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1 on, LV end-diastolic dimension, LV mass, and right ventricular systolic pressure.
2 ted for approximately 60% of the increase in right ventricular systolic pressure.
3 ertrophy, pulmonary vascular remodeling, and right ventricular systolic pressure.
4 se PASMCs was correlated with an increase in right ventricular systolic pressure.
5 ere associated with reversible increments of right ventricular systolic pressure.
6 h-fat diet develop PAH as judged by elevated right ventricular systolic pressure.
7 aled NO, or intravenous iloprost in reducing right ventricular systolic pressure.
8 T gradient (51.4 to 21.7 mm Hg, P<0.001) and right ventricular systolic pressure (72.8 to 47.3 mm Hg,
9 monary hypertension, judged by regression of right ventricular systolic pressure and hypertrophy and
10                   At 17 weeks postinfection, right ventricular systolic pressure and liver and lung e
11 asurements indicated modest increases in the right ventricular systolic pressure and right ventricle
12 atment significantly attenuated elevation of right ventricular systolic pressure and right ventricula
13                 S100A4/Mts1 mice had greater right ventricular systolic pressure and right ventricula
14 (-/-) mice developed significantly increased right ventricular systolic pressure and substantial pulm
15 odel, A-17 treatment significantly decreased right ventricular systolic pressure and total pulmonary
16            The Tg(+) mice exhibited elevated right ventricular systolic pressures and right ventricul
17 tation, LV ejection fraction, LV dimensions, right ventricular systolic pressure) and exercise variab
18 In the PH mouse model, A-17 and A-21 reduced right ventricular systolic pressure, and all antagomirs
19   All rats developed pulmonary hypertension (right ventricular systolic pressure approximately 100 mm
20 had lower plasma adiponectin, and had higher right ventricular systolic pressure associated with righ
21  mice, sildenafil attenuated the increase in right ventricular systolic pressure but without a signif
22 ypertension ( approximately 118% increase in right ventricular systolic pressure) but not polycythemi
23 pha(-/-) mice exhibited significantly higher right ventricular systolic pressure compared with wild-t
24 sure cardiac parameters, including estimated right ventricular systolic pressure (ERVSP), and a full
25                     In PPH patients, resting right ventricular systolic pressure fell after NO inhala
26 6.3 to 10.9 +/- 6.7 mm Hg (P<0.001), and the right ventricular systolic pressure fell from 71.6 +/- 2
27  of echocardiogram data, pHTN was defined as right ventricular systolic pressure greater than or equa
28                        A subset of mice with right ventricular systolic pressure &gt;30 mm Hg exhibited
29                In patients with an estimated right ventricular systolic pressure&gt;35 mm Hg, suggestive
30 s score (hazard ratio 1.55), higher baseline right ventricular systolic pressure (hazard ratio 1.11),
31 dence interval, 0.17-0.50; P<0.001), resting right ventricular systolic pressure (hazard ratio, 1.03;
32 gurgitation (hazard ratio, 1.29), and higher right ventricular systolic pressure (hazard ratio, 1.3)
33 nuated the acute hypoxia-induced increase in right ventricular systolic pressure in anesthetized mice
34 cular lesions, fasudil also markedly reduced right ventricular systolic pressure in late-stage rats.
35        We observed a significant increase in right ventricular systolic pressure in Prkg1(-/-) mice i
36          Hypoxia caused similar increases in right ventricular systolic pressure in wild-type and HO-
37 pressing nuRFP alone) attenuated MCT-induced right ventricular systolic pressure increase, right vent
38 emia, medications, aortic regurgitation, and right ventricular systolic pressure), increased the c-st
39 eart catheterization in patients in whom the right ventricular systolic pressure is calculated to be
40 th pulmonary hypertension, but estimation of right ventricular systolic pressure is often inaccurate.
41                               Controls had a right ventricular systolic pressure &lt; 40 mm Hg (if estim
42 ios (Th1/Tc1) (P=0.009), while in those with right ventricular systolic pressure&lt;35 mm Hg, a lower va
43 rols (normal echocardiography with estimated right ventricular systolic pressure, &lt;35 mm Hg; n = 122)
44 t ventricular systolic pressure, peak stress right ventricular systolic pressure, metabolic equivalen
45 and MCT+AP rats developed PH with respective right ventricular systolic pressures of 40.2 +/- 1.5 and
46 onse to treatment at 6 months was defined by right ventricular systolic pressure or MPAP as significa
47 , indexed LV end-systolic dimension, resting right ventricular systolic pressure, peak stress right v
48 % O(2)) exhibited a significant reduction in right ventricular systolic pressure (placebo versus sild
49 lower LV ejection fraction, and high resting right ventricular systolic pressure predicted worse outc
50 iety of Thoracic Surgeons score and baseline right ventricular systolic pressure) provided incrementa
51 ressures (Ppa = 41 +/- 3 mm Hg) (p < 0.001), right ventricular systolic pressures (Prv,s = 45 +/- 2 m
52 on and remodeling, as evidenced by decreased right ventricular systolic pressure, ratio of right vent
53 e pulmonary hypertension, judged by elevated right ventricular systolic pressure, right ventricular h
54 nhibitor resulted in prevention of increased right ventricular systolic pressure, right ventricular h
55 ia-treated rats with established PH improved right ventricular systolic pressures, right ventricular
56                                              Right ventricular systolic pressure (RVSP) (mean +/- SD)
57 e wall curvature ratio for prediction of the right ventricular systolic pressure (RVSP) in patients c
58                                  The rise in right ventricular systolic pressure (RVSP) normally obse
59 lated altitude of 17,000 ft for 5 weeks, and right ventricular systolic pressure (RVSP) was measured.
60 ndexed LV end-systolic diameter (LVESD), and right ventricular systolic pressure (RVSP) were 62 +/- 2
61  mice were found to have the same life span, right ventricular systolic pressure (RVSP), and lung his
62 itral effective regurgitant orifice, resting right ventricular systolic pressure (RVSP), exercise met
63                                              Right ventricular systolic pressure (RVSP), RV weight, a
64 cardiac echocardiography and measurements of right ventricular systolic pressure (RVSP).
65 erwent Doppler echocardiography to determine right ventricular systolic pressure (RVSP).
66 nary hypertension (PHT) (RVEF 31.4 +/- 9.6%, right ventricular systolic pressure [RVSP] 76.5 +/- 26.2
67 y hypertension with exaggerated elevation of right ventricular systolic pressure, significant right v
68         In the lungs of these mice with high right ventricular systolic pressure, the expression of p
69  hypertension associated with an increase in right ventricular systolic pressure, thickening of the p
70 AP-Cav to MCT rats significantly reduced the right ventricular systolic pressure to 30.1 +/- 1.3 mm H
71                                  Preexercise right ventricular systolic pressure was assessed by Dopp
72 s score, degree of aortic regurgitation, and right ventricular systolic pressure) was 0.64 (95% confi
73       Society of Thoracic Surgeons score and right ventricular systolic pressure were 2+/-3 and 15+/-
74   The Society of Thoracic Surgeons score and right ventricular systolic pressure were 3.3+/-3 and 31+
75 rifice, indexed LV end-diastolic volume, and right ventricular systolic pressure were 4+/-1%, 62+/-3%
76 n fraction, mean aortic valve gradients, and right ventricular systolic pressure were 7+/-6, 58+/-6%,
77  determined that age, renal dysfunction, and right ventricular systolic pressure were independently a
78                               Wide ranges of right ventricular systolic pressure were observed in mic
79                               Despite higher right ventricular systolic pressures with chronic hypoxi

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