ライフサイエンスコーパス: rhinovirus infection

1 asthma hospitalizations independent of human rhinovirus infection.

2 y subjects and treated with anti-IL-33 after rhinovirus infection.

3 n the asthmatic and healthy airways during a rhinovirus infection.

4 specific IgE antibodies was detected during rhinovirus infection.

5 elates to exacerbations of asthma related to rhinovirus infection.

6 models of allergic airways disease and human rhinovirus infection.

7 of clinical illness that is associated with rhinovirus infection.

8 as been the lack of a small-animal model for rhinovirus infection.

9 xtracts from Echinacea angustifolia roots on rhinovirus infection.

10 th and the development of a murine model for rhinovirus infection.

11 f URT infections or symptoms associated with rhinovirus infection.

12 toms and airway inflammation associated with rhinovirus infection.

13 infection by RSV group B, and no prior human rhinovirus infections.

14 d cost-effective means to reduce the risk of rhinovirus infections.

15 in nasal samples in natural and experimental rhinovirus infections.

16 l during H1N1 infections compared with human rhinovirus infections (38 vs. 21%; odds ratio, 2.6; 95%

17 erval, 1.8-9; P < 0.001), but rates of human rhinovirus infection (90% each) and other viral infectio

18 depletion after allergen challenge or during rhinovirus infection abrogated exacerbation of inflammat

19 Following rhinovirus infection, all subjects developed nasal sympt

20 , which was induced by allergen challenge or rhinovirus infection and conditioned pDCs for proinflamm

21 lation and again on Days 5, 15, and 42 after rhinovirus infection and DNA was extracted.

22 We used a human experimental model of rhinovirus infection and novel airway sampling technique

23 ent study collected individual data on human rhinovirus infection and sensitization to Alternaria and

24 f host double-stranded DNA (dsDNA) following rhinovirus infection and the exacerbation of type-2 alle

25 um was collected before and repeatedly after rhinovirus infection and virus and bacterial loads measu

26 Deficient interferon responses to rhinovirus infection are present in childhood in asthmat

27 Rhinovirus infections are frequently followed by seconda

28 Infant respiratory syncytial virus and rhinovirus infections are known to be associated with an

29 Rhinovirus infections are the dominant cause of asthma e

30 Respiratory syncytial virus (RSV) and rhinovirus infections are the most common significant in

31 Rhinovirus infection at an early age has been associated

32 udies found that, during poliovirus or human rhinovirus infection, AUF1 is cleaved by the viral prote

33 dren with histories of wheezing illness with rhinovirus infection before the third birthday had lower

34 Rhinovirus infections cause exacerbations of asthma.

35 m people with asthma are more susceptible to rhinovirus infection caused by deficient induction of th

36 vels of SRp20 relocalization observed during rhinovirus infection compared to poliovirus.

37 s a proinflammatory cytokine up-regulated by rhinovirus infection during acute exacerbations of asthm

38 Clinically significant rhinovirus infection during infancy was more strongly as

39 Rhinovirus infection enhances detection of specific bact

40 mportant implications in the pathogenesis of rhinovirus infections, especially initiation of the host

41 SLPI and elafin significantly reduced after rhinovirus infection exclusively in subjects with COPD w

42 Early-life human rhinovirus infection has been linked to asthma developme

43 s of the cytokine dysregulation that follows rhinovirus infection have implicated monocytic lineage c

44 ue of the lungs has been demonstrated during rhinovirus infection in allergic individuals.

45 y increased IgE responsiveness 3 weeks after rhinovirus infection in atopic asthmatics.

46 The prevalence of rhinovirus infection in children is high and presents a

47 airway interferon production in response to rhinovirus infection in children who are asthmatic, atop

48 on of tICAM453 was efficacious in preventing rhinovirus infection in chimpanzees subsequently challen

49 Our findings suggest that rhinovirus infection in COPD alters the respiratory micr

50 s have features similar to those observed in rhinovirus infection in humans, including augmentation o

51 ate lymphoid cells (ILC2s) was expanded with rhinovirus infection in neonatal but not mature mice.

52 Compared with mature mice, rhinovirus infection in neonatal mice increased lung IL-

53 Nevertheless, the role of rhinovirus infection in the initiation of asthma remains

54 asthma exacerbation-related cytokines and by rhinovirus infection in vitro.

55 effects of inhaled fluticasone propionate on rhinovirus infection in vivo, in a mouse model.

56 We sought to examine the prevalence of rhinovirus infections in relation to the atopic status o

57 Rhinovirus infection induced a dose- and time-dependent

58 nd polyinosinic-polycytidylic acid and human rhinovirus infection induced a potent antiviral protecti

59 In parallel, we found that rhinovirus infection induced a rapid increase of intrace

60 Rhinovirus infection induced a time- and dose-dependent

61 We sought to evaluate whether rhinovirus infection interferes with the mechanisms of a

62 dence that early activation of p38 kinase by rhinovirus infection is a key event in regulation of vir

63 Rhinovirus infection is a leading cause of exacerbation

64 Along with other respiratory viruses, rhinovirus infection is also a cause of exacerbations of

65 Rhinovirus infection is followed by significantly increa

66 Rhinovirus infection is now recognized as an important c

67 However, the inflammatory response to rhinovirus infection is poorly understood.

68 Here we describe three novel mouse models of rhinovirus infection: minor-group rhinovirus infection o

69 Rhinovirus infection of airway epithelium induces ICAM-1

70 sociated with viral load during experimental rhinovirus infection of asthmatic patients.

71 models of rhinovirus infection: minor-group rhinovirus infection of BALB/c mice, major-group rhinovi

72 Rhinovirus infection of human primary BECs induced IL-33

73 tory T (Treg) cells, and we examined whether rhinovirus infection of the respiratory tract can block

74 To test the hypothesis that rhinovirus infection of the upper airway may be associat

75 ovirus infection of BALB/c mice, major-group rhinovirus infection of transgenic BALB/c mice expressin

76 To investigate the effects of rhinovirus infection on the intracellular redox balance,

77 Rhinovirus infection or dsRNA stimulation increased thym

78 ovirus RNA shedding, duration or severity of rhinovirus infections, or occurrence of rhinovirus RNA i

79 rial infections, but it is not known whether rhinovirus infections precipitate secondary bacterial in

80 After rhinovirus infection, secondary bacterial infection was

81 ith atopic asthma are not at greater risk of rhinovirus infection than healthy individuals but suffer

82 t people with asthma are more susceptible to rhinovirus infection than people without the disease and

83 epidemiology and clinical manifestations of rhinovirus infection that have occurred as a result of t

84 After rhinovirus infection, there is a rise in bacterial burde

85 At 15 days after rhinovirus infection, there was a sixfold increase in 16

86 Rhinovirus infection triggers acute asthma exacerbations

87 lergic airway hypersensitivity, we show that rhinovirus infection triggers dsDNA release associated w

88 lular redox balance, we also studied whether rhinovirus infection triggers the production of reactive

89 First rhinovirus infection was associated more frequently with

90 o contribute to the symptoms and sequelae of rhinovirus infection, we investigated the role of p38 ki

91 Following experimental rhinovirus infection, we observed increased respiratory

92 Symptoms of LRT associated with rhinovirus infection were significantly more severe (p=0

93 fficient for neutrophilic inflammation after rhinovirus infection, whereas macrophages treated with I

94 The innate immune response to rhinovirus infection, which may play an important role i

95 ene-environment interactions (GEIs), such as rhinovirus infections, will improve detection of asthma