ライフサイエンスコーパス: right heart

1 ansverse aortic arch, and compression of the right heart.

2 scular resistance, and increased load on the right heart.

3 sualizing interventional procedures from the right heart.

4 , a Notch ligand expressed in the developing right heart.

5 chronic cardiac damage, mainly involving the right heart.

6 Each subject, after catheterization of the right heart and a radial artery, was exposed in an envir

7 e conclude that the transfer function of the right heart and lungs is equivalent to a delay and seque

8 Right heart and transseptal catheterization were perform

9 rt, but its effects on the physiology of the right heart are incompletely understood.

10 use its development and consequences for the right heart are now seen as mainstay abnormalities that

11 oxidized lipids or fibrin deposition within right heart biopsy (RHB) specimens.

12 vival is possible, based on the principle of right heart bypass, whereby the ventricle pumps blood on

13 led study, 60 patients with fibrotic IIP and right heart catheter confirmed PH were randomized 2:1 to

14 ccounting for deaths and withdrawals, paired right heart catheter data were available for analysis in

15 ontrast transesophageal echocardiography and right heart catheter studies.

16 essure (PAP) was measured with an indwelling right heart catheter.

17 hould be referred to a specialist centre for right-heart catheterisation and pulmonary angiography.

18 d retrospectively all US veterans undergoing right heart catheterization (2007-2012) in the Veterans

19 In patients with right heart catheterization (30 HF-PH, 14 PVOD), similar

20 nary capillary wedge pressure </=15 mm Hg at right heart catheterization (allele frequency, 0.66; odd

21 57 patients with normal mPAP) who underwent right heart catheterization (RHC) and three-directional

22 and pulmonary hypertension (PH) diagnosed by right heart catheterization (RHC) are independent risk f

23 Right heart catheterization (RHC) is commonly performed

24 Right heart catheterization (RHC) is commonly used in th

25 Right heart catheterization (RHC) must be performed in p

26 ure (mPAP) value of at least 25 mm Hg during right heart catheterization (RHC).

27 nts known or suspected to have PAH underwent right heart catheterization and adenosine stress MR imag

28 consent, 35 PAH patients were evaluated with right heart catheterization and cardiac magnetic resonan

29 In vivo measurements were assessed using right heart catheterization and cardiac MRI.

30 ved from 60 COPD patients who underwent both right heart catheterization and computed tomography in a

31 spectively evaluated 53 PPH patients who had right heart catheterization and cycle ergometer CPET stu

32 onsecutive patients, undergoing simultaneous right heart catheterization and DE.

33 mptoms of congestive heart failure underwent right heart catheterization and Doppler-echocardiography

34 -17 years, 19 women) undergoing simultaneous right heart catheterization and echocardiographic imagin

35 In 50 patients with simultaneous right heart catheterization and echocardiographic imagin

36 II; Thoratec Inc; n=18) were evaluated with right heart catheterization and echocardiography preoper

37 Right heart catheterization and echocardiography were pe

38 upine cycle ergometer test with simultaneous right heart catheterization and echocardiography.

39 able patients who underwent first diagnostic right heart catheterization and from a prospective cohor

40 utinely underwent detailed reassessment with right heart catheterization and noninvasive testing at 3

41 All measurements, including data from right heart catheterization and norepinephrine requireme

42 nts undergoing clinically indicated left and right heart catheterization and same day research cardia

43 pulmonary edema, invasive techniques such as right heart catheterization and the sampling of alveolar

44 Hemodynamic variables obtained by right heart catheterization and transpulmonary thermodil

45 modynamic evaluation was performed by serial right heart catheterization and transthoracic echocardio

46 omboembolic pulmonary hypertension underwent right heart catheterization and-after digital subtractio

47 isk factors using systematic confirmation on right heart catheterization are lacking.

48 Echocardiography and right heart catheterization are the principal modalities

49 All patients underwent right heart catheterization at baseline and were followe

50 underwent simultaneous echocardiography and right heart catheterization at rest and during exercise.

51 0 years; n=14 HFpEF; n=12 control) underwent right heart catheterization at rest, during supine exerc

52 Twenty-six patients underwent preoperative right heart catheterization before PTE.

53 d vital status for all patients referred for right heart catheterization between 1998 and 2014.

54 ve patients with heart failure who underwent right heart catheterization between 2000 and 2005.

55 Laboratory, echocardiography, and right heart catheterization data collected from 205 pati

56 distress syndrome who had complete baseline right heart catheterization data from the Fluid and Cath

57 h heart failure, we retrospectively assessed right heart catheterization data in 162 consecutive pati

58 nship between screening echocardiography and right heart catheterization determinations of pressure,

59 HA) class III or IV symptomatology underwent right heart catheterization for determination of baselin

60 Patients undergoing right heart catheterization for evaluation of pulmonary

61 ographic techniques for the determination of right heart catheterization hemodynamic variables in pat

62 Patients underwent right heart catheterization immediately followed by tran

63 The significance of right heart catheterization in critically ill patients i

64 cic echocardiography and following up with a right heart catheterization in patients in whom the righ

65 rt the routine use of fluid challenge during right heart catheterization in patients with risk factor

66 phic parameters and hemodynamics obtained by right heart catheterization in PH with AF.

67 failure and may obviate the need for routine right heart catheterization in potential heart transplan

68 valence of exercise-induced PH determined by right heart catheterization in scleroderma spectrum diso

69 cteristics of echocardiography compared with right heart catheterization in the determination of syst

70 We studied 233 patients assigned to right heart catheterization in the Vasodilation in the M

71 66.4 [9.9] years; 3.3% female) who underwent right heart catheterization in this cohort study, Td and

72 Fluid challenge at the time of right heart catheterization is easily performed, safe, a

73 In conclusion, right heart catheterization is necessary to confirm POPH

74 Right heart catheterization is the gold standard for ass

75 Cardiac output measurement via right heart catheterization is used extensively for hemo

76 Pericardial tamponade was suggested by right heart catheterization measurements and diagnosed b

77 Using right heart catheterization measurements, mild PH was de

78 operative transthoracic echocardiography and right heart catheterization measurements.

79 ents (simultaneously measured mPAP) and with right heart catheterization measurements.

80 progress in echocardiography and biomarkers, right heart catheterization remains the only test that c

81 We suggest that right heart catheterization should be reserved for patie

82 ations of variables collected during resting right heart catheterization that best predicted abnormal

83 ients with advanced heart failure undergoing right heart catheterization to assess cardiac transplant

84 sthoracic echocardiography further underwent right heart catheterization to confirm the diagnosis of

85 Subjects underwent right heart catheterization to define LV pressure-volume

86 went invasive haemodynamic measurements with right heart catheterization to define Starling and left

87 Sixty healthy subjects underwent right heart catheterization to measure age- and sex-rela

88 Left and right heart catheterization using MR guidance is feasibl

89 ditional baseline characteristics, including right heart catheterization variables, were not consiste

90 The confirmed PAH prevalence on right heart catheterization was 2.1% (95% confidence int

91 in beta-thalassemia patients as confirmed on right heart catheterization was 2.1%, with an approximat

92 The PCWP obtained from right heart catheterization was compared with diastolic

93 Right heart catheterization was employed in one study to

94 Right heart catheterization was performed and serum uric

95 Right heart catheterization was performed immediately af

96 Left and right heart catheterization was performed in 7 swine wit

97 To investigate this hypothesis, right heart catheterization was performed in eight males

98 Right heart catheterization was performed using a pressu

99 h severe aortic stenosis and a preprocedural right heart catheterization were assessed.

100 s with PAH or ILD-associated PH confirmed by right heart catheterization were included in the study.

101 Simultaneous echocardiography and right heart catheterization were prospectively performed

102 ing transient RBBB pattern in lead V1 during right heart catheterization were studied.

103 clinic with transthoracic echocardiogram and right heart catheterization within 1 year.

104 ke on SPECT myocardial perfusion imaging and right heart catheterization within 4 wk were studied pro

105 e hypothesized that a fluid challenge during right heart catheterization would identify occult pulmon

106 ed to medical therapy with a sham procedure (right heart catheterization) versus medical therapy and

107 and left ventricular (LV) filling pressures (right heart catheterization) were measured under varying

108 ents who had coronary physiology assessment, right heart catheterization, and echocardiography perfor

109 not demonstrated any sustained benefits from right heart catheterization, and some studies have even

110 diac studies, including echocardiography and right heart catheterization, are key elements in the ass

111 sure product was also determined by means of right heart catheterization, as an index of the RV MVO2,

112 operation [n=6]) were evaluated monthly with right heart catheterization, CMR, and computed tomograph

113 utine invasive hemodynamic ramp testing with right heart catheterization, during which LVAD speeds we

114 ts were more than 15000 adults who underwent right heart catheterization, including 12232 in the Vete

115 ury during interventional procedures such as right heart catheterization, pacemaker implantation, inv

116 Diagnosis was confirmed by right heart catheterization, ventilation-perfusion lung

117 erization," "Swan-Ganz catheterization" and "right heart catheterization," and restricting the result

118 ce on the basis of PC-MRI in comparison with right heart catheterization-based measurements by a medi

119 onse to vasodilator testing require invasive right heart catheterization.

120 g hemodynamics underwent subsequent exercise right heart catheterization.

121 who were referred for a clinically indicated right heart catheterization.

122 thered data on IPAH patients who underwent a right heart catheterization.

123 -time MRI (MR fluoroscopy) to guide left and right heart catheterization.

124 PCWP was measured with right heart catheterization.

125 tients with pulmonary hypertension underwent right heart catheterization.

126 nits; or (3) inpatient status at the time of right heart catheterization.

127 ients with unexplained dyspnea who underwent right heart catheterization.

128 s bag technique in adult patients undergoing right heart catheterization.

129 sure >35 mm Hg on echocardiogram underwent a right heart catheterization.

130 terminal telopeptide of type I collagen) and right heart catheterization.

131 AH using Optical Coherence Tomography during Right Heart catheterization.

132 anently implanted in all participants during right heart catheterization.

133 iratory rise in right atrial pressure during right heart catheterization.

134 Twenty patients with PH underwent right heart catheterization: mean pulmonary artery press

135 study patients were divided into 2 groups by right heart catheterization: no PH (mean pulmonary arter

136 of cardiologists were more likely to undergo right-heart catheterization (adjusted odds ratio, 2.9 [C

137 weeks after therapy; patients also underwent right-heart catheterization and LSM at these time points

138 as demonstrated by pulmonary hypertension at right-heart catheterization at days 21 to 35 and major r

139 Peak VO2 outperforms clinical variables, right-heart catheterization data, exercise time, and oth

140 apillary wedge pressure waveform obtained by right-heart catheterization during 5 different loading c

141 dopamine-derived radioactivity who underwent right-heart catheterization had a decreased cardiac extr

142 diography were performed simultaneously with right-heart catheterization in 51 consecutive patients (

143 e anesthetized rat with a recently developed right-heart catheterization technique.

144 us Doppler echocardiographic examination and right-heart catheterization were performed in 44 patient

145 They also underwent right-heart catheterization, intra-aortic balloon pumpin

146 Using right-heart catheterization, we evaluated the acute pulm

147 Right-heart catheterization, with its associated disadva

148 ss hemodynamic variables were measured using right-heart catheterization.

149 ange; P <.05) in the 6 patients restudied by right-heart catheterization.

150 Pulmonary hypertension was determined by right-heart catheterization.

151 ertension, but definitive diagnosis requires right-heart catheterization.

152 ams (at 1, 2, 3, 4, 6, 9, and 12 months) and right heart catheterizations were performed after LVAD i

153 ly evaluated with serial echocardiograms and right heart catheterizations.

154 Specialized dual thermistor right heart catheters were constructed using a second th

155 e), which reflects LV preload independent of right heart congestion and pericardial restraint, was si

156 lume was associated with higher incidence of right heart dilatation.

157 achieve this goal and assessed children with right heart disease.

158 tensive patients with PE but with indicia of right heart dysfunction (by biomarkers or imaging) const

159 Two readers evaluated signs of right heart dysfunction at CT pulmonary angiography, mea

160 On the basis of the findings of right heart dysfunction on echocardiograms, computed tom

161 on between clot burden measures and signs of right heart dysfunction.

162 hat in this community-based study of SDB and right heart echocardiographic features, RV wall thicknes

163 valve replacement, moderate or severe TR and right heart enlargement are independently associated wit

164 that may predict magnitude of resolution in right heart enlargement.

165 Bisoprolol delayed time to right heart failure (P<0.05).

166 amine reduces the incidence of postoperative right heart failure (RHF) in pediatric heart transplant

167 o associated with increased unadjusted early right heart failure (RHF).

168 n, retroperitoneal vascular constriction and right heart failure - has shown that serotonin and tachy

169 the case of a young man who developed acute right heart failure after combined heart and kidney tran

170 sidered when evaluating patients with severe right heart failure after PPM or ICD implantation.

171 Underlying causes of right heart failure and baseline hemodynamics did not pr

172 relentlessly progressive disease leading to right heart failure and death.

173 nt, the disorder progresses in most cases to right heart failure and death.

174 a progressive disease that ultimately causes right heart failure and death.

175 reased work of the right ventricle may cause right heart failure and liver congestion.

176 evealed a combination of left heart failure, right heart failure and moderate-to-severe tricuspid reg

177 c abnormalities that reflect the severity of right heart failure and predict adverse outcomes in pati

178 lung inflammation, vascular remodeling, and right heart failure and reverses hypoxic pulmonary hyper

179 One patient died from right heart failure at 3 months.

180 26 critically ill adult patients with acute right heart failure defined by echocardiographic criteri

181 OF REVIEW: To review recent publications on right heart failure developing early and late after impl

182 early to remove fluid and reduce preload if right heart failure develops.

183 have provided good evidence about predicting right heart failure early after LVADs, though how to pre

184 Patients with right heart failure from cor pulmonale were classified a

185 Patients with pulmonary hypertension and right heart failure have a high risk of clinical deterio

186 k of progressive tricuspid regurgitation and right heart failure in patients with moderate or lesser

187 Right heart failure is a cause of morbidity and mortalit

188 Right heart failure is an important cause of morbidity a

189 ure early after LVADs, though how to predict right heart failure late after LVAD is still unclear as

190 cyclin can be life-saving when perioperative right heart failure occurs due to exacerbation of pulmon

191 determined to be the direct cause of death (right heart failure or sudden death) in 37 (44%) patient

192 ents with PAH and 75.7% of those who died of right heart failure received parenteral prostanoid thera

193 evices is associated with improved outcomes, right heart failure remains a considerable challenge.

194 Despite improved outcomes, right heart failure remains a significant challenge to s

195 ention that left heart failure has received, right heart failure remains understudied both at the pre

196 perience progressive symptoms of dyspnea and right heart failure resulting in significant morbidity a

197 Twelve patients died or developed right heart failure secondary to pulmonary hypertension

198 cured from explanted hearts of patients with right heart failure served as novel comparison samples.

199 1) How is right heart failure syndrome best defined?

200 s with dyspnea, exercise intolerance, and/or right heart failure who have elevated pulmonary artery s

201 ped for treating patients with severe TR and right heart failure with prohibitive surgical risk.

202 Lower extremity edema, venous stenosis, right heart failure, and deep venous thrombosis occurred

203 th pulmonary hypertension, hypoxemia, and/or right heart failure, and may offer a new therapeutic app

204 The main causes of death included sepsis, right heart failure, and multiorgan failure.

205 nt with bisoprolol delays progression toward right heart failure, and partially preserves RV systolic

206 or adverse events included bleeding, stroke, right heart failure, and percutaneous lead infection.

207 nts included postoperative bleeding, stroke, right heart failure, and percutaneous lead infection.

208 Echocardiogram showed acute right heart failure, and pulmonary perfusion scan demons

209 ve clearer evidence now for predicting early right heart failure, and treating it in those patients w

210 antly left heart failure in combination with right heart failure, and tricuspid regurgitation; and (i

211 This leads to reduced cardiac output, right heart failure, and ultimately death.

212 t estimates for bleeding, stroke, infection, right heart failure, arrhythmias, and rehospitalizations

213 e pulmonary hypertension precipitating acute right heart failure, despite administration of milrinone

214 anifest pulmonary veno-occlusive disease and right heart failure, detectable at 8 months of age.

215 pertension, along with frequently associated right heart failure, is extremely challenging.

216 th respect to medical therapies for treating right heart failure, there is evidence for the use of bo

217 ar and biventricular assist devices, such as right heart failure, valvular regurgitation, cardiac arr

218 riuretic peptide levels, and the presence of right heart failure.

219 terized by pulmonary vascular remodeling and right heart failure.

220 ients with severe pulmonary hypertension and right heart failure.

221 te treatment strategies for PH and resultant right heart failure.

222 ypoxic pulmonary hypertension and ultimately right heart failure.

223 pulmonary arterial tree, eventually leads to right heart failure.

224 nt of pulmonary hypertension, and associated right heart failure.

225 No deaths were associated with right heart failure.

226 in pulmonary vascular resistance leading to right heart failure.

227 -recognized but treatable etiology of severe right heart failure.

228 primary graft nonfunction or intraoperative right heart failure.

229 viduals present with dyspnoea or evidence of right heart failure.

230 TV) is increasing and results in intractable right heart failure.

231 years with death usually due to progressive right heart failure.

232 e progression of tricuspid regurgitation and right heart failure.

233 y vascular resistance, eventually leading to right-heart failure and death.

234 ications (one constrictive pericarditis, two right heart failures without underlying infection, and o

235 systolic HF, CXL-1020 reduced both left and right heart filling pressures and systemic vascular resi

236 Syncope in PAH is associated with worsening right heart function and is an independent predictor of

237 hysiology may be useful during assessment of right heart function and pulmonary pressures before tran

238 f venous return and its interaction with the right heart function as it relates to mechanical ventila

239 autoimmune disease that can affect left and right heart function directly through inflammation and f

240 tative two-dimensional methods for assessing right heart function that are both well established and

241 In the postoperative period, changes in right heart function will depend on preexisting pulmonar

242 ine nonconsecutive patients with compromised right heart function, pulmonary hypertension, and severe

243 n leads to clinically significant changes in right heart function.

244 is inflammation is associated with decreased right heart function.

245 regional wall motion (RWM), and analysis of right heart function.

246 function, pulmonary vascular remodeling, and right heart function.

247 -term outcome; namely, exercise capacity and right heart function.

248 ular resistance, which results in diminished right-heart function due to increased right ventricular

249 icting results, and the impact of SDB on the right heart has not been investigated in the general pop

250 ge demonstrating that diseases affecting the right heart have been shown to have the same clinical co

251 Clinical bedside evaluations and right heart hemodynamic assessments can alert clinicians

252 ative diastolic mitral inflow velocities and right heart hemodynamic data in 39 consecutive patients

253 The ECGs, echocardiograms, and right heart hemodynamic data were reviewed to determine

254 Improvements in right heart hemodynamics and exercise capacity were conf

255 Accurate noninvasive evaluation of right heart hemodynamics is an essential component of th

256 ciation class III HF underwent assessment of right heart hemodynamics, gas exchange, and first-pass r

257 n together for an accurate interpretation of right heart hemodynamics.

258 naling, and reversed vascular remodeling and right-heart hypertrophy in vivo.

259 ul navigation of the aorta, left atrium, and right heart, including detailed understanding of relatio

260 rt the need for future studies on TR and the right heart, including whether concomitant treatment of

261 h global function and catheterization of the right heart indexes.

262 how these shock states perturb venous return/right heart interactions.

263 e venous system and its interaction with the right heart may be more useful.

264 Right heart measurements, made without knowledge of clin

265 Among right heart metrics, RVESRI demonstrated the best test-r

266 al area were strongly connected to the other right heart metrics.

267 egistrants supported with dual inotropes and right heart monitoring had a higher risk of adverse even

268 n=5), tetralogy of Fallot (n=1), hypoplastic right heart (n=1), and common arterial trunk (n=1).

269 ntribute significantly to common variants of right heart obstructive disease.

270 the authors discuss the emerging concepts of right heart pathobiology in PAH.

271 ority, 86 of 109 (79%), had CHD resulting in right heart pressure or volume overload.

272 lure has little effect on cardiac output and right heart pressures because of compensatory conduit fu

273 As a result, cardiac output decreased and right heart pressures increased only after superimposed

274 pulmonary venous flow), cardiac output, and right heart pressures were measured at matched paced hea

275 Several large clinical databases with right heart/pulmonary catheterization data were analyzed

276 2.7 L/min/m2; RV % area change, 24 vs. 33%), right heart remodeling (right atrial area index, 17.0 vs

277 TV deformations and their association with right heart remodeling differ between AF-TR and left-sid

278 a in smooth muscle on pulmonary vascular and right heart responses to chronic hypoxia.

279 id valves (n=16) were studied in an in vitro right heart simulator.

280 Little is known about reduction in right heart size after closure of ASD.

281 cardiogram findings of sinus tachycardia and right heart strain are common in Pneumocystis carinii pn

282 Significant right heart structural reverse remodeling takes place im

283 ffect of sleep-disordered breathing (SDB) on right heart structure and function is controversial.

284 rtension is associated with abnormalities of right heart structure and function that contribute to th

285 Growth of the PV annulus and right heart structures in patients with critical PS afte

286 asty in infants with critical and severe PS, right heart structures increase in size at a rate that p

287 Visualization of right heart structures was rated significantly (P < .05)

288 No significant changes were found in the right heart structures.

289 hemodynamically unstable patients with acute right heart syndrome has not been previously described.

290 in weight, filling pressures of the left and right heart, systemic vascular resistance, and echocardi

291 n, a patent foramen ovale, and free-floating right-heart thrombus are echocardiographic markers that

292 ses on the function of the venous system and right heart under normal and stressed conditions.

293 In combination with echocardiographic right heart variables, also available from routine echoc

294 protected from the effects of hypoxia on the right heart, vascular remodeling, and raised serum endot

295 specified cardiac targets were imaged from a right heart venue.

296 Mean contrast medium attenuation in the right heart was significantly (P < .001) higher in the s

297 the basal septal myocardium to re-enter the right heart where it is exchanged for a suture.

298 ficient attenuation for visualization of the right heart, while streak artifacts from high-attenuatio

299 esigned to optimize the visualization of the right heart with echocardiography with case examples wil

300 he interaction of the pathologically altered right heart with the anatomically-supposedly-normal left