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1 4 mm (left to right shunts) and 10 +/- 3 mm (right to left shunts).
2  and do not allow quantitative assessment of right to left shunt.
3 the pulmonary capillary bed through anatomic right-to-left shunts.
4 r impair the ability of inhaled NO to reduce right-to-left shunting.
5 han both patients with PFO and those with no right-to-left shunt (7.7 vs. 8.6 vs. 9.3 kPa, respective
6                       PAVMs provide anatomic right-to-left shunts, allowing systemic venous blood to
7 vations suggest a causal association between right-to-left shunts and migraine with aura.
8                                              Right-to-left shunts are also associated with stroke and
9 FO size, hypermobile septum, and presence of right-to-left shunt at rest are associated with clinical
10 ciated hypermobile septum, and presence of a right-to-left shunt at rest have all been proposed as ma
11 esence of neither a hypermobile septum nor a right-to-left shunt at rest was detected more often in t
12 via convective and evaporative heat loss, so right-to-left shunted blood flow through a patent forame
13                             Exercise-induced right-to-left shunting can be detected by noninvasive, c
14      In total, 163 of 432 patients (38%) had right-to-left shunts consistent with a moderate or large
15 lactic treatments, and had moderate or large right-to-left shunts consistent with the presence of a P
16      Improvement is related to the degree of right-to-left shunt created.
17  preventive medications, and had significant right-to-left shunt defined by transcranial Doppler.
18 ubmaximal exercise testing despite increased right-to-left shunting during exercise.
19 ree patients with a patent foramen ovale and right-to-left shunt flow while breathing at F(i)O(2) = 1
20 chnique has been used to detect and quantify right-to-left shunt for more than 50 years.
21 ontan circulation that results in a residual right-to-left shunt has improved operative survival rate
22 ation between migraine with aura and cardiac right-to-left shunts has been reported.
23 th low oxygen saturation (indicating greater right-to-left shunting); higher transferrin iron saturat
24 have severe migraine associated with a large right-to-left shunt in whom closure of the atrial defect
25  This trial confirmed the high prevalence of right-to-left shunts in patients with migraine with aura
26                   It is useful for detecting right-to-left shunts in settings in which transesophagea
27 ad placement (n=10) followed by intracardiac right-to-left shunt (n=5).
28 y elevated pulmonary vascular resistance and right-to-left shunting of blood through a systemic-to-pu
29  increased pulmonary vascular resistance and right-to-left shunting of deoxygenated blood.
30     We investigated the effect of closure of right-to-left shunts on migraine symptoms.
31  paradoxically migrate through physiological right-to left shunts, or they may arise directly from th
32 characterized by pulmonary vasoconstriction, right-to-left shunt pathophysiology and systemic hypoxem
33  In contrast to angioplasty, a fenestration (right-to-left shunt) reduced TCPC pressure at the cost o
34                              An intracardiac right-to-left shunt (RLS) could allow larger fat particl
35 81; P=0.026) and in those with a substantial right-to-left shunt size (hazard ratio, 0.19; 95% confid
36 han 37 years and in those with a substantial right-to-left shunt size, deserves further investigation
37                        It is unknown whether right-to-left shunting through PFO increases during exer
38 ation then may link cardiac and extracardiac right-to-left shunts to migraine aura.
39 ial pressure during exercise, resulting in a right-to-left shunt via a patent foramen ovale (PFO).
40                 The presence of a PFO with a right-to-left shunt was confirmed with transesophageal e
41 ial for interatrial flow, this may lead to a right-to-left shunt, which becomes physiologically appar

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