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1 ene under standard conditions (30 degrees C, rich medium).
2 gbuABC) allowed DeltadacA mutants to grow in rich medium.
3 41 when the bacteria were grown in phosphate-rich medium.
4 uring growth in minimal medium compared with rich medium.
5 per cell in cells grown in either minimal or rich medium.
6 f SER3 is tightly repressed during growth in rich medium.
7 or induction of phenazine gene expression in rich medium.
8 CS) was stimulated by pheromone signaling in rich medium.
9 ximately 3% of yeast genes in cells grown in rich medium.
10 dent when cells approach stationary phase in rich medium.
11  exposure to NCS, although grown in nutrient-rich medium.
12  bacillus Calmette-Guerin on minimal but not rich medium.
13 t on the in vitro growth of V. cholerae in a rich medium.
14  long as those of cells growing in phosphate-rich medium.
15 stems normally repressed in cells growing in rich medium.
16  strain is half that of the parent strain in rich medium.
17 y a slight increase in autophagy in nitrogen-rich medium.
18 ndergo a similar invasive growth response in rich medium.
19 , 17 percent were essential for viability in rich medium.
20 m and are unable to form colonies on complex rich medium.
21 s) using a chemically defined, growth factor-rich medium.
22 cts of nup184 mutants when grown in nutrient-rich medium.
23 ic complete medium, but were able to grow on rich medium.
24 vasive filaments which penetrate the agar in rich medium.
25 in stationary-phase cultures after growth in rich medium.
26  a temperature-sensitive-lethal phenotype in rich medium.
27 ned in early-stationary-phase cells grown in rich medium.
28  hydroxamate to cultures growing in nutrient-rich medium.
29 on of oxyR in E. coli growing aerobically in rich medium.
30 old) over a broad range of cell densities in rich medium.
31 l growth rate (mu) and maximum growth (A) in rich medium.
32  growing in a minimal medium than those in a rich medium.
33 d in short-term maintenance in starvation or rich medium.
34 e) complex when yeast are grown in phosphate-rich medium.
35 ressed at early stationary phase in nutrient-rich medium.
36  of the mutant was even more pronounced in a rich medium.
37 toplasmic when yeast were grown in phosphate-rich medium.
38  detected for the other csp genes in defined rich medium.
39 RNA accumulation after cold-shock in defined rich medium.
40 ntial or strongly advantageous for growth in rich medium.
41 ng filamentation at 37 degrees C in nutrient-rich medium.
42 ed for survival of engineered bacterium on a rich medium.
43  is repressed by NilR and growth in nutrient-rich medium.
44 ld reduced for cells grown in minimal versus rich medium.
45 nes necessary for optimal growth in nutrient-rich medium.
46   Unexpectedly, the strain was not viable on rich medium.
47 thesis but replication competent in vitro in rich medium.
48 on in Clostridium difficile during growth in rich medium.
49 w about 30% slower than the parent strain in rich medium.
50 ble but switched at high rates when grown in rich medium.
51 re but had no detectable effect on growth in rich medium.
52 rmed on mid-logarithmic-phase cells grown in rich medium.
53 ptor and declined to a greater extent in LDL-rich medium.
54 ors not influenced during growth in nutrient-rich medium.
55  that germinate when transferred to nutrient-rich medium.
56 cpA isogenic mutant strain grown in nutrient-rich medium.
57 found in TC during rapid growth in a complex rich medium.
58 ole in germination of C. difficile spores in rich medium.
59 on, and during growth in the laboratory on a rich medium.
60 er low-iron conditions and repressed in iron-rich medium.
61 ined glucose-containing medium or a nutrient-rich medium.
62 n the cytoplasm of Escherichia coli grown in rich medium.
63  expression compared to growth in a nutrient-rich medium.
64 ts involving 67 different metabolic genes in rich medium.
65 tion of Caco-2 cells for 3 days in a peptide-rich medium (4 mM Gly-Gln replacing 4 mM Gln).
66                     If cells are returned to rich medium after they have committed to meiosis, the tr
67 fluence the rate of growth or spreading on a rich medium, AldA was required for growth on a minimal m
68 le, sup70-65, induces pseudohyphal growth on rich medium, an inappropriate nitrogen starvation respon
69 ssue culture monolayers but normal growth in rich medium anaerobically.
70 the cooperative network of 204 regulators in Rich Medium and a subset of them in four different envir
71 s collected from PANC-1 cells grown in serum-rich medium and at 24 h following the removal of serum.
72 erichia coli nucleoid during rapid growth in rich medium and following an induced amino acid starvati
73 as required for full Ribi gene expression in rich medium and for its modulation in response to glucos
74 or metabolic changes that occur in growth in rich medium and host cells but not minimal medium.
75 ed fitness defects in vitro during growth in rich medium and in gnotobiotic mice colonized with defin
76 an glyoxalase II (Glx2) was overexpressed in rich medium and in minimal medium containing zinc, iron,
77 er meiosis, in the shape of budding cells in rich medium and in the morphology of filamentous growth
78 sm when budding yeast are grown in phosphate-rich medium and is unphosphorylated and localized to the
79 letion mutant had a growth defect in protein-rich medium and mimicked the phenotype of a generated Ta
80  These mutants fail to form aerial hyphae on rich medium and most are defective in antibiotic product
81 romoter causes cell-cycle arrest in nutrient-rich medium and promotes developmental events, such as t
82 analyze the growth capacity under amino-acid-rich medium and provide evidence that amino acid prefere
83  WT actin cells under standard conditions in rich medium and rescued the mitochondrial phenotype resu
84 with salt stabilized the DeltadacA mutant in rich medium and restored cefuroxime resistance.
85 e to nutrients, moving quickly through G1 in rich medium and slowly in poor medium.
86 rown microaerobically and anaerobically in a rich medium and soluble and membrane proteins of strain
87 prfA cultures grown at 30 or 37 degrees C in rich medium and that the lack of PrfA exacerbates the ch
88                    The repressive effects of rich medium and the stimulatory effects of ppGpp were al
89 e when cells entered the stationary phase on rich medium and was replenished upon restoration of favo
90 hich are likely to be required for growth on rich medium and which represent potential therapeutic ta
91 rs in the absence of major growth defects in rich medium and with classical virulence-associated phen
92 tribute to selection of lasR mutants both on rich medium and within the CF airway, supporting a key r
93 e cycle that includes vegetative swarming on rich medium and, upon starvation, aggregation to form fr
94 0.1% of the tatC mutant cells were motile in rich medium, and <0.01% were motile in vir induction med
95  studies in minimal growth conditions and LB rich medium, and from clinical tests that identify V.cho
96 low temperature, glucose as a carbon source, rich medium, and high osmolarity.
97 ntly higher levels in minimal medium than in rich medium, and rpoS expression was similarly elevated.
98 in growth rate, moving quickly through G1 in rich medium, and slowly in poor medium.
99 ted by specific nutrients in the medium, but rich medium appears to suppress pzX formation.
100  maize (Zea mays) seedlings cultivated in Se rich medium are also presented.
101 growth and chromosome segregation defects in rich medium are suppressed by a reduction of replication
102      Bacteria were first grown in a nutrient-rich medium at 26 degrees C to establish a baseline of g
103 sly in sup70-65 mutants cultured in nitrogen-rich medium at 30 degrees C.
104 aZ inhibited cell division and was lethal in rich medium at high induction levels and in minimal medi
105           The mutant derivative grew well in rich medium but did not grow in minimal medium supplemen
106 logarithmic growth phase in cells growing in rich medium but poorly expressed in late logarithmic and
107  encodes sigma(S), does not affect growth in rich medium but severely decreases L. pneumophila intrac
108 himurium was unimpaired on aerobic growth in rich medium but showed enhanced sensitivity in vitro to
109  short in blue light when grown on a sucrose-rich medium but tall when grown on sucrose-deficient med
110 HI mutants of zevA and zevB grew normally in rich medium but were defective for colonization in a mou
111 n of Sis1 is dispensable for rapid growth on rich medium, but is required for [RNQ+] maintenance, dis
112  was also expressed as a single mRNA in iron-rich medium, but its expression was reduced approximatel
113 I transcription during exponential growth in rich medium, but the mechanism of this regulation has re
114  cells in the exponential phase of growth in rich medium by artificial induction of the synthesis of
115 her kinase in vivo inhibits GCN2 function in rich medium by reducing tRNA binding activity.
116 Fe(III) reductase activities were induced in rich medium by the addition of cAMP to aerobically growi
117       When grown on a soft agar surface in a rich medium, cells of Salmonella typhimurium elongate, p
118 on for 106 transcription factors profiled in rich medium conditions data from over 500 expression exp
119 reatest in cells grown at low temperature in rich medium, conditions in which the growth defect was m
120  medium and high in cells growing rapidly in rich medium, consistent with the previously recognized r
121 rement for this pathway even when growing in rich medium containing DAP.
122 ced cells expressing the Mal61/HA protein to rich medium containing glucose produces a decrease in ma
123 wed that YmgB represses biofilm formation in rich medium containing glucose, decreases cellular motil
124 erely attenuated for aerobic growth, even in rich medium containing supplemental amino acids, and exh
125 tamate or casamino acids as well as nutrient-rich medium containing yeast extract, peptone, and aceta
126 I when P. pastoris is cultured in a nutrient-rich medium containing yeast extract, peptone, and metha
127 id induction of gadA and gadBC observed when rich-medium cultures enter stationary phase corresponds
128 charomyces cerevisiae populations growing in rich medium for 1,000 generations.
129  acids (e.g., phenylalanine) and thus, needs rich medium for cell growth.
130                                When grown in rich medium, greater than 50% of the minCD mutant cells
131                             The cells in the rich medium grew faster, and expression of the majority
132 sources and hyperaccumulation of glycogen in rich medium high in Pi.
133               Cultures were grown in glucose-rich medium in the presence or absence of 20 mM benzoate
134  MYO1 (the only myosin II gene) is lethal on rich medium in the W303 strain background and causes slo
135 cytosine methyltransferase, during growth in rich medium in vitro.
136  transposon-containing cells are selected on rich medium, insertions occur at both attTn7 sites with
137 Salmonella enterica, SP induction of RpoS in rich medium is >30 fold and includes effects on both tra
138 The pus7 deletion strain, although viable in rich medium, is growth-disadvantaged under certain condi
139 0%) has no obvious phenotypic consequence in rich medium, it is difficult to study their functions.
140 ucleolus than in the nucleoplasm, whereas on rich medium, it was more evenly distributed between the
141 ng GerO were defective in germination with a rich medium, KCl, L-asparagine, and a 1:1 chelate of Ca(
142 on in the UPEC strain UTI89 during growth in rich medium (LB medium) and urine and during infection o
143  adaptation to four laboratory environments (rich medium, low glucose, high salt, and a nonfermentabl
144                      However, for cells in a rich medium, Lrp levels dropped 3- to 4-fold during the
145 ession of lrp during logarithmic growth in a rich medium may be due to low ppGpp levels under these c
146 plays a role in NO, as mukB mutants grown in rich medium often exhibit FtsZ rings on top of diffuse,
147                    When cells are grown in a rich medium on agar instead, they elongate, produce more
148                              When grown in a rich medium on agar, many bacteria elongate, produce mor
149 tative bacteria that can swim are grown in a rich medium on an agar surface, they become multinucleat
150                               Upon growth in rich medium, only slight protein export defects were obs
151 lation after growth into stationary phase in rich medium or after osmotic challenge.
152 wo molybdoenzyme activities when cultured in rich medium or glucose-minimal medium.
153 ting) in cells grown in a chemically defined rich medium or in a minimal medium.
154 s into the culture supernatant when grown in rich medium or in vir induction minimal medium.
155 jected to nutritional or osmotic stress in a rich medium or to nitrogen exhaustion had large and dyna
156 of mitochondrial iron in cells grown in iron-rich medium overexpressing MFT1 or MFT2 show a 2-5-fold
157  cell populations (those incubated in a 5-HT-rich medium overnight) and in unloaded populations and w
158 sted temperature, osmolarity, carbon source, rich medium, oxygen levels, pH, amino acids, solid mediu
159 ium but that in plant oil minimal medium and rich medium, phaB3 seems to be unexpressed.
160                     When cells were grown in rich medium, PhaZ1 was sufficient to account for intrace
161  pulsed-laser ablation of OMS-2 in an oxygen-rich medium produces a three-dimensional nanostructured
162  mutant strains were grown on a conventional rich medium (R2YE, containing high sucrose), inactivatio
163 aromyces cerevisiae culture during growth in rich medium rapidly decreases by 40 to 60% when the cell
164    Rhodobacter sphaeroides grown in a Mn(II)-rich medium replaces the intrinsic Mg(II) ion with an EP
165  osmolarity, glucose as a carbon source, and rich medium repressed transcription of all three operons
166 ic induction of the PHO5 acid phosphatase in rich medium requires the transcriptional activators Pho4
167                              Growth to SP in rich medium results in a 30-fold induction of RpoS, alth
168                 Evolving faster migration in rich medium results in slow growth and fast swimming, wh
169 ghout the growth of the bacterial culture in rich medium revealed a dramatic increase in all four NTP
170 073 grown statically to exponential phase in rich medium, revealing the strategies this pathogen uses
171 he generation of common and stereochemically rich medium-sized benzo-fused sultams via complementary
172 Such plasmids are stably maintained, even in rich medium, so optimising biomass production and yields
173 ntially in time after exposure to a nutrient-rich medium, so that the CaDPA amount inside the trapped
174 low-growth conditions, but were sensitive to rich medium, suggesting that the dense packing of initia
175 hat did not fix nitrogen, and grew slowly in rich medium, suggesting that the locus was a cyc gene en
176  binding, were expressed at higher levels in rich medium, suggesting that, upon exposure to a rich en
177 d in wild-type M. smegmatis, using selective rich medium supplemented with DAP unless there was an ex
178                       E. coli cells grown in rich medium that contained all amino acids and low amoun
179 d into nitrogen-limited medium from nutrient-rich medium, the cell size increased two- to threefold,
180     As Fis levels increase in cells grown in rich medium, the positioning of its binding site, overla
181 s strongly induced simply upon switch from a rich medium to a minimal medium in the complete absence
182 se pathways when transferred from amino acid-rich medium to amino acid-deficient medium.
183 epeated cycles of single colony isolation on rich medium to generate lineages that are forced through
184   When irradiated log phase WT cells were in rich medium, two TS domains were evident: a fast-repaire
185                                  In nutrient-rich medium, UgtP is distributed more or less uniformly
186 ls to those grown to midexponential phase in rich medium under aerobic conditions.
187 he resulting mutants revealed that growth in rich medium under low-O(2) conditions was indistinguisha
188 icant selective disadvantage just growing on rich medium under normal conditions.
189 ccumulate PHB when it is grown in a nutrient-rich medium (up to 23% of the cell dry weight in dextros
190                                           In rich medium, V. cholerae expresses almost exclusively th
191 dition, regulation of citB(Lm) expression in rich medium was growth phase dependent; during exponenti
192            Endospore formation in amino-acid-rich medium was significantly defective and germination
193 tationary-phase cultures in glucose-depleted rich medium was truncated by switching the cells to phos
194 es, spore germination, and optimal growth on rich medium, was predictive of the contributions of two
195                                           In rich medium we detected transcripts for 97% and 87% of t
196   Genes required for viability of E. coli in rich medium were identified on a whole-genome scale usin
197  PBPs possess largely redundant functions in rich medium, when divalent cations are limiting, PBP1 is
198 lta with respect to ste11(+) derepression in rich medium, whereas the opposite relationship, gcn5Delt
199  due to indicator accumulation in the Ca(2+)-rich medium, which induces an increase in the fluorescen
200 pletely spliced during exponential growth in rich medium, which suggests that meiosis is not the only
201 ysine-supplemented defined medium or complex rich medium, while the BCG mutants grow only on lysine-s
202        C. albicans yeast cells were grown in rich medium with 2% glucose.
203          In contrast, a MC grown in nutrient rich medium with addition of SCB had a lower microbial d
204                                           In rich medium with high amino acid content further incubat
205                                Supplementing rich medium with short, branched-chain fatty acids or de
206 ons confer sensitivity to growth in nutrient-rich medium (YES) that is accompanied by nuclear poly(A)

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