus A (aOR 3.5, P = .34), and rhinovirus C (aOR 2

31 on of rhinovirus C (OR, 2.85; P < 0.001) and rhinovirus A (OR, 2.92; P < 0.001), as well as, to a les

32 We report a case of chronic infection with rhinovirus A type 33 in a 43-year-old male with CF which

33 We have shown that rhinovirus, a cause of asthma exacerbation, colocalizes

34 The severity and rates of rhinovirus acute respiratory illnesses (ARIs) in adults

35 novirus (adjusted OR, 1.7; 95% CI, 1.1-2.7), rhinovirus (adjusted OR, 1.6; 95% CI, 1.1-2.3), and huma

36 justed OR, 4.8; P = .014), and wheezing with rhinovirus (adjusted OR, 5.0; P = .035).

37 ma, early-life exposure to tobacco smoke and rhinovirus aetiology were early-life risk factors for la

38 ive samples; this risk was also present with rhinovirus alone (aHR for mortality, 2.6; 95% CI, 1.2-5.

39 th control mice treated with fluticasone and rhinovirus alone and improved viral clearance without ha

40 tection when feasible, even for detection of rhinovirus alone.

41 Rhinovirus and IgE act in concert to promote asthma exac

42 e asthma AMs and was associated with reduced rhinovirus and imiquimod-induced IFN responses by these

43 zumab treatment were stimulated ex vivo with rhinovirus and influenza in the presence or absence of I

44 me of the differences observed between human rhinovirus and other enterovirus infections.

45 l/throat swabs were collected and tested for rhinovirus and other viruses by using RT-PCR.

46 ut was antagonized by enteroviruses, such as rhinovirus and poliovirus, which cleave C3 using their 3

47 ar cells, taken at age 11 years, to viruses (rhinovirus and respiratory syncytial virus; RSV) and bac

48 xperimentally the recombination potential of rhinoviruses and analyze recombination sites.

49 Rhinoviruses and coronaviruses were the most frequently

50 Interspecies recombination is even rarer for rhinoviruses and mostly is related to ancient events whi

51 ole of specific respiratory viruses, such as rhinoviruses and respiratory syncytial virus, in asthma

52 h increased susceptibility to infection with rhinoviruses and with changes in the composition of micr

53 yncytial virus, human metapneumovirus, human rhinovirus, and adenovirus) were not associated with acq

54 detections of parainfluenza, coronaviruses, rhinovirus, and adenovirus, especially in children, requ

55 mily protein receptors for poliovirus, human rhinovirus, and coxsackievirus B (CVB) serve to bind the

56 iral effect against hepatitis C virus, human rhinovirus, and coxsackievirus B3.

57 l virus, parainfluenza viruses, coronavirus, rhinovirus, and human metapneumovirus, represent a consi

58 al virus, adenovirus, human metapneumovirus, rhinovirus, and influenza virus but not parainfluenza vi

59 virus, metapneumovirus, parainfluenza virus, rhinovirus, and respiratory syncytial virus were detecte

60 including encephalomyocarditis virus, human rhinovirus, and respiratory syncytial virus.

61 for early inflammatory responses induced by rhinovirus, and TLR2(+) macrophages are sufficient to co

62 Omalizumab improved IFN-alpha responses to rhinovirus, and within the omalizumab group, greater IFN

63 Omalizumab treatment increased rhinovirus- and influenza-induced PBMC and rhinovirus-in

64 Minor-group rhinoviruses are more immunogenic in laboratory studies,

65 Rhinoviruses are the major cause of asthma exacerbations

66 Rhinoviruses are the most common causes of the common co

67 eas additional effort is needed in regard to rhinovirus, as well as other respiratory tract viruses.

68 We sought to determine rhinovirus-associated ARI rates in adults presenting for

69 can reduce the likelihood of presenting with rhinovirus-associated ARIs.

70 There were 7 rhinovirus-associated ED visits and 3 hospitalizations p

71 mouse airway epithelial cells infected with rhinovirus at 33 degrees C vs. 37 degrees C.

72 P < 0.001), as well as, to a lesser extent, rhinovirus B (OR, 1.98; P = 0.019).

73 za viruses, respiratory syncytial virus, and rhinoviruses being the most frequent and with the highes

74 ignaling drove the impaired IFN responses to rhinovirus by asthmatic alveolar macrophages (AMs); the

75 = .09), rhinovirus A (aOR 3.5, P = .34), and rhinovirus C (aOR 2.9, P = .57).

76 e significantly associated with detection of rhinovirus C (OR, 2.85; P < 0.001) and rhinovirus A (OR,

77 Members of rhinovirus C (RV-C) species are more likely to cause whe

78 Isolates of rhinovirus C (RV-C), a recently identified Enterovirus (

79 infection of the respiratory epithelium with rhinovirus can antagonize tolerance to inhaled antigen t

80 Rhinoviruses cause the common cold and exacerbations of

81 se, IL-33 levels were persistently high, and rhinovirus challenge exacerbated the type 2 inflammatory

82 umor-specific cytotoxicity of the poliovirus/rhinovirus chimera PVSRIPO is facilitated by Raf-ERK1/2

83 raspecies recombination gives rise to viable rhinovirus chimeras in the polyprotein coding region and

84 Patients with rhinovirus, compared with virus-negative ARI, were more

85 The 25 children with greater than 7000 rhinovirus copies/mL (most sensitive cutoff) benefitted

86 human metapneumovirus, adenovirus, and human rhinoviruses, coronaviruses, and bocavirus.

87 ettings and identify factors associated with rhinovirus detection.

88 ions were usually symptomatic; bocavirus and rhinovirus detections were often asymptomatic.

89 compound that had been developed as an anti-rhinovirus drug.

90 cies rhinovirus/rhinovirus or rhinovirus/non-rhinovirus enterovirus recombinants are fully viable.

91 athogens such as poliovirus, coxsackievirus, rhinovirus, enterovirus 71 and foot-and-mouth disease vi

92 with increasing age and infection with RSV, rhinovirus, enterovirus, adenovirus, and bocavirus.

93 ILI episodes: influenza A (104/556, 18.7%), rhinovirus/enterovirus (82/556, 14.7%), coronavirus and

94 Positive rhinovirus/enterovirus FA targets revealed patterns loos

95 ay (FA) respiratory panel was used to detect rhinovirus/enterovirus in respiratory specimens; suspect

96 ombination is described frequently among non-rhinovirus enteroviruses but appears to be sporadic in r

97 ombination is described frequently among non-rhinovirus enteroviruses, it seems to occur more rarely

98 Most frequently detected were rhinoviruses/enteroviruses (RV/EV) (21%) and influenza v

99 y, and ELISA, we measured T-cell activation, rhinovirus expression, T-cell death, and eosinophil cyst

100 After confirming rhinovirus from nasopharyngeal aspirate by using PCR, 79

101 Here, we show that human rhinovirus (HRV) 3A also recruited PI4KIIIbeta to replic

102 for associations with asthma and with human rhinovirus (HRV) and respiratory syncytial virus (RSV) w

103 Respiratory syncytial virus (RSV) and human rhinovirus (HRV) are the most common viruses associated

104 First, infection of HeLa cells with human rhinovirus (HRV) induced the phosphorylation of PKD.

105 d whole blood samples during natural RSV and rhinovirus (hRV) infection (acute versus early recovery

106 Human rhinovirus (HRV) infection is a common trigger for child

107 thma exacerbations are associated with human rhinovirus (HRV) infections, and more severe exacerbatio

108 aetiological agent of the common cold, human rhinovirus (HRV) is the leading cause of human infectiou

109 situation with different serotypes of human rhinovirus (HRV) is unclear.

110 n all respiratory samples positive for human rhinovirus (HRV) or negative for all respiratory viruses

111 Human rhinovirus (HRV) replication triggers exacerbation of as

112 nfections, caused by viruses including human rhinovirus (HRV), influenza virus, and respiratory syncy

113 ith acute respiratory infection due to human rhinovirus (HRV; n = 52) or respiratory syncytial virus

114 Human rhinoviruses (HRV) comprise 3 species representing more

115 Human rhinoviruses (HRV) have been linked to the development o

116 Human rhinoviruses (HRVs) are a major trigger of asthma exacer

117 Human rhinoviruses (HRVs) are one of the main causes of virus-

118 Human rhinoviruses (HRVs) commonly precipitate asthma exacerba

119 Human rhinoviruses (hRVs) were detected in 25.5% of 1232 nasal

120 teristics and clinical significance of human rhinoviruses, human coronaviruses, human metapneumovirus

121 events with influenza virus, coronaviruses, rhinoviruses, human metapneumovirus, respiratory syncyti

122 at the 17q21 genetic locus who wheeze during rhinovirus illnesses have a greatly increased likelihood

123 tions, a mechanism through which IgE impairs rhinovirus immunity and underlies asthma exacerbations w

124 (respiratory syncytial virus in 51.4%, human rhinovirus in 11.7%).

125 airway epithelial cells infected with human rhinovirus in asthma, but whether this abnormality occur

126 ects of omalizumab on IFN-alpha responses to rhinovirus in PBMCs were examined.

127 4, IL-5, IL-13, and IL-33 are all induced by rhinovirus in the asthmatic airway in vivo and relate to

128 The most common pathogens were human rhinovirus (in 9% of patients), influenza virus (in 6%),

129 urthermore, M catarrhalis detected alongside rhinovirus increased the likelihood of experiencing cold

130 catarrhalis, respectively, with detection of rhinovirus increasing the risk of detecting bacteria wit

131 In contrast to RSV, rhinovirus induced T-cell activation without the involve

132 Without moDCs, rhinovirus induced T-cell proliferation of both CD4 and

133 In animal models of AAD and rhinovirus-induced AAD exacerbations, pDCs were recruite

134 he magnitude of type 2 inflammation during a rhinovirus-induced acute exacerbation; however, only ant

135 ted against the development of allergen- and rhinovirus-induced airway hyperreactivity and decreased

136 creasing PP2A activity with AAL(S) abolished rhinovirus-induced airway hyperreactivity, eosinophil in

137 ncreasing viral loads, fluticasone inhibited rhinovirus-induced airway inflammation as evidenced by s

138 macrophages are required and sufficient for rhinovirus-induced airway inflammation in vivo.

139 wild-type, OVA-treated C57BL/6 mice blocked rhinovirus-induced airway responses, whereas transfer of

140 the requirement and sufficiency of TLR2 for rhinovirus-induced airway responses.

141 uggest that fluticasone treatment suppresses rhinovirus-induced airways inflammation in vivo but also

142 33 and type 2 cytokines are induced during a rhinovirus-induced asthma exacerbation in vivo.

143 and bronchoalveolar lavage before and after rhinovirus-induced asthma exacerbations.

144 y represent potential therapeutic targets of rhinovirus-induced asthma exacerbations.

145 ndings indicate that the regulation of human rhinovirus-induced B-cell responses could be a novel app

146 re increased in the lung microenvironment on rhinovirus-induced COPD exacerbation in vivo.

147 We have also shown that rhinovirus-induced cytokine expression is abolished in T

148 Also, the incidence of rhinovirus-induced episodes, which comprised 80% of all

149 etermine the relationship between ORMDL3 and rhinovirus-induced ER stress and type I IFN in human leu

150 ociation between higher ORMDL3 and increased rhinovirus-induced HSPA5 and type I IFN gene expression.

151 Paradoxically, FOXA3 inhibited rhinovirus-induced IFN production, IRF-3 phosphorylation

152 ween bacterial infection and the severity of rhinovirus-induced illnesses.

153 basophils in promoting, or else prolonging, rhinovirus-induced inflammation in atopic asthmatics.

154 es and transcription factors involved in the rhinovirus-induced interferon production and inflammator

155 rfactant protein C expression and suppressed rhinovirus-induced IP-10 secretion, which was associated

156 fects by increasing PP2A activity in AAD and rhinovirus-induced lung inflammation, which might potent

157 After allergen exposure, rhinovirus-induced neutrophilic and eosinophilic airway

158 Rhinovirus-induced ORMDL3 expression in primary leucocyt

159 The degree of rhinovirus-induced ORMDL3, HSPA5 and IFNB1 expression va

160 d rhinovirus- and influenza-induced PBMC and rhinovirus-induced pDC IFN-alpha responses in the presen

161 sufficient to recapitulate many features of rhinovirus-induced type-2 immune responses and asthma pa

162 ment of the first acute, moderate-to-severe, rhinovirus-induced wheezing episode in young children.

163 ncing their first acute, moderate-to-severe, rhinovirus-induced wheezing episode.

164 Rhinovirus-induced wheezing is an important risk factor

165 , and SOCS1-deficient mice, SOCS1 suppressed rhinovirus induction of interferons.

166 infection, fluticasone treatment suppressed rhinovirus induction of type I and III IFNs in the airwa

167 human T cells and ILC2s with supernatants of rhinovirus-infected BECs strongly induced type 2 cytokin

168 phoid cells (ILC2s) with the supernatants of rhinovirus-infected bronchial epithelial cells (BECs) to

169 The latter phenotype was replicated in rhinovirus-infected human AECs, suggesting that anti-IL-

170 depletion after allergen challenge or during rhinovirus infection abrogated exacerbation of inflammat

171 , which was induced by allergen challenge or rhinovirus infection and conditioned pDCs for proinflamm

172 lation and again on Days 5, 15, and 42 after rhinovirus infection and DNA was extracted.

173 We used a human experimental model of rhinovirus infection and novel airway sampling technique

174 ent study collected individual data on human rhinovirus infection and sensitization to Alternaria and

175 f host double-stranded DNA (dsDNA) following rhinovirus infection and the exacerbation of type-2 alle

176 Rhinovirus infection at an early age has been associated

177 m people with asthma are more susceptible to rhinovirus infection caused by deficient induction of th

178 Rhinovirus infection enhances detection of specific bact

179 Early-life human rhinovirus infection has been linked to asthma developme

180 ue of the lungs has been demonstrated during rhinovirus infection in allergic individuals.

181 y increased IgE responsiveness 3 weeks after rhinovirus infection in atopic asthmatics.

182 Our findings suggest that rhinovirus infection in COPD alters the respiratory micr

183 ate lymphoid cells (ILC2s) was expanded with rhinovirus infection in neonatal but not mature mice.

184 Compared with mature mice, rhinovirus infection in neonatal mice increased lung IL-

185 Nevertheless, the role of rhinovirus infection in the initiation of asthma remains

186 asthma exacerbation-related cytokines and by rhinovirus infection in vitro.

187 effects of inhaled fluticasone propionate on rhinovirus infection in vivo, in a mouse model.

188 nd polyinosinic-polycytidylic acid and human rhinovirus infection induced a potent antiviral protecti

189 Rhinovirus infection is a leading cause of exacerbation

190 sociated with viral load during experimental rhinovirus infection of asthmatic patients.

191 Rhinovirus infection of human primary BECs induced IL-33

192 tory T (Treg) cells, and we examined whether rhinovirus infection of the respiratory tract can block

193 Rhinovirus infection or dsRNA stimulation increased thym

194 Rhinovirus infection triggers acute asthma exacerbations

195 lergic airway hypersensitivity, we show that rhinovirus infection triggers dsDNA release associated w

196 Following rhinovirus infection, all subjects developed nasal sympt

197 udies found that, during poliovirus or human rhinovirus infection, AUF1 is cleaved by the viral prote

198 Following experimental rhinovirus infection, we observed increased respiratory

199 fficient for neutrophilic inflammation after rhinovirus infection, whereas macrophages treated with I

200 y subjects and treated with anti-IL-33 after rhinovirus infection.

201 n the asthmatic and healthy airways during a rhinovirus infection.

202 specific IgE antibodies was detected during rhinovirus infection.

203 asthma hospitalizations independent of human rhinovirus infection.

204 Rhinovirus infections are the dominant cause of asthma e

205 ovirus RNA shedding, duration or severity of rhinovirus infections, or occurrence of rhinovirus RNA i

206 ene-environment interactions (GEIs), such as rhinovirus infections, will improve detection of asthma

207 infection by RSV group B, and no prior human rhinovirus infections.

208 d cost-effective means to reduce the risk of rhinovirus infections.

209 sociated with development of asthma, but how rhinovirus influences the immune response is not clear.

210 ted with inherent deficient IFN responses to rhinovirus, inhaled corticosteroids might interact syner

211 uced sputum was collected at baseline before rhinovirus inoculation and again on Days 5, 15, and 42 a

212 To further assess the rhinovirus intra- and interspecies recombination potenti

213 Rhinovirus is associated with a substantial number of ED

214 Rhinovirus is linked to asthma exacerbations and chronic

215 The primary interaction analysis examined rhinovirus load.

216 how the interaction of increased ORMDL3 and rhinovirus may predispose to asthma.

217 anation for how different types (strains) of rhinoviruses may elicit different cell responses that di

218 espiratory syncytial virus (RSV), influenza, rhinovirus, metapneumovirus, and adenovirus was highly a

219 were calculated and rhinovirus-positive and rhinovirus-negative patients were compared.

220 the first severe respiratory syncytial virus/rhinovirus-negative wheezing episode (adjusted OR, 8.0;

221 (UTR) interspecies rhinovirus/rhinovirus or rhinovirus/non-rhinovirus enterovirus recombinants are f

222 ected and examined for the presence of human rhinovirus or enterovirus using the FilmArray Respirator

223 eight (73%) of 11 children were positive for rhinovirus or enterovirus.

224 ng negative association with age; especially rhinovirus or parainfluenza virus detection showed posit

225 or both compared with isolated detection of rhinovirus (OR, 2.0 [95% CI, 1.0-4.1]; P = .04).

226 ry virus types (respiratory syncytial virus, rhinoviruses, other picornaviruses, coronaviruses 229E a

227 its and hospitalizations were calculated and rhinovirus-positive and rhinovirus-negative patients wer

228 m severely ill patients nationwide, who were rhinovirus-positive or enterovirus-positive in hospital

229 mature BALB/c mice, neonatal infection with rhinovirus promotes an IL-25-driven type 2 response, whi

230 with high frequency, whereas no interspecies rhinovirus recombinants could be recovered.

231 s regarding the mechanism and limitations of rhinovirus recombination.

232 l approaches to improve current knowledge on rhinovirus recombination.

233 ings demonstrate that in mouse airway cells, rhinovirus replicates preferentially at nasal cavity tem

234 -16-induced interferon production, increased rhinovirus replication and impaired TLR3 expression in b

235 Anti-IL-33 decreased rhinovirus replication and increased IFN-lambda levels a

236 Rhinovirus replication was higher and IFN-lambda levels

237 ed 5' untranslated region (UTR) interspecies rhinovirus/rhinovirus or rhinovirus/non-rhinovirus enter

238 y of rhinovirus infections, or occurrence of rhinovirus RNA in asymptomatic infants.

239 ifferences emerged among the study groups in rhinovirus RNA load during infections, duration of rhino

240 irus RNA load during infections, duration of rhinovirus RNA shedding, duration or severity of rhinovi

241 Both rhinovirus (RV) and polyinosinic-polycytidilic acid [pol

242 and III production associated with enhanced rhinovirus (RV) and respiratory syncytial virus (RSV) re

243 would shorten the frequency and duration of rhinovirus (RV) illnesses in children with allergic asth

244 Mice were infected intranasally with rhinovirus (RV) immediately after O2 exposure.

245 ay epithelial cells are the major target for rhinovirus (RV) infection and express proinflammatory ch

246 Rhinovirus (RV) infection in asthma induces varying degr

247 Rhinovirus (RV) infection of 6-d-old mice, but not matur

248 Rhinovirus (RV) infections are closely linked to exacerb

249 Rhinovirus (RV) infections are common and have the poten

250 Some children with rhinovirus (RV) infections wheeze, but it is unknown whe

251 Rhinovirus (RV) is a major cause of common cold and an i

252 in causative agent of bronchiolitis, whereas rhinovirus (RV) is most commonly detected in wheezing ch

253 y-life respiratory syncytial virus (RSV) and rhinovirus (RV) lower respiratory tract infections (LRTI

254 Early life rhinovirus (RV) wheezing illnesses and aeroallergen sens

255 Rhinovirus (RV), a virus responsible for a majority of c

256 ined as respiratory syncytial virus (RSV) or rhinovirus (RV), including coinfections with other virus

257 Most strains of rhinovirus (RV), the common cold virus, replicate better

258 FN-beta and IFN-lambda1) induction following rhinovirus (RV)-16 infection.

259 The RNA rhinoviruses (RV) encode 2A proteases (2A(pro)) that con

260 allergen-challenged mice received intranasal rhinovirus-(RV)-1B followed by lung TSLP immunostaining.

261 Rhinoviruses (RVs) are a major cause of symptomatic resp

262 Rhinoviruses (RVs) are the major triggers of asthma exac

263 Rhinoviruses (RVs) are ubiquitous respiratory pathogens

264 Human rhinoviruses (RVs) of the A, B, and C species are define

265 Human rhinoviruses (RVs), comprising three species (A, B, and

266 Rhinoviruses (RVs), respiratory enteroviruses (EVs), inf

267 irus-induced attacks of asthma are caused by rhinoviruses (RVs).

268 irulence.IMPORTANCE Most colds are caused by rhinoviruses (RVs).

269 In the absence of rhinovirus, S pneumoniae was associated with increased c

270 In the presence of rhinovirus, S pneumoniae was associated with increased m

271 valuated bacterial detection during the peak rhinovirus season in children with and without asthma to

272 a preparation of a common cold virus (human rhinovirus serotype 2, HRV-A2) and (ii) the analysis of

273 may contribute to the very large numbers of rhinovirus serotypes that coexist while differing in vir

274 rovide evidence of differential virulence of rhinovirus species (RV).

275 a subset known as the minor-group members of rhinovirus species A (RV-A) are correlated with the ince

276 Rhinovirus species and type were determined by partial g

277 such as enterovirus species groups A to H or rhinovirus species groups A to C.

278 lyprotein region between members of the same rhinovirus species, but not between members of different

279 Regardless of rhinovirus status, H influenzae was not associated with

280 ay did not detect any other enteroviruses or rhinoviruses tested and did detect divergent strains of

281 Most isolates of human rhinovirus, the common cold virus, replicate more robust

282 nce against virus infections to both RSV and rhinovirus; there were no differences in antibacterial r

283 arly and later life and then challenged with rhinovirus to model disease onset, progression, and chro

284 Furthermore, the ability of rhinoviruses to disrupt and interfere with epithelial ti

285 New evidence indicates that rhinovirus-triggered asthma exacerbations become more se

286 We have used IRES elements from human rhinovirus type 2 (HRV2) and foot-and-mouth disease viru

287 irus (PV) (Sabin) vaccine containing a human rhinovirus type 2 (HRV2) IRES, is demonstrating early pr

288 irus (PV) (Sabin) vaccine containing a human rhinovirus type 2 (HRV2) IRES.

289 We show that serum nAb against many rhinovirus types can be induced by polyvalent, inactivat

290 pes.IMPORTANCE Genetic variation among human rhinovirus types includes unexpected diversity in the ge

291 Rhinovirus was associated with CAP among adults (AF, 0.9

292 ature-dependent IFN induction in response to rhinovirus was dependent on the MAVS protein, a key sign

293 Among 2351 enrollees, rhinovirus was detected in 247 (11%).

294 Rhinovirus was detected in 74% (n = 80/108) of the child

295 Rhinovirus was detected most often; multiple viruses wer

296 Rhinovirus was the most frequently detected RV (42%).

297 Given the clinical importance of rhinovirus, we sought to determine whether it had any un

298 factor that is required for cytotoxicity by rhinoviruses, which cause the common cold.

299 monocytes overexpressing ORMDL3 responded to rhinovirus with increased IFNB1 and HSPA5.

300 ailable assays that detect enteroviruses and rhinoviruses without distinguishing between the two, inc