ライフサイエンスコーパス: ripple

1 on of the membrane potential ("intracellular ripple").

2 ic propagating Ca(2+) release events (Ca(2+) ripples).

3 s responded more consistently from ripple to ripple.

4 pleats, folds, blisters, and liquid crystal ripples.

5 es such as theta oscillations and sharp-wave ripples.

6 llations, spontaneous ripples, and synthetic ripples.

7 of replay events from non-replay-associated ripples.

8 pal activity patterns, so called hippocampal ripples.

9 quence spiking ("replays") during sharp wave ripples.

10 n the selection of CA1 PCs during sharp-wave ripples.

11 states like theta oscillations or sharp-wave ripples.

12 uency oscillations, the so-called sharp-wave/ripples.

13 pyramidal cells during sharp-wave associated ripples.

14 atum radiatum) rat CA1 PCs during sharp-wave ripples.

15 , whereas another subset is inhibited during ripples.

16 were most active during tasks and sharp wave/ripples.

17 r the time-dependent behaviours of intrinsic ripples.

18 y delayed in time and did not interfere with ripples.

19 interneurons aborted spontaneously occurring ripples.

20 coupling of slow oscillations, spindles, and ripples.

21 ogical blockade of GABAA receptors abolished ripples.

22 -8 Hz) and the other by irregular sharp-wave ripples.

23 propagating Ca(2+) spikes during sharp-wave ripples.

24 ns, theta bursts, and hippocampal sharp-wave ripples.

25 ns during sleep, concurrent with hippocampal ripples.

26 tical delta waves and hippocampal sharp-wave ripples.

27 s of identified CA1 pyramidal neurons during ripples.

28 mechanism keeping most neurons silent during ripples.

29 a higher occurrence of ripples than of fast ripples.

30 t lines, making them unlike terrestrial wind ripples.

31 ripple rates that were higher than those of ripples.

32 primary cortical areas, displayed localized ripple (100 to 150 hertz) oscillations during sleep, con

33 During physiological ripples (100-200 Hz), few pyramidal cells fire together

34 lations spanning the high gamma (50-125 Hz), ripple (125-250 Hz) and fast ripple (250-500 Hz) frequen

35 ciated delta (0.5-4 Hz), theta (4-12 Hz) and ripple (150-250 Hz) oscillations; and (2) stabilization

36 ion were spectrally similar to physiological ripples (150-250Hz).

37 ppocampal place cell ensembles occurs during ripples [16-19].

38 ma (50-125 Hz), ripple (125-250 Hz) and fast ripple (250-500 Hz) frequency bands using intracranial r

39 subcortical deactivation specifically after ripples [27].

40 ided into fast ripples (FRs; 250-500 Hz) and ripples (80-250 Hz), and spikes in pre- and postresectio

41 Besides assigning sharp-wave/ripples a crucial role for replay generation and thus me

42 ting ChIA-PET and Hi-C data sets showed that RIPPLE accurately predicts interactions among enhancers

43 wolves have had effects on Yellowstone that ripple across the entire structure of the food web that

44 coordinated interactions between hippocampal ripple activity and ACC neural firings.

45 enetic stimulation of MnR neurons suppressed ripple activity and inhibition of these neurons increase

46 MnR) is important for regulating hippocampal ripple activity and memory consolidation.

47 ow ACC activity is influenced by hippocampal ripple activity during sleep.

48 ity of ACC neurons are activated just before ripple activity during the sleep state, but not during t

49 d showed that subsequent sleep periods where ripple activity was perturbed by timed electrical stimul

50 eurons becomes active just after hippocampal ripple activity, and that electrical stimulation of the

51 ty and inhibition of these neurons increased ripple activity.

52 layed a further activation immediately after ripple activity.

53 showed increased activity before hippocampal ripple activity; moreover, a subpopulation (17%) display

54 Moreover, the number of ripples after learning predicts subsequent memory perfor

55 SPW-R-triggered fMRI maps show that ripples aligned to the positive peak of their SPWs have

56 The correlation between micrometer-scale ripple alignment and atomic-scale arrangement of exfolia

57 on of ACC neurons correlated positively with ripple amplitude, and the same neurons were excited upon

58 tion maps, we develop an ensemble version of RIPPLE and apply it to generate interactions in five hum

59 modulated the number of induced high gamma, ripple and fast ripple detections in the studied structu

60 Furthermore, the induced high gamma, ripple and fast ripple responses discriminated the encod

61 clusters of bursting cells, but HFOs in the ripple and the fast ripple range are vastly intermixed.

62 h of MWC, because the light scattered by the rippled and smooth metal sidewall can be confined inside

63 interactions between hippocampal sharp-wave ripples and ACC neurons in a state-dependent manner.

64 Importantly, sharp-wave ripples and associated activation appear to regulate act

65 g learning are "replayed" during hippocampal ripples and contribute to the consolidation of episodic

66 mergence of two forms of HFOs reminiscent of ripples and fast ripples recorded in vivo from normal an

67 role of single cells in the subiculum during ripples and found that, dependent on their subtype, they

68 sand on Earth produces decimeter-wavelength ripples and hundred-meter- to kilometer-wavelength dunes

69 ate a critical role of the MnR in regulating ripples and memory consolidation.

70 Together, the data indicate that deficits in ripples and neuronal synchronization occur before overt

71 Yet it is unclear whether ripples and other hippocampal neural events influence en

72 Inhibition of CA3 activity suppressed ripples and replays in CA1 regardless of behavioral stat

73 ic stimulation completely blocked sharp wave ripples and strongly suppressed the power of both slow o

74 gradually increased their activity prior to ripples and were suppressed during the population bursts

75 Here, we document the appearance of ripples and wrinkles in biofilms grown from three specie

76 tial oscillations associated with sharp-wave ripples, and controlled the phase of action potentials.

77 iking during theta oscillations, spontaneous ripples, and focal optically induced high-frequency osci

78 exploration, elevated their association with ripples, and showed increased bursting and temporal coac

79 lated during theta oscillations, spontaneous ripples, and synthetic ripples.

80 campal replay occurs during local sharp-wave ripples, and the associated neocortical replay tends to

81 p-the thalamo-cortical spindles, hippocampal ripples, and the cortical slow oscillations-is thought t

82 aired rats to examine age-related changes in ripple architecture, ripple-triggered spike variance, an

83 Sharp-wave ripples are brief ( approximately 70 ms) high-frequency

84 o test the assumption that SOs, spindles and ripples are functionally coupled in the hippocampus.

85 Ripples are high-frequency oscillations associated with

86 Ripples are ideally suited for memory consolidation [14,

87 Our study predicts that ripples are not persistent oscillations but result from

88 Little is known about how ripples are regulated by other brain regions.

89 Sharp-wave ripples are transient oscillatory events in the hippocam

90 y 200 Hz) of the hippocampus, referred to as ripples, are believed to be important for consolidation

91 port for the use of the propagation of these ripples as a proxy for remote measurements of sediment t

92 This reactivation occurred in ripple-associated awake replay of place cell sequences e

93 In particular, hippocampal sharp-wave ripple-associated neural activation is important for thi

94 tructures is the cause for the nucleation of ripples at the edges that grow towards the center of the

95 Thus, the ripple band could not help to disambiguate the underlyin

96 Scar was assessed for sequential movement of ripple bars, during sinus rhythm or pacing, which were d

97 y a model for seeded elongation featuring a "rippled beta-sheet" interface between seed fibril and do

98 high-frequency (>80 Hz) oscillations called ripples-both during sleep [9, 10] and awake deliberative

99 Thus, MECIII input to CA1 is crucial for ripple bursts and long-range replays specifically in qui

100 state, but roles of neural inputs to CA1 in ripple bursts and replays are unknown.

101 Here we show that ripple bursts in CA1 and medial entorhinal cortex (MEC)

102 CIII input to CA1 during quiet awake reduced ripple bursts in CA1 and restricted the spatial coverage

103 Chains of ripples (ripple bursts) in CA1 have been reported to co-occur wit

104 ic increase in the DMN fMRI signal following ripples, but not following other hippocampal electrophys

105 find increases in ongoing DMN activity after ripples, but not in other RSNs.

106 which on Earth include water-worked current ripples, but on Mars instead form by wind because of the

107 e p-bits, and we present results for a 4-bit ripple carry adder with 48 p-bits and a 4-bit multiplier

108 y reducing gamma oscillations and sharp wave ripples, changes associated with a decrease in extinctio

109 ed spindle co-occurrence and frontal spindle-ripple co-occurrence, eventually resulting in increased

110 etworks, including alterations in sharp wave-ripple complexes.

111 is of the temporal alignment between SPW and ripple components reveals well-differentiated SPW-R subt

112 However, ripple-contingent training also expedites learning, sugg

113 lash leads to a mass displacement toward the ripple crests.

114 re, an external input, mimicking hippocampal ripples, delivered to the cortical network results in in

115 h young rats, the rate of ripple occurrence (ripple density) is reduced in aged rats during postbehav

116 umber of induced high gamma, ripple and fast ripple detections in the studied structures, which was g

117 rve three-color domains with three different ripple directions, which meet at a core.

118 re remains a lack of understanding regarding ripple domains and their topological defects formed on m

119 Here we explore configuration of ripple domains and their topological defects in exfoliat

120 This fascinating configuration of ripple domains may result from the intrinsic hexagonal s

121 e origin was believed to be the formation of ripple domains.

122 TF mutations induce rippling downstream effects by simultaneously altering t

123 terneurons dynamically constrains individual ripple duration.

124 inhibition, pyramidal cell excitability, and ripple dynamics.

125 lography (UEC), we report the observation of rippling dynamics in suspended monolayer graphene, the p

126 selected for during viral evolution or are a ripple effect from the primary function.

127 osphere, they are paradoxically analogous to ripples emerging on granular beds submitted to viscous s

128 Comparisons Ripple et al. (2014) used to demonstrate increased fruit

129 ) hypothesis, not sufficiently considered by Ripple et al., exists and is better supported by availab

130 Hippocampal replay during sharp-wave ripple events (SWRs) is thought to drive memory consolid

131 ocampal-entorhinal circuit during sharp wave ripple events (SWRs) that occur during sleep or rest.

132 Sharp-wave ripple events generated in the hippocampus have been imp

133 Third, replay-associated sharp-wave-ripple events in the local field potential exhibited sub

134 single-unit activity surrounding sharp-wave ripple events were examined in the CA1 region of the hip

135 Ripple events were followed by a prominent afterhyperpol

136 campal input, such as mediated by sharp wave-ripple events, cortical slow oscillations, and synaptic

137 etwork activity during theta oscillations or ripple events, respectively.

138 o a shorter distance corresponding to single ripple events.

139 triple coupling of slow oscillation-spindle-ripple events.

140 SOs), sleep spindles may cluster hippocampal ripples for a precisely timed transfer of local informat

141 tion of hippocamposeptal fibers at theta and ripple frequencies, we elicit postsynaptic GABAergic res

142 Second, mean ripple frequency during prebehavior and postbehavior res

143 harp wave ripples, which are associated with ripple frequency fluctuation of the membrane potential (

144 neurons is both necessary and sufficient for ripple-frequency current and rhythm generation.

145 Instead, they suggest that ripple-frequency excitation leading inhibition shapes in

146 rrents give rise to a major component of the ripple-frequency oscillation in the local field potentia

147 e smallest functional unit that can generate ripple-frequency oscillations is a segment of a dendrite

148 rvalbumin-positive basket cells, which start ripple-frequency spiking that is phase-locked through re

149 : detection of interictal ripples (Rs), fast ripples (FRs), and VHFOs; resective surgery; and at leas

150 rospectively, marked HFOs, divided into fast ripples (FRs; 250-500 Hz) and ripples (80-250 Hz), and s

151 These results constrain competing models of ripple generation and indicate that temporally precise l

152 dy proposes a novel mechanism of hippocampal ripple generation consistent with a broad range of exper

153 en excitatory and inhibitory neurons support ripple generation in the intact hippocampus.

154 re inconsistent with models of intracellular ripple generation involving perisomatic inhibition alone

155 is work, we develop a computational model of ripple generation, motivated by in vivo rat data showing

156 s been postulated by computational models of ripple generation.

157 Individual ripple geometry was recently imaged using scanning tunne

158 nce between the linear periodic potential of rippled graphene and the C60 surface mobility, we demons

159 dimensional (quasi-1D) C60 nanostructures on rippled graphene.

160 In the epileptic hippocampus, fast ripples (>200 Hz) reflect population spikes (PSs) from c

161 While spiking during ripples has been extensively studied, our understanding

162 , motivated by in vivo rat data showing that ripples have a broad frequency distribution, exponential

163 Sharp-wave associated ripples have been shown to be necessary for the consolid

164 Ripples have the potential to be used as modern 'wind va

165 net excitatory input to CA1, while the post-ripple hyperpolarization varies proportionately.

166 hereby adsorbates are carried by propagating ripples in a motion similar to surfing.

167 ticipation during sleep and awake sharp-wave ripples in freely moving rats.

168 owledge of and control over the curvature of ripples in freestanding graphene are desirable for fabri

169 Intrinsic ripples in freestanding graphene have been exceedingly d

170 Ripples in graphene are extensively investigated because

171 d, significant changes in characteristics of ripples in older animals that could impact consolidation

172 thought to originate from periodic nanoscale ripples in the graphene sheet, which enhance puckering a

173 , these modes are altered, forming potential ripples in the local density of states, due to intrinsic

174 erometric imaging attributes this finding to ripples in the membrane that stiffen the graphene sheets

175 tions even at 4.2 K and of the vital role of ripples in the pinning potential, giving new insights in

176 ably, spindles were found to in turn cluster ripples in their troughs, providing fine-tuned temporal

177 tal evidence supports the role of sharp-wave ripples in transferring hippocampal information to the n

178 induced defect coalescence and to long range rippling in graphene.

179 ced high-frequency oscillations ("synthetic" ripples) in freely moving mice.

180 The formation of zigzag directional ripple is consistent with our theoretical model that tak

181 ritic excitation of pyramidal neurons during ripples is countered by shunting of the membrane and pos

182 that the subthreshold depolarization during ripples is uncorrelated with the net excitatory input to

183 frustration associated with an isotropic to rippled lamellar liquid-crystal transition.

184 scale (mm to m) sedimentary structures (e.g. ripple lamination, cross-bedding) have received a great

185 of eyes with advanced GA and CNV revealed a rippled layer of basal laminar deposits in an area of RP

186 Our data suggest that IID and PID ripple-like oscillations (150-250Hz) in human epileptic

187 staple motifs, which self-organize into five ripple-like stripes on the surface of the barrel-shaped

188 pendent bending behaviours, from spontaneous rippling (<5 atomic layers) to homogeneous curving (~ 10

189 Ripple mapping (RM) displays each electrogram at its 3-d

190 Ripple mapping (RM) displays every electrogram deflectio

191 Ripple mapping can be used to identify conduction channe

192 hythm or ventricular pacing and reviewed for ripple mapping conducting channel identification.

193 Ablation was performed along all identified ripple mapping conducting channels (median 18 lesions) a

194 apped in 3 patients and colocated within the ripple mapping conducting channels identified.

195 A median of 2 ripple mapping conducting channels were seen within each

196 Ripple mapping displays every deflection of an electrogr

197 We prospectively used CARTO-based ripple maps to identify conducting channels as a target

198 ng" and plastic changes, regulate subsequent ripple-mediated consolidation of spatial memory during s

199 fluence on wind speed and direction and that ripple movement likely reflects steered wind direction f

200 before inferring regional wind patterns from ripple movement or dune orientations on the surface of M

201 The peak amplitudes of ripples observed are in excellent agreement with the exp

202 in specific temporal order during sharp-wave ripples observed in quiet wakefulness or slow wave sleep

203 First, compared with young rats, the rate of ripple occurrence (ripple density) is reduced in aged ra

204 dle coupling and is accompanied by decreased ripple occurrence.

205 In contrast, ripples occurring at the trough of their SPWs relate to

206 We experimentally demonstrate that ripples on graphene are formed along the zigzag directio

207 our foreheads to crinkly plant leaves, from ripples on plastic-wrapped objects to the protein film o

208 in ripple-spindle coupling without affecting ripple or spindle incidence.

209 anoparticles, and nanoparticles with surface ripples or a 'raspberry' surface morphology.

210 The percentage of resected FRs, ripples, or spikes in pre-ECoG did not predict outcome.

211 e tuned to a narrower range of phases of the ripple oscillation relative to young animals.

212 ential place cell activity during sharp-wave ripple oscillations (SWRs).

213 Given the connection between ripple oscillations and memory consolidation, we investi

214 on, we investigated whether the structure of ripple oscillations and ripple-triggered patterns of sin

215 We examine here whether these ripple oscillations are altered over the course of the l

216 be observed during theta and high-frequency ripple oscillations in the hippocampal CA1 region and is

217 s in the medial prefrontal cortex (mPFC) and ripple oscillations in the hippocampus is thought to und

218 Second, high-frequency ripple oscillations of local field potentials in the hip

219 elay between intracellular and extracellular ripple oscillations varies systematically with membrane

220 High-frequency ripple oscillations, observed most prominently in the hi

221 tion leading inhibition shapes intracellular ripple oscillations.

222 ed-loop optogenetic disruption of sharp wave-ripple oscillations.

223 driving of parvalbumin-positive cells evoked ripple oscillations.

224 ltered morphology compared to WT EBs, with a rippled outer surface and a smaller size due to decrease

225 volvement of neuromodulatory pathways in the ripple phenomenon mediated by long-range interactions.

226 ected to play a key role in understanding of ripple physics in graphene and other two-dimensional mat

227 g cells, but HFOs in the ripple and the fast ripple range are vastly intermixed.

228 rly in response to prolonged high-frequency (ripple range) stimulation.

229 d a hypersynchronous onset pattern with fast ripple rates that were higher than those of ripples.

230 zures presented with higher ripple than fast ripple rates.

231 of contextual emotional memory occurs during ripple-reactivation of hippocampus-amygdala circuits.

232 orms of HFOs reminiscent of ripples and fast ripples recorded in vivo from normal and epileptic rats,

233 The ripple reduction is associated with less bursty firing a

234 Sharp-wave ripples represent a prominent synchronous activity patte

235 Ca(2+) ripples resemble Ca(2+) waves in terms of local propagat

236 ore, the induced high gamma, ripple and fast ripple responses discriminated the encoded and the affec

237 eolian sand beds exhibit regular patterns of ripples resulting from the interaction between topograph

238 associated with an irregularly thickened and rippled retinal pigment epithelium band in 2 eyes.

239 Chains of ripples (ripple bursts) in CA1 have been reported to co-

240 These findings suggest that ripple-ripple coupling supports hippocampal-association

241 th surgical outcome: detection of interictal ripples (Rs), fast ripples (FRs), and VHFOs; resective s

242 diodes by introducing a spontaneously formed ripple-shaped nanostructure of ZnO and applying an amine

243 hippocampus, replay occurs within sharp wave-ripples: short bouts of high-frequency activity in area

244 nsistent depolarization, often exceeding pre-ripple spike threshold values, current pulse-induced spi

245 rdination surpasses the normal physiological ripple-spindle coupling and is accompanied by decreased

246 wever, whether learning-induced increases in ripple-spindle coupling are necessary for successful mem

247 nsistent with the hypothesized importance of ripple-spindle coupling in memory consolidation, post-tr

248 that contextual fear conditioning increased ripple-spindle coupling in mice.

249 eliminated this learning-induced increase in ripple-spindle coupling without affecting ripple or spin

250 In order to decouple ripple-spindle oscillations, here we chemogenetically in

251 Sharp-wave-ripple (SPW-R) complexes are believed to mediate memory

252 tial sequences during hippocampal sharp wave-ripple (SPW-R) events of quiet wakefulness and sleep is

253 es of rest and sleep, it exhibits sharp-wave/ripple (SPW/R) complexes, which are short episodes of in

254 Because sharp wave-ripples (SPW-R) orchestrate both retrospective and prosp

255 Hippocampal sharp wave-ripples (SPW-Rs) and associated place-cell reactivations

256 vations peaked during hippocampal sharp wave-ripples (SPW-Rs) and involved a subgroup of BLA cells po

257 Sharp-wave ripples (SPW-Rs) in the hippocampus are implied in memor

258 d or younger) identifies potential wind-drag ripple stratification formed under a thin atmosphere.

259 hape, and both directionality and associated ripple structure reflected the segmentation of the maze.

260 uring high frequency (100-250 Hz) sharp wave ripple (SWR) activity in a manner that likely drives syn

261 uring high frequency (100-250 Hz) sharp-wave ripple (SWR) activity in a manner that probably drives s

262 Hippocampal sharp-wave-ripple (SWR) events have been linked to this consolidati

263 Hippocampal activity during awake sharp-wave ripple (SWR) events is important for spatial learning, a

264 Hippocampal sharp-wave ripple (SWR) events occur during both behavior (awake SW

265 During sharp-wave ripple (SWR) events, hippocampal neurons express sequenc

266 Sharp-wave ripples (SWRs) are high-frequency local field potential

267 Sharp-wave ripples (SWRs) are high-frequency oscillations that gene

268 Hippocampal sharp-wave ripples (SWRs) are highly synchronous oscillatory field

269 ronal activity during hippocampal sharp wave-ripples (SWRs) is essential in memory formation.

270 IGNIFICANCE STATEMENT Hippocampal sharp-wave ripples (SWRs) occur both in the awake state during beha

271 nal apoE4-KI phenotypes involving sharp-wave ripples (SWRs), hippocampal network events critical for

272 ions in the hippocampus, known as sharp-wave ripples (SWRs), synchronise the firing behaviour of grou

273 incidences of sleep spindles and sharp-wave ripples (SWRs), typically associated with cortical plast

274 In contrast, during sharp-wave ripples (SWRs), when representations of experience are t

275 ncrease spiking during sharp wave-associated ripples (SWRs).

276 both types of seizures presented with higher ripple than fast ripple rates.

277 were associated with a higher occurrence of ripples than of fast ripples.

278 tion for Promoters and Long-range Enhancers (RIPPLE), that integrates published Chromosome Conformati

279 e endogenous DMN fluctuations to hippocampal ripples, thereby linking network-level resting fMRI fluc

280 ged animals responded more consistently from ripple to ripple.

281 her the structure of ripple oscillations and ripple-triggered patterns of single-unit activity are al

282 age-related changes in ripple architecture, ripple-triggered spike variance, and spike-phase coheren

283 detected in 23 of 40 patients and ultrafast ripples (UFRs; 1,000-2,000Hz) in almost half of investig

284 ed in-plane strain through the nucleation of ripples under both tensile and compressive loading condi

285 at might activate even small-scale bedforms (ripples) under certain contemporary wind regimes.

286 Interictal very fast ripples (VFRs; 500-1,000Hz) were detected in 23 of 40 pa

287 Coupling between hippocampal and neocortical ripples was strengthened during sleep following learning

288 pplocation-best described as an atomic scale ripple-was proposed to explain deformation in two-dimens

289 ain trajectories, whose length is close to a ripple wavelength and whose splash leads to a mass displ

290 ccurring SWRs and the generation of periodic ripples, we selectively manipulated different components

291 hat, when a group of MnR neurons was active, ripples were absent.

292 In contrast, PID ripples were associated with depolarizing synaptic input

293 We found that IID ripples were associated with rhythmic gamma-aminobutyric

294 Linear stability analysis suggested that the ripples were Kelvin-Helmholtz Instabilities.

295 Action potentials during and outside ripples were orthodromic, arguing against ectopic spike

296 ectively used to transmit information during ripples, whereas the firing probability in regular firin

297 ons in CA1 pyramidal cells during sharp wave ripples, which are associated with ripple frequency fluc

298 Rather, these structures resemble fluid-drag ripples, which on Earth include water-worked current rip

299 spontaneous spindles in nesting hippocampal ripples within their excitable troughs, stimulation in-p

300 Interfering with ripples would then result in a dynamic compensatory resp