ライフサイエンスコーパス: right hemisphere

1 tained responses over the left than over the right hemisphere.

2 get-specific connections we obtained for the right hemisphere.

3 and dorsolateral prefrontal cortices of the right hemisphere.

4 al and subcortical limbic targets within the right hemisphere.

5 spectrum disorders group, especially in the right hemisphere.

6 gh normal or enhanced activity in the intact right hemisphere.

7 areas, and their anatomic homologues in the right hemisphere.

8 , and precuneus along the mesial wall of the right hemisphere.

9 on was significantly weaker in the undamaged right hemisphere.

10 belt ACFs, indicating neuroplasticity in the right hemisphere.

11 rontal lobes, mesolimbic connections and the right hemisphere.

12 hippocampal region were only observed in the right hemisphere.

13 is activation appeared to be stronger in the right hemisphere.

14 reased negativity of N170 amplitude over the right hemisphere.

15 responses in a voice-sensitive region in the right hemisphere.

16 ferentially modulate voice processing in the right hemisphere.

17 asing signals than their counterparts in the right hemisphere.

18 I fiber pathways are better preserved in the right hemisphere.

19 unced age-related loss of gray matter in the right hemisphere.

20 arietal cortex and early visual areas of the right hemisphere.

21 ft hemisphere and a relative increase in the right hemisphere.

22 ination between stimuli observed only in the right hemisphere.

23 temporal cortex (pSTC), particularly in the right hemisphere.

24 ced movement representations in the dominant right hemisphere.

25 pairs plus a lateral temporal region in the right hemisphere.

26 or cingulate, and orbitofrontal areas in the right hemisphere.

27 e infusion pipette into the SI cortex of the right hemisphere.

28 atter three regions, BP was decreased in the right hemisphere.

29 in control is not restricted to the left or right hemisphere.

30 gion and the inferior parietal lobule in the right hemisphere.

31 ence, driven by a reduction in volume in the right hemisphere.

32 cortical thinning in prefrontal areas of the right hemisphere.

33 tes along dorsal and ventral pathways in the right hemisphere.

34 MOA) of SLF II, SLF III, and IFOF within the right hemisphere.

35 tors of recovery are at play in the left and right hemisphere.

36 hemisphere but to attention networks in the right hemisphere.

37 oxelwise comparisons of activity in left and right hemispheres.

38 5) demonstrated asymmetric left greater than right hemisphere (18)F-AV1451 uptake in three of five pa

39 ups had language function lateralized to the right hemisphere (54.5%) or dispersed bilaterally (27.3%

40 ctivation in both regions as well as reduced right hemisphere activation in the posterior temporal re

41 Right hemisphere activation in these two groups occurred

42 amage or healthy ageing results in increased right-hemisphere activation in homologous regions to tho

43 AgCC participants showing significantly more right hemisphere activations than controls or than indiv

44 sed, indicating that homologous areas in the right hemisphere actively contribute to language functio

45 ctivity only for contralateral memory items; right hemisphere activity reflected VSTM load regardless

46 es of mediated priming have failed to find a right hemisphere advantage for processing distantly link

47 hout the occipital and temporal lobes with a right hemisphere advantage.

48 s that semantic activation is broader in the right hemisphere, affording it an advantage over the lef

49 For the right hemisphere, age [beta = -0.678, t(-3.087), P = 0.0

50 decreased fractional anisotropy (FA) in the right hemisphere and a subnetwork with increased mean di

51 sented on the left producing activity in the right hemisphere and accurate memory for items previousl

52 Patients who had damage to the left or right hemisphere and age-matched control participants re

53 ses in the left hemisphere, in 1 case in the right hemisphere and in 1 case bilateral.

54 nsight-related coarse semantic coding in the right hemisphere and internally focused attention preced

55 hen lexical information is introduced to the right hemisphere and must subsequently be transferred to

56 heory emphasizing a dominant function of the right hemisphere and others supporting an interhemispher

57 he lingual and inferior temporal gyri of the right hemisphere and regression of participation of the

58 implanted with F98 glioblastoma cells in the right hemisphere and scanned 9-15 d later.

59 on a combination of visual expertise in the right hemisphere and semantics in the left hemisphere.

60 group occurred by T = 1 h in the ICH injured right hemisphere and T = 2 h in the contralateral hemisp

61 -greater-than-parietal P3b topography in the right hemisphere and the highest P3a amplitude at fronta

62 initially projected to and processed in the right hemisphere and the left hemisphere, respectively.

63 g of the cortex of the lateral aspect of the right hemisphere and the medial aspect of the left, as w

64 the left visual field is represented in the right hemisphere and vice versa.

65 and frontal operculum/insular cortex of the right hemisphere and, to a lesser extent, in the anterio

66 occipital and parietal areas of the left and right hemispheres and were short lived: they were observ

67 l MR hemisphere activation and both definite right-hemisphere and bilateral language dominance.

68 owed prolonged response profiles (all in the right hemisphere), and fast profiles (all but one in the

69 ns immediately surrounding the lesion in the right hemisphere, and also, surprisingly, in correspondi

70 al sensory motor cortex, particularly in the right hemisphere, and surrounding association areas (Bro

71 ital regions, the mesial frontal lobe of the right hemisphere, and the cuneus and precuneus in the le

72 dren, the left hemisphere is larger than the right hemisphere, and the normal pattern of fronto-occip

73 greater extent when applied to left than to right hemisphere, and was more disruptive when applied c

74 at 40 962 homologous points in the left and right hemispheres, and the trajectory of change in asymm

75 to-parietal and default mode networks in the right hemisphere; and (iii) increased intrahemispheric c

76 her order language functions mediated by the right hemisphere are essential to an accurate understand

77 have previously suggested that the left and right hemispheres are specialized for controlling differ

78 motor lateralization, in which the left and right hemispheres are specialized for different aspects

79 tion that increased activation of homologous right hemisphere areas supports aphasia recovery after l

80 ontal regions and the anterior insula of the right hemisphere as well as an expansive negative networ

81 ain damage as measured by weight loss of the right hemisphere at 22 days after HI and by gross and mi

82 in both temporal lobes, and rely more on the right hemisphere auditory regions, particularly right su

83 in nonspeech acoustic signals lateralize to right-hemisphere auditory areas, whereas rapid temporal

84 nspeech stimuli, and it is not known whether right-hemisphere auditory cortex is dominant for coding

85 Right-hemisphere auditory cortex was 100% more accurate

86 f a lexically ambiguous word have reported a right hemisphere benefit.

87 (left hemisphere) and orbitofrontal cortex (right hemisphere); bilateral precuneus, posterior cingul

88 ntralateral and ipsilateral responses in the right hemisphere but maintained or enhanced contralatera

89 with brain injuries or stroke in the left or right hemisphere, but not in the postcentral gyrus as th

90 pplied left hemisphere anodal-excitatory and right hemisphere cathodal-inhibitory tDCS, compared to s

91 (chi2 = 11.90, P = 0.003), especially in the right hemisphere (chi2 = 13.67, P = 0.001).

92 Heschl's gyrus, insula, and striatum in the right hemisphere, clearly different from the lesion patt

93 y more than control subjects, specifically a right hemisphere cluster encompassing the putamen, insul

94 We observed higher metabolism in the right hemisphere compared to the left and a positive cor

95 e demonstrating consistently elevated within-right hemisphere connectivity.

96 as and prefrontal attention areas; increased right-hemisphere connectivity; reduced connectivity in t

97 A bias was also found in favor of the right hemisphere consistent with functional attentional

98 isual information across saccades underlying right-hemisphere constructional apraxia.

99 EEG activity over central electrodes in the right hemisphere, contralateral to the PA-induced, compe

100 control), acceleration time (associated with right hemisphere control), and speed equivalent to contr

101 during sham stimulation and stimulation to a right-hemisphere control brain region.

102 In contrast, right-hemisphere cortical regions involved in visuospati

103 resolving the activity imbalance with their right hemisphere counterparts, thus leading to persisten

104 iparetic stroke patients with left (LHD) and right hemisphere damage (RHD).

105 e to movement control, showing that left and right hemisphere damage produce different effects on mov

106 fying features of movement trajectory, while right hemisphere damage produces deficits in achieving a

107 We now propose that left and right hemisphere damage should also produce different de

108 individuals with left hemisphere damage and right hemisphere damage whereas the Multistep Object Use

109 lesional arm of stroke patients with left or right hemisphere damage, provided a critical test of our

110 rrors in movement extent were greatest after right hemisphere damage.

111 our predictions, patients with left, but not right, hemisphere damage showed reduced modulation of ac

112 l subjects and patients with either left- or right-hemisphere damage performed targeted single-joint

113 cation of initial trajectory features, while right-hemisphere damage would produce deficits in final

114 Following right-hemisphere damage, a specific disorder of motor aw

115 d to be differentially affected by left- and right-hemisphere damage.

116 d an important dissociation between left and right hemisphere damaged patients.

117 measured attention and motor deficits in 44 right hemisphere-damaged patients with a first-time stro

118 exhibited the expected match effect, whereas right-hemisphere-damaged participants showed no effect o

119 f Kurzban et al. in light of our findings on right-hemisphere-damaged patients, who show increasing a

120 This dissociation was more evident in the right hemisphere, demonstrating functional lateralizatio

121 Despite the complete loss of her right hemisphere (di- and telencephalon) at birth, the p

122 However, a right-hemisphere difference was found in the N1 amplitud

123 tion in the discordant context, and, for the right hemisphere, discordant context information actuall

124 possibility of light-based interventions for right hemisphere disorders of spatial attention.

125 gion of the anterior cingulate cortex in the right hemisphere displayed greater thickness in SuperAge

126 Therefore, right hemisphere dominance during stimulus-driven shifts

127 Right hemisphere dominance for visuospatial attention is

128 attended location produced a more widespread right hemisphere dominance in frontal, parietal, and tem

129 The anatomy and right hemisphere dominance of neglect follow from the an

130 volved in tutor song memory, while there was right hemisphere dominance of neuronal activation in HVC

131 (i.e., greater likelihood of bilaterality or right hemisphere dominance) in this cohort compared with

132 Here we show strong right-hemisphere dominance for coding the speech envelop

133 found that good readers indicated consistent right-hemisphere dominance in auditory cortex for all me

134 The right-hemisphere dominance model introduces a functional

135 frontoparietal network that conforms to the right-hemisphere dominance model.

136 o competing models: the orientation bias and right-hemisphere dominance models.

137 esponse involves the coordinated action of a right hemisphere dominant ventral frontoparietal network

138 lective attention is widely considered to be right hemisphere dominant.

139 atial attention in both visual fields evoked right-hemisphere dominant activity in temporoparietal ju

140 nd occipitotemporal regions (for faces), and right hemisphere dorsolateral prefrontal regions during

141 d that reduced normalized streamlines in the right-hemisphere dorsolateral prefrontal cortex-sensorim

142 stimulation, brain activity increased in the right hemisphere during negative emotion and was localiz

143 In contrast, the NN uniquely engage the right hemisphere during the M400.

144 effective connectivity between the left and right hemispheres during repetition of auditory and visu

145 By contrast, left-side onset patients (LPD; right hemisphere dysfunction) would show impaired global

146 sus right side of the body (LPD, predominant right-hemisphere dysfunction; RPD, predominant left-hemi

147 The load dependence of right hemisphere effects argues that memory-dependent an

148 the current prevailing theory suggests that right hemisphere engagement is ineffective or even malad

149 robust, emphasizing the contribution of the right hemisphere, especially the frontal lobe, to readin

150 te an electrophysiological response over the right hemisphere exactly at 1.2 Hz (6 Hz/5).

151 The dominant right hemisphere exhibited early visual discrimination b

152 ine whether local grey matter volumes in the right hemisphere explained additional variance in langua

153 recuneus was maintained across the lifespan (right hemisphere: F = 7.69, P < 0.001; left hemisphere:

154 Only patients with frontal damage in the right hemisphere failed to correct for this discrepancy

155 ed system can successfully reorganize to the right-hemisphere following left-hemisphere brain damage.

156 en reading span scores and activation in the right hemisphere for both types of ambiguous words sugge

157 ately stronger in the left compared with the right hemisphere for place but not for voicing or manner

158 ing and accounting for limb dynamics and the right hemisphere for stabilizing limb position through i

159 emisphere for linguistic probes and over the right-hemisphere for non-linguistic probes.

160 ctivation that was not direction specific in right hemisphere frontal regions (FEF, SFG, MFG).

161 l cortex and/or the balance between left and right hemisphere functions.

162 Increased activation of the right hemisphere has been observed after left hemisphere

163 ntrolateral prefrontal cortex (VLPFC) in the right hemisphere, has been implicated to serve as a gene

164 area in the left, and Wernicke's area in the right hemisphere) have been reported without evident rep

165 ubjects, each ROI in each hemisphere (except right-hemisphere hMT) showed significant selectivity for

166 A right-hemisphere homolog (rVWFA) shows similarly positio

167 lated separately for Broca's area versus its right-hemisphere homolog and Wernicke's area versus its

168 phere homolog and Wernicke's area versus its right-hemisphere homolog.

169 ity of this left language region extended to right-hemisphere homologs was positively associated with

170 of correlated activity between the left and right hemispheres, however little is known about regiona

171 showed activation of the Broca's area in the right hemisphere in 3/4 cases of low grade gliomas (LGG)

172 We entrained the left versus right hemisphere in accordance to two different coupling

173 The role of the right hemisphere in aphasia recovery after left hemisphe

174 LD activation in the insula only; and in the right hemisphere in both the insular and temporal cortic

175 gyrus to the superior parietal lobule in the right hemisphere in healthy controls, at-risk mental sta

176 s between hemispheres rather than a dominant right hemisphere in the intact human brain.

177 are consistent with research implicating the right hemisphere in the representation of contextually r

178 The anterior insular cortex of the right hemisphere, in particular its dorsal subregion, wa

179 cal diseases or injuries that can affect the right hemisphere, including stroke, traumatic brain inju

180 mine whether local grey matter volume in the right hemisphere independently contributes to aphasia ou

181 had subcortical infarcts only and seven had right-hemisphere infarcts.

182 eurological syndrome following predominantly right hemisphere injuries and is characterized by both s

183 lower spans are more likely to involve show right hemisphere involvement in the processing of the am

184 nduces a switch in spatial representation in right hemisphere IPS from contralateral to full-field co

185 pe information at different time scales: the right hemisphere is thought to be specialized in process

186 categorize stimuli while the left-eye (i.e. right-hemisphere) is used to inspect novel items and ini

187 We studied 27 patients with acute right hemisphere ischaemic stroke and 24 neurologically

188 ate in an emotional empathy task after acute right hemisphere ischemic stroke.

189 cal measurements of CC size with left versus right hemisphere language activation in 74 normal subjec

190 nvestigate the contribution of both left and right hemisphere language areas in recovery from aphasia

191 Although older evidence suggested that right hemisphere language homologues compensate for dama

192 The right hemisphere language network seems to be important

193 , but fMRI may be more sensitive than IAT to right hemisphere language processing.

194 tional MR hemisphere activation and definite right-hemisphere language dominance.

195 emporo-parietal junction was correlated with right hemisphere lateralization of language.

196 ure of spatial neglect and may relate to its right hemisphere lateralization.

197 ground, whereas the 4-8 Hz coupling remained right-hemisphere lateralized in both conditions.

198 ound that disruption of either region in the right hemisphere led to greater selection of both gains

199 Fourteen stroke patients with right hemisphere lesions and contralesional paralysis we

200 techniques, we explored the effect of acute right hemisphere lesions in 18 patients on perceived ang

201 e errors with secondary saccades, those with right hemisphere lesions often failed to do so.

202 e spatial neglect most commonly occurs after right hemisphere lesions, damage to diverse areas within

203 normal observers and two male patients with right-hemisphere lesions and previous histories of spati

204 ction, which is more robust in patients with right-hemisphere lesions.

205 targets were presented on left visual field-right hemisphere (LVF-RH) trials.

206 Alternatively, the right hemisphere may actively contribute to language fun

207 lesions, damage to diverse areas within the right hemisphere may lead to neglect, possibly through d

208 l neural processing of pitch in the left and right hemispheres may enable the audio-vocal system to d

209 Wistar rats were subjected to right hemisphere middle-cerebral artery occlusion and re

210 However, within the HC group, the larger the right hemisphere MiOG volume, the better the performance

211 of patients with either lOFC (predominantly right hemisphere), mOFC/vmPFC, or dorsomedial prefrontal

212 ate long-latency causal interactions between right-hemisphere motor areas and the left M1 (lM1).

213 ial diffusivity profile of white matter in a right hemisphere network of white matter regions in keta

214 Right-hemisphere networks are important for both spatial

215 ral lobe perisylvian cortices, predominantly right-hemisphere occipital and occipitotemporal regions

216 ural differences were observed solely in the right hemisphere of patients with freezing of gait.

217 The right hemisphere of the brain was seen during injection

218 In the right hemisphere of the brain, the pericalcarine gyrus w

219 e given, structural adaptation in the intact right hemisphere of the brain.

220 Although the left and right hemispheres of our brains develop with a high degr

221 as injected intracerebroventricularly in the right hemisphere on postnatal day 6 at 30min prior to th

222 ) and grey matter density (in the unaffected right hemisphere) on language laterality.

223 thod to align FreeSurfer-registered left and right hemispheres onto a common template in order to cha

224 The greatest differences were in the right hemisphere (P = 0.006).

225 visual stimuli, via an asymmetric effect on right-hemisphere parieto-occipital alpha-power.

226 Previously, assessments of right-hemisphere patients with hemispatial neglect have

227 Here we examined the ability of right-hemisphere patients with neglect to maintain atten

228 Results provide evidence that the right hemisphere plays a specific and important role in

229 gs suggest that the grey matter structure of right hemisphere posterior dorsal stream language homolo

230 ptica patients and human complement into the right hemisphere preferentially turned to the right at 7

231 ndings suggest that cortical thinning in the right hemisphere produces disturbances in arousal, atten

232 th ASD also showed underconnectivity between right-hemisphere pSTS, a region known for processing spe

233 Conversely, connection strengths between right hemisphere regions became weaker after training.

234 plore the joint involvement of occipital and right hemisphere regions in a visual-based phonological

235 showed that joint speech recruits additional right hemisphere regions outside the classic speech prod

236 with cortical thickness in two left and four right hemisphere regions, as follows: bilateral temporal

237 slexics showed less GMV in multiple left and right hemisphere regions, including left superior tempor

238 ructural adaptation in similar (overlapping) right hemisphere regions.

239 nstructional apraxia patients' damage to the right-hemisphere regions involved in remapping locations

240 is highly heritable and, specifically in the right hemisphere, regulated oligogenically with linkages

241 ions of the human brain, particularly in the right hemisphere, remains poorly understood.

242 phere represents the right side, whereas the right hemisphere represents both sides of the sensorium.

243 position may be lateralized to the left and right hemispheres, respectively.

244 +/- 6.1 and 48.7 +/- 8.5 Hz for the left and right hemispheres, respectively.

245 ound that indicated size/distance, LO in the right hemisphere responded significantly more to the sma

246 ed behaviours involved partially overlapping right hemisphere reward circuit regions including putame

247 It has been suggested that the right hemisphere (RH) has a privileged role in the proce

248 egmental) content (e.g., [1-3]), whereas the right hemisphere (RH) is more sensitive to prosodic (sup

249 rges under stimulus-guided attention: in the right hemisphere (RH), visual maps IPS0, IPS1, and IPS2

250 whereas in infants, it is lateralized to the right hemisphere (RH).

251 rongly related to structural deficits in the right hemisphere's locomotor network involving prefronta

252 subjects also received inhibitory rTMS over right hemisphere S1 and the vertex (control).

253 at different time scales, with the left and right hemispheres sampling at short (25 ms; 40 Hz) and l

254 3 amplitude at left hemisphere, midline, and right hemisphere scalp locations was affected by the sta

255 In contrast, hV4 of the right hemisphere showed expanded response properties.

256 This may be important as right-hemisphere spatial abilities may underlie our abil

257 is to achieve optimal separation of left and right hemisphere ssSEPs.

258 Reading impairments after left or right hemisphere stroke are common yet receive little at

259 esions were significantly more impaired than right hemisphere stroke patients without uncinate fascic

260 We tested 52 patients with neglect after right hemisphere stroke, and conducted an anatomical ana

261 0 individuals with left neglect secondary to right hemisphere stroke.

262 l empathy in 30 patients with acute ischemic right hemisphere stroke.

263 gical evidence that attention deficits after right-hemisphere stroke arise in part from hyper-excitat

264 Hemispatial neglect following right-hemisphere stroke is a common and disabling disord

265 sion behavior mapping analysis of a group of right-hemisphere stroke patients supported this hypothes

266 Sixteen right-hemisphere stroke patients were recruited, all of

267 using both visuospatial and verbal tasks in right-hemisphere stroke patients with anosognosia (n = 1

268 Right-hemisphere stroke patients with constructional apr

269 y disorder and hemispatial neglect following right-hemisphere stroke.

270 PTN expression were on average higher in the right hemisphere, suggesting that asymmetric NPTN expres

271 In addition, the data suggest that the right hemisphere superior temporal gyrus is particularly

272 These effects appeared in the right hemisphere, supporting lateralization and top-down

273 bservations: a first model proposes that the right hemisphere sustains production and comprehension o

274 of the contralesional hemisphere (i.e., the "right-hemisphere-take-over" theory).

275 One aphasic case displayed higher right-hemisphere tangle density despite greater left-hem

276 ecline in cortical thickness with age in the right hemisphere than in the left on the lateral surface

277 of nonadjacent patterns was stronger in the right hemisphere than in the left, and may reflect an ef

278 male and female birds, were stronger in the right hemisphere than in the left, and that right-side r

279 more precise and mature more rapidly in the right hemisphere than in the left.

280 ree-way functional-anatomical network in the right hemisphere that could either brake or completely s

281 posterior cortical network, particularly in right hemisphere, that prepares the saccade system for r

282 In the right hemisphere the pattern of sex differences was diff

283 ispheres, contrary to the predictions of the right hemisphere theory of metaphor.

284 These results support the "brain's right-hemisphere" theory, which predicts that the right-

285 he differential contribution of the left and right hemisphere to executing a grasping movement with t

286 rs generally performed worse than those with right hemisphere tumors.

287 significantly different between the left and right hemisphere (Type I: right > left, Type II, III: le

288 f 45 patients with unilateral strokes in the right hemisphere underwent cognitive testing for neglect

289 ional relationship between the LC-NE system, right-hemisphere ventral attention network, and P300 EEG

290 (face) influences on neuronal responses in a right-hemisphere voice-sensitive region in the anterior

291 The length of the planum temporale in the right hemisphere was the main predictor of language late

292 Activity in the right-hemisphere was not correlated with damage in the l

293 vs cathodal) and electrode location (left vs right hemisphere) was tested in a series of separate sin

294 We obtained activation only in the right hemisphere Wernicke's area in 4/5 of the cases.

295 s within the left (P < 0.05), but not in the right, hemisphere when compared to controls.

296 ital gyrus was unilaterally activated in the right hemisphere while the cuneus was bilaterally activa

297 onal connectivity between IPS and FEF in the right hemisphere with early visual areas was stronger fo

298 waves are generally larger and lead from the right hemisphere with only moderate covariation of ampli

299 us pallidus and orbitofrontal regions of the right hemisphere (with the left hemisphere not analyzed

300 ences in tensor metrics between the left and right hemispheres within or between the two groups.