ライフサイエンスコーパス: right ventricular outflow tract

1 omografts has improved reconstruction of the right ventricular outflow tract.

2 of a significant delay in the anterolateral right ventricular outflow tract.

3 grams (2.21+/-0.67 versus 0 mV); (2) delayed right ventricular outflow tract activation (82+/-18 vers

4 phic ventricular ectopy originating from the right ventricular outflow tract and cardiomyopathy and t

5 ant, this is to be expected with the complex right ventricular outflow tract and pulmonary artery ana

6 e border region of scar most commonly in the right ventricular outflow tract and right ventricle basa

7 ventricular tachycardia originating from the right ventricular outflow tract, and (c) ventricular tac

8 e related to the tricuspid annulus, proximal right ventricular outflow tract, and anterior/inferior-a

9 previous endocarditis, in situ stents in the right ventricular outflow tract, and presence of outflow

10 evidence of CA compression with simultaneous right ventricular outflow tract angioplasty and CA angio

11 have had a definitive primary repair; their right ventricular outflow tracts are characterized by mi

12 ocardiographic criteria can help distinguish right ventricular outflow tract arrhythmias originating

13 ded by the presence of lower-risk idiopathic right ventricular outflow tract arrhythmias with left bu

14 erval/Deltax=105+/-24 versus 7+/-5 ms/cm) at right ventricular outflow tract borders.

15 are used primarily for reconstruction of the right ventricular outflow tract, both in children with c

16 There was no difference in E12 in the right ventricular outflow tract compared with the right-

17 Implantation of the Melody valve for right ventricular outflow tract conduit dysfunction can

18 In patients with right ventricular outflow tract conduit dysfunction, TPV

19 lacement has emerged as a viable therapy for right ventricular outflow tract conduit dysfunction.

20 re pulmonary valve function in patients with right ventricular outflow tract conduit dysfunction; the

21 d adult patients (median age, 19 years) with right ventricular outflow tract conduit obstruction or r

22 was approved for implantation in obstructed right ventricular outflow tract conduits in 2010 after a

23 tion approval for treatment of dysfunctional right ventricular outflow tract conduits in patients >/=

24 United States for placement in dysfunctional right ventricular outflow tract conduits.

25 s, Minnesota) in patients with dysfunctional right ventricular outflow tract conduits.

26 Surgical right ventricular outflow tract cryoablation was perform

27 Peak Doppler gradient across the right ventricular outflow tract decreased from 41.9 +/-

28 ional area change (HR, 0.94 per 1%; P=0.02), right ventricular outflow tract diameter (HR, 1.08 per 1

29 t represents a transformative technology for right ventricular outflow tract dysfunction with the pot

30 t therapy for the treatment of postoperative right ventricular outflow tract dysfunction.

31 re than mild regurgitation, and 4 had a mean right ventricular outflow tract gradient >/=30 mm Hg.

32 in their RV:Ao pressure ratio (P<0.001) and right ventricular outflow tract gradient (P=0.004) than

33 to good effect with significant reduction in right ventricular outflow tract gradient and the RV:Ao r

34 A higher right ventricular outflow tract gradient at discharge (P

35 The median peak right ventricular outflow tract gradient was 37 mm Hg be

36 A conduit prestent and lower discharge right ventricular outflow tract gradient were associated

37 ly reduced RV:Ao pressure ratio (P=0.02) and right ventricular outflow tract gradients (P</=0.001).

38 VT originated from the right ventricular outflow tract in 10 patients, the righ

39 ars restricted to the anterior subepicardial right ventricular outflow tract in 11 patients (group B)

40 ts (82%) after myocardial infarction, in the right ventricular outflow tract in 13 of 15 patients (87

41 similar among the 3 groups, occurring in the right ventricular outflow tract in 82%.

42 s 56 patients had either a shunt or a patent right ventricular outflow tract intentionally left in pl

43 entricular tachycardia) originating from the right ventricular outflow tract is an underappreciated c

44 d-transposition of the great arteries (12%), right ventricular outflow tract lesions (10%), l-transpo

45 studies were performed to image the left and right ventricular outflow tract (LVOT and RVOT) forward

46 abnormal electric activity in the epicardial right ventricular outflow tract may be beneficial in pat

47 ral crest cells is of critical importance in right ventricular outflow tract morphogenesis.

48 Indications for reoperation included right ventricular outflow tract obstruction (19), branch

49 l exercise capacity, there was mild residual right ventricular outflow tract obstruction (mean gradie

50 derwent 11 reoperations, including relief of right ventricular outflow tract obstruction (n=5), pulmo

51 ssociation between the use of paroxetine and right ventricular outflow tract obstruction (relative ri

52 Right ventricular outflow tract obstruction (RVOTO) is a

53 sible association between paroxetine use and right ventricular outflow tract obstruction and between

54 ular stent implantation for the treatment of right ventricular outflow tract obstruction are often le

55 increase in the risk of Ebstein's anomaly (a right ventricular outflow tract obstruction defect) in i

56 jects) and between the use of paroxetine and right ventricular outflow tract obstruction defects (odd

57 The prevalence of right ventricular outflow tract obstruction defects was

58 ging therapy for pulmonary regurgitation and right ventricular outflow tract obstruction in selected

59 Treatment of right ventricular outflow tract obstruction is possible

60 en (median age, 12.9 years) with significant right ventricular outflow tract obstruction underwent BM

61 Right ventricular outflow tract obstruction was managed

62 to early complete repair include removal of right ventricular outflow tract obstruction, alleviation

63 In some cases of left or right ventricular outflow tract obstruction, early inter

64 All reinterventions in this series were for right ventricular outflow tract obstruction, highlightin

65 st recent echocardiograms were evaluated for right ventricular outflow tract obstruction.

66 ic effects of PPVI over BMS in patients with right ventricular outflow tract obstruction.

67 persion of repolarization are present in the right ventricular outflow tract of BrS patients.

68 n in vitro and a chronic animal model of the right ventricular outflow tract of postoperative tetralo

69 ical study did not demonstrate injury to the right ventricular outflow tract or pulmonic valve.

70 gical pulmonary valve replacement in dilated right ventricular outflow tracts, permitting lower risk,

71 activity were then evaluated in response to right ventricular outflow tract PVCs with fixed short, f

72 ngenital heart defects in children requiring right ventricular outflow tract reconstruction typically

73 ocytes, as well as conduction slowing in the right ventricular outflow tract region.

74 (range, 0.4-7 years), 32 patients underwent right ventricular outflow tract reintervention for obstr

75 anatomical mapping was performed in both the right ventricular outflow tract (RVOT) and aortic sinus

76 ial recording/pacing catheters placed in the right ventricular outflow tract (RVOT) and right ventric

77 (MAPs) were recorded simultaneously from the right ventricular outflow tract (RVOT) and the right ven

78 istance 1 mm) was placed epicardially on the right ventricular outflow tract (RVOT) before video-assi

79 Due to recurrent right ventricular outflow tract (RVOT) dysfunction, pati

80 All had a right ventricular outflow tract (RVOT) gradient >50 mm H

81 r patching (TAP) and thereafter allow normal right ventricular outflow tract (RVOT) growth.

82 The shape of right ventricular outflow tract (RVOT) has been assumed

83 of an anomalous coronary artery crossing the right ventricular outflow tract (RVOT) in patients with

84 RMVT typically arises from the right ventricular outflow tract (RVOT) in patients witho

85 ver, several studies have indicated that the right ventricular outflow tract (RVOT) is likely to be t

86 t comorbidity in patients with postoperative right ventricular outflow tract (RVOT) obstruction or pu

87 g left ventricular outflow tract (LVOT) from right ventricular outflow tract (RVOT) origin in patient

88 Right ventricular outflow tract (RVOT) reconstruction wi

89 ntricle to the physiological sequelae of the right ventricular outflow tract (RVOT) reconstruction.

90 act (LVOT) reconstructions (31 adult) and 43 right ventricular outflow tract (RVOT) reconstructions (

91 d 28 consecutive patients who presented with right ventricular outflow tract (RVOT) tachycardia.

92 etween idiopathic and cardiomyopathy-related right ventricular outflow tract (RVOT) ventricular arrhy

93 ty in guiding ablation of difficult-to-treat right ventricular outflow tract (RVOT) ventricular tachy

94 During ablation of right ventricular outflow tract (RVOT) VT, 12-lead ECG p

95 The right ventricular outflow tract (RVOT) was the most comm

96 with an anomalous coronary artery across the right ventricular outflow tract (RVOT), "small pulmonary

97 axis morphology are usually localized to the right ventricular outflow tract (RVOT), presumably below

98 cardias are focal in origin, localize to the right ventricular outflow tract (RVOT), terminate in res

99 ntly around the tricuspid annulus and in the right ventricular outflow tract (RVOT).

100 to patients with nonconduit outflow tracts (right ventricular outflow tract [RVOT]) has the potentia

101 clinical entity of an isolated subepicardial right ventricular outflow tract scar serving as a substr

102 ith risk factors for early primary repair by right ventricular outflow tract stenting (stent).

103 Right ventricular outflow tract stenting of symptomatic

104 ery pressure (sPAP) (r = 0.880; p < 0.0001), right ventricular outflow tract stroke volume (r = 0.660

105 asia/cardiomyopathy (ARVD/C) from those with right ventricular outflow tract tachycardia (RVOT-VT).

106 d in 35 (76%) of 46 patients with idiopathic right ventricular outflow tract tachycardia and in seven

107 A diagnosis of idiopathic right ventricular outflow tract tachycardia or indetermi

108 omes have been described, such as idiopathic right ventricular outflow tract tachycardia, idiopathic

109 exhibiting heart malformations involving the right ventricular outflow tract-the same region affected

110 cuspid regurgitant velocity (TRV, ms) to the right ventricular outflow tract time-velocity integral (

111 multipolar catheters were positioned in the right ventricular outflow tract to map the tachycardia f

112 rdia with left bundle branch block excluding right ventricular outflow tract ventricular tachycardia.

113 entified on the anterior right free wall and right ventricular outflow tract, which increased after f

114 al substrate was observed exclusively in the right ventricular outflow tract with the following prope