ライフサイエンスコーパス: right atrium

1 ive cardiomyocyte cell cycle activity in the right atrium).

2 nd 19+/-9%, 18+/-3% [P<0.001, <0.001] in the right atrium).

3 xtent of fibrosis was more pronounced in the right atrium.

4 reased homogeneously throughout the left and right atrium.

5 postero-inferior left atrium and the caudal right atrium.

6 ion time, CTni, through the remainder of the right atrium.

7 oltage areas ("scars") in the posterolateral right atrium.

8 pplied to transect critical isthmuses in the right atrium.

9 iod and APD are closely related in the human right atrium.

10 st frequently of right coronary arteries and right atrium.

11 and severe in the right coronary artery and right atrium.

12 inear ablation directed at the inferolateral right atrium.

13 aticity of pacemaker cells isolated from cat right atrium.

14 erature in vivo just prior to entry into the right atrium.

15 calculated at 3360 endocardial sites in the right atrium.

16 rant circuits could be identified within the right atrium.

17 multiple multipolar electrodes placed in the right atrium.

18 ed mapping with the ablation catheter in the right atrium.

19 hyarrhythmia most often contained within the right atrium.

20 in was bound to PLL), and reinfused into the right atrium.

21 t the junction of the superior vena cava and right atrium.

22 long-term central venous catheters into the right atrium.

23 rastimuli at one to four pacing sites in the right atrium.

24 ganized T-tubules and topography than in the right atrium.

25 catheters in the coronary sinus and lateral right atrium.

26 terclockwise inductions were from the smooth right atrium.

27 cent sites within the coronary sinus and the right atrium.

28 ll as prolapse of the ruptured head into the right atrium.

29 t ventricle of the heart and dilation of the right atrium.

30 rial pacemaker (LAP) cells isolated from cat right atrium.

31 rial pacing lead positioned in the posterior right atrium.

32 located in the left atrium and 52.2% in the right atrium.

33 5.1+/-40.2 (n=12) expression ratio of SAN to right atrium.

34 sting active gene transcription in the human right atrium.

35 electrical impedance of blood in-vivo in the right atrium.

36 rse of the right coronary artery through the right atrium.

37 cells (hCDCs) grown from neonatal and infant right atrium.

38 an SAN, and the conduction pathways into the right atrium.

39 o phospholamban protein ratio in SAN than in right atrium.

40 ventricle, and a bipolar pacing wire in the right atrium.

41 A latex balloon was advanced into the right atrium.

42 Mean AF cycle length was longer in the right atrium (109.2+/-5 ms) than in the left atrium (85.

43 ) than inferior SAN (138+/-24 ms; P=0.01) or right atrium (164+/-33 ms; P=0.001) and was associated w

44 rine or saline placebo was injected into the right atrium 2 min after the start of precordial compres

45 re: left ventricle (4), right ventricle (3), right atrium (2), right atrium/superior vena cava (1), a

46 ar the pulmonary vein ostia (4) and from the right atrium (2), whereas adenosine-sensitive AT arose f

47 the position error was 1.9+/-0.9 mm for the right atrium, 2.7+/-1.2 mm for the right ventricle, 1.8+

48 al flutter; (4) another delay on the lateral right atrium (283+/-52 ms); and (5) typical atrial flutt

49 the HF group and increased minimally in the right atrium (3+/-3%, 17+/-5% [P=NS, P=0.001]).

50 xolone prolonged electrogram duration in the right atrium (39.7+/-4.2 to 42.3+/-4.3 ms; P=0.01) and r

51 left atrium: 76 with TATS, 30 without TATS; right atrium: 42 with TATS, 58 without TATS).

52 often coming from the atrial septum via BB (right atrium, 62%; left atrium, 67%).

53 nd LAFW was significantly higher than in the right atrium (7.8+/-0.3 Hz; P<0.001).

54 left atrium (65+/-16 vs 41+/-9 mL/m(2)), and right atrium (78+/-17 vs 56+/-17 mL/m(2); P<0.01 for all

55 cine or saline placebo was injected into the right atrium after 5 mins of untreated ventricular fibri

56 e serving as a placebo was injected into the right atrium after 7 mins of untreated ventricular fibri

57 right coronary arteries and arteries of the right atrium after ETRA infusion for 5 days.

58 ally practical third electrode placed in the right atrium along the atrial septum (RSP).

59 endocardial activation from the high lateral right atrium, along with a step-wise reduction in heart

60 ved 20 mL/kg saline (vehicle) bolus into the right atrium and 0.01 mL/kg/min i.v., beginning 20 mins

61 n these 22 patients, including 15 located in right atrium and 10 in the left atrium.

62 on zones (mean duration, 57 +/- 16 ms in the right atrium and 53 +/- 23 ms in the left atrium) observ

63 re obtained before venous cannulation of the right atrium and after myocardial reperfusion.

64 nade, and death caused by perforation of the right atrium and aorta by a stent after embolization fro

65 leads were placed in the lateral wall of the right atrium and at the roof of the left atrium in Bachm

66 s were significantly higher expressed in the right atrium and atrioventricular node compared with lef

67 W potentials were more often observed at the right atrium and Bachmann's bundle (P<0.001).

68 chieved by simultaneously pacing at the high right atrium and coronary sinus ostium at an identical r

69 Dual-site right atrial pacing from the high right atrium and coronary sinus ostium can suppress indu

70 single-site right atrial pacing modes (high right atrium and coronary sinus ostium) and the long-ter

71 until the beat that initiated flutter, when right atrium and coronary sinus were activated in sequen

72 ical stimulation was performed from the high right atrium and CSd, and bipolar recordings were obtain

73 ine alone, with similar findings during high right atrium and distal coronary sinus pacing.

74 Epinephrine administration via the right atrium and femoral vein resulted in significant in

75 sponse to pacing were recorded from the high right atrium and from the distal coronary sinus.

76 Right atrium and LA burst pacing (burst) (cycle length 5

77 ajority of IART circuits involve the lateral right atrium and may be successfully ablated by creating

78 Fs found in the nearest atrium (rho=0.96 for right atrium and rho=0.92 for left atrium) and the DF gr

79 therapeutic hypothermia system placed in the right atrium and set at a target of 32 degrees C.

80 nd 15 catheter tip malpositions (nine in the right atrium and six in the right axillary vein).

81 left atrial appendages, the junction of the right atrium and superior vena cava, crista terminalis,

82 and a strictly delimited cluster between the right atrium and superior vena cava.

83 effective refractory period between the high right atrium and the coronary sinus ostium pacing sites

84 enlargement of the right ventricle (RV) and right atrium and tricuspid regurgitation.

85 cava flow passed almost exclusively into the right atrium and tricuspid valve; a small amount that wa

86 e, reduced RV function (manifest as a larger right atrium and ventricle and lower RV stroke work inde

87 comparing proliferation in the normotensive right atrium and ventricle and pressure-overloaded left

88 The acute effect of pacing the right atrium and ventricle may be detrimental to both sy

89 on of the pulmonary artery, left atrium, and right atrium, and a flow probe was positioned around the

90 Blood samples were taken from the LA, right atrium, and femoral vein at baseline and at 15 min

91 ng various H(2)R mutants, at the isolated gp right atrium, and in GTPase assays for activity on recom

92 nces in pressure between inferior vena cava, right atrium, and left atrium were found.

93 in lateral (p < 0.01) and septal (p = 0.03) right atrium, and proximal (p = 0.02) and distal (p < 0.

94 globin saturation in the superior vena cava, right atrium, and pulmonary artery (SVO2) was measured b

95 tion was the lowest toward the apex from the right atrium, and the anteroseptal portion was the highe

96 ng tantalum-impregnated blood clots into the right atrium, and the rabbits were radiographed to locat

97 minute, under ventricular fibrillation, the right atrium-aorta system was significantly the most eff

98 rculatory support systems were compared: (1) right atrium-aorta, extracorporeal membrane oxygenation

99 , revealed clockwise LLR involving the lower right atrium around the IVC in 7 patients, figure-of-8 d

100 ation capacity was also greatest in neonatal right atrium as evidenced by c-kit(+), NKX2-5(+), NOTCH1

101 mitral regurgitation and rapidly pacing the right atrium at 640 bpm for >8 weeks.

102 Animals were paced in the right atrium at a rate of 130 beats per minute (bpm).

103 Animals were paced via the right atrium at a rate of 130 bpm.

104 Configurational changes were noted in the right atrium at pacing sites 17 mm from the distal pole.

105 Standard leads were placed in the right atrium, at the right ventricular apex, and in a co

106 atrial myocardial structural changes in the right atrium, atrial fibrillation (AF) is a disease of v

107 pping (interelectrode distances 2 mm) of the right atrium, Bachmann's bundle, the left atrioventricul

108 he intact rat, cariporide, injected into the right atrium before chest compression was started (after

109 elate conduction block in the isthmus of the right atrium between the inferior vena cava and the tric

110 noted in the left atrium, whereas 8% of the right atrium burns had char.

111 or blood viscosity continuously in the human right atrium by a dedicated central venous catheter equi

112 brane oxygenation return blood flow upon the right atrium by considering the physiologic effects duri

113 guinea-pig left ventricle, left atrium, and right atrium, carbenoxolone increased R(j) by 28+/-9%, 2

114 Lactic acid injected into the right atrium caused a transient decrease in arterial blo

115 e congenital aneurysm or diverticulum of the right atrium caused repeated attacks of supraventricular

116 tly (all P < .05) greater in systole for the right atrium (CNR, 8.9 vs 7.5; image quality, 438 vs 91)

117 Islet-1(+) cells in the right atrium coexpressed the sinoatrial node pacemaker c

118 ure gradient between the right ventricle and right atrium (delta P) and cardiac output (CO).

119 e, systolic pressure gradient between RV and right atrium (DeltaPRV-RA), tricuspid regurgitation velo

120 in resident hCPCs, we isolated and expanded right atrium-derived CPCs from all patients (n=103) acro

121 tion, whereas the transmural pressure of the right atrium did not change.

122 Local capture of the right atrium during AF was demonstrated for at least one

123 on and prevents upregulation of miR-1 in the right atrium during coronary artery bypass graft.

124 n at the posteromedial (sinus venosa region) right atrium during counterclockwise and clockwise atria

125 ne placebo were injected as a bolus into the right atrium during CPR.

126 Pacing was performed at the high right atrium during type I AF in 24 patients in the elec

127 d left atrial response to pacing at the high right atrium during type I AF in humans.

128 ide or placebo in a dose of 3 mg/kg into the right atrium either 5 mins before or at 8 mins after ons

129 grams kg-1), given as a rapid bolus into the right atrium, elicited a burst of action potentials in t

130 g-1), administered as a rapid bolus into the right atrium, elicited a transient burst of action poten

131 of phenylbiguanide (PBG, 100 microg/kg) into right atrium elicits differential responses in the two p

132 ound that ERG is most abundant in the medial right atrium, especially in the trabeculae and the crist

133 biguanide (2-5 micrograms) injected into the right atrium, evoked a similar respiratory and cardiac r

134 % sodium chloride) was administered into the right atrium, femoral vein, or endotracheal tube.

135 os of the coronary sinus and the low lateral right atrium for both counterclockwise and clockwise flu

136 r catheterization of the inferior vena cava, right atrium, foramen ovale, and left atrium with a guid

137 requency) via two electrodes attached to the right atrium from 109 +/- 7.3 to 170 +/- 9.8 beats min-4

138 Furthermore, in dogs with AF, the right atrium had a significantly higher nerve density th

139 odgment of an undersized prosthesis into the right atrium had developed.

140 pacing was performed from four sites in the right atrium (high and low trabeculated and smooth right

141 rograms were recorded from the anterolateral right atrium, His bundle position, and coronary sinus.

142 Catheters were targeted to the right atrium, His bundle, and right ventricle of 10 mong

143 polar recordings were obtained from the high right atrium, His bundle, posterior triangle of Koch, an

144 more readily inducible by APDs from the high right atrium (HRA) than the coronary sinus (CS).

145 corded at the isthmus and either high or low right atrium (HRA, LRA) during overdrive pacing to 160 m

146 enous bolus injection of anandamide near the right atrium immediately elicited the pulmonary chemoref

147 ) and blood temperature (T) as obtained from right atrium impedance measurements: Viscosity(imp)=(-15

148 y sinus in 3 patients, to the posterolateral right atrium in 1 patient, and to the left atrial (LA) s

149 ms and monophasic action potentials from the right atrium in 35 patients with spontaneous, sustained

150 .1%), right atrial-SVC junction in 6 (9.1%), right atrium in 8 (12.1%), inferior vena cava in 1 (1.5%

151 FIRM analysis revealed sources in the right atrium in 85% of patients (1.8 +/- 1.3) and in the

152 ocated in the remainder of the morphological right atrium in all but 1 patient.

153 ICE probe was positioned in the right atrium in all patients, in the pulmonary artery in

154 Dobutamine was infused into the right atrium in an amount of 5 microg.kg.min.

155 t sinus node (RSN) in the innervated remnant right atrium in cardiac transplant patients were compare

156 The right atrium in CHD supports a variety of IART mechanism

157 corded from five equally spaced sites in the right atrium in patients undergoing electrophysiology st

158 Four patients had right atrium incisional atrial flutter (AFL), and 6 had

159 NOS-1 gene transfer to the guinea pig right atrium increased protein expression and NOS-1 immu

160 lutter, whereas pacing from the trabeculated right atrium induces clockwise flutter.

161 he site of induction: Pacing from the smooth right atrium induces counterclockwise flutter, whereas p

162 2), right atrium/superior vena cava (1), and right atrium/inferior vena cava (1).

163 e; a small amount that was refluxed from the right atrium into the inferior vena cava subsequently pa

164 inus ostium atrial timing during low lateral right atrium IP at cycle length of 600 ms through 300 ms

165 ased, whereas the transmural pressure of the right atrium is not changed.

166 he slow AV node pathway in the posteroseptal right atrium is the preferred therapeutic approach in pa

167 unication between the left ventricle and the right atrium, is presented.

168 tion of either form of flutter is in the low right atrium isthmus.

169 to evaluate the impact of an incision in the right atrium joining the lateral tunnel suture line and

170 thod to position the catheter tip within the right atrium just as accurately (average, 1.9 +/- 1.3 cm

171 This compensatory response of the right atrium likely plays an important role in preventin

172 d in the RAA, CS, pulmonary artery (PA), low right atrium (LRA), and across the SP.

173 d activation of plasma AII and ET in plasma, right atrium, lung, and renal medulla which was further

174 incremental pacing (IP) from the low lateral right atrium may distinguish slow conduction from comple

175 ent foramen ovale, increased pressure in the right atrium may result in widening of the foramen and c

176 rial systole, indicating that coronary sinus-right atrium muscle continuity is likely the primary cau

177 Coronary sinus-right atrium muscle continuity was indirectly evaluated

178 ed in the coronary sinus (n = 29) and in the right atrium (n = 10).

179 EBW mostly occurred at right atrium (N=105, 48%) and left atrioventricular groo

180 de, and were more commonly identified in the right atrium (n=25) than in the right ventricle (n=5).

181 atedly at the identical anatomic site in the right atrium (n=8 swine).

182 hrough near the CS os, 1 patient in the high right atrium near the presumed insertion of Bachmann's b

183 The SA node region was isolated from the right atrium of guinea pigs between birth and 38 months

184 ansmural and continuous can be placed in the right atrium of normal pigs.

185 second study, infusion of PROLI/NO into the right atrium of sheep with induced pulmonary hypertensio

186 r Lenti.EF1alpha-eGFP was transferred to the right atrium of Spague-Dawley (SD) rats and acetylcholin

187 l left hepatic vein draining directly to the right atrium of the donor heart, which was discovered du

188 were tested in vivo with injection into the right atrium of the endogenous heart.

189 sequence of activation on the surface of the right atrium of the Langendorff-perfused sheep heart dur

190 The constructs were transfected into the right atrium of transplanted hearts.

191 chycardia-induced HF pacing, either from the right atrium or high right ventricular free wall.

192 ava, through drainage into the hepatic vein, right atrium or left atrium.

193 site atrial pacing was performed at the high right atrium or the coronary sinus ostium.

194 n of 0.06 ml/kg oleic acid solution into the right atrium over a 30-min period.

195 roposterior left atrium (p < 0.01), superior right atrium (p < 0.05), and inferior right atrium (p <

196 perior right atrium (p < 0.05), and inferior right atrium (p < 0.05).

197 tion to standard atrial pacing from the high right atrium, pacing from novel sites like the interatri

198 ) patients, was patent at the entry into the right atrium (PFO) in 62 patients (61.4% of patients wit

199 An 8F AcuNav probe was introduced into right atrium, pulmonary artery, and coronary sinus.

200 ncreased cardiac output and pressures in the right atrium, pulmonary artery, and pulmonary capillary

201 from the multipolar catheters located in the right atrium (RA) and coronary sinus (CS).

202 found in 12 of 20 optical recordings of the right atrium (RA) and in all (n=19) recordings of the le

203 usculature has electrical connections to the right atrium (RA) and left atrium (LA) and forms an RA-L

204 The spread of activation between the right atrium (RA) and left atrium (LA), particularly alo

205 er was navigated to preselected sites in the right atrium (RA) and right ventricle (RV) in the first

206 ize and function of the left atrium (LA) and right atrium (RA) are related closely with the prognosis

207 y from the coronary sinus (CS) and posterior right atrium (RA) during AF.

208 greater than the sinus rate from the lateral right atrium (RA) during control, followed by 2 periods

209 ccurred along BB and IPP, resulting in an LA-right atrium (RA) frequency gradient of 5.7+/-1.4 HZ: Le

210 Electrogram recordings were made from a wide right atrium (RA) to left atrium (LA) bipole and digital

211 e compared: cryoablation of the inferomedial right atrium (RA), and a more extensive modified RA maze

212 lower-loop reentry (LLR), involved the lower right atrium (RA), as manifested by early breakthrough i

213 Transvenous leads were implanted into the right atrium (RA), coronary sinus, and left pulmonary ar

214 ent to the inferior and medial aspect of the right atrium (RA).

215 thdrawal of the sheath and catheter into the right atrium (RA).

216 LA), with fibrillatory conduction toward the right atrium (RA).

217 se, 14 had complex ATs involving the lateral right atrium (RA).

218 alence of GP in the posterior surface of the right atrium (RA).

219 maps were constructed of 11 tachycardias (6 right atrium [RA], 4 left atrium [LA] and 1 biatrial).

220 ere higher in SAN than in atria, with SAN to right atrium ratios of 6.1+/-0.9 and 4.6+/-0.6 (n=12), r

221 iables, pericardial effusion and an enlarged right atrium remained predictors of adverse outcomes.

222 The effective refractory period at the high right atrium remained unchanged with dual-site atrial pa

223 Tumour thrombus into the vena cava or right atrium requires thoracotomy and hypothermic circul

224 Enhancement of corresponding currents in the right atrium resulted in shortened action potential dura

225 Similarly, pacing in the right atrium resulted in significant changes in amplitud

226 Bs were delivered initially from the lateral right atrium, scanning diastole with a 10-ms decrement u

227 ig heart in tissue samples from left atrium, right atrium, septum, left ventricle and right ventricle

228 (4), right ventricle (3), right atrium (2), right atrium/superior vena cava (1), and right atrium/in

229 aced animals (P=0.05) and was greater in the right atrium than in the left atrium (P=0.004).

230 uting and hyperinnervation was higher in the right atrium than in the left atrium.

231 were paced from endocardial sites low in the right atrium, the P waves in ECG leads II, III, and aVF

232 From the right atrium, the whole LAA cavity could not be seen in

233 levels: the junction between vena cavae and right atrium; the tricuspid annulus; or between TV leafl

234 was seen in the posteromedial (sinus venosa) right atrium; this was manifested by the presence of dou

235 dults, BMP10 expression is restricted to the right atrium, though ventricular hypertrophy is accompan

236 s applied to the posteroseptal or mid-septal right atrium to lower the ventricular rate in atrial fib

237 RV dynamics or the adaptive response of the right atrium to maintain RV filling.

238 Blood was withdrawn from the right atrium to measure pH.

239 vely, with no significant difference between right atrium to pulmonary artery versus total cavopulmon

240 had a coronary sinus diverticulum and one a right atrium to right ventricle diverticulum.

241 -pulmonary artery connection in 135 (51.7%); right atrium to right ventricle in 25 (9.6%); and total

242 as a function of coupling interval, from the right atrium to the interatrial area and finally to the

243 enesis of the tricuspid valve connecting the right atrium to the right ventricle and both an atrial s

244 atrium (high and low trabeculated and smooth right atrium) to assess efficacy at inducing atrial flut

245 /-3.5 [SEM] kg) were subjected to 60 mins of right atrium-to-aortic, hypothermic (28 degrees C) CPB.

246 Fontan surgery at a median age of 7.9 years: right atrium-to-pulmonary artery connection in 135 (51.7

247 g a preformed autologous blood clot into the right atrium using a 7-French introducer sheath inserted

248 d and individual lesions were created in the right atrium using radiofrequency energy (30 W/48 degree

249 and membrane potential in 25 isolated canine right atrium, using previously described criteria of the

250 erior AVC, whereas right pSHF contributes to right atrium, ventral left atrium, and inferior AVC.

251 10 patients (1.8+/-1.1 per patient; 7 in the right atrium versus 12 in the left atrium; 15 extrapulmo

252 positioning the balloon catheter in the mid right atrium via a femoral venous approach.

253 on consecutive days, were delivered into the right atrium via a multiport pulmonary artery catheter d

254 rant circuits nor wave fronts activating the right atrium via BB.

255 The mean LGE burden for left atrium and right atrium was 23.9+/-1.6% and 15.9+/-1.8%, respective

256 GAP43-positive and TH-positive nerves in the right atrium was 470+/-406 and 231+/-126 per mm(2), resp

257 firmed that active BMP10 secreted from mouse right atrium was in the prodomain-bound form.

258 Catheter tip high in the right atrium was marginally associated with better cathe

259 of paced P-wave body surface mapping in the right atrium was obtained by estimating the area size of

260 A snare was placed around the aorta, and the right atrium was paced.

261 After weaning from CPB, the right atrium was recannulated with a two-stage, dual-lum

262 man plasma and BMP10 secreted from the mouse right atrium were fully active.

263 re, and burst pacing protocols from the high right atrium were performed at baseline, during isoprote

264 Electrograms from the right atrium were recorded.

265 ) present primarily in the atrial septum and right atrium were responsible for maintenance of AF.

266 pathways may be located in the posteroseptal right atrium, where slow pathway modification is perform

267 kwise flutter, 18 were from the trabeculated right atrium, whereas all the counterclockwise induction

268 and 29.2 +/- 6.1 pmol.mg-1 of protein in the right atrium, which were both significantly lower (P < 0

269 onary sinus via femoral sheaths and into the right atrium with fluoroscopic guidance.