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2 We propose that programmed cell death in the rhombencephalic neural crest is modulated by pre-pattern
5 g pronephric duct as well as a subset of the rhombencephalic neural crest, which will form the Schwan
6 kines induce selective apoptosis of discrete rhombencephalic neural crest-associated cellular populat
7 ration of neural crest occurs in the preotic rhombencephalic neural tube even after laser irradiation
8 plate originate from discrete subdomains of rhombencephalic neuroectoderm expressing Wnt1; that chor
9 wed that ethanol augments apoptosis in fetal rhombencephalic neurons and co-treatment with alpha-lipo
10 developing serotonin (5-HT) and other fetal rhombencephalic neurons in rats by augmenting apoptosis.
12 the anti-apoptotic effects of S100B on fetal rhombencephalic neurons were linked to the activation of
13 optosis was augmented significantly in fetal rhombencephalic neurons when they were treated with 50 m
14 monstrated that a 24-hour treatment of fetal rhombencephalic neurons with 75 mM ethanol caused a 3-fo
15 d the effects of LA on ethanol-treated fetal rhombencephalic neurons with regard to oxidative stress
16 ted that ethanol augments apoptosis in fetal rhombencephalic neurons, and that the increased apoptosi
23 mpartments of the mesencephalon and the main rhombencephalic regions, including the cerebellum, were
25 cells and unidentified neurons in the middle rhombencephalic reticular nucleus (MRRN), displayed dela
26 neurons, particularly those in the posterior rhombencephalic reticular nucleus (PRRN), either adapted
27 gamma2 was the most abundant subunit in all rhombencephalic structures during development and in adu
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