ライフサイエンスコーパス: right-atrial

1 ented controls (n=6 for left atrial, n=4 for right atrial).

2 During this period, 523 catheters (276 right atrial, 155 left atrial, 68 common atrial, and 24

3 ccurrence of atrial fibrillation between the right atrial AAI and AAT groups (P=0.8).

4 ASc score was 0 to 1 in 82%, 8% had previous right atrial ablation, whereas all had at least 1 antiar

5 patients, with 15 of 67 terminations due to right atrial ablation.

6 Average distance of the repeated right atrial ablations was 3.92+/-0.5 mm.

7 breakthrough waves if they were the earliest right atrial activated site.

8 C) atrial flutter (Afl) are used to describe right atrial activation around the tricuspid valve in th

9 Right atrial activation was examined 3 days after surger

10 ficacy is similar between femoral venous and right atrial administration.

11 During right atrial and endocardial pacing, AP rise time (10%-9

12 d 43 without OSA [apnea-hypopnea index <5]), right atrial and left atrial voltage distribution, condu

13 ure, right atrial minimal volume, as well as right atrial and left ventricular longitudinal strain re

14 ranolazine (5 mumol/L) in coronary-perfused right atrial and left ventricular preparations isolated

15 m capture threshold data had higher baseline right atrial and right ventricular capture thresholds an

16 Right atrial and right ventricular dimensions correlated

17 easing severity of RV dysfunction as well as right atrial and RV enlargement were also associated wit

18 d associated with increased mortality as did right atrial and RV enlargement, but not RV dysfunction.

19 transfemoral approach, baseline TR severity, right atrial and RV size and RV function were evaluated

20 Right atrial and ventricular volumes were calculated bef

21 atrial pacing (n=6 for left atrial, n=4 for right atrial) and sham instrumented controls (n=6 for le

22 the time of first AF recurrence at both the right atrial appendage (161+/-22 vs 167+/-26 ms, P=0.05)

23 onsists of a shock between electrodes in the right atrial appendage (RAA) and coronary sinus (CS).

24 nd their enzymatic sources in samples of the right atrial appendage (RAA) from 303 patients undergoin

25 enates and isolated atrial myocytes from the right atrial appendage (RAA) of patients undergoing card

26 ) energy of the standard lead configuration, right atrial appendage (RAA) to coronary sinus (CS), was

27 We measured AFCL at the right atrial appendage and distal coronary sinus before

28 Biopsies were obtained from the right atrial appendage before and after aortic cross-cla

29 nts, cardiac myocytes were isolated from the right atrial appendage during CABG.

30 CSCs were isolated from the right atrial appendage harvested and processed during su

31 lized myofibers prepared from samples of the right atrial appendage obtained from nondiabetic (n = 13

32 slices from rat left ventricle and from the right atrial appendage of patients undergoing elective c

33 ing routine cardiac surgical procedures from right atrial appendage tissue discarded from 2 age group

34 Right atrial appendage was collected from 8 male patient

35 Arterioles dissected from right atrial appendage were studied with video microscop

36 We hypothesized that partial clipping of the right atrial appendage would increase the blood flow to

37 Three of these were right atrial appendage-to-right ventricle APs, and epica

38 2 mm) was inserted into the incision in the right atrial appendage.

39 ously into the distal coronary sinus and the right atrial appendage.

40 /-7 micro m, n=71) were dissected from human right atrial appendages at the time of cardiac surgery a

41 Human CPCs from the right atrial appendages from children of different ages

42 METHODS AND Human CPCs from the right atrial appendages from children of different ages

43 techniques and immunoconfocal microscopy in right atrial appendages from patients with ischemic hear

44 ed glycoproteins were identified in left and right atrial appendages from the same patients.

45 HCRAs were dissected from right atrial appendages obtained from patients during ca

46 ardiopulmonary bypass, paired samples of the right atrial appendages were obtained before venous cann

47 Methods and Results-- Right atrial appendages were obtained from AF patients u

48 HCAs were dissected from right atrial appendages.

49 Right-atrial appendages from control sinus rhythm patien

50 r end-diastolic area index <10.0 cm(2)/m(2), right atrial area <20 cm(2), and right ventricular fract

51 P/PCWP was associated with increasing median right atrial area (23, 26, 29 cm2, respectively; P<0.005

52 (hazard ratio [HR], 1.02 per 1 ms; P=0.046), right atrial area (HR, 1.05 per 1 cm(2); P=0.02), right

53 Pericardial effusion (p = 0.003) and indexed right atrial area (p = 0.005) were predictors of mortali

54 Pericardial effusion (p = 0.017), indexed right atrial area (p = 0.012) and the degree of septal s

55 change, 24 vs. 33%), right heart remodeling (right atrial area index, 17.0 vs. 12.1 cm(2)/m), and RV-

56 en consumption of greater than 15 mL/min/kg, right atrial area of less than 18 cm2, cardiac index of

57 In comparison with jet area or jet/right atrial area ratio, the VCW showed better correlati

58 RVESRI and right atrial area were strongly connected to the other r

59 d extent, RV volumes/mass/ejection fraction, right atrial area, peak Vo(2), and age at repair.

60 on of echocardiographic variables, including right atrial area, right ventricular fractional area cha

61 of the maximal jet area to the corresponding right atrial area.

62 ) were reduced, whereas left ventricular and right atrial beta2-AR and Gi (guanine nucleotide binding

63 ithout a history of AF consented to left and right atrial biopsies and a pre-operative peripheral blo

64 Right atrial biopsies were collected before cardiopulmon

65 Mice inheriting both transgenes exhibited right atrial cardiomyocyte cell cycle activity and a con

66 ty in left atrial cardiomyocytes, but not in right atrial cardiomyocytes.

67 ft circumflex (LCx) catheter (n=11) or via a right atrial catheter in animals with an LCx occluder (n

68 Right atrial CFE area was reduced by LA ablation, from 2

69 and linear lesions, and LA ablation reduced right atrial CFE area.

70 Ultrasound imaging showed that partial right atrial clipping led to a significant increase in l

71 n ex ovo culture setup, we performed partial right atrial clipping on embryonic day 8 chick embryos.

72 cyte proliferation and myocardial mass after right atrial clipping was also observed in embryos with

73 Cerebral cooling lagged behind right atrial cooling.

74 les and 1 patient with a left ventricular-to-right atrial device did not recover.

75 ysmal group, there was a significant left-to-right atrial DF gradient, with DF highest at the PV/left

76 y resuscitation hemodynamics over time, mean right atrial diastolic pressure was 9 +/- 0.6 mm Hg with

77 increase of right atrial pressure (RAP) and right atrial dilatation.

78 -system was observed in patients with marked right atrial dilation within the Fontan group.

79 mias who had the Fontan procedure had larger right atrial dimension than those without arrhythmias (6

80 ve duration and P-wave dispersion and larger right atrial dimension than those without the arrhythmia

81 dispersion were significantly correlated to right atrial dimension within the Fontan group (r=0.55,

82 ormal, or low, and decreased with increasing right atrial dimensions (r=-0.62, P=0.006).

83 Right atrial dimensions, RV dimensions, and RV systolic

84 bances and 19 of 31 (61%) subjects exhibited right atrial disturbances.

85 simultaneous high-resolution mapping of the right atrial endo- and epicardial wall during AF in huma

86 Aortic, right atrial, endotracheal pressure, intracranial pressu

87 Coronary venous and right atrial enhancement were evaluated to assess whethe

88 Pericardial effusion, right atrial enlargement and septal displacement are ech

89 ve cardiomegaly due to right ventricular and right atrial enlargement at later stages of the disease.

90 rare and is associated with advanced age and right atrial enlargement.

91 imension, and 25 of 89 patients (28.1%) with right atrial enlargement.

92 In 13 other open-chest dogs, right atrial ERP was determined before and after occlusi

93 d a concomitant reduction in the severity of right atrial fibrosis, despite the presence of a similar

94 d counterclockwise isthmus-dependent (CCWID) right atrial flutter (AFL) and to attempt to correlate F

95 Right atrial flutter with positive flutter waves in the

96 ine the incidence and mechanisms of atypical right atrial flutter.

97 Most right atrial flutters with positive flutter wave on surf

98 hanism of the TI isthmus-dependent clockwise right atrial flutters.

99 ary vein foci, becoming markedly smaller for right atrial foci, especially those near the sinoatrial

100 rresponded to craniocaudal activation of the right atrial free wall.

101 in atrial fibrillation (AF), with a left-to-right atrial frequency gradient during AF in isolated sh

102 that human AF would also manifest a left-to-right atrial frequency gradient.

103 Activity from multiple IC neurons in the right atrial ganglionated plexus was recorded in eight a

104 Cx40 is associated with human right atrial gap-junctional resistivity such that increa

105 imental data suggested the potential role of right atrial GP in the AF initiation and maintenance.

106 atheters were successfully positioned at the right atrial, His bundle, and right ventricular target s

107 ly did so at the cost of systemic venous and right atrial hypertension, and the long-term effects of

108 In patients with postoperative right atrial incisional scar and flutter, multiple ablat

109 Twenty-nine patients with single right atrial incisional scars undergoing ablation for sc

110 with atrial flutter (AFL) and postoperative right atrial incisional scars, we sought to assess if th

111 al intensity-time curves were obtained after right atrial injection of gadoteridol (0.025 mmol/kg) wi

112 non-myelinated cardiopulmonary afferents by right atrial injection of phenylbiguanide (PBG).

113 (2) The C-fibre response to right-atrial injection of capsaicin (0.5 microg kg(-1))

114 conduction in both vagus nerves was blocked, right-atrial injection of capsaicin elicited augmented b

115 Right atrial injections of PBG excited nine of eleven CV

116 ulmonary C-fibre afferents are stimulated by right atrial injections of phenylbiguanide (PBG).

117 ibre afferent-evoked excitation of CVPNs, by right-atrial injections of phenylbiguanide (PBG).

118 paralleling symptoms of the human condition right atrial isomerism.

119 oon positioned at the superior vena cava and right atrial junction (SVC-RAJ) reduces sodium or water

120 Right atrial, left atrial, systolic and mean arterial pr

121 left atrial (LA) lesions, and two after just right atrial lesions.

122 ined in dogs with LBBB-failing hearts during right atrial, LV, and BiV stimulation.

123 Three types of right atrial macro-re-entrant circuits were identified:

124 ntain View, California), to perform left and right atrial mapping and radiofrequency ablation of atri

125 Electroanatomic right atrial maps were obtained during 15 MacroATs in 13

126 racic echocardiography revealed an irregular right atrial mass and moderate to severe pericardial eff

127 tween coronary perfusion pressure (aortic to right atrial mean decompression-phase pressure) and cere

128 ysis, age, sex, pulmonary systolic pressure, right atrial minimal volume, as well as right atrial and

129 ether exercise is able to influence left and right atrial morphology and function also in female athl

130 Left and right atrial myocardial deformation was assessed by two-

131 Right atrial myocardium was obtained from 141 consecutiv

132 NCX-mediated Ca2+ influx in isolated canine right atrial myocytes by approximately 60%, but had no s

133 ted from the AP and VP recorded after 4 s of right atrial occlusion.

134 re instances, catheter- or surgically- based right atrial or sinus node modification may be helpful,

135 to-epicardial wavefronts were studied during right atrial or ventricular endocardial pacing.

136 e was less than 10 mm Hg in 49 patients, the right atrial oxygen saturation was less than 70% in 97 p

137 with OSA had lower atrial voltage amplitude (right atrial, P=0.0005; left atrial, P=0.0001), slower c

138 al, P=0.0001), slower conduction velocities (right atrial, P=0.02; left atrial, P=0.0002), and higher

139 In contrast, hearts from both right atrial-paced HF dogs and an additional 4 noninstru

140 rwent left bundle-branch ablation and either right atrial pacing (190 to 200 bpm) for 6 weeks (DHF) o

141 gs underwent left-bundle branch ablation and right atrial pacing (200 beats/min) for 6 weeks (DHF) or

142 Fast-rate right atrial pacing (600 bpm) was used to induce and mai

143 obtained from dogs with AF induced by rapid right atrial pacing (n=6 for left atrial, n=4 for right

144 Reconstructions were performed during right atrial pacing and nine cycles of VT.

145 or descending coronary artery occlusion with right atrial pacing at 150 bpm.

146 to 200 bpm) for 6 weeks (DHF) or 3 weeks of right atrial pacing followed by 3 weeks of resynchroniza

147 We performed rapid right atrial pacing in 6 dogs for 111+/-76 days to induc

148 AVNS was delivered via a right atrial pacing lead positioned in the posterior rig

149 and varied little with isoproterenol or high right atrial pacing rate.

150 Right atrial pacing was performed in 41 subjects with co

151 rded at baseline and after LV epicardial and right atrial pacing with high-resolution Doppler and con

152 ight ventricular pacing but synchronous with right atrial pacing.

153 re studied and subjected to 5 hours of rapid right atrial pacing.

154 model of sustained AF produced by prolonged right atrial pacing.

155 Four human left-right atrial pairs were subjected to whole-genome expres

156 Right atrial peak atrial longitudinal strain and peak at

157 Canine right atrial preparations (n=7) were optically mapped.

158 ramural optical mapping of coronary-perfused right atrial preparations revealed that adenosine (10 mu

159 ed SCr best predicted death in patients with right atrial pressure <10 mm Hg.

160 iuretic peptide (BNP), and hemodynamics with right atrial pressure <8 mm Hg and cardiac index >2.5 mg

161 alization of right ventricular function with right atrial pressure <8 mm Hg and cardiac index >2.5 to

162 and 40 mg groups, respectively; P<0.05) and right atrial pressure (-2.0+/-0.4, -3.7+/-0.4, and -3.5+

163 Tolvaptan also reduced right atrial pressure (-4.4 +/- 6.9 mm Hg [p < 0.05], -4

164 to approximately 2-fold greater increases in right atrial pressure (10+/-4 versus 6+/-3 mm Hg; P=0.02

165 4 versus 5.1+/-1.9 L/min; P=0.01), increased right atrial pressure (12+/-5 versus 4+/-1 mm Hg; P=0.00

166 ary vascular resistance (by 29%; P=0.03) and right atrial pressure (by 40%; P=0.007), but with only m

167 t ventricle (HR, 10.5; P=0.0429), and higher right atrial pressure (HR, 1.3 per 1 mm Hg; P=0.0016).

168 76; 95% CI, 1.76-12.88; P=0.0021), increased right atrial pressure (HR, 1.34; 95% CI, 0.95-1.90; P=0.

169 A paradoxical inspiratory rise in right atrial pressure (in contrast to the normal fall du

170 using a Cox proportional hazards model: mean right atrial pressure (mRAP) more than or equal to 14 mm

171 levels of DHEA-S were associated with lower right atrial pressure (P = 0.02) and pulmonary vascular

172 istance, mean pulmonary artery pressure, and right atrial pressure (P</=0.001, 0.003, 0.017, and 0.03

173 eart Association functional class (P=0.009), right atrial pressure (P=0.037), and stroke volume (P=0.

174 dds ratio, 5.3; P<0.001), whereas changes in right atrial pressure (P=0.36) and pulmonary capillary w

175 wedge pressure (PCWP) (32 to 14 mm Hg), and right atrial pressure (RA) (19 to 9 mm Hg).

176 Diastolic dysfunction was defined as right atrial pressure (RAP) >/=15 mm Hg (right ventricul

177 dditional pediatric hemodynamic cutpoints of right atrial pressure (RAP) >12 mm Hg or pulmonary capil

178 ocardiographic and invasive measures of mean right atrial pressure (RAP) (r = 0.863; p < 0.0001), sys

179 Although right atrial pressure (RAP) and pulmonary capillary wedg

180 nce of AF was associated with an increase of right atrial pressure (RAP) and right atrial dilatation.

181 he predictive value of coronary fistulae and right atrial pressure (RAP) score (comprising the tricus

182 2.44, p = 0.577; Q = 14.64, I(2) = 79.51%), right atrial pressure (WMD: 1.01 mmHg, 95%CI: -0.93, 2.9

183 Elevated SCr was associated with higher right atrial pressure and lower cardiac index.

184 Syncopal patients presented with higher right atrial pressure and lower cardiac outputs with low

185 Changes in hemodynamic values (except for right atrial pressure and mean pulmonary artery pressure

186 pressure, pulmonary vascular resistance, and right atrial pressure and provided incremental prognosti

187 s performed during exercise, included higher right atrial pressure and pulmonary capillary wedge pres

188 modynamic changes and additionally decreased right atrial pressure and pulmonary vascular resistance.

189 tolic excursion correlated with WHO-FC, mean right atrial pressure and survival (P<0.05).

190 use of the modified Bernoulli equation, with right atrial pressure assumed to be 10 mm Hg.

191 nal class at diagnosis (P < 0.001), and high right atrial pressure at diagnosis (P = 0.002).

192 Breathing NO decreased the mean right atrial pressure by 12 +/- 3%, mean pulmonary arter

193 High pulmonary vascular resistance and right atrial pressure by invasive hemodynamic measuremen

194 ent of invasively derived measurements, mean right atrial pressure cardiac index, and mixed venous ox

195 ts randomized to the PAC arm (n = 194), only right atrial pressure correlated weakly with baseline SC

196 Right atrial pressure decreased 52% (P=0.012), pulmonary

197 hysiology was defined as inspiratory rise in right atrial pressure during right heart catheterization

198 ary wedge pressure fell from 31 to 18 mm Hg, right atrial pressure from 15 to 8 mm Hg, and SVR from 1

199 parameters also improved with a reduction in right atrial pressure from 22 mm Hg at baseline, to 9 mm

200 >/=40%, exercise PCWP >/=25 mm Hg, and PCWP-right atrial pressure gradient >/=5 mm Hg.

201 odynamic variables such as cardiac index and right atrial pressure have consistently been associated

202 Right atrial pressure increased by 2.5 +/- 1.8 mm Hg (P=

203 nary vascular resistance, cardiac index, and right atrial pressure may be used to stratify risk of de

204 s with primary pulmonary hypertension (PPH), right atrial pressure may exceed left atrial pressure du

205 .49; P<0.01) per 10-mL/m(2) decrease and for right atrial pressure was 1.05 (95% confidence interval,

206 In the 123 patients, the right atrial pressure was less than 10 mm Hg in 49 patie

207 Exercise right atrial pressure was the highest in MI+DD followed

208 regurgitation, low cardiac index, and raised right atrial pressure were associated with poor survival

209 alk distance, stroke volume index (SVI), and right atrial pressure were independently associated with

210 SVI and right atrial pressure were the hemodynamic variables tha

211 perfusion pressure (diastolic; aortic minus right atrial pressure) and cerebral perfusion pressure (

212 coronary perfusion pressure (CPP) (aortic - right atrial pressure) without epinephrine (A-CPR 21 +/-

213 pressure (pulmonary capillary wedge pressure-right atrial pressure), which reflects LV preload indepe

214 cluded exercise tolerance, functional class, right atrial pressure, and vasodilator response to adeno

215 vement of 6MWD, pulmonary arterial pressure, right atrial pressure, cardiac index and pulmonary vascu

216 d Health Organization functional class, mean right atrial pressure, cardiac index, and mixed venous o

217 ffect on pulmonary-capillary wedge pressure, right atrial pressure, heart rate, or cardiac output.

218 indicators of RV-PV function (i.e., resting right atrial pressure, mean PA pressure, pulmonary vascu

219 Active treatment significantly lowered right atrial pressure, mean pulmonary artery pressure, a

220 e TR and EDPR gradients, in conjunction with right atrial pressure, provide Doppler estimates of pulm

221 In controls subjects, right atrial pressure, pulmonary arterial pressure, and

222 8) at 3 months (p=0.035), with no changes in right atrial pressure, pulmonary arterial pressure, or p

223 Hemodynamic determinants included elevated right atrial pressure, reduced pulmonary artery pulse pr

224 e tissue disease, functional class III, mean right atrial pressure, resting systolic blood pressure a

225 , diffusing capacity of carbon monoxide, and right atrial pressure.

226 valuation of right ventricular function, and right atrial pressure.

227 pression end-diastolic arterial pressure and right atrial pressure.

228 fect on heart rate, mean aortic pressure, or right atrial pressure.

229 radient (=pulmonary capillary wedge pressure-right atrial pressure; r=0.67; P=0.003), suggesting reli

230 nd a significant interaction between SCr and right atrial pressures (interaction P<0.0001); increased

231 Arterial and right atrial pressures and end-tidal CO2 were measured.

232 sure, whereas diastolic left ventricular and right atrial pressures decreased significantly and propo

233 Aortic and right atrial pressures were measured with micromanometer

234 ssure, pulmonary arterial pressure, left and right atrial pressures, intracranial pressure, body temp

235 Nitrite reduced right atrial pressures, with no effect on cardiac output

236 ar dysfunction on echocardiography, ratio of right atrial/pulmonary capillary wedge pressure, hemoglo

237 ication (TR severity, right ventricular, and right atrial quantification) with simultaneous respirome

238 In Cx40(+/+) hearts, spontaneous right atrial (RA) activation showed a focal breakthrough

239 Right atrial (RA) and right ventricular (RV) chamber dim

240 Right atrial (RA) and skeletal muscle(SM) was harvested

241 prospectively characterize the reduction in right atrial (RA) area and right ventricular (RV) volume

242 the culprit coronary lesion is distal to the right atrial (RA) branches, augmented RA contractility e

243 l fibrillation (AF), and left atrial (LA)-to-right atrial (RA) DF gradients have been identified in b

244 e PFO and prominent eustachian valve (EV) or right atrial (RA) filamentous strands were found more fr

245 This study was done to characterize human right atrial (RA) flutter (AFL) using noncontact mapping

246 A canine right atrial (RA) linear lesion model was used to produc

247 of non-cavotricuspid isthmus (CTI)-dependent right atrial (RA) or left atrial (LA) flutter circuits.

248 In 5 of 8 patients who converted to NSR, right atrial (RA) pacing was performed for 3 minutes in

249 Elevated right atrial (RA) pressure is a risk factor for mortalit

250 Elevated right atrial (RA) pressure is an established prognostic

251 pertension has been associated with elevated right atrial (RA) pressure.

252 Right atrial (RA) size is important in screening, diagno

253 olerance and effectiveness of overdrive high right atrial (RA), dual-site RA and support (DDI or VDI)

254 evated central venous pressure (expressed as right atrial [RA] area, RA pressure, and ratio of RA to

255 ntry required shorter fluoroscopy times than right atrial re-entry, which entailed a longer intramyoc

256 Right atrial reentrant tachycardia resulting from lower

257 atrial flutter (n=7), non-isthmus-dependent right atrial reentry (n=7), and 1 focal atrial tachycard

258 59.4+/-7.6% versus 61.9+/-6.8%; P<0.01), and right atrial reverse remodeling occurred (pPVR versus mP

259 When the right atrial/right ventricular interferences are compare

260 ling secondary to atrioventricular block and right atrial samples from 130 patients undergoing cardia

261 ), and [Ca(2+)](i) (Fluo-3) were measured in right atrial samples from 76 sinus rhythm (control) and

262 n of Rac1 and NOX2-NADPH oxidase activity in right atrial samples from patients who developed postope

263 Chronic outcomes were associated with higher right atrial saturations, use of electroanatomic mapping

264 ntional discontinuation of CRT (1%), loss of right atrial sensing (1%), and ventricular oversensing (

265 2- and 3-chamber devices displayed impaired right atrial sensing.

266 Catheter ablation of the right atrial septum attenuated interatrial conduction wi

267 In 2 patients, pacing the high right atrial septum near the presumed site of Bachmann's

268 before and after sequential ablation of the right atrial septum, targeting interatrial conduction zo

269 adiofrequency ablated lesions applied to the right atrial septum.

270 AC <23% (P<0.001), TAPSE <17 mm (P=0.02), or right atrial short axis/BSA >/=25 mm/m(2) (P=0.04) at ba

271 action potentials were recorded at multiple right atrial sites and during different basic cycle leng

272 or paroxysmal atrial fibrillation (PAF); if right atrial sites are important; and what the long-term

273 Human right atrial specimens were obtained during routine cong

274 ation resulted in shorter A-H intervals than right atrial stimulation (73+/-3 ms versus 99+/-3 ms; P<

275 During right atrial stimulation, the anterior and posterior app

276 Right atrial streak artifact focally traversed the right

277 visualization of the guide wire and positive right atrial swirl sign using the subcostal four-chamber

278 Immediately before defibrillation: 1) right atrial systolic/diastolic pressures (mm Hg) were l

279 o characterize the circuit of macroreentrant right atrial tachycardia (MacroAT) in patients after sur

280 Right atrial tachycardias included cavotricuspid isthmus

281 dias involving the left atrium compared with right atrial tachycardias.

282 n less successful in eliminating left versus right atrial tachycardias.

283 ternal chiller to 10 degrees C, cerebral and right atrial temperatures were reduced by 0.49 +/- 0.09

284 xternal chiller to 4 degrees C, cerebral and right atrial temperatures were reduced by 0.61 +/- 0.18

285 R included advanced age, female sex, greater right atrial than left atrial enlargement and lower syst

286 d nonsystemic TEC, defined as Fontan conduit/right atrial thrombus or pulmonary embolus.

287 ocardial oxidative stress were quantified in right atrial tissue from 104 consecutive patients with m

288 Right atrial tissue was also collected from humans with

289 Right atrial tissue was harvested pre- and post-CP/Rep f

290 Left and right atrial tissue was obtained from dogs with AF induc

291 NP and BNP in hypoxic compared with normoxic right atrial tissue.

292 l heart disease (univentricular heart with a right atrial to right ventricle bioprosthesis in 3, Ebst

293 Isometrically contracting isolated human right atrial trabeculae were exposed to MIF (100 ng/mL)

294 Lead placement was two right atrial, two RV, and one LV.

295 -3.6 versus 26.7+/-6.9 mL/m(2); P<0.001) and right atrial volume index (15.66+/-3.09 versus 20.47+/-4

296 Patients had dilated right-sided chambers (right atrial volume index, 44 +/- 19 mL/m(2); RV end-dia

297 6), reflux grade of 3 or higher (OR = 2.63), right atrial volume of greater than or equal to 106 cm(3

298 ar area was more closely correlated with the right atrial volume than right ventricular end-systolic

299 IART only, seven had both, and one had a low right atrial wall tachycardia that could not be entraine

300 perative mapping of the endo- and epicardial right atrial wall was performed during (induced) AF in 1