ライフサイエンスコーパス: rib cage

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1 eletal defects in cervical vertebrae and the rib cage.

2 area of apposition of the diaphragm with the rib cage.

3 heating were examined in samples of porcine rib cage.

4 e zone of apposition of the diaphragm to the rib cage.

5 ity, supporting its role in the extension of rib cages.

6 elephants and manatees, which have extended rib cages.

7 ly, transformation of cervical segments, and rib cage abnormalities.

8 nk lesions (lateral abdominal wall below the rib cage, above the iliac crest).

9 alized along the entire vertebral column and rib cage and are linked to defective formation of cartil

10 ns regarding the nature of the ;Hox code' in rib cage and axial skeleton development are revealed.

11 e probably because of greater recruitment of rib cage and expiratory muscles (p = 0.004) and because

12 Other defects in the skull, lung, rib cage and long bones are likely to be the result of t

13 An arrowhead lodged between the rib cage and the left scapula was the probable cause of

14 ogenin gene knock-in (ki) developed a normal rib cage and were viable, therefore demonstrating functi

15 They had brachycephaly, small rib cages, and homeotic skeletal transformations with in

16 the diaphragm (t(di)), circumference of the rib cage (c(di)), and CSA(di) increased with height and

17 due to respiratory failure from a diminished rib cage circumference.

18 0.0015) and 48.7+/-4.3L/min with soft manual rib cage compression (p = 0.0002).

19 Hard manual rib cage compression improved mucus clearance in animals

20 the effects of two different types of manual rib cage compression on expiratory flow and mucus cleara

21 Soft manual rib cage compression slightly worsened static lung elast

22 reased to 28.4+/-5.2L/min during hard manual rib cage compression vs. 15.9+/-2.2 and 16.6+/-2.8L/min

23 Conversely, soft manual rib cage compression was not effective and potentially u

24 zed with early expiratory phase (hard manual rib cage compression) and soft and gradual rib cage comp

25 uring the late expiratory phase (soft manual rib cage compression).

26 he lungs during no treatment and soft manual rib cage compression, -0.28 +/- 0.61 and -0.15+/-0.95mm/

27 During hard manual rib cage compression, mucus moved toward the glottis (1.

28 /-2.8L/min without treatment and soft manual rib cage compression, respectively (p = 0.0006).

29 l rib cage compression) and soft and gradual rib cage compressions applied during the late expiratory

30 age compressions were tested: Hard and brief rib cage compressions synchronized with early expiratory

31 Two types of manual rib cage compressions were tested: Hard and brief rib ca

32 The normal decrease in the rib cage contribution to the tidal volume during phasic

33 We investigated whether the rib cage defect was due to the failure of the early acti

34 A rib cage defect was observed in Myf5-deficient mice, whi

35 By predisposing to rib cage deformation and reduced end-expiratory lung vol

36 these subjects necessitates abnormally large rib cage displacements during exercise, which may be a s

37 acement of the lateral portions of the lower rib cage during inspiration.

38 to 10.0 MHz transducer placed over the lower rib cage in the mid-axillary line.

39 ely from somitic mesoderm, patterning of the rib cage is complicated by its derivation from two disti

40 arge frequencies of the SMUs were higher and rib cage movement greater during HF-SCS compared to spon

41 plitude during HF-SCS was adjusted such that rib cage movement matched (Protocol 2).

42 ge phenotype, chondrocytes isolated from the rib cages of developing rat embryos were evaluated for t

43 to the Myf5 locus (Myf5(myg-ki)) rescued the rib cage truncation in the Myf5-null mutant, hence demon

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