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1 shifted registers reminiscent of programmed ribosomal frameshifting.
2 oacyl synthetase recognition, and programmed ribosomal frameshifting.
3 requirements and mechanism of programmed -1 ribosomal frameshifting.
4 promote significant levels of programmed -1 ribosomal frameshifting.
5 rocess may also have an impact on programmed ribosomal frameshifting.
6 tidyltransferase center affect programmed -1 ribosomal frameshifting.
7 l antiviral agents that target programmed -1 ribosomal frameshifting.
8 PKs to decipher the mechanism of programmed ribosomal frameshifting.
9 y alter the efficiency of -1, but not of +1, ribosomal frameshifting.
10 protein can function as a transactivator of ribosomal frameshifting.
11 s mainly due to PA-X, which was expressed by ribosomal frameshifting.
12 d open reading frame ("X-ORF"), accessed via ribosomal frameshifting.
13 through, ribosome biogenesis, and programmed ribosomal frameshifting.
14 All four pseudoknots cause -1 programmed ribosomal frameshifting.
15 active TK (TK-low phenotype), evidently via ribosomal frameshifting.
16 cane yellow leaf virus (ScYLV) stimulates -1 ribosomal frameshifting.
17 structure are required for the programmed -1 ribosomal frameshifting.
18 ading frames required for programmed -1 mRNA ribosomal frameshifting.
19 tau and a truncated gamma that is created by ribosomal frameshifting.
20 1) regulates the efficiency of programmed -1 ribosomal frameshifting.
21 eing a truncated version of tau arising from ribosomal frameshifting.
22 nce of a novel structure that can facilitate ribosomal frameshifting.
23 d signals that are involved in programmed -1 ribosomal frameshifting (-1 PRF) are typically two-stemm
24 determinants of stimulation of -1 programmed ribosomal frameshifting (-1 PRF) by RNA pseudoknots are
32 d related alphaviruses utilize programmed -1 ribosomal frameshifting (-1 PRF) to synthesize the viral
34 oit one such mechanism, termed -1 programmed ribosomal frameshifting (-1 PRF), to engineer ligand-res
36 vious studies have identified operational -1 ribosomal frameshifting (-1 RF) signals in eukaryotic ge
41 pe and increased efficiency of programmed -1 ribosomal frameshifting and conferred paromomycin sensit
42 the genomic mRNA was critical for sufficient ribosomal frameshifting and EIAV replication, while conc
43 rts a trans-dominant effect on programmed -1 ribosomal frameshifting and killer virus maintenance.
45 nsferase activity, stimulating programmed -1 ribosomal frameshifting and promoting virus propagation
46 er refine the relationship between efficient ribosomal frameshifting and pseudoknot structure and sta
47 oted increased efficiencies of programmed -1 ribosomal frameshifting and rendered cells unable to mai
48 of the potential link between -1 programmed ribosomal frameshifting and response of a pseudoknot (PK
49 " model in which viruses use both programmed ribosomal frameshifting and translational attenuation to
50 not just unconventional initiation, but also ribosomal frameshifting and/or imperfect repeat DNA repl
53 molecular mechanisms governing programmed -1 ribosomal frameshifting are almost identical from yeast
54 ope whose expression results from incidental ribosomal frameshifting at a sequence element within the
55 f functional antizyme requires programmed +1 ribosomal frameshifting at the 3' end of the first of tw
56 mutation that increased the efficiency of -1 ribosomal frameshifting at the L-A virus frameshift site
59 he molecular mechanisms governing programmed ribosomal frameshifting by using two viruses of the yeas
61 in testing the hypothesis that programmed -1 ribosomal frameshifting can be used to control cellular
62 he basis of studies using cell-free systems, ribosomal frameshifting can explain this ability to expr
64 identification of novel frameshift proteins, ribosomal frameshifting, coding sequence detection and t
66 of translational recoding events (programmed ribosomal frameshifting, codon redefinition and translat
67 t killer virus phenotype, suggesting that -1 ribosomal frameshifting does not occur after the peptidy
68 there is an unusually high level, 15%, of +1 ribosomal frameshifting due to features of the nascent p
69 pe 1 (HIV-1) has an absolute requirement for ribosomal frameshifting during protein translation in or
70 It is generally believed that significant ribosomal frameshifting during translation does not occu
72 t signals, promoting increased programmed -1 ribosomal frameshifting efficiencies and subsequent loss
73 e inhibitors, anisomycin and sparsomycin, on ribosomal frameshifting efficiencies and the propagation
75 in of Rous sarcoma virus (RSV) requires a -1 ribosomal frameshifting event at the overlap region of t
76 lyses of alphavirus genomes suggested that a ribosomal frameshifting event occurs during translation
77 e RNA sequence that directs a programmed, +1 ribosomal frameshifting event required for Gag-Pol trans
78 t al. describe a novel, antibiotic-dependent ribosomal frameshifting event that activates translation
80 that a specific conformation is required for ribosomal frameshifting, further implying a specific int
81 li an autoregulatory mechanism of programmed ribosomal frameshifting governs the level of polypeptide
83 ghly accurate, a number of cases of directed ribosomal frameshifting have been reported in RNA viruse
84 nals are associated with sites of programmed ribosomal frameshifting, hopping, termination codon supp
86 ecific mRNA elements required for sufficient ribosomal frameshifting in equine anemia infectious viru
87 ation, specifically inhibits Ty1-directed +1 ribosomal frameshifting in intact yeast cells and in an
88 that provide one of the signals required for ribosomal frameshifting in mouse mammary tumor virus hav
89 h is a mutant of the pseudoknot required for ribosomal frameshifting in mouse mammary tumor virus, ha
91 fluenza virus virulence protein generated by ribosomal frameshifting in segment 3 of influenza virus
92 e cis-acting elements that promote efficient ribosomal frameshifting in the -1 (5') direction have be
95 these drugs also change the efficiency of -1 ribosomal frameshifting in yeast and mammalian in vitro
98 totiviruses, the efficiency of programmed -1 ribosomal frameshifting is critical for ensuring the pro
101 y support the mechanistic hypothesis that -1 ribosomal frameshifting is enhanced by torsional resista
103 e slippery sequence and stem-loop to promote ribosomal frameshifting is influenced by the flanking up
107 iae double-stranded RNA virus, programmed -1 ribosomal frameshifting is responsible for translation o
113 shift/slippage site, which is important for ribosomal frameshifting, is shown here to limit reverse
117 ed exclusively as a Gag-Pol fusion either by ribosomal frameshifting or by read-through of the gag st
118 pathogenic RNA viruses and retroviruses use ribosomal frameshifting or stop codon readthrough to reg
119 unclear, a novel viral protein expressed by ribosomal frameshifting, PA-X, was found to play a major
120 ing mRNA elements that promote programmed -1 ribosomal frameshifting present a natural target for the
121 Coronavirus (SARS-CoV) employ programmed -1 ribosomal frameshifting (PRF) for their protein expressi
124 Translational control through programmed ribosomal frameshifting (PRF) is exploited widely by vir
125 nse and activating a unique -2/-1 programmed ribosomal frameshifting (PRF) signal for the expression
129 putative feline immunodeficiency virus (FIV) ribosomal frameshifting pseudoknot (PK) has been investi
130 s on killer virus maintenance, programmed -1 ribosomal frameshifting, resistance/hypersensitivity to
135 frames, the over-reading of stop codons via ribosomal frameshifting, the existence of an antizyme an
138 A1 undergo highly efficient +1/-2 programmed ribosomal frameshifting to generate previously undescrib
139 Many pathogenic viruses use programmed -1 ribosomal frameshifting to regulate translation of their
140 isiae killer virus system uses programmed -1 ribosomal frameshifting to synthesize its gene products.
141 part of its life cycle, termed programmed -1 ribosomal frameshifting, to produce the required ratio o
142 e codons and/or the process of programmed -1 ribosomal frameshifting used by viruses to control their
143 distribution of recoding with a focus on the ribosomal frameshifting used for gene expression in bact
145 doknots in controlling the extent of -1-type ribosomal frameshifting, we determined the crystal struc
146 th sequences that trigger genuine programmed ribosomal frameshifting; we have experimentally confirme
147 ally mimic these RNA structures to induce +1 ribosomal frameshifting when annealed downstream of the
148 te tRNA slippage is the driving force for +1 ribosomal frameshifting while the presence of a 'hungry
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