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1 cells indicated IMD pathway activation upon rickettsial infection.
2 tress-induced toxic nucleotide levels during rickettsial infection.
3 the protective inflammatory response during rickettsial infection.
4 CTLs appear to be crucial to recovery from rickettsial infection.
5 s was not the explanation for the control of rickettsial infection.
6 and prevented the suppression of the initial rickettsial infection.
7 te production and suppression of the initial rickettsial infection.
8 ntibody-mediated killing confers immunity to rickettsial infection.
9 s of the immune response, remains unclear in rickettsial infections.
10 educe inflammatory damage to the host during rickettsial infections.
11 ole early during the immune response against rickettsial infections.
12 (a scavenger of extracellular NO) during the rickettsial infection alleviated the suppression of the
14 r interleukin-12 (IL-12) p40 production upon rickettsial infection and were more potent in priming na
15 charide exhibited suppression of the initial rickettsial infection, and the suppression was relieved
17 d against the major infected target cells of rickettsial infections, endothelial cells and macrophage
20 ions may be as common as spotted fever group rickettsial infections in febrile patients from central
21 hermore, NK cells are involved in preventing rickettsial infection-induced endothelial cell damage, p
24 on alleviated the suppression of the initial rickettsial infection observed in appropriately treated
25 travelers returning from sub-Saharan Africa, rickettsial infection, primarily tick-borne spotted feve
26 activity was more critical to recovery from rickettsial infection than were the effects of IFN-gamma
27 , in relevant animal models that mimic human rickettsial infections, there is reciprocal immunologica
28 product of NO) were produced and the initial rickettsial infection was suppressed in cultures of L929
30 factors associated with the tick response to rickettsial infection, we utilized differential-display
31 TLR4 signaling developed overwhelming, fatal rickettsial infections when given an inoculum that was n
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